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From: Long-Term Ritonavir Exposure Increases Fatty Acid and Glycerol Recycling in 3T3-L1 Adipocytes as
Compensatory Mechanisms for Increased Triacylglycerol Hydrolysis
Endocrinology. 2010;151(5):2097-2105. doi:10.1210/en.2009-1364
Endocrinology | Copyright © 2010 by the Endocrine Society
From: Long-Term Ritonavir Exposure Increases Fatty Acid and Glycerol Recycling in 3T3-L1 Adipocytes as
Compensatory Mechanisms for Increased Triacylglycerol Hydrolysis
Endocrinology. 2010;151(5):2097-2105. doi:10.1210/en.2009-1364
Endocrinology | Copyright © 2010 by the Endocrine Society
From: Long-Term Ritonavir Exposure Increases Fatty Acid and Glycerol Recycling in 3T3-L1 Adipocytes as
Compensatory Mechanisms for Increased Triacylglycerol Hydrolysis
Endocrinology. 2010;151(5):2097-2105. doi:10.1210/en.2009-1364
Endocrinology | Copyright © 2010 by the Endocrine Society
From: Long-Term Ritonavir Exposure Increases Fatty Acid and Glycerol Recycling in 3T3-L1 Adipocytes as
Compensatory Mechanisms for Increased Triacylglycerol Hydrolysis
Endocrinology. 2010;151(5):2097-2105. doi:10.1210/en.2009-1364
Endocrinology | Copyright © 2010 by the Endocrine Society
From: Long-Term Ritonavir Exposure Increases Fatty Acid and Glycerol Recycling in 3T3-L1 Adipocytes as
Compensatory Mechanisms for Increased Triacylglycerol Hydrolysis
Endocrinology. 2010;151(5):2097-2105. doi:10.1210/en.2009-1364
Endocrinology | Copyright © 2010 by the Endocrine Society
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