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A Class for Foreign MD Students Spine Trauma Dr. Yue Wang 王 跃 MD, PhD Department of Orthopedic Surgery The First Affiliated Hospital, college of Medicine, ZheJiang University 浙江大学医学院附属第一医院骨科 Anatomy of the Spine  Cervical spine, C,  Thoracic Spine, T, 12  Lumbar Spine, L, 5  Sacrum, S, 5, fused  Coccyx, 3-4, fused AP and Lateral View Osteology: The Vertebra  The shape of the vertebrae changes! C T L The Cervical vertebrae atlas, C1 C3-7 axis, C2 The lumbar spine The thoracic and lumbar vertebrae Articular processes Pars Superior Articular Process Pars interarticularis Zygapophyseal Joint (Facet Joint) Inferior Articular Process Arthrology  Intervertebral Discs  Fibrocartilaginous joint  Increase in size from C to L  Ratio remains the same  Make up 20-30% of length of column The intervertebral disc Two components: nucleus and anulus fibrosus Major Ligaments of the Spine  Anterior Longitudinal Ligament - ALL  Dense band along anterior and lateral surface of the vertebral bodies from C2 to sacrum  Posterior Longitudinal Ligament – PLL  Runs along posterior surface of vertebral bodies (anterior to spinal canal)  Ligamentum flavum  Supraspinous lig. and interspinous lig. Ligaments Ligaments of the spine Spinal Cord Part of the CNS along with brain  Contained within vertebral canal  Extends from cranium to 1st-2nd lumbar vertebrae  Lumbar roots & sacral nerves for a “horse-like tail” called cauda equina  2 plexuses   Brachial, lumbosacral Nerves Each vertebrae has a nerve that exits either below or above it  31 pairs of spinal nerves  8 cervical nerves  12 thoracic nerves  5 lumbar  5 sacral  1 coccygeal  The thoracic spine Spinal Injuries  Suspected spinal injuries  High speed crash: MVA  Unconscious  Multiple injuries  Neurologic  Spinal deficit pain/tenderness Spinal injury • 5% worsen neurologically at hospital • Protection is a priority • Detection is a secondary priority • Spinal evaluation complicated by multiple traumae; Mechanism of Injury  Axial compression: Compress/crush the C spine; tends to fracture the vertebra into pieces  Distraction  Pull apart the spinal elements  Usually associated with flexion or extension forces Mechanism of Injury Mechanism of Injury  Flexion:  Severe forward bending  In cervical spine, often leads to neurological damage  Extension:  Severe backward bending  Not always associated with neurological trauma Mechanism of Injury Mechanism of Injury  Shear: Mechanism of Injury  Rotational Rescue  Save the life: ABC;  Evaluate/identify a patient with spinal injury;  Using a spinal board;  Determine appropriate disposition; Classification of Neurologic Injury Frankel Score  A: Complete loss of motor and sensory function  B: Only sensory function remains  C: Motor function is present but of no practical use (i.e., can move legs but not walk)  D: Motor function impaired (i.e.can walk but not with normal gait)  E: No neuro impairment noted Diagnosis Spinal injury • Physical examinations; • X-ray; • CT; • MRI; • Electromyogram (EMG); C Spine Injury  The C spine is involved in more than half of all the patients with traumatic spine injuries  Stability is a crucial key point to determine the choice of management, and it is determined by the integrity of the anatomical structures constituting the cervical spine  Lesions with spinal cord involvement are unstable One of the most common pitfalls in patients with cervical spine injury is missed or delayed diagnosis!  Reasons: 1) inadequate radiographs (44%) ; 2) misinterpretation of adequate radiographs (47%) Goals of Management  Decompression  Alignment  Stabilization  Reconstruction, if necessary Adequate management starts from  A proper classification of the injury type  Assessment of stability of the affected segment Instability : “the loss of the ability of the spine under physiological loads to maintain relationships between vertebrae in such a way that the spinal cord or nerve roots are not damaged or irritated and deformity or pain does not develop. Stable or unstable      Lesions with spinal cord involvement are severe and unstable and require surgical management. On the other hand, lesions without spinal cord involvement can evolve toward a stable or unstable lesion Lesions that mainly involve bone structures may determine temporary instability. Successful fracture healing (with bony callus formation) and subsequent stabilization of these lesions may be obtained with reduction and immobilization. On the other hand, ligamentous or osteo-ligamentous lesions may cause irreversible instability with secondary dislocation. Imaging Evaluation  Alignment/Bones /Soft Tissue  Look for  Normal atlanto-occipital alignment  Predental space 3 mm or less  Prevertebral soft tissue space less than 5 mm anterior to C3  Spinal canal plain film AP diameter 13 mm or greater  Any horizontal translation of one vertebra on the next  Fanning of the space between spinous processes  Fracture of any bone Upper C spine trauma  Jefferson’s fracture  Burst fracture of rings of C1  Sometimes without neurological deficit  Hangman’s fracture  Involves C2  Sudden hyperextension of head and neck forces vertebrae against spinal cord  Complete neurological loss Radiograph Odontoid fractures  Type 1 fractures at tip  Type 2 fractures at waist  Type 3 fractures at base MRI Lower C Spine Fractures n Facet joint dislocation  May occur without fracture  Possible spinal canal compromise Thoraco-lumbar fractures  axial   may result in anterior discoligamentous disruption and posterior compression fractures of facets, laminae, or spinous processes rotation   the posterior ligamentous and osseous elements fail in tension hyperextension   Axial load may result in a burst fracture flexion/distraction   compression Combine compressive forces and flexion/distraction mechanisms and are highly unstable shear  Shear forces produce severe ligamentous disruption and are often associated with spinal cord injury Classifications  White and Panjabi defined clinical instability of the spine : Loss of the ability of the spine under physiologic loads to maintain relationships between vertebrae in such a way that there is neither damage nor subsequent irritation to the spinal cord or nerve root and, in addition, there is no development of incapacitating deformity or pain from structural changes. Denis’ 3 columns theory & Classifications  minor and major injuries:  minor injuries  included fractures of the articular, transverse, and spinous processes as well as the pars interarticularis.  Major injuries  were divided into compression fractures, burst fractures, flexiondistraction (seat-belt) injuries, and fracture dislocations Wedge/compression fractures These injuries may involve both endplates(A), the superior endplate only(B), the inferior endplate only (C), or a buckling of the anterior cortex with both endplates intact (D) Burst fractures      Five subgroups: type A fracture is characterized by #’s of both the superior and inferior end plates, usually as a result of an axial load. The types B and C burst fractures involve the superior and inferior end plates, respectively. result from an axial load coupled with a flexion load. An axial and a rotational load give rise to a type D burst fracture. The type E fracture is characterized by widened pedicles with a burst and lateral flexion injury. Flexion-Distraction fractures (Seat-belt/Chance Fractures) Fracture dislocations General guidance Treatment based on Denis classification  Wedge  most Fractures treated with external orthosis  surgical indications:  failure of brace  progressive deformity  >50% height loss  >30° kyphosis  Burst  canal Fractures compromise & neuro deficit should be treated with surgical decompression  Consider non-operative if:  neuro intact  kyphosis <20-30°  residual spinal canal > 50%  anterior body height > 50% of posterior height  posterior column intact  purely boney injury  show stability in supine/upright x-rays in brace  Seat-belt/Chance Fractures     usually unstable if purely osseous injury  consider trial of bracing and close serial xray f/u if ligamentous injury  healing less predictable, probably best to do posterior fusion Fracture-Dislocation     generally all unstable and require surgery associated with >75% neuro injury (1/2 of which are complete), dural tears, exposed/avulsed roots usually posterior fusion (early anterior fusion dangerous since associated visceral injury is common) staged anterior decompression if necessary Case 1 Post-operative surgery Case 2 Post-operative Thanks! The Rock Mountain, 2012