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Chapter 5: Sensation and Perception
Chapter Outline
1.
2.
3.
4.
5.
Common features of sensation and perception
The chemical senses: Smell and taste
The tactile or cutaneous senses: Touch, pressure, pain,
vibration
The auditory sense: Hearing
The visual sense: Sight
© John Wiley & Sons Canada, Ltd.
Basic Definitions
 Sensation—the act of
using our sensory
systems to detect
environmental stimuli
 Perception—
recognizing and
identifying sensory
stimulus
Raw Sensory Data
Vision
Light waves
Hearing
Sound waves
Smell
Airborne chemicals
Taste
Food chemicals
Touch
Pressure
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Common Features of Sensation and
Perception
 What do we need to do to the raw sensory data so
that our brain can understand it?
 Sensory receptor cells—specialized cells that
convert a specific form of environmental stimuli
into neural impulses.
 Sensory transduction—the process of converting
a specific form of sensory data into a neural impulse
that our brain can read
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Sensory Receptor Cells
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Thresholds: Testing the Limits
 Absolute threshold is the smallest amount of a
stimulus that one can detect
 For example, what is the dimmest light you can
see?
 Difference threshold (or JND)—the minimal
difference needed to notice a difference between two
stimuli
 For example, when do you perceive a difference in
change of volume in your ear?
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Sensory Adaptation
 Constant stimulation decreases the number of
sensory messages sent to the brain, which causes
decreased sensation. Why? We can’t afford to waste
attention on unchanging stimuli
 Examples:
A crying baby will wake us, but not a thunderstorm that
might be even louder
 You notice how tight your pants are when you first put
them on but over the course of the day they seem looser
(you don’t register the sensation of the tightness)
 You do not notice how much perfume you put on; at
first it seems good but then you do not smell it so you
put on more and more…

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Processing Sensory Information
 Bottom-up processing—the raw sensory data is sent
to the brain and your brain uses all of that data to
build a perception

You take 1,000s of data points of visual stimuli and put them
together to create an image of your mother
 Top-down processing—you use previously learned
information to help recognize and interpret the data
coming into your brain

You recognize some of those data points and immediately
match them to your previous knowledge about your mother’s
face
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Perceptual Set
 Perceptual set is the
readiness to interpret a
certain stimulus in a certain
way
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How Do We Smell?
 Chemicals, called odorants, are carried through
the air and reach the 5 million receptor cells
located at the top of each nasal cavity
o
Olfactory receptor neurons
 The receptor cells turn that molecule into a neural
impulse (transduction) and send that impulse to
the olfactory bulb (smell centre in the brain)
o
Lock-and-key binding
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Human Olfactory System
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How Do We Taste?
 Chemical substances in the food we eat dissolve in
saliva and fall into the crevices between the bumps
(papillae) of the tongue
 The bumps hold our taste buds
 Each taste bud contains 60 to 100 sensory receptor
cells for taste
 Taste buds are our receptor cells for taste
 The taste buds translate the chemical message into a
neural impulse and send that impulse to the
thalamus and, eventually, the cerebral cortex
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A Taste Bud
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Five Taste Receptors
1.
2.
3.
4.
5.
Sweet
Sour
Bitter
Salt
Umami—the taste of monosodium glutamate
(MSG)
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Eating Is More than Taste and Smell
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Smell and Taste Disorders
 Anosmia—inability to smell
 Ageusia—inability to taste
 Both
are usually caused by head trauma
 Other conditions:
 Reflex epilepsy—a seizure occurs only after
exposure to a specific odour
 Migraine headaches—specific odours can trigger
migraines
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Tactile or Cutaneous Senses
 The tactile or somatosensory system is a
combination of skin senses:
 Pressure, touch, temperature, vibration, pain
 The tactile senses rely on a variety of receptors
located in different parts of the skin
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Sensory Receptors in the Skin
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Different Sensory Receptors
 Free nerve endings


Located near the surface of the skin
Function: Detect touch, pressure, or pain
 Meissner’s corpuscles


Located in fingertips, lips, and palms (hairless skin areas)
Function: Transduce information about sensitive touch
 Merkel’s discs


Located near the surface of the skin
Function: Transduce information about light to moderate pressure against
the skin
 Ruffini’s end organs


Located deep in the skin
Function: Register heavy pressure and movement of the joints
 Pacinian corpuscles


Located deep in the skin
Function: Respond to vibrations and heavy pressure.
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Steps to Perceiving Touch
Sensory neurons
register pressure
2. Information sent to
the spinal cord, then
the thalamus
(telephone operator)
3. Information is then
sent to the
somatosensory cortex
that registers the
sensation
1.
1.
Tactile information is
processed
contralaterally
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Two Pathways of Pain
 Fast pathway pain (myelinated pathway)—sharp,
localized pain is felt quicker because it travels along
myelinated neurons to the brain
 Slow burn (unmyelinated pathway)—nagging,
burning pain is slower to be felt because it travels
along unmyelinated pathways
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Why Do We Feel or Not Feel Pain?
 Gate control theory—patterns of neural activity can
actually close a “gate” that prevents messages from
reaching parts of the brain where they are perceived
as pain
 Pain threshold
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Chronic Pain
 Chronic pain is the most common abnormality
associated with the somatosensory system
 Endorphins and enkephalins are naturally
occurring pain-killing chemicals in the brain
They can be found in opiates such as morphine or
heroine
 They are produced naturally through intense physical
activity, sex, or intense stress (endogenous opiates)

 Cingulotomy—destruction of the cingulate cortex

An extreme form of neurosurgery to relieve chronic
pain
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Pain Abnormalities
 Familial dysautonomia—rare genetic condition
associated with an inability to detect pain or
temperature
 Phantom limb sensations—tactile hallucinations of
touch, pressure, vibration, and pain in the body part
that no longer exists
 “Mirror box” therapy
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Properties of Sound
 Sound waves—vibrations of the air in the frequency
of hearing
 Frequency—the number of cycles per second in a
wave
 Determines pitch of sound
 Measured in units called Hertz (Hz), which
represent cycles per second
 Amplitude—the magnitude (height of a wave)
 Determines
loudness
 Measured in units called decibels (dB)
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How the Ear Hears
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Hearing
1. Sound waves enter the outer
ear
2. Hits the eardrum (tympanic
membrane)
 Thin
membrane that moves
with sound waves
3. Passes into the middle ear
 Contains
the 3 smallest bones
(or ossicles) in the human
body: maleus (hammer),
incus (anvil), and stapes
(stirrup)
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Hearing
4.
5.
6.
Stapes hits the oval window and
creates vibrations that move
fluid in the cochlea
 Oval window—membrane
separating the ossicles and
the inner ear
The vibrations move the basilar
membrane (in the cochlea),
which is covered with sensory
receptors called hair cells
As hair cells move, neural
impulses are created and sent
to the brain
© John Wiley & Sons Canada, Ltd.
Different Theories of Hearing
 Place theory
Vibration of the basilar membrane (BM) at different places
results in different pitches/frequencies
 Near the oval window (where BM is thinner)—higher
frequencies; lower frequencies occur farther from the oval
window
 Frequency theory
 Different sound frequencies are converted into different
rates of action potentials or firing
 High-frequency sounds produce a more rapid firing than do
low-frequency sounds
 Different firing rates contribute to sound perception of low
frequency tones

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Sound Localization
 General loudness—louder sounds seem closer
 Loudness in each ear—the ear closer to the sound
hears a louder noise than the ear farther from the
sound
 Timing—sound waves will reach the ear closer to the
source of the sound before they reach the ear farther
away
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Hearing and the Brain
 Cochlea  brainstem  thalamus  auditory cortex
 auditory association areas in the cortex
 Tonotopic map—information transmitted from
different parts of the cochlea is projected to specific
parts of the auditory cortex
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Auditory Abnormalities
 Deafness
 Can
be genetic or caused by infection, physical
trauma, or exposure to toxins, including overdose
of common medications such as Aspirin
 Tinnitus—ringing in the ear
 Usually due to abnormalities in the ear
 One of every 200 people experiences tinnitus
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Stimulus for Vision
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Steps Involved in Vision
1. Light waves enter the cornea
 Cornea
is protective outer layer
2. Passes through the pupil
 The
pupil is a small opening in
the eye
3. Passes through the lens
 The
lens focuses the light
waves
4. Projected onto the retina
 Retina
contains all of the
receptor cells (rods and cones)
© John Wiley & Sons Canada, Ltd.
Steps Involved in Vision
The rods and cones
transduce the light waves
into a neural impulse.
 Rods
5.



Used for periphery and night vision
Not as acute as cones (i.e., fuzzy
vision)
Many more rods than cones (over
100 million)
 Cones



Used for central and colour vision
Very acute (i.e., very clear)
 The fovea in the centre of the
eye is all cones
Not as many cones (4–5 million)
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Steps Involved in Vision
6.
The neural impulses are
sent to the optic nerve
• The optic nerve contains
the axons of 1 million
ganglion cells that extend
through the wall of the
retina and go to the brain.
7.
The optic nerve carries
messages from each eye to
the visual cortex (occipital
lobe)
© John Wiley & Sons Canada, Ltd.
Why Do We See in Colour?
 Trichomatic theory—There are three different
sensors for colour and each type responds to a
different range of wavelengths of light
 We see more than three colours, which is he variety of
colours arise from combining the three colours
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Opponent-Process Theory
 The activation of one cone (at retinal level) inhibits
another cone
 This theory explains colour vision at the level of the
ganglion cells
 Ganglion cells are arranged in opposing cells: redgreen, yellow-blue, black-white
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Afterimages
• Opponent-process theory may explain colour
afterimages: continual viewing of red weakens the
ability to inhibit green; remove red and you see green
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Afterimages (blank page to see effect)
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Colour Blindness
• Most people who are colour blind cannot distinguish
between red and green; they would see only a random
pattern of dots in this figure.
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“What” and “Where” Pathways
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“What” Pathway
 Visual agnosia—damage to
the “what” pathway; cannot
recognize objects
 Prosopagnosia—a form of
visual agnosia in which
people cannot recognize
faces
© John Wiley & Sons Canada, Ltd.
“Where” Pathway
 Hemi-neglect—damage to
the “where” pathway; people
ignore one side of their
visual field


Examples of effects: apply
makeup to only one side of
face, shave only one side of
face, eat food on only one side
of plate
People with damage to the
right side of their “where”
pathways neglect the left side
of their visual field
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Top-Down Processing
 The process by which we organize small pieces of sensory experience
into meaningful wholes
 Gestalt principles
 Visual information is organized into coherent images
 Gestalt means whole or totality
 The whole is more than the sum of its parts
 Proximity—stimuli near to one another tend to be grouped together
 Similarity—stimuli resembling one another tend to be grouped
together
 Continuity—stimuli falling along the same plane tend to be grouped
together
 Good form—stimuli forming a shape tend to be grouped together
while those that do not remain ungrouped
 Closure—we tend to fill in small gaps in objects so that they are still
perceived as whole objects
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Impossible Figure
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Grouping Rules
 Proximity—we group nearby
figures together
• Similarity—figures
similar to each other are
grouped together
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Closure
 Closure—tendency to
perceive incomplete figures
as whole and complete
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Depth Perception
 Binocular cues are cues from both eyes
 Convergence—the tendency of the eyes to move toward
each other as we focus on objects up close
 Binocular disparity—different images of objects are cast
on the retinas of each eye
When images are very different, we perceive object is close by
 When images are similar, we perceive object is farther away

© John Wiley & Sons Canada, Ltd.
Monocular Cues
 Monocular cues are cues from one eye
 Interposition—when one object blocks part of another
from our view, we see the blocked object as farther away.
 Elevation—we see objects that are higher in our visual
plane as farther away than those that are lower.
 Texture gradient—we can see more details of textured
surfaces, such as the wood grain on a restaurant table,
that are closer to us.
 Linear perspective—parallel lines seem to converge in the
distance.
 Shading—we are accustomed to light, such as sunlight,
coming from above us. We use differences in the shading
of light from the top to the bottom of our field of view to
judge size and distance of objects.
© John Wiley & Sons Canada, Ltd.
Monocular Cues




Clarity or arial perspective—we tend to see closer objects with
more clarity than objects that are further away. Realizing that
this is sometimes referred to as a fog or smog effect will clarify
what we mean by it.
Familiar size—once we have learned the sizes of objects, such
as people or restaurant plates, we assume that they stay the
same size, so objects that look smaller than usual must be
farther away than usual
Relative size—when we look at two objects we know are about
the same size, if one seems smaller than the other, we see it as
farther away than the other.
Motion Parallax—if we look out the side window of a moving
car or train, objects that are closer to us appear to move past us
more quickly than do objects that are further away (and very
distant objects (like mountains) sometimes do not appear to
move at all).
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Monocular Cues and Illusions
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Pavement Patty
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Perceptual Constancy
 Size constancy—we perceive
objects as the same size
regardless of the distance
from which it is viewed
 Shape constancy—we see an
object as the same shape no
matter from what angle it is
viewed
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Shape and Size Constancy: The Ames Room
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Magnetic Hill, Moncton
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Visual Abnormalities
 Blindness—About 278,000 people in Canada suffer
from visual impairments, of which about 108,000
are legally blind
 Amblyopia—a loss of visual abilities in a weaker eye

Caused by abnormal development of the brain’s visual cortex
due to a failure to receive visual stimulation from both eyes by
the age of six
 Strabismus—lack of coordinated movement of both
eyes; can lead to amblyopia; affects about 2 percent
of the population
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