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Gastrointestinal Disorders in Pediatric Patients Revised, Summer 2009 Cleft Lip and Cleft Palate Etiology- Failure of maxillary and median nasal processes to fuse during embryonic development Remember the psycho-social implications for these children and families Assessment Unilateral, bilateral, midline Treatment Surgical repair done ASAP Rule of 10 > 10#, 10 weeks, 10 HGB Multidisciplinary team Homecare by the family prior to surgery – – – – E-enlarge opening in nipple S-stimulate suck reflex S-swallow fluids appropriately R-rest when infant signals Pre-op Teaching Remind parents that defect is operableshow photographs of corrected clefts Introduce cup, spoon feeding devices (see your book for feeding tips) Explain restraints Post-Op Prevent trauma to suture line – Do not allow to suck! – Facilitate breathing – Maintain nutrition Reduce pain to minimize crying Prevent infection – Cleanse suture lines as ordered Referrals to appropriate team members Esophageal Atresia/ Tracheoesophageal fistula Failure of the esophagus to totally differentiate – 4-5th wk gestation Both are malformations of ESOPHAGUS Cause is unknown Assessment 3C’s -coughing, choking, cyanosis when feeding Respiratory difficulties Drooling Inability to pass suction catheter, NG @ birth Abdominal distention if fistula present Management Early diagnosis Ultra sound Radiopaque catheter inserted in the esophagus to illuminate defect on X-ray Surgical repair- thoracotomy Anastomose ends of esophagus if possible (may need 2 stage repair) Ligate fistula Pre-Op Maintain airway – – – – Keep NPO- administer IV fluids Elevate HOB 30 degrees Suction PRN Gastrostomy for feedings Prevent aspiration pneumonia – Suction – HOB 30 degrees – Prophylactic antibiotics Post-Op Maintain airway Maintain nutrition Prevent trauma Monitor growth and development Gastroesophageal Reflux Disease (GERD) The cardiac/lower esophageal sphincter (AKA LES) and lower portion of the esophagus are weak, allowing regurgitation of gastric contents back into the esophagus. Assessment: Infant Regurgitation almost immediately after each feeding when the infant is laid down Excessive crying, irritability FTH Risk for: – aspiration (pneumonia) – Apnea – Development of respiratory problems (asthma) Assessment: Child Heartburn Abdominal pain Cough, recurrent pneumonia Dysphagia Diagnosis Ph of secretions in esophagus <7.0=acid Barium Swallow and visualization of any esophageal abnormalities Management & Nursing Care Nutritional needs Positioning – PRONE (supine worsens GERD) Medications – H2 receptor antaqgonists (-tidine) – Cholinergics – metoclopramide (Reglan) – Proton pump inhibitors – (-prazole) CPR instruction for parents/caregivers Possible Nissen Fundoplication Diarrhea/Gastroenteritis Severe A disturbance of the intestinal tract that alters motility and absorption and accelerates the excretion of intestinal contents. 3-30 stools/day!!! Most infectious diarrheas in this country are caused by Rotovirus, but can be c.diff Clinical Manifestations Increase in peristalsis Large volume stools (loose, watery, green) Increase in frequency of stools with cramps, nausea, vomiting Urge with small stool present Increased heart & resp. rate, decreased tearing and fever Complications Dehydration – Mucus membranes dried, cracked – Decreased elasticity of skin – Depressed fontanels, eyes sunken – Decreased urinary output, dark Metabolic Acidosis – pH <7.35 – HCO3 =/<22mEq/L Diagnosis Stool culture -causative organism -O&P ABG’s to diagnose Metabolic Acidosis Treatment & Nursing Care Contact isolation Treat cause Weigh daily Monitor I&O, assess for dehydration Skin care Fluid and electrolyte balance – Oral rehydration – IV rehydration (RL or D5NS) Appendicitis Inflammation of the lumen of the appendix which becomes quickly obstructed causing edema, necrosis and pain. Clinical Manifestations Pain – Vague – Periumbilical – Rebound tenderness No bowels sounds “silent abdomen” Anorexia with or without vomiting Diarrhea Increased temperature If ruptures/perforates, there is immediate relief of pain followed by high fever and dehydration Diagnosis WBC <15-20,000 Rebound tenderness at McBurney’s point Abdominal ultrasound or xray - fecalith Management and Nursing Care: Pre-Op NPO, IV Comfort measures, knee chest position Antibiotics Thermal therapy – Ice pack No elimination Patient education for post-op – +/- NG tube – Penrose drain vs open wound bed Management and Nursing Care: Post-Op NPO, IVs Antibiotics Analgesia Patient teaching – Wound care – Open vs laproscopic – No contact sports, PE, lifting until released by surgeon Pyloric Stenosis Pyloric sphincter Incidence Possible genetic predisposition Assessment Vomiting: character?? Constant hunger and fussiness Distended upper abdomen Visible peristaltic waves Hypertrophied pylorus No pain Weight loss Dehydration and electrolyte imbalance Diagnosis History and physical Abdominal ultrasound Laboratory data Pre-op care Restore fluid and electrolyte balance – NPO –I&O – Urine specific gravity Parental support – Guilt – think they are “bad parents” – Emphasize structural problem not parental feeding technique Management and Nursing Care Pylorotomy via laproscopy I&O Feeding Position – HOB elevated slightly Surgical site infection free Patient teaching – s/s recurrence Critical Thinking A 4 week old infant with a history of vomiting after feeding has been hospitalized with a tentative diagnosis of pyloric stenosis. Which of these actions is priority for the nurse? – – – – Begin an intravenous infusion Measure abdominal circumference Orient family to unit Weigh infant Intussuception Most commonly seen in infants 3-12 months but can occur in older child Bowel “telescopes” within itself usually at ileocecal valve Assessment Pain – colicky, knee chest position Vomiting – can contain stool Stools – “currant jelly” Dehydration Serious complications Diagnosis Abdominal xray = intraperitoneal AIR Abdominal ultrasound Therapeutic Intervention Hydrostatic Surgery reduction Post-op care NPO with NG tube Monitor bowel sounds and passage of stool Gradual introduction of fluids and solids Hirschsprung’s Disease Congenital disorder of nerve cells in lower colon Assessment Failure to pass meconium Vomiting with reluctance to feed Bowel assessment Breath If in older child: Constipation Offensive ribbon-like stools History of REGULAR laxative use Palpable fecal mass Diagnosis History & Physical Barium enema (X-ray) Rectal biopsy- absence of ganglionic cells in bowel mucosa Management Surgical intervention –One stage = resection –Two stage Temporary diverting colostomy with resection Re-anastomosis and takedown of colostomy Nursing Care: Pre-op – Cleanse bowel – Neomycin per rectum – Patient/parent teaching re: ostomy Post-op – NPO – N/G tube, IV fluids – No rectal thermometers, monitor VS – Monitor bowel sounds and abdominal girth – Patient/parent teaching Incision care, s/s infection Pain management ?colostomy teaching Volvulus & Malrotation Assessment- pain, bilious vomiting, S & S bowel obstruction Treatment- surgery to prevent ischemia Nursing Care- same as Intussuception and Hirschsprung’s Failure to Thrive (FTH) Assessment- low growth for age, developmental delays, apathy Diagnosis- History to determine organicvs- non-organic Nursing Care- Teaching on nutrition feeding techniques, feeding cues, praise Community resources Celiac Disease Assessment- Growth pattern, GI pattern Treatment- Dietary restrictions Nursing Care- monitor for dehydration, encourage compliance with dietary restrictions, provide support groups for patient and caregiver Diagnosis Measure fetal fat Duodenal biopsy Screen IgA Complications Hypocalcemia Osteomalacia Osteoporosis Depression