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Transcript
Chapter 4
Atrial Rhythms
1
Objectives






Explain the concepts of altered automaticity, triggered activity,
and reentry.
Explain the terms bigeminy, trigeminy, quadrigeminy, and run
when used to describe premature complexes.
Describe the ECG characteristics, possible causes, signs and
symptoms, and initial emergency care for premature atrial
complexes (PACs).
Explain the difference between a compensatory and
noncompensatory pause.
Explain the terms wandering atrial pacemaker and multifocal
atrial tachycardia.
Describe the ECG characteristics, possible causes, signs and
symptoms, and initial emergency care for wandering atrial
pacemaker (multiformed atrial rhythm).
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2
Objectives






Describe the ECG characteristics, possible causes, signs and
symptoms, and initial emergency care for multifocal atrial
tachycardia (MAT).
Describe the ECG characteristics, possible causes, signs and
symptoms, and initial emergency care for atrial tachycardia
(AT).
Discuss preexcitation syndrome and name its three major forms.
Explain the terms paroxysmal atrial tachycardia (PAT) and
paroxysmal supraventricular tachycardia (PSVT).
List four examples of vagal maneuvers.
Discuss the indications and procedure for synchronized
cardioversion.
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3
Objectives




Describe the ECG characteristics, possible causes, signs
and symptoms, and initial emergency care for
atrioventricular nodal reentrant tachycardia (AVNRT).
Describe the ECG characteristics, possible causes, signs
and symptoms, and initial emergency care for
atrioventricular reentrant tachycardia (AVRT).
Describe the ECG characteristics, possible causes, signs
and symptoms, and initial emergency care for atrial flutter.
Describe the ECG characteristics, possible causes, signs
and symptoms, and initial emergency care for atrial
fibrillation (AFib).
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4
Introduction

Atria

Thin-walled, lowpressure chambers
 Receive blood from
systemic circulation
and lungs
 “Atrial kick”
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5
Atrial Dysrhythmias: Mechanisms

Atrial dysrhythmias reflect abnormal electrical
impulse formation and conduction in the atria.
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6
Atrial Dysrhythmias: Mechanisms

Atrial dysrhythmias may occur because of:

Altered automaticity
 Triggered activity
 Reentry

Altered automaticity and triggered activity are
disorders in impulse formation

Reentry is a disorder in impulse conduction
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7
Altered Automaticity

Can occur in:



Normal pacemaker cells
Myocardial working cells that do not normally
function as pacemaker sites
These cells depolarize and initiate impulses
before a normal impulse
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8
Triggered Activity

Results from abnormal electrical impulses
that sometimes occur during repolarization
(afterdepolarizations), when cells are
normally quiet

Requires a stimulus to initiate depolarization
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9
Reentry (Reactivation)

A condition in which an impulse returns to
stimulate tissue that was previously
depolarized
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10
Reentry (Reactivation)

Reentry requires:



A potential conduction
circuit or circular
conduction pathway
A block within part of the
circuit
Delayed conduction with
the remainder of the
circuit
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11
Reentry (Reactivation)

Reentry results in a
single premature beat or
repetitive electrical
impulses resulting in
short periods of rapid
rhythms
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12
Atrial Dysrhythmias

Most atrial dysrhythmias are not lifethreatening


Some are associated with extremely fast
ventricular rates
An excessively rapid heart rate may compromise
cardiac output
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13
Premature Complexes

Premature beats may be produced by:




Atria
AV junction
Ventricles
Premature beats appear early, that is, they
occur before the next expected beat
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14
Premature Complexes

Premature beats are identified by their site of
origin



Premature atrial complexes (PACs)
Premature junctional complexes (PJCs)
Premature ventricular complexes (PVCs)
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15
Premature Complexes — Patterns

Pairs (coupled)


“Runs” or “bursts”


Every other beat is a premature beat
Trigeminy


Three or more premature beats in a row
Bigeminy


Two premature beats in a row
Every third beat is a premature beat
Quadrigeminy

Every fourth beat is a premature beat
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16
Premature Atrial Complexes


Occur when an irritable site within the atria
discharges before the next SA node impulse
is due to discharge
The P wave of a PAC may be:




Biphasic (partly positive, partly negative)
Flattened
Notched
Pointed
 Lost
in the preceding T wave
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17
PACs—
How Do I Recognize Them?
Rate
Usually within normal range, but depends on
underlying rhythm
Rhythm
Regular with premature beats
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18
PACs—
How Do I Recognize Them?
S = SA node;
P waves
= atrial beat
Premature (occurring earlier than the next expected sinus P
wave), positive (upright) in lead II, one before each QRS
complex, often differ in shape from sinus P waves–may be
flattened, notched, pointed, biphasic, or lost in the preceding T
wave
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19
PACs—
How Do I Recognize Them?
PR
interval
May be normal or prolonged depending on the prematurity of the
beat
QRS
duration
Usually 0.10 sec or less but may be wide (aberrant) or absent,
depending on the prematurity of the beat; the QRS of the PAC is
similar in shape to those of the underlying rhythm unless the PAC
is abnormally conducted
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20
PACs—
How Do I Recognize Them?
Sinus tachycardia at 111 bpm with three PACs
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21
PACs—
How Do I Recognize Them?
Rate
Usually within normal range, but depends on underlying rhythm
Rhythm
P waves
Regular with premature beats
Premature (occurring earlier than the next expected sinus P
wave), positive (upright) in lead II, one before each QRS
complex, often differ in shape from sinus P waves–may be
flattened, notched, pointed, biphasic, or lost in the preceding T
wave
PR
interval
QRS
duration
May be normal or prolonged depending on the prematurity of the
beat
Usually 0.10 sec or less but may be wide (aberrant) or absent,
depending on the prematurity of the beat; the QRS of the PAC is
similar in shape to those of the underlying rhythm unless the PAC
is abnormally conducted
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22
Compensatory/Noncompensatory Pause

A “noncompensatory” (incomplete) pause
often follows a PAC

Represents the delay during which the SA node
resets its rhythm for the next beat
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23
Compensatory/Noncompensatory Pause

To determine whether a pause following a premature
complex is compensatory or noncompensatory:

Measure the distance between three normal beats
 Compare that distance between three beats, one of which includes
the premature complex
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Compensatory/Noncompensatory Pause

The pause is termed “noncompensatory”
(incomplete) if the normal beat following the
premature complex occurs before it was
expected


When the distance is NOT the same
The pause is “compensatory” (complete) if
the normal beat following the premature
complex occurs when expected

When the distance is the same
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25
Aberrantly Conducted PACs

PACs associated with a wide QRS complex
are called “aberrantly conducted” PACs

Indicates conduction through ventricles is
abnormal
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26
Nonconducted PACs

A PAC may occur very prematurely and close
to the T wave of the preceding beat


Only a P wave may be seen with no QRS after it
(appearing as a pause)
This is a “nonconducted” or “blocked” PAC

P wave occurred too early to be conducted
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27
PACs—
What Causes Them?





Emotional stress
Congestive heart
failure
Acute coronary
syndromes
Mental and physical
fatigue
Atrial enlargement





Digitalis toxicity
Valvular heart
disease
Electrolyte
imbalance
Hyperthyroidism
Stimulants: caffeine,
tobacco, cocaine
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28
PACs—
What Do I Do About Them?



Occasional PACs usually do not require
treatment
Frequent PACs may initiate episodes of atrial
fibrillation, atrial flutter, or PSVT
Frequent PACs are treated by correcting the
underlying cause:

Stress reduction
 Reduced consumption of caffeine-containing
beverages
 Treatment of congestive heart failure
 Correction of electrolyte imbalance
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29
Wandering Atrial Pacemaker

Multiformed atrial rhythm


Updated term for the rhythm formerly known as
wandering atrial pacemaker
Size, shape, and direction of P waves vary
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30
Wandering Atrial Pacemaker—
How Do I Recognize It?
Rate
Usually 60–100 bpm, but may be slow. If the rate is more than 100
bpm, the rhythm is termed multifocal (or chaotic) atrial tachycardia
Rhythm
May be irregular as pacemaker site shifts from SA node to ectopic
atrial locations and AV junction
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31
Wandering Atrial Pacemaker—
How Do I Recognize It?
P Waves
Size, shape, and direction may change from beat to beat
PR Interval
Variable
QRS
Usually 0.10 sec or less unless an intraventricular conduction
delay exists
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32
Wandering Atrial Pacemaker—
How Do I Recognize It?
Rate
Usually 60–100 bpm, but may be slow. If the rate is greater than
100 bpm, the rhythm is termed multifocal (or chaotic) atrial
tachycardia.
Rhythm
May be irregular as the pacemaker site shifts from the SA node to
ectopic atrial locations and the AV junction
P Waves
Size, shape, and direction may change from beat to beat.
At least three different P wave configurations (seen in the same
lead) are required for a diagnosis of wandering atrial pacemaker
or multifocal atrial tachycardia.
PR Interval
Variable
QRS
Usually 0.10 sec or less unless an intraventricular conduction
delay exists
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Wandering Atrial Pacemaker—
What Causes It?

May be observed in normal, healthy hearts
(particularly in athletes) and during sleep

May also occur with some types of organic
heart disease and with digitalis toxicity
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34
Wandering Atrial Pacemaker—
What Do I Do About It?

Usually produces no signs and symptoms
unless associated with a slow rate

If the rhythm occurs because of digitalis
toxicity, the drug should be withheld
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35
Multifocal Atrial Tachycardia—
How Do I Recognize It?

When wandering atrial pacemaker is associated
with a ventricular rate greater than 100 bpm, the
rhythm is called “multifocal atrial tachycardia”
(MAT) or chaotic atrial tachycardia
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36
Multifocal Atrial Tachycardia—
What Causes It?

Most often seen in:







Severe COPD
Hypoxia
Acute coronary syndromes
Digoxin toxicity
Rheumatic heart disease
Theophylline toxicity
Electrolyte imbalances
• Hypokalemia
• Hypomagnesemia
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37
Multifocal Atrial Tachycardia—
What Do I Do About It?

Treatment is directed at the underlying cause

If patient is symptomatic but you are
uncertain rhythm is MAT:


Vagal maneuvers, adenosine
If you know the rhythm is MAT and the patient
is symptomatic, consult cardiologist
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Vagal Maneuvers







Coughing
Squatting
Breath-holding
Carotid sinus massage
Application of a cold
stimulus to the face
Valsalva’s maneuver
Gagging
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Carotid Sinus Massage
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Carotid Sinus Massage
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Carotid Sinus Massage
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Adenosine

Can interrupt reentry pathways that involve
the AV node

Rapid onset of action

Short half-life
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43
Supraventricular Arrhythmias

Begin above the bifurcation of the bundle of
His

Includes rhythms that begin in the:



SA node
Atrial tissue
AV junction
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Types of Supraventricular Tachycardias
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Atrial Tachycardia

Atrial tachycardia is a series of rapid beats
from an atrial ectopic focus

This rapid atrial rate overrides the SA node and
becomes the pacemaker
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Atrial Tachycardia

Atrial tachycardia


3 or more PACs in a row at a rate of more than
100 bpm
Paroxysmal atrial tachycardia (PAT)

Atrial tachycardia that starts or ends suddenly
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47
Atrial Tachycardia
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Atrial Tachycardia—
How Do I Recognize It?
Atrial tachycardia is shown that ends spontaneously with the
abrupt resumption of sinus rhythm.
The P waves of the tachycardia (rate: about 150 bpm) are
superimposed on the preceding T waves.
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49
Atrial Tachycardia—
How Do I Recognize It?
Rate
100–250 bpm
Rhythm
Regular
P Waves
One P wave precedes each QRS complex in lead II; P
waves differ in shape from sinus P waves; an isoelectric
baseline is usually present between P waves; if the atrial
rhythm originates in the low portion of the atrium, P waves
will be negative in lead II. With rapid rates, it is difficult to
distinguish P waves from T waves
PR Interval
May be shorter or longer than normal
QRS
0.10 sec or less unless an intraventricular conduction
delay exists
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50
Atrial Tachycardia

Paroxysmal atrial tachycardia with block
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Atrial Tachycardia—
What Causes It?

Stimulant use





Caffeine, albuterol, theophylline, cocaine
Infection
Electrolyte imbalance
Acute illness with excessive catecholamine
release
Myocardial infarction
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52
Atrial Tachycardia—
What Do I Do About It?

Possible signs and symptoms:








Asymptomatic
Palpitations
Fluttering sensation in the chest
Chest pressure
Dyspnea
Fatigue
Dizziness or lightheadedness
Syncope or near-syncope
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53
Atrial Tachycardia—
What Do I Do About It?

If symptomatic due to rapid rate:





Vagal maneuvers
Adenosine drug of choice
Calcium channel blockers
Beta-blockers
Amiodarone
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Amiodarone

Coronary and peripheral vasodilator

Suppresses SA node function

Prolongs PR, QRS, and QT intervals

Slows conduction at the AV junction
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Amiodarone

Hypotension and bradycardia most common
adverse effects


Slow infusion rate or discontinue if seen
Additive effect with other medications that
prolong the QT interval
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Synchronized Cardioversion

Delivery of an electrical shock to the heart
timed to occur during QRS
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57
Cardioversion—Indications

Tachycardias (except sinus tachycardia) with
a ventricular rate greater than 150 bpm that
have a clearly identifiable QRS complex
(such as some narrow QRS tachycardias and
ventricular tachycardia)
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Cardioversion—Procedure
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Cardioversion—Procedure
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Cardioversion—Procedure
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Cardioversion—Procedure
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Cardioversion—Procedure
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Cardioversion—Procedure
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AV Nodal Reentrant Tachycardia
(AVNRT)


Most common type of SVT
Caused by reentry in the area of the AV node
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AVNRT—
How Do I Recognize It?

AVNRT is usually precipitated by a PAC
 Electrical circuit allows impulse to spin
around in a circle
 Results
in a very rapid and regular rhythm
• Rate: 150–250 bpm
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AVNRT—
How Do I Recognize It?
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AVNRT—
How Do I Recognize It?
Rate
150–250 bpm
(usually 180 to 200 bpm in adults)
Rhythm
Regular
P Waves
Usually not identifiable; P wave usually hidden in
T wave of preceding beat
PR Interval
Not measurable
QRS
0.10 sec or less unless an intraventricular
conduction delay exists
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Paroxysmal Supraventricular
Tachycardia (PSVT)

Regular, narrow-QRS tachycardia that starts
or ends suddenly

P waves usually hidden in T waves of
preceding beats
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AVNRT—
What Causes It?


Common in individuals with no structural
heart disease
Triggers:

Hypoxia
 Stress
 Anxiety
 Caffeine
 Smoking
 Sleep deprivation
 Many medications
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AVNRT—
What Causes It?

AVNRT also occurs in individuals with:





COPD
Coronary artery disease
Valvular heart disease
Congestive heart failure
Digitalis toxicity
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AVNRT—
What Do I Do About It?

Possible signs and symptoms:





Palpitations
(common)
Lightheadedness
Neck vein pulsations
Syncope or nearsyncope
Dyspnea





Weakness
Nervousness, anxiety
Chest pain or pressure
Signs of shock
Congestive heart failure
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AVNRT—
What Do I Do About It?

Stable patient



Oxygen, IV access, cardiac monitor
Vagal maneuvers
Adenosine
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AVNRT—
What Do I Do About It?

If the patient is unstable:




Oxygen, IV access, cardiac monitor
Consider medication administration
Consider sedation
• If the patient is awake and time permits
Synchronized cardioversion
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AV Reentrant Tachycardia
(AVRT)

Atrioventricular reentry tachycardia (AVRT)
involves a pathway of impulse conduction
outside the AV node and bundle of His
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AV Reentrant Tachycardia
(AVRT)

Preexcitation


Impulse begins above the ventricles but travels via
a pathway other than AV node and bundle of His
Supraventricular impulse excites the ventricles
earlier than normal
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AV Reentrant Tachycardia
(AVRT)
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WPW—
How Do I Recognize It?

Short PR interval

Delta wave

QRS widening

Secondary ST
segment and T
wave changes
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WPW—
How Do I Recognize It?
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WPW—
How Do I Recognize It?
Rate
Usually 60–100 bpm, if the underlying rhythm is
sinus in origin
Rhythm
Regular, unless associated with atrial fibrillation
P Waves
Normal and positive in lead II unless WPW is
associated with atrial fibrillation
PR Interval
If P waves are observed, less than 0.12 sec
QRS
Usually greater than 0.12 second; slurred
upstroke of the QRS complex (delta wave) may
be seen in one or more leads
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AVRT

Three main types of tachydysrhythmias that
occur in WPW:



AVRT (most common)
Atrial fibrillation
Atrial flutter
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WPW—
What Causes It?


Common cause of tachydysrhythmias in
infants and children
Accessory pathway in WPW is likely to be
congenital in origin
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AVRT—
What Do I Do About It?

Common signs and symptoms associated
with AVRT:

Palpitations
 Lightheadedness
 Shortness of breath
 Anxiety
 Weakness
 Dizziness
 Chest discomfort
 Signs of shock
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AVRT—
What Do I Do About It?

Consultation with a cardiologist is
recommended

Stable but symptomatic patient


Oxygen, IV access, and IV medications such as
amiodarone
Unstable patient

Synchronized cardioversion
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84
Atrial Flutter

Ectopic atrial rhythm in
which an irritable site
fires regularly at an
extremely rapid rate

Type I atrial flutter

Type II atrial flutter
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Atrial Flutter—
How Do I Recognize It?

Atrial flutter with
2:1 conduction

Atrial flutter with
4:1 conduction
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Atrial Flutter—
How Do I Recognize It?
Rate
Atrial rate 250–450 bpm, typically 300 bpm; ventricular rate
variable — determined by AV blockade; ventricular rate will
usually not exceed 180 bpm due to intrinsic conduction rate
of AV junction
Rhythm
Atrial regular, ventricular regular or irregular depending on AV
conduction/blockade
P Waves
No identifiable P waves; saw-toothed “flutter” waves are
present
PR Interval
Not measurable
QRS
0.10 sec or less but may be widened if flutter waves are
buried in the QRS complex or an intraventricular conduction
delay exists
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Atrial Flutter—
What Causes It?

Atrial flutter is usually a paroxysmal rhythm
precipitated by a PAC


May last for seconds to hours and occasionally
lasts 24 hours or more
Chronic atrial flutter is unusual

Rhythm usually converts to sinus rhythm or atrial
fibrillation, on its own or with treatment
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Conditions Associated
with Atrial Flutter






Hypoxia
Pulmonary embolism
Chronic lung disease
Mitral or tricuspid
valve stenosis or
regurgitation
Pneumonia
Complication of
myocardial infarction






Ischemic heart
disease
Cardiomyopathy
Hyperthyroidism
Digitalis or
quinidine toxicity
Cardiac surgery
Pericarditis/myoca
rditis
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Atrial Flutter—
What Do I Do About It?

Cardiologist consult recommended

If rapid ventricular rate, control ventricular
response

If rapid ventricular rate and serious signs and
symptoms, synchronized cardioversion
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Atrial Fibrillation (AFib)
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Atrial Fibrillation—
How Do I Recognize It?
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92
Atrial Fibrillation—
How Do I Recognize It?

AFib can occur with complete AV block

Ventricular rhythm will be slow and regular
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93
Atrial Fibrillation—
How Do I Recognize It?
Rate
Atrial rate usually 400–600 bpm;
ventricular rate variable
Rhythm
Ventricular rhythm usually irregularly irregular
P Waves
No identifiable P waves, fibrillatory waves present;
erratic, wavy baseline
PR Interval
Not measurable
QRS
0.10 sec or less but may be widened if an
intraventricular conduction delay exists
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Atrial Fibrillation—
What Causes It?

Can occur in patients with or without
detectable heart disease or related symptoms

Increased stroke risk



Atria do not contract effectively
Blood pools within the atria, forming clots
Clot dislodges and moves to artery in the brain
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Conditions Associated with Atrial
Fibrillation












Idiopathic (no clear cause)
Hypertension
Ischemic heart disease
Advanced age
Rheumatic heart disease
Cardiomyopathy
Congestive heart failure
Congenital heart disease
Sick sinus syndrome
WPW syndrome
Pericarditis
Pulmonary embolism












Chronic lung disease
After surgery
Diabetes
Stress
Sympathomimetics
Excessive caffeine
Hypoxia
Hypokalemia
Hypoglycemia
Systemic infection
Hyperthyroidism
Electrocution
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96
Atrial Fibrillation—
What Do I Do About It?

Cardiologist consult recommended

If rapid ventricular rate, control ventricular
response

If rapid ventricular rate and serious signs and
symptoms, synchronized cardioversion

Anticoagulation recommended if AFib has been
present for 48 hours or longer
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Questions?
98