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Transcript 
Smooth Muscle Cell Apoptosis Promotes Vessel
Remodeling and Repair via Activation of Cell Migration,
Proliferation, and Collagen Synthesis
by Haixiang Yu, Murray C.H. Clarke, Nichola Figg, Trevor D. Littlewood, and Martin
R. Bennett
Arterioscler Thromb Vasc Biol
Volume 31(11):2402-2409
October 19, 2011
Copyright © American Heart Association, Inc. All rights reserved.
Diphtheria toxin (DT) administration promotes vascular smooth muscle cell (VSMC) apoptosis
after vessel surgery.
Haixiang Yu et al. Arterioscler Thromb Vasc Biol.
2011;31:2402-2409
Copyright © American Heart Association, Inc. All rights reserved.
Vascular smooth muscle cell (VSMC) apoptosis increases vessel wall area after vessel ligation.
Haixiang Yu et al. Arterioscler Thromb Vasc Biol.
2011;31:2402-2409
Copyright © American Heart Association, Inc. All rights reserved.
Vascular smooth muscle cell (VSMC) apoptosis increases medial and neointimal cell
proliferation.
Haixiang Yu et al. Arterioscler Thromb Vasc Biol.
2011;31:2402-2409
Copyright © American Heart Association, Inc. All rights reserved.
Vascular smooth muscle cell (VSMC) apoptosis before vessel ligation prevents neointimal
formation.
Haixiang Yu et al. Arterioscler Thromb Vasc Biol.
2011;31:2402-2409
Copyright © American Heart Association, Inc. All rights reserved.
Vascular smooth muscle cells (VSMCs) undergoing apoptosis cause release of cytokines that
promote cell proliferation, migration, and matrix synthesis.
Haixiang Yu et al. Arterioscler Thromb Vasc Biol.
2011;31:2402-2409
Copyright © American Heart Association, Inc. All rights reserved.
Apoptotic vascular smooth muscle cells (VSMCs) cause release of interleukin-6 (IL-6) in
conditioned media.
Haixiang Yu et al. Arterioscler Thromb Vasc Biol.
2011;31:2402-2409
Copyright © American Heart Association, Inc. All rights reserved.
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