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Pharmacology of Antivirals for HZV and VSV Skin Infections – Practice Questions Block 7 - Pharmacology 1. Which of the following correctly describes the mechanism of action for nucleoside analogues for the treatment of viral skin infections? A. Nucleoside analogues are converted to nucleotide triphosphates and competitively inhibit viral DNA polymerase. B. Nucleoside analogues are converted to nucleotide triphosphates and competitively inhibit viral RNA polymerase. C. Nucleoside analogues are converted to nucleotide triphosphates and competitively inhibit host cell DNA polymerase. D. Nucleoside analogues are converted to nucleotide triphosphates and competitively inhibit host cell RNA polymerase. E. Nucleoside analogues are converted to ribonucleotide triphosphates and competitively inhibit viral RNA polymerase. 2. Which of the following antiviral medications would be most toxic to a patient in renal failure? A. B. C. D. 3. Acyclovir Famciclovir Pentciclovir Valacyclovir The most common side effect reported by patients when using topical antimicrobials is… A. B. C. D. E. Headache Nausea Vomiting Steven-Johnson’s Syndrome Application site reactions 4. Which of the following statements is true concerning the concomitant use of corticosteroids with antivirals? A. Concomitant use of corticosteroids and antivirals has been shown to resolve postherpetic neuritis. B. Concomitant use of corticosteroids and antivirals has been shown to resolve acute neuritis. C. Concomitant use of corticosteroids and antivirals has been shown to shorten VZV infection. D. Concomitant use of corticosteroids and antivirals has been shown to shorten HSV infection. E. Concomitant use of corticosteroids and antivirals has been shown to resolve postherpetic neuritis and acute neuritis. 1 Pharmacology of Antivirals for HZV and VSV Skin Infections – Practice Questions Block 7 - Pharmacology 5. Which of the following best describes the mechanism of action of Docosanol? A. Inhibition of fusion between the HSV capsid and the HSV envelope preventing viral entry. B. Inhibition of fusion between the plasma membrane and the HSV capsid preventing viral entry. C. Inhibition of fusion between the plasma membrane and the HSV envelope preventing viral entry. D. Inhibition of fusion between the plasma membrane and the HSV envelope preventing vial assembly before budding. E. Inhibition of fusion between the plasma membrane and the HSV capsid preventing viral assembly before budding. 2