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10.4 Special Senses
Smell
– olfactory organs
Taste – taste buds
Hearing/Equilibriuim – Ears
Sight – eyes
Spidey Sense
10.5 Sense of Smell
 Olfactory
receptors and taste receptors are
chemoreceptors, which means that chemicals
dissolved in liquids stimulate them.
Olfactory organs
 Chemicals called
odorant molecules
stimulate various sets
of olfactory receptor
proteins, and
therefore various sets
of olfactory receptor
cells, to send a signal
of a detected odor
to the brain.
Olfactory Nerve Pathways
 Stimulated
olfactory receptor cells send
impulses along their axon which synapse with
neurons located in the olfactory bulb.
In the olfactory bulb, the impulses are analyzed
and some impulses travel along olfactory tracts
to the limbic system.
Olfactory Stimulation
 The
several hundred types of receptor
cells can code for many thousands of
odors when they signal the brain in
groups it is like a code of sorts
 The
sense of smell adapts rapidly, but the
adaptation to one scent does not
diminish the sensitivity to new odors.
TASTE
10.6 Sense of Taste
 Taste
buds are the special organs of taste. The
10,000 or so taste buds are located primary on
the surface of the tongue and are associated
with tiny elevations called papillae.
10.6 Sense of Taste
Taste Receptors
 Each taste bud is composed of 50-150 taste cells,
which function as receptor cells, the taste cells
(gustatory cells).
 Each taste cell is replaced on average every ten
days.
Taste




Papaillae: tiny elevations on the surface of the tongue.
Taste Cell: (AKA Gustatory Cells) receptor cells in taste
buds
Taste Pore: opening in the taste bud
Taste hairs: which extend from the cells, are the
sensitive parts of the receptor cells.
Taste
 Network
of nerve fibers (page 270): interwoven
and wrapped around taste cells. Stimulation of a
receptor cell triggers an impulse on a nearby
nerve fiber, and an impulse travels to the brain.
 Food
is received by tongue triggers nerve
impulses take impulse to the brain

Chemicals (in food) must be dissolved in saliva in
order to be tasted.

Food molecules bind to specific receptor proteins
embedded in the taste hairs. The pattern of the
receptor types that bind food molecules and
generate sensory impulses on nearby nerve fibers is
interpreted as a particular taste sensation.

*The chemical senses of smell and taste arise from
molecules from the environment that bind receptors
on neurons specialized as sensory receptors.
Taste Sensations
Five basic taste sensations
 Sweet (tip of tongue)
 Sour (margins/sides of tongue)
 Bitter (back of tongue)
 Salty (widely distributed)
 Umami (MSG)

Flavor results from one or a combination of the
primary sensations. Flavor is a result of more than
just the ‘taste’. Smell, touch, texture, temperature,
etc. also contribute.
Hearing
10.7 Sense of Hearing
 The
ear is divided into three parts: outer,
middle, and inner.
Outer (External) Ear
 Auricle/Pinna:
Collects sound waves
traveling through the air, and directs them
down into the external auditory meatus.
 External Auditory Meatus: Funnels sound
waves to the eardrum
 Eardrum/Tympanic Membrande: Separates
outer from middle ear. Converts sound
waves into vibrations
Middle Ear Parts:

Malleus (hammer)attaches to eardrum

Incus (anvil)connects malleus to
stapes

Stapes (stirrup)attaches to the oval
window
Ear drum wiggles…Malleus which
touches eardrum wiggles….Incus
which touches malleus
wiggles….Stapes which touches
incus wiggles…sound wave is
amplified X22
Auditory Ossicles: Three bones connecting
eardrum to the inner wall of the inner ear
Middle Ear Parts:
 Oval
window:
Leads to the inner
ear. Vibration of
the stapes at the
oval window moves
a fluid in the inner
ear, which
stimulates the
hearing receptors.
Inner Ear (Bony Labyrinth)
 Semicircular
equilibrium.
 Cochlea:
Canals: provides a sense of
function in hearing.
 Simplification
of what is in your book: The
cochlea contains hair cells. The hair cells
synapse with neurons which carry the impulse to
the brain.
Middle Ear



Stapes which touches oval window touches the
inner ear fludi filled structures. The sound waves
are then transferred to mechanical fluid waves.
Oval window touches the
The pitch is deteremined by how far wave
travelsing to coclia.
There are hair cells in the coclia. The hair cells
synapes (ie send messges) to sensory neurons.
So Far:
Sound wave-eardrum-malleas-incus-stapes
TMI
Semicircular Canals



Fluid fills
There are “rocks” in semicircular canals that
roll around.
If you spin and get waves, the waves keep
going even though you have stopped.
TMI
Hearing Nerve Pathways

Cranial Nerve –nerve
receives information from
the semicircular canal and
cochlea.

The nerve goes to the
inferior colliculus which
interpret the sound and
say do we need to fight or
have a reflex or not.

If not, input is passed on to
the thalamus. Thalamus
sends impulse onto
temporal lobe to
determine res
Steps of hearing







The external ear picks up sound waves and
focuses them onto the eardrum.
The eardrum vibrates and converts the sound
impulses to mechanical waves.
The three ossicles move in response to the
eardrum vibration, amplifying the sound.
The stapes pushes on the oval window.
Fluid waves causes membranes within the
cochlea to move, bending hair cells
The hair cells bending causes a nerve impulse.
The nerve impulse is carried to the temporal lobes
by nerve fibers where they are interpreted.
Sight
ALZHEIMERS' EYE TEST
Count every ' F ' in the following
text:
FINISHED FILES ARE THE RE
SULT OF YEARS OF SCIENTI
FIC STUDY COMBINED WITH
THE EXPERIENCE OF YEARS...
10.9 Sense of Sight
Visual Accessory Organs:
 Eyelid:
four layers of the eyelid: skin, muscle,
connective tissue, and conjunctiva.
 Conjunctiva:
mucous layer that lines the inner
surfaces of the eyelids.
 Lacrimal
3
gland: secretes tears
Layers of the Eye: Outer (fibrous) layer, a middle
(vascular) layer, and an inner (nervous) layer.
Outer Layer Of Eye

Lateral rectus: helps control
muscles of the eye.

Cornea: transparent window of
the eye. Helps focus light rays.

Sclera: white portion of the
eye. Protects the eye and is an
attachment for muscles.

Optic nerve: carries the
impulse to the brain.
Middle Layer of Eye

Choroid Coat: produce melanin which
absorbs light and helps keep inside of eye
dark.

Ciliary muscle: cause lens to flex in order to
focus.

Lens: adjusts shape to focus.

Iris: colored part of the eye.

Aqueous humor: water fluid secreted by
the ciliary body

Pupil: hole that lets light in
Inner Layer of Eye

Retina: contains visual receptor
cells. The retina is thin and
delicate, but its structure is quite
complex.

Fovea centralis: region of the
retina that produces the
sharpest vision.

Optic disc: where nerve fibers
from the retina leave the eye
and join the optic nerve.

Vitreous humor: supports
internal parts of the eye and
helps maintain its shape
GLAUCOMA
sual Defects/Diseases
Glaucoma
Aqueous humor forms faster than it is removed
Fluid in eye increases and the fovea is compressed, leading to
restricted vision
Nyctalopia  Night Blindness
Congenital
Injury
Malnutrition (Vitamin A)
Nyctalopia
al Defects/Diseases
Glaucoma
Aqueous humor forms faste
Fluid in eye increases and th
restricted vision
Nyctalopia  Night Blindness
Congenital
Injury
Malnutrition (Vitamin A)
Astigmatism
retinitis pigmentosa, a disorder in which the rod cells in the retina gradually
lose their ability to respond to the light.
Astigmatism
isual Defects/Diseases
Glaucoma
Aqueous humor forms faster than it is removed
Fluid in eye increases and the fovea is compressed, leading to
restricted vision
Nyctalopia  Night Blindness
Congenital
Injury
Malnutrition (Vitamin A)
Astigmatism
Light passing through the cornea is not all focused at a single
point
Distorted image on the retina
Due to uneven curvature of the anterior surface of the cornea
Myopia nearsightedness
Eyeball is too long
Visual Defects/Diseases
Glaucoma
Aqueous humor forms faster than it is rem
Fluid in eye increases and the fovea is com
restricted vision
Nyctalopia  Night Blindness
Congenital
Injury
Malnutrition (Vitamin A)
Astigmatism
Light passing through the cornea is not all
point
Distorted image on the retina
Due to uneven curvature of the anterior su
Myopia nearsightedness
Eyeball is too long
Far object images are blurred
Corrective lenses are concave
Hyperopia farsightedness
Visual Defects/Diseases
Malnutrition (Vitamin A)
Astigmatism
Light passing through the cornea
Visual Defects/Diseases
point
Distorted image on the retina
Due to uneven curvature of the a
Myopia nearsightedness
Eyeball is too long
Far object images are blurred
Corrective lenses are concave
Hyperopia farsightedness
Eyeball is too short
Near objects are blurred
Corrective lenses are convex
Presbyopia
Visual Defects/Diseases
Glaucoma
Aqueous humor forms faster than it is removed
Fluid in eye increases and the fovea is compressed, leading to
restricted vision
Nyctalopia  Night Blindness
Congenital
Injury
Malnutrition (Vitamin A)
Astigmatism
Light passing through the cornea is not all focused at a single
point
Distorted image on the retina
Due to uneven curvature of the anterior surface of the cornea
Myopia nearsightedness
Eyeball is too long
Far object images are blurred
Corrective lenses are concave
Hyperopia farsightedness
Eyeball is too short
Near objects are blurred
Corrective lenses are convex
Presbyopia
Occurs with advancing age as the ciliary muscles lose their
contractive powers & lens loses some of it elasticity.
Eye can’t focus as well or as efficiently.
Cataracts
Common in older people
Visual Defects/Diseases
Malnutrition (Vitamin A)
Astigmatism
Light passing through the cornea is not all focused at a
point
Distorted image on the retina
Due to uneven curvature of the anterior surface of the
Myopia nearsightedness
Eyeball is too long
Far object images are blurred
Corrective lenses are concave
Hyperopia farsightedness
Eyeball is too short
Near objects are blurred
Corrective lenses are convex
Presbyopia
Occurs with advancing age as the ciliary muscles lose t
contractive powers & lens loses some of it elasticity.
Eye can’t focus as well or as efficiently.
Cataracts
Common in older people
Occurs when the lens becomes cloudy and opaque
Visual Defects/Diseases
Blindness


Worldwide, blindness is most commonly caused by
changes in the cornea
Cataracts are a leading cause of blindness

Visual Defects/Diseases

Glaucoma



Nyctalopia  Night Blindness










Eyeball is too short
Near objects are blurred
Corrective lenses are convex
Presbyopia



Eyeball is too long
Far object images are blurred
Corrective lenses are concave
Hyperopia farsightedness


Light passing through the cornea is not all focused at a single point
Distorted image on the retina
Due to uneven curvature of the anterior surface of the cornea
Myopia nearsightedness


Congenital
Injury
Malnutrition (Vitamin A)
Astigmatism


Aqueous humor forms faster than it is removed
Fluid in eye increases and the fovea is compressed, leading to
restricted vision
Occurs with advancing age as the ciliary muscles lose their contractive
powers & lens loses some of it elasticity.
Eye can’t focus as well or as efficiently.
Cataracts


Common in older people
Occurs when the lens becomes cloudy and opaque