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CARE OF THE PEDIATRIC PATIENT WITH COGNITION OR PERCEPTION PROBLEMS Elizabeth Allen RN, MSN LEARNING OBJECTIVES Describe the neurologic anatomic and physiologic differences between infants and children and adults Identify aspects of the pediatric neurologic assessment including Glasgow Coma Scale and signs of meningitis Identify and create a treatment plan for pediatric neurologic infectious diseases Compare pediatric traumatic brain injuries for different age groups Describe pediatric pain management ANATOMIC DIFFERENCES Brain & spinal cord Cranial Nerves Head to toe Cranial bones Present, must mature Myelination Intact from birth Reflexes Large head Brain growth Not complete Head vs. chest size Blood brain barrier NEUROLOGICAL DEVELOPMENT Neurological system of the infant is incompletely developed. First year the neurons become myelinated. Myelinization occurs in a cephalocaudal and proximodistal manner. NEUROLOGIC ASSESSMENT Cerebellar Noting Functioning milestones Fine and gross motor skill Behavioral assessment Waking activity Responses to environment Social interaction Cranial nerves Motor system Head control Reflexes Primitive reflexes Stepping Tonic-Neck Moro/Startle Sucking NEUROLOGIC ASSESSMENT Brudzinski’s sign Kernig’s sign Nuchal rigidity - neck Photophobia ICP LOC Glasgow Coma Scale Glasgow Coma Scale Including Pediatric Assessment INCREASED INTRACRANIAL PRESSURE Increased ICP creates decreased CPP (Cerebral Perfusion Pressure) Causes Head injury Bleeding Space occupying lesion Infection Hydrocephalus Infant Signs & Symptoms Fontanels (bulging) Sutures (widened) Scalp veins (dilated) Sunset eyes Older child Signs & Symptoms Normal ICP < 20 mmHg Headache Nausea, vomiting Decreased LOC, irritability Pupil changes Sunset eyes Posturing Cushing’s Triad (Late sign) AMBLYOPIA Amblyopia: Reduced vision in 1 or both eyes From visual deprivation of eye Most often from strabismus Visual assessment and correction by age 6 years Strabismus NEUROLOGIC INFECTIOUS DISEASES Bacterial Meningitis Infant Signs & Symptoms Poor feeding/suck Vomiting High-pitched cry Bulging fontanel Fever or hypothermia Poor muscle tone Bacterial Meningitis Child & Adolescent Signs & Symptoms Abrupt onset Fever & chills HA Nuchal rigidity Vomiting Irritability Seizures Photophobia/alterations in sensorium BACTERIAL MENINGITIS Nursing Interventions Droplet Isolation! Maintain patent airway, ventilation support prn Post-LP care Assessment Increased ICP Nutrition Pain control Monitor IV antibiotic therapy Monitor for complications- seizures, immobility VIRAL MENINGITIS (ASEPTIC MENINGITIS) Infant and Toddler Signs & Symptoms Irritability Lethargy Vomiting Change in appetite Children & Adolescent Signs & Symptoms Preceded by nonspecific febrile illness HA Malaise Muscle aches N&V Photophobia Nuchal/spinal rigidity VIRAL MENINGITIS Treated symptomatically Hospitalized? Decease stimulation Hydration Comfort measures NEUROLOGIC INFECTIOUS DISEASES Reye Syndrome Usually develops after mild viral illness Strongly associated with Aspirin (salicylates) use in viral infections Acute encephalopathy and fatty infiltration of the liver and other organs Poor liver function Hypoglycemia Prolonged Prothrombin time Developmental and neurologic deficits may occur REYE SYNDROME Stages Progress with decreasing LOC 1. Vomiting and drowsiness, listlessness 2. Personality changes: irritable, aggressive 3. Disorientation: confusion, irrational, combative 4. Delirium, seizures, coma, loss of deep tendon reflexes, respiratory arrest Symptoms of Reye Syndrome in infants do not follow a typical pattern Symptoms appear most commonly after a viral illness (National Reye Syndrome Foundation, 2013) REYE SYNDROME Signs & Symptoms Abrupt change in LOC Vomiting Liver enzymes, ammonia levels elevated Blood sugars low PT prolonged Bilirubin remains normal Liver biopsy shows small fat deposits REYE SYNDROME Nursing Interventions Monitor ICP Fluid restrictions Neurological assessments Monitor lab values Ammonia Glucose Blood gases Provide emotional support Medications Vitamin K Phenytoin Corticosteroids Family education ACCIDENTS & HEAD INJURIES Shaken baby syndrome Head trauma Falls Child abuse MVA Near-Drowning SHAKEN BABY SYNDROME Pathophysiology – central nervous system injury from repeated coup and contra-coup injury Symptoms blindness or eye damage delay in normal development seizures damage to the spinal cord Retinal hemorrhage paralysis brain damage death SHAKEN BABY SYNDROME Maintain cardiopulmonary function Prevent complications- Secondary Brain Injury Promote recovery Maximize developmental function Provide emotional support Role of Social Work Discharge planning and home care teaching Resources HEAD INJURY Mild to Severe Highest rate of death in adolescents 15-19 years, then <5 years Side effects of trauma = cerebral edema & increased ICP Major cause falls Child abuse, shaken baby syndrome <1yr. Other causes MVA Bicycle, skateboard, snowboard, skiing Alcohol or drug-related MVAs, sports injuries in teens HEAD INJURY Primary Brain Injury Develop at time of trauma Direct blow Inc. ICP Apnea Loss of consciousness Secondary Brain Injury Results as response to injury Few hours to weeks post injury Inc. ICP can result in irreversible brain damage by decreased CPP (cerebral perfusion pressure) HEAD INJURY Nursing Interventions Assessment Seizure precautions Decrease environment stimulus Coordinate rehabilitation Coordinate resource services Administer meds as ordered Corticosteroids Seizure medications Fosphenytoin Phenobarbital Child and Family Education OTHER HEAD INJURIES Skull fractures Contusion Concussion http://www.cdc.gov/headsup/basics/index.html Subdural hematoma Epidural hematoma SEIZURE DISORDER Most are idiopathic Genetic factors- associated with syndromes Head injury Stroke/cerebrovascular disorders Metabolic disturbances Electrolyte Hypoglycemia Renal failure Hepatic failure Hypoxia SEIZURE DISORDER Chronic disorder characterized by recurrent seizures, result of underlying brain abnormality Intractable seizures Can suffer from poor self-esteem, academic failure, poor social relationships Anti-epileptic Drugs For some surgery may be an option to control seizures Prolonged, uncontrolled seizures often result in developmental delays, neurological damage SEIZURE DISORDER Priority Nursing Diagnoses Risk for aspiration Risk for injury r/t type of seizure and possible loss of consciousness Altered family processes r/t having child with chronic illness Nursing Interventions Keep calm A = Airway Safety Seizure Medications Benzodiazepine (lorazepam, intranasal midazolam) Phenobarbital Tegretol Assessment Provide emotional support for patient/family Resource information and F/U CONGENITAL NEUROLOGIC PROBLEMS Neural tube defects Spina bifida occulta Meningocele Myelomeningocele Hydrocephalus Cerebral Palsy SPINA BIFIDA Congenital neural tube defect affects head and spinal column Approximately 1500 births in US per year Higher the defect the greater the neurologic dysfunction Cause unknown (possibly chemicals, medications, maternal low folic acid levels, genetic) SPINA BIFIDA Pre Op Nursing Care Sac care Position Assess motor function Assess bowel and bladder function Nutrition/Hydration Post Op Nursing Care Monitor VS Assess Infection CSF Leak ICP Positions Pain management Education SPINA BIFIDA Associated Musculoskeletal Talipes (clubfoot), dislocated hip, scoliosis, kyphosis neurogenic bladder, hydronephrosis, renal damage, UTI, incontinence Genitourinary Gastrointestinal Hydrocephalus cognitive deficit, visual perceptual problems, sensory dysfunction, paralysis, muscle weakness, feeding difficulties, swallowing problems, sleep apnea Integumentary constipation, impaction, incontinence Neurologic Problems Skin breakdown related to immobility, incontinence Psychosocial Latex sensitivity/ allergy SPINA BIFIDA Nursing Interventions Assistance with bowel and bladder function Promote Mobility Maintain skin integrity Positioning & turning Provide emotional support Resources Child and Family Education HYDROCEPHALUS Imbalance between production and absorption of CSF Most common congenital defect (50%) Etiology Congenital Acquired from meningitis, trauma, hemorrhage in premature infant Idiopathic (50%) If untreated can cause permanent brain damage related to increased ICP HYDROCEPHALUS Ventriculoperitoneal Shunt HYDROCEPHALUS Signs & Symptoms All Children Vomiting Lethargy Cheyne-Stokes respiratory pattern Infant Inc. head circumference Split cranial sutures High-pitched cry Bulging fontanels Irritability when awake Seizures Signs & Symptoms Toddlers & Older Children Setting-sun eyes Seizures Irritability Papilledema Decreased LOC Inc. B/P HA Difficulty with balance & coordination HYDROCEPHALUS Priority Nursing Diagnoses Altered tissue perfusion (cerebral) High risk for infection Risk for impaired skin integrity r/t large size of head and inability to move Nursing Care Ventriculoperitoneal Shunt Provide post-op care Assess Infection ICP HOB? Antibiotic therapy Provide emotional support Child and Family Education CEREBRAL PALSY Non-progressive motor and posture dysfunction secondary to anoxic damage to motor centers in fetal or infant brain( up to 2 yrs.) 70% prenatal (fetal), 20% perinatal, 10% <2 years 2- 2.5:1000 Abnormal muscle tone and lack of coordination 4 types: spastic, dyskinetic, ataxic & mixed Symptoms depend on area of brain involved CEREBRAL PALSY All infants that show developmental delays, feeding difficulties, abnormal muscle tone should be evaluated Turn infant’s head to one side, persistent asymmetric tonic neck reflex beyond 6 mo. Indicates pathologic condition, if any primitive reflexes persist should suspect CP Other complications: Intellectual disabilities, vision impairment, hearing loss, delays in speech and language, seizures CEREBRAL PALSY Priority Nursing Diagnoses Impaired physical mobility Self-care deficit Impaired verbal communication Altered nutrition: less than body requirements High risk for injury r/t neuromuscular, perceptual, or cognitive impairments Fatigue Nursing Interventions Provide adequate nutrition Maintain skin integrity Promote physical mobility Developmental progression Safety Emotional support Team effort Child and Family Education DEVELOPMENTAL DEFICITS Pervasive Developmental Disorders Autistic spectrum disorders Asperger’s syndrome, Rett’s disorder, childhood disintegrative disorder Approximately 1% of children, 1 in 100-150 Neurodevelopmental Disorder Screening at well checks Early intervention PERVASIVE DEVELOPMENTAL DISORDERS Priority Nursing Diagnoses Communication, verbal, impaired Social interaction, impaired Injury, risk for Caregiver role strain, risk for Coping, family, compromised COGNITIVE DISORDERS Learning Disabilities Alteration in information reception and processing Not low IQ Mental Retardation Intellectual functioning and adaptive behavior Congenital developmental disability Low IQ Trisomy 21 (Down Syndrome) COGNITIVE DEFICITS Expected Outcomes: Learning Disabilities Will compensate through new strategies Mental Retardation Reach highest level of independence Encourage optimal family use of resources COGNITIVE DEFICITS Nursing Care: Language, Motor Delays = Risk for Learning Disabilities Refer for testing Early Developmental Testing Identify cognitive delays Support Group Referral Help Families Set goals: life and learning skills Promote self-esteem Partner with Families Plan health interventions together PEDIATRIC PAIN MANAGEMENT Non-pharmacological Distraction Play Position Heat/cold Guided Imagery Sucrose Pharmacological Non-narcotic Acetaminophen Ibuprofen (>6 months) NO aspirin Narcotic Morphine Fentanyl Acetaminophen with codeine