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Transcript
Intrapartal Complications
Complications of the:
Powers
Passageway
Passenger
Placenta
Complications of the Powers
 Dystocia
 Dysfunctional or uncoordinated uterine contractions that result in a
prolongation of labor
Normal labor curve
Abnormal labor curve
Dysfunctional Labor
Hypertonic
Hypotonic
Phase of Labor
Latent (< 4 cm)
Active (> 4 cm)
Symptoms
↑freq
Risks
Fetal Distress-early Infection, Exhaustion,
in labor process
Hemorrhage,
Late fetal distress
Treatment
Rest Uterus
Stimulate Uterus
R/O CPD, Breech, etc.
Medications
Morphine, Stadol
Pitocin
& intensity of ↓ freq & intensity of
contractions, pain
contractions,
 ↓effectiveness
No progress in labor
Nursing Care
Hypertonic Dystocia
Hypotonic Dystocia
•Bedrest
•Sedation to promote relaxation
and reduce pain
•Careful monitoring of mother
and fetus
•Relaxation techniques
•Pain management
•LOTS OF ENCOURAGEMENT
•Careful monitoring of mother
and fetus
•Offer warm shower
•Relaxation techniques
•Assist with AROM and careful
monitoring of fetus
•Prepare to start Oxytocin infusion
•LOTS OF ENCOURAGEMENT
Precipitous Labor
 < 3 hours; Rapid Dilatation and Decent
 Risks
 Mom: genital tract lacerations, abruptio placentae,
postpartum hemorrhage
 Fetus: meconium-stained fluid, bruising, cerebral
trauma
 Treatment—safe passage of fetus through
perineal support, calm atmosphere, careful
assessment postpartum of both mom and
baby
 Treatment, if Hx of precipitous labor
 Induce w/SROM
 BE READY
Preterm Labor (PTL) = < 37
weeks
 PTL is the #1 perinatal and neonatal problem in
US.
 A major goal of Healthy People 2020
Reduce PTL rate in US to 7.6 %
In 2011, 12.8 % of all babies were born
preterm (all-time high  )
Rate is INCREASING, not decreasing
Maternal Causes
 Race, SES, Age,
< High School Education
Unmarried
 Smoking
 History of
 AB’s
 LBW/PTL
 Metabolic Disease
 Alcohol in excess
 UTI in 3rd Trimester
 Illicit Drugs eg. Cocaine,
heroine
 DES Exposure
 Poor Nutrition
 Anemia
 Exposure to Toxins
 Low Wt. Gain in PG
 Domestic Abuse
 Infections
 PIH
 Short interpregnancy interval
 Hx of Heart Disease
 Type 1 or 2 Diabetes
Other Factors
 Fetal
Multiple
gestation
Macrosomia
Polyhydramnios
Early
Engagement
Fetal Distress
 Placental
Previa
Abruption
Risks
 Mother
If Placenta is
cause
 severe
hemorrhage and
Shock
 Fetus
RDS and other
complications of
prematurity
Hypoxia if the
problem is
placental
Symptoms
 Uterine Activity
 Cx q 10” for 1 hour
 w/ or w/o pain
 Cervical changes
 Discomfort
 Dilatation of >2cm
 Abdominal Cramping; w/ or
w/o diarrhea
 Effacement of >80%
 Dull, low back pain
 Vaginal Discharge
 Painful menstrual-like cramps
 Thicker or thinner
 Suprapubic pain
 Sudden spotting or blood,
brown or colorless discharge
 Urinary frequency
 ↑ amt.; malodorous
 SROM
 Pelvic pressure
Treatment
 Bedrest
 Hydration
 Antibiotics(if evidence of infection)
 Analgesic
 May be used in conjunction with tocolytics
Tocolytic drugs
 Magnesium Sulfate—MgSO4 (IV)
 Bolus of 4-6 Gms over 15-30 min,
then 1-4 gm/hour till contractions stop.
 Maternal Mg serum level for effectiveness in
tocolysis is 5.5-7.5mg/dL
 Follow all nursing care r/t MgSO4 discussed earlier
 Nifedipine (Procardia; Ca++ channel
blocker)
 10-20 mg PO; 20 mg q6 hr x 24 hrs; 20mg q8 hrs
 Because mechanism of action is different from betaadrenergic agonists, it might be used in conjunction
with terbutaline or ritodrine.
Tocolytic drugs cont’d.
 Indomethacin (Indocin)-
used for short-term management of PTL
especially if Beta adrenergic agonists
failed. Best to use for <5-7 days. As a
prostaglandin inhibitor, it helps to stop
contractions and prevent release of
Oxytocin.
 po or pr: 25-50 mg q6hr for 48hr.
Discontinue if birth is imminent or likely
to occur within 24hr.
Tocolytic drugs cont’d
 17 Alpha-Hydroxyprogesterone Caproate
Used only with single gestation pregnancies
Acts to relax smooth muscle ie pregnant
uterus
Administered to prevent PTL
Used for long-term management of PTL
administered weekly
(17 P) IM injection given z-track slowly over 35 min, to minimize discomfort best to ice the
injection site prior to administration
Side Effects & Complications of
Magnesium Sulfate
 Magnesium Sulfate
 SIDE EFFECTS~Mom: flushing, drowsiness, muscle
weakness, blurred vision, N& V
 COMPLICATIONS~Mom: pulmonary edema, respiratory
depression or arrest, cardiac arrest, profound hypotension,
hyporeflexia
 COMPLICATIONS Neonate: hypermagnesemia
Side Effects & Complications
of Calcium Channel Blockers
 SIDE EFFECTS~ MOM: flushing,
tachycardia
 COMPLICATIONS~MOM: profound
hypotension, possible decrease in
uteroplacental perfusion
Side Effects & Complications
of Prostaglandin Inhibitors~
Indomethacin
 SIDE EFFECTS~ MOM: epigastric pain, nausea &
vomiting
 COMPLICATIONS~MOM: GI bleeding, renal failure
 COMPLICATIONS Neonate: premature closure of the
ductus arteriosus, necrotizing enterocolitis, intracranial
hemorrhage
Tocolytics: Beta2 Adrenergic
Agonists
 Terbutaline/Brethine
 SQ
 .25 mg q 20-30” for 2 hrs
 .25 mg q 3-4 hrs
 SQ Pump
 0.03-.01 mg/hr
 Max = 3 mg/24 hr
 PO
 2.5 – 5.0 mg Q 4-6 hrs
Tocolytics: Beta2 Adrenergic
Agonists
Lots of SIDE EFFECTS
 Maternal
 SOB, tachypnea, pulmonary
edema
 Chest pain, ↓ B/P, Palpitations
 Fluid retention, ↓ Urine
 Tremors, Muscle cramps, H/A
 Hyperglycemia, hypokalemia,
hypocalcemia, metabolic acidosis
 N/V
 Fetal
 Tachycardia
 Hyperinsulinemia
 Hyperglycemia (Fetus)
 Hypoglycemia (Neonate)
 Hyperbilirubinemia
 Hypotension
Nursing Care w/Tocolytics
 Monitor IV rates CAREFULLY
 Continuous EFM—record q 15 minutes
 If FHR> 180 bpm, STOP beta adrenergic agonists
 Call MD
 Maternal VS and Cxs;
 record q 15” until stable then q 30”
 Notify MD if P > 120,
 STOP meds if:
P > 120, > 6 PVC’s/min, systolic > 180, diastolic < 40,
c/o chest pain, SOB
Nursing Care w/Tocolytics
 Strict I& O
 Bedrest—Left Lateral
 Lung sounds---Pulmonary edema
 Daily Weights
 Urine for Glucose
 Serum Electrolytes
 EMOTIONAL SUPPORT
PTL—Home Therapy
 Timing of taking oral medications
 Palpate contractions
 No heavy lifting, nipple stimulation, intercourse
 Quit work—take LOA
 May have uterine home monitoring
 Teach symptoms of PTL early in pregnancy
Premature Rupture of
Preterm PROM—rupture
before 37 weeks
Membranes
gestation
Diagnosis
Nitrazine paper, pH strip- color change??
Fern test
Risks
Maternal: Chorioamnionitis/endometritis
Fetal: PTL/Prematurity
Stress of PROM may stimulate surfactant production
and thus ↓ incidence of RDS
Treatment of preterm PROM
 If infection noted  Deliver
 If w/o infection; conservative mgmt
 VS q 4 especially noting elevated temp
 CBC, vaginal culture on admission
 Frequent BPP—assess amt of amniotic fluid
 Assess for uterine tenderness, any vaginal leaking
 Prophylactic antibiotics for 48 hrs often given
 Modified bedrest (NO WORK)
 NOTHING in Vagina; No Intercourse or tub bath
Treatments
 Corticosteroids stimulates
surfactant production,  risk
of NEC, & IVH in Fetus
 Betamethasone
(Celestone) = 12 mg IM
x 2 doses 24 hours apart
Wait 1 week and
repeat
 Dexamethasone
(Decadron) = 6 mg IM
X 4 doses 12 hours apart
 Fetal Kick Counts
 Choose time of day to
sit quietly
 Count to 10
If < 10 movements
in 12 hrs Call MD
 After meals, Count 4
movements
If < 4 movements in
2 hrs  Call MD
Complications of the
Passageway
 Cephalopelvic Disproportion (CPD)
 Risks
 Uterine Rupture
 Assisted Delivery cervical/vaginal lacerations
 Trauma to fetal head,
 Fracture, CNS damage
 Treatment  Cesarean Section
Complications of the
Passenger
 Malpresentation
Tranverse Lie
Breech
Brow/Face
 Multiple Gestation
 IUFD
 Fetal Distress
 Shoulder Dystocia
Multiple Gestation (Twins +)
 Increase risk of PTL, Malpresentation, PIH,
Maternal Hemorrhage
 ↑ incidence d/t fertility treatments
 Most common is twins
 1/85 births is a twin
Twins
 Monozygotic—33% of all twins
 1 egg + 1 sperm= “Identical”
 Variations
 2 amnions/2 chorions 30%
(Dichorionic/diamniotic)
 2 amnions & 1 chorion—68%
(monochorionic/diamniotic)
 1 amnion & 1 chorion –2%
(monochorionic/monoamniotic)
 MOST COMPLICATIONS
 Twin-to-twin transfusion
 Dizygotic— 67% of all twins
 2 eggs = 2 sperm = “Fraternal”
 2 ovums + 2 placentas = 2 babies
Risk for Multiple Gestation
 Family HX
 Increased maternal age
 Increased parity
 Conceiving within 1 month of stopping OC
 Increased frequency of Coitus
Risks
 Maternal
 PTL
 Cardiac stress
 Anemia
 PIH
 Fetal
 Congenital
anomalies
 Monozygotic
twin-to-twin
tranfusion
 Polyhydramnios
Polycythemic
 Placenta previa
Anemic
 Dysfunctional Labor
 Abnormal
Presentation
Umbilical Cords
intertwined
Management
 Antepartum
 U/S early to confirm
twins
 > # of office visits
 ↑ caloric needs—see
dietician
 ↑ rest
 Assess for infection
 Monitor fetal status
 U/S, NST’s, BPP
 Intrapartum
 Monitor twins
 1 tocotransducter
 2 U/S transducers or 1
U/S transducer and 1
scalp electrode
 Maternal VS, IV’s
 Vag delivery with C/Sec
back up
 2 OB’s/Peds/RN’s
 May have 1 baby
vaginally and 1 baby by
C/Section
Management
 Postpartum
 Assess CLOSELY for
Uterine Atony
 Emotional Support
 Support with
Breastfeeding
 If Triplets or Quads or +++
 C/Section is delivery method
of choice
 Referrals to social
worker/PHN
Morgan, Sam, & Ben
Shoulder Dystocia-an obstetrical
emergency
 An intrapartum event that occurs when the
infant’s head has been delivered, but the
shoulders remain wedged behind the mother’s
pubic bone
 Risk factors
Macrosomic babies are most at risk
 GDM, Obesity, hx of previous LGA baby or
previous shoulder dystocia
 Shoulder dystocia may occur when the woman
has no risk factors.
Management
 Position in McRobert’s position
Legs and thighs flexed up to her
abdomen with the head of the bed
lowered
Apply suprapubic pressure
Apply pressure directly over they
symphysis pubis to aid in dislodging
the fetal shoulder
Complications of Shoulder
Dystocia
 Permanent injury to baby
 Brachial plexus injury (caused by excessive traction on
fetal head)
 Fractured clavicles
 Asphyxia
 Neurologic damage
 Maternal Complications
 Heavy bleeding after delivery
 Tearing of the uterus, vagina, cervix or rectum
 Bruising of the bladder
Other Fetal Complications
 IUFD—Intrauterine Fetal Demise
 Often detected by absent fetal movement
 Nursing Dx: Grieving, Altered family processes,
Ineffective individual coping
 Goal: a supportive, pain-free delivery with resources
available to make this a special memory for the family

Cause is often unknown or there is some
physiological maladaptation such as placenta
previa/abruptio, maternal diabetes, or severe
renal disease, profound congenital anomalies.

Risks to mother -- prolonged retention of dead
fetus can lead to DIC

Diagnosis is based on absence of fetal heart
tones and/or ultrasound

Usually labor will begin on its own, if not, labor
will be induced within 2 weeks of documented
demise

During labor, the woman is often sedated and an
epidural is initiated soon after the onset of
contractions so that labor is made as painless as
possible.
 The couple may or may not wish to see the baby at that time,
some may want to hold their baby. Treat them with respect. If the
parents do not wish to see the baby, the baby should be baptized
(if parents are Catholic), pictures taken, identification bands made
out, foot prints taken, a lock of hair cut, and a weight and length
recorded. This information is given to the family in a sealed
envelope for them to open whenever they wish. If parents wish to
hold the baby, give them some privacy and be near to answer any
questions.
 Post-partally, offer the parents to be transferred off the maternity
unit, and allow the father to stay as much as possible. Call pastor,
priest, or rabbi for support. Refer to support groups, such as
Resolve, Share, or Compassionate Friends.
 Studies have shown that parental grief after a stillbirth is aided if
the parents name the baby, see the baby, hold the baby, and bury
the baby.
 BE COMPASSIONATE, IT IS OKAY TO CRY WITH THE FAMILY.
TRY TO FORGET YOUR OWN DISCOMFORT IN ORDER FOR
THE FAMILY TO EXPRESS THEIRS.
Complications of the Placenta
Placenta Previa
Placenta Abruptio
Placenta Accreta
Umbilical Cord Prolapse
Placenta
Previa
 Types
 Low Implantation
 Partial Previa
 Complete Previa
 PAINLESS VAGINAL
BLEEDING in the 2nd3rd Trimester
 Dx
 Ultrasound
 Management
 Hospitalized, Bedrest
 Tocolysis, if contracting
 C/Sec
 NO VAGINAL EXAMS
Placenta Abruptio
 Types
 Covert/Concealed
 Overt/Partial
 Overt/Complete
 Symptoms
 Knife-like pain w/concealed
 Shock
 Varying amt. of bleeding
 DX
 Fetal Distress
 U/S or CAT Scan
 Treatment
 Emergency C/Sec
Placenta Accreta
 Placenta adheres to uterine myometrium
It attaches itself too deeply into the
lining of the uterus
 Maternal hemorrhage is often severe
Does not respond to treatment for P/P
hemorrhage
 Often results in hysterectomy
Umbilical Cord
Prolapse
Extremely critical obstetrical
situation
Cord protudes from cervix
into vagina
Seen in breech and when
presenting part is
unengaged
Position Mom
Knee-chest, Trendelenburg,
elevate hips
Sterile gloved hand—hold
presenting part off cord
EMERGENCY C/SECTION,
O2, ↑IV flow rate
Other Complication
Amniotic Fluid Embolism
 Pathophysiology
 Amniotic fluid enters
maternal circulation→
pulmonary capillaries
 Tiny emboli form →
pulmonary vasospasm
→Hypoxemia and Acute
Right-sided Heart Failure
 Symptoms
Restlessness
Chills
Pallor
 Vernix and Lanugo
↓ B/P, ↑Pulse, ↑
Resp.
 DIC may develop
Dyspnea
Chest Pain
Amniotic Fluid Embolism
 Medical
Management
Drugs
Morphine
 Nursing
 Follow orders
 Semi-fowler’s position
 Oxygen
 Medication
Aminophyllie
 Blood Products
Digoxin
I & O
Cortisone
 If undelivered C/Sec
 STAY WITH MOTHER if
suspect AFE
Nurses must be alert
to symptoms of what
can go wrong and
take initial steps to
enhance the health
of the mother and
the baby.