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1. A. B. C. D. * E. 2. A. B. C. D. * E. 3. A. B. C. D. * E. 4. A. B. C. D. E. * 5. A. B. C. D. E. * 6. A. B. C. D. E. * 7. A. * B. C. D. E. 8. A. * B. C. All the following medicines are hepatoprotective agents except: Carsil Silibor Legalon Amicacinum Arginine All the following medicines are hepatoprotective agents except: Essenciale Silibor Legalon Atenolol Hepadif All the following medicines are hepatoprotective agents except: Carsil Silibor Legalon Lomusol Hepabene All the following medicines are interferons except: Intron Pegasis Reaferon Leukinferon Cycloferon All the following medicines are interferons except: Intron Roferon Reaferon Leukinferon Ceftriaxon All the following medicines are interferons except: Intron Roferon Reaferon Laferobion Legalon All the hepatitis have parenteral route of transmission except: A B C D TTV All the hepatitis have parenteral route of transmission except: E B C D. E. 9. A. * B. C. D. E. 10. A. * B. C. D. E. 11. A. B. C. D. E. * 12. A. B. C. D. E. * 13. A. B. C. D. E. * 14. A. B. C. D. E. * 15. A. * B. C. D. E. D TTV All the hepatitis have parenteral route of transmission except: A and E B and D C and B D and C TTV All the hepatitis have parenteral route of transmission except: A B C D TTV As etiotropic therapy of acute and chronic viral hepatitis B utillize: Corticosteroid Immunomodulate preparations Cytostatics Antibiotics Antiviral preparations As etiotropic therapy of sharp and chronic viral hepatitis B utillize: Corticosteroid Immunomodulate preparations Cytostatics Antibiotics Antiviral preparations Basic principles of antiviral therapy for viral hepatitis. Individual selection of dose and rhythm of application of preparations Duration of introduction of preparations Control of amount of erytrocytes, leucocytes and thrombocytes, in blood Control of iron level in blood All the above Basic principles of antiviral therapy for viral hepatitis. Individual selection of dose and rhythm of application of preparations Duration of introduction of preparations Control of amount of erytrocytes, leucocytes and thrombocytes, in blood Control of iron level in blood All the above Before the appearance of jaundice in a patient 16 years old, during 2 days there were an increasing of the body temperature (38,5 °C), headache, dull ache disturbed whole body. Name the variant of pre-icteric period of viral hepatitis for this patient? Influenza-like Astenovegetative Artralgic Dyspeptic Allergic 16. A. * B. C. D. E. 17. A. B. * C. D. E. 18. A. * B. C. D. E. 19. A. B. * C. D. E. 20. A. B. * C. D. E. 21. A. B. * C. D. E. 22. A. B. * C. D. E. 23. A. B. * C. Choose the indexes of efficiency of interferon therapy. Disappearance| of markers of viral replication Improvement of the general state Normalization of the liver size Disappearance of icterus All the above Choose the indexes of efficiency of interferon therapy. Improvement of the general state Normalization of activity of ALaT Normalization of the liver size Disappearance of icterus All the above Choose the indexes of efficiency of interferon therapy. Disappearance| of markers of viral replication Improvement of the general state Normalization of the liver size Disappearance of icterus All the above Choose the indexes of efficiency of interferon therapy. Improvement of the general state Normalization of activity of ALaT Normalization of the liver size Disappearance of icterus All the above Choose the indexes of efficiency of interferon therapy. Improvement of the general state Normalization of activity of ALaT Normalization of the liver size Disappearance of icterus All the above Choose the remedies for etiotropic therapy for viral hepatitis. Antibiotics Interferon Probiotics Vaccine Normal human immunoprotein Choose the remedies for etiotropic therapy for viral hepatitis. Antibiotics Interferon Probiotics Vaccine Normal human immunoprotein Choose the remedies for etiotropic therapy for viral hepatitis. Antibiotics Interferon Probiotics D. E. 24. A. * B. C. D. E. 25. A. * B. C. D. E. 26. A. * B. C. D. E. 27. A. * B. C. D. E. 28. A. * B. C. D. E. 29. A. * B. C. D. E. 30. A. 31. A. B. C. D. E. * Vaccine Normal human immunoprotein Choose the remedies for etiotropic therapy of viral hepatitis. Ribavirin Vaccine Normal human immunoprotein Hepatoprotector Glucocorticoid Choose the remedies for etiotropic therapy of viral hepatitis. Ribavirin Vaccine Normal human immunoprotein Hepatoprotector Glucocorticoid Chronic course is common for viral hepatitis except: A B C D B+C Chronic course is common for viral hepatitis except: A B C D B+C Chronic course is common for viral hepatitis except: A B C D B+C Chronic course is common for viral hepatitis except: A B C D B+C Contra-indications for antiviral therapy of viral hepatitis. Contra-indications for antiviral therapy of viral hepatitis. ecompensatory cirrhosis of liver Autoimmune disease Alcoholism and other drug addictions Coinfection by HIV All the above 32. A. 33. A. B. C. D. E. * 34. A. 35. A. B. * C. D. E. 36. A. B. * C. D. E. 37. A. B. * C. D. E. 38. A. B. C. D. E. * 39. A. B. C. D. E. * 40. A. B. Contra-indications for antiviral therapy of viral hepatitis. Contra-indications for antiviral therapy of viral hepatitis. ecompensatory cirrhosis of liver Autoimmune disease Alcoholism and other drug addictions Coinfection by HIV All the above Diagnosed a patient: chronic hepatitis in the stage of integration. What markers will be in patient in this stage disease? Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing of stercobilin of excrements. What is the type of icterus? Haemolitic Parenchymatous Transport Extraliver Mechanical Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing of stercobilin of excrements. What is the type of icterus? Haemolitic Parenchymatous Transport Extraliver Mechanical Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing of stercobilin of excrements. What is the type of icterus? Haemolitic Parenchymatous Transport Extraliver Mechanical Etiotropic therapy of viral hepatitis is. Ribavirin Interferon Inductors of interferon Zefix All the above. Etiotropic therapy of viral hepatitis is. Ribavirin Interferon Inductors of interferon Zefix All enumerated. Factors which are indications of successful interferon therapy in HV infections are all, except. Level of ALaT not more than 2-3 norm Low titre of HCV after the treatment C. D. E. * 41. A. B. C. D. E. * 42. A. B. C. D. E. * 43. A. * B. C. D. E. 44. A. B. C. D. E. * 45. A. * B. C. D. E. 46. A. B. C. D. E. * 47. A. B. Absence of cholestasis 2th and 4th genotypes of HCV Expressed fibrosis Factors which are indications of successful interferon therapy in HV infections are all, except. Level of ALaT not more than 2-3 norm Low titre of HCV after the treatment Absence of cholestasis 2th and 4th genotypes of HCV Expressed fibrosis Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical supervision after isolation. A medical supervision during 6 month Biochemical inspection Abstain from hard physical load Temporal contra-indications for prophylactic inoculations All the above Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical supervision after isolation. Medical supervision during 6 months, periodic biochemical inspections. Control bacteriological examinations Full labor investigation To continue prophylactic inoculations Supervision is not needed Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical supervision after isolation. A medical supervision during 6 month Biochemical inspection Abstain from hard physical load Temporal contra-indications for prophylactic inoculations All the above Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical supervision after isolation. Medical supervision during 6 months, periodic biochemical inspections. Control bacteriological examinations Full labor investigation To continue prophylactic inoculations Supervision is not needed Indirect action of interferon therapy are all except. Influenza-like syndrome Nausea Depression Intensification of autoimmune diseases Progress of fibrosis Indirect action of interferon therapy are all except. Influenza-like syndrome Nausea C. D. E. * 48. A. B. C. D. E. * 49. A. B. C. D. E. * 50. A. B. C. D. E. * 51. A. B. C. D. E. * 52. A. B. C. D. E. * 53. A. B. C. D. E. * 54. A. B. C. Depression Intensification of autoimmune diseases Progress of fibrosis Indirect action of interferon therapy are all except. Influenza-like syndrome Nausea Depression Intensification of autoimmune diseases Progress of fibrosis Indirect action of interferon therapy are all except. Influenza-like syndrome Nausea Depression Intensification of autoimmune diseases Progress of fibrosis Indirect action of interferon therapy. Influenza-like syndrome Nausea Itching Para-hypnosis All the above Indirect action of interferon therapy. Influenza-like syndrome Nausea Itching Para-hypnosis All the above Indirect action of interferons. Flatulence Diarrhea Nausea Depression All the above Indirect action of interferons. Flatulence Diarrhea Nausea Depression All the above On the average 15 to 30 % of all population of the planet suffer from some pathology of liver. Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the world there are about 2 million people with acute viral hepatitis. What % of all cases will develop chronic form. 100 % 50 % 25 % D. * E. 55. A. B. C. D. * E. 56. A. B. C. D. * E. 57. A. * B. C. D. E. 58. A. * B. C. D. E. 59. A. * B. C. D. E. 60. A. * B. C. D. E. 61. A. B. 10 % 1% On the average 15 to 30 % of all population of the planet suffer from some pathology of liver. Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the world there are about 2 million people with acute viral hepatitis. What % of all cases will develop chronic form. 100 % 50 % 25 % 10 % 1% On the average 15 to 30 % of all population of the planet suffer from some pathology of liver. Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the world there are about 2 million people with acute viral hepatitis. What % of all cases will develop chronic form. 100 % 50 % 25 % 10 % 1% That characteristic of a partial answer of interferon therapy are all, except. Disappearance of markers of viral replication Normalization of activity of ALaT is upon completion of course of therapy Disappearance of icterus Normalization the state of patient Normalization of the size of liver That characteristic of a partial answer of interferon therapy are all, except. Disappearance of markers of viral replication Normalization of activity of ALaT is upon completion of course of therapy Disappearance of icterus Normalization the state of patient Normalization of the size of liver That characteristic of a partial answer of interferon therapy are all, except. Disappearance of markers of viral replication Normalization of activity of ALaT is upon completion of course of therapy Disappearance of icterus Normalization the state of patient Normalization of the size of liver That characteristic of a partial answer of interferon therapy are all, except. Disappearance of markers of viral replication Normalization of activity of ALaT is upon completion of course of therapy Disappearance of icterus Normalization the state of patient Normalization of the size of liver The characteristic of an unsteady answer of interferon therapy are. Disappearance of markers of viral replication upon completion of course of therapy Normalization of activity of ALaT during the course of therapy C. D. E. * 62. A. B. C. D. E. * 63. A. B. C. D. E. * 64. A. B. C. D. E. * 65. A. B. C. D. E. * 66. A. B. C. D. E. * 67. A. B. C. D. E. * 68. A. B. C. D. E. * 69. An origin of relapse in next 6 months Disappearance of icterus All the above The characteristic of an unsteady answer of interferon therapy are. Disappearance of markers of viral replication upon completion of course of therapy Normalization of activity of ALaT during the course of therapy An origin of relapse in next 6 months Disappearance of icterus All enumerated The characteristic of an unsteady answer of interferon therapy are. Disappearance of markers of viral replication upon completion of course of therapy Normalization of activity of ALaT during the course of therapy An origin of relapse in next 6 months Disappearance of icterus All the above The criteria for application of etiotropic therapy in viral hepatitis is. Protracted motion of HBV, HVD Any form of HV Biochemical activity Presence of virus replication All the above. The criteria for application of etiotropic therapy for the patient with HCV. Clinical displays are insignificant Icterus is absent Moderate biochemical activity There is anti-HCV in blood RNA of HCV + The criteria for application of etiotropic therapy in viral hepatitis is. Protracted motion of HBV, HVD Any form of HV Biochemical activity Presence of virus replication All the above. The criteria for application of etiotropic therapy for the patient with HCV. Clinical displays are insignificant Icterus is absent Moderate biochemical activity There is anti-HCV in blood RNA of HCV + Types of answer for interferon therapy are. Stable remission Unsteady Partial answer Absence of answer All the above Types of answer for interferon therapy are. A. B. C. D. E. * 70. A. B. C. D. E. * 71. A. B. * C. D. E. 72. A. B. * C. D. E. 73. A. B. * C. D. E. 74. A. * B. C. D. E. 75. A. * B. C. D. E. 76. A. * B. C. D. Stable remission Unsteady Partial answer Absence of answer All the above Types of answer for interferon therapy are. Stable remission Unsteady Partial answer Absence of answer All the above What group of infectious diseases hepatitis A belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis A belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis A belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis B belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis B belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis B belong to: External covers Intestinal Blood Wound E. 77. A. * B. C. D. E. 78. A. * B. C. D. E. 79. A. * B. C. D. E. 80. A. * B. C. D. E. 81. A. * B. C. D. E. 82. A. * B. C. D. E. 83. A. B. * C. D. E. 84. A. B. * Transmissive What group of infectious diseases hepatitis C belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis C belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis C belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis D belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis D belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis D belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis E belong to: External covers Intestinal Blood Wound Transmissive What group of infectious diseases hepatitis E belong to: External covers Intestinal C. D. E. 85. A. B. * C. D. E. 86. A. * B. C. D. E. 87. A. * B. C. D. E. 88. A. * B. C. D. E. 89. A. B. * C. D. E. 90. A. B. * C. D. E. 91. A. B. * C. D. E. 92. Blood Wound Transmissive What group of infectious diseases hepatitis E belong to: External covers Intestinal Blood Wound Transmissive What is incubation period for hepatitis A: 45 days 180 days 360 days 90 days 25 days What is incubation period for hepatitis A: 45 days 180 days 360 days 90 days 25 days What is incubation period for hepatitis A: 45 days 180 days 360 days 90 days 25 days What is incubation period for hepatitis B: 45 days 180 days 360 days 90 days 25 days What is incubation period for hepatitis B: 45 days 180 days 360 days 90 days 25 days What is incubation period for hepatitis B: 45 days 180 days 360 days 90 days 25 days What is incubation period for hepatitis B: A. B. * C. D. E. 93. A. * B. C. D. E. 94. A. * B. C. D. E. 95. A. B. C. D. * E. 96. A. B. C. D. * E. 97. A. B. C. D. * E. 98. A. B. C. D. * E. 99. A. * B. C. D. 45 days 180 days 360 days 90 days 25 days What is mechanism of transmission in viral hepatitis C: Contact Transmissive Alimentary Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis D: Contact Transmissive Alimentary Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis E: Contact Sexual Alimentary Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis A: Contact Transmissive Vertical Fecal-oral Air-drop What is mechanism of transmission in salmonelosis: Contact Transmissive Alimentary Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis A: Contact Transmissive Vertical Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis B: Contact Transmissive Alimentary Fecal-oral E. 100. A. * B. C. D. E. 101. A. * B. C. D. E. 102. A. B. C. D. * E. 103. A. B. C. * D. E. 104. A. B. C. * D. E. 105. A. B. C. * D. E. 106. A. B. C. D. E. * 107. A. B. Air-drop What is mechanism of transmission in viral hepatitis C: Contact Transmissive Alimentary Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis D: Contact Transmissive Alimentary Fecal-oral Air-drop What is mechanism of transmission in viral hepatitis E: Contact Sexual Alimentary Fecal-oral Air-drop What is transmissive factor for hepatitis A: Blood Semen Water Air Milk of mother What is transmissive factor for hepatitis A: Blood Semen Water Air Milk of mother What is transmissive factor for hepatitis A: Blood Semen Water Air Milk of mother What is transmissive factor for hepatitis B: Food Milk Water Air Milk of mother What is transmissive factor for hepatitis B: Food Milk C. D. E. * 108. A. B. C. D. E. * 109. A. B. C. D. * E. 110. A. B. C. D. * E. 111. A. B. C. D. * E. 112. A. B. C. D. * E. 113. A. B. C. D. * E. 114. A. B. Water Air Milk of mother What is transmissive factor for hepatitis B: Food Milk Water Air Milk of mother What laboratory and instrumental examinations are needed for confirming the diagnosis of viral hepatitis. Complete analysis of blood Ultrasound of abdominal region Determination of activity of aminotransferase Determination of antigen of viruses Duodenal probing What laboratory and instrumental examinations are needed for confirming the diagnosis of viral hepatitis. Complete analysis of blood Ultrasound of abdominal region Determination of activity of aminotransferase Determination of antigen of viruses Duodenal probing What laboratory and instrumental examinations are needed for confirming the diagnosis of viral hepatitis. Complete analysis of blood Ultrasound of abdominal region Determination of activity of aminotransferase Determination of antigen of viruses Duodenal probing What laboratory and instrumental examinations are needed for confirming the diagnosis of viral hepatitis. Complete analysis of blood Ultrasound of abdominal region Determination of activity of aminotransferase Determination of antigen of viruses Duodenal probing What laboratory work-up is needed for confirming the diagnosis of viral hepatitis. Total analysis of blood Determination of level of bilirubin Determination of activity of aminotransferase Determination of markers of HV in IFA All the above What laboratory work-up is needed for confirming the diagnosis of viral hepatitis. Total analysis of blood Determination of level of bilirubin C. D. * E. 115. A. B. C. D. * E. 116. A. B. C. D. * E. 117. A. B. C. D. E. * 118. A. B. C. D. E. * 119. A. B. C. D. E. * 120. A. B. C. D. E. * 121. A. B. * C. D. Determination of activity of aminotransferase Determination of markers of HV in IFA All the above What laboratory work-up is needed for confirming the diagnosis of viral hepatitis. Total analysis of blood Determination of level of bilirubin Determination of activity of aminotransferase Determination of markers of HV in IFA All enumerated What laboratory work-up is needed for confirming the diagnosis of viral hepatitis. Total analysis of blood Determination of level of bilirubin Determination of activity of aminotransferase Determination of markers of HV in IFA All the above When is interferon therapy effective in the the patient. Normalization of the state of patient Normalization of activity of ALaT upon completion of course of therapy Disappearance of icterus Normalization of the size of liver The markers of viral replication, are determined upon completion of course of therapy When is interferon therapy effective in the the patient. Normalization of the state of patient Normalization of activity of ALaT upon completion of course of therapy Disappearance of icterus Normalization of the size of liver The markers of viral replication, are determined upon completion of course of therapy When is interferon therapy effective in the the patient. Normalization of the state of patient Normalization of activity of ALaT upon completion of course of therapy Disappearance of icterus Normalization of the size of liver The markers of viral replication, are determined upon completion of course of therapy When is interferon therapy effective in the the patient. Normalization of the state of patient Normalization of activity of ALaT upon completion of course of therapy Disappearance of icterus Normalization of the size of liver The markers of viral replication, are determined upon completion of course of therapy Combinations of loss of body mass, sweating, recurrent аpthus stomatitis and girdle herpes and lymphadenopathy, allowed a physician to assume HIV-infection. It can be discovered at an additional inspection: Increased correlation of CD4/CD8 lymphocyte Diminished correlation of CD4/CD8 lymphocyte Change of neutrophil formula to the left Increased correlation of T8/T4 lymphocyte E. 122. A. * B. C. D. E. 123. A. * B. C. D. E. 124. A. B. C. D. E. * 125. A. B. C. D. E. * 126. A. B. C. D. E. 127. A. B. Diminished correlation of T8/T4 of lymphocyte A sick entered permanent establishment with complaints about general weakness, increase of temperature, pain in throat. Objectively: the mucus cell of retropharynx is bright red, on oral cavity are raids of gum-blush, taken off easily, discovered enlargement of all groups of lymphnode, 2-3cm in a diameter, dense, elastic little painful, not soldered between itself. Liver is enlarged on 3cm, spleen – on 2cm. In blood present leucocytosis and lymphomonocytosis. What is probable diagnosis? Infectious mononucleosis Diphtheria Acute leukosis Quinsies Adenovirus infection A sick entered permanent establishment with complaints about general weakness, increase of temperature, pain in throat. Objectively: the mucus cell of retropharynx is bright red, on oral cavity are raids of gum-blush, taken off easily, discovered enlargement of all groups of lymphnode, 2-3cm in a diameter, dense, elastic little painful, not soldered between itself. Liver is enlarged on 3cm, spleen – on 2cm. In blood present leucocytosis and lymphomonocytosis. What is probable diagnosis? Infectious mononucleosis Diphtheria Acute leukosis Quinsies Adenovirus infection A youth with the catarrhal phenomena found out enlargement of axillary and submandibular lymphatic node, hyperplasia of oral cavity with the magnificent raid of gum-blush on them as points and spots, presence of mucus cell pouring out, increased liver and spleen size.What additional researches must be appointed to do for diagnosis? A test to HIV IFA for the exposure of antibodies toHIV IFA for the exposure of antibodies to the virus of Epstien-Bar Analysis in the presence of mononuclear antibodies All the above A youth with the catarrhal phenomena found out enlargement of axillary and submandibular lymphatic node, hyperplasia of oral cavity with the magnificent raid of gum-blush on them as points and spots, presence of mucus cell pouring out, increased liver and spleen size.What additional researches must be appointed to do for diagnosis? A test to HIV IFA for the exposure of antibodies toHIV IFA for the exposure of antibodies to the virus of Epstien-Bar Analysis in the presence of mononuclear antibodies All the above Call the groups of possible risk of HIV infection: Only homo- and bisexual, prostitutes and other persons who conduct disorderly sexual life: Only drug addicts who enter drugs parenterally Only recipeint of blood, its preparations, sperm and organs Only patients with venereal diseases and parenteral viral hepatitis and from the HIV infected mothers All enumerated Choose the criteria for post contact prophylaxis of HIV infection. A medical failure during working| with the HIV |patient Birth of child by the HIV infected | mother C. D. E. * 128. A. B. C. D. E. * 129. A. B. C. D. E. * 130. A. B. * C. D. E. 131. A. B. C. * D. E. 132. A. B. * C. D. E. 133. A. B. C. * D. E. 134. A. Violence Blood transfusion All the above| Choose the criteria for post contact prophylaxis of HIV infection. A medical failure during working| with the HIV |patient Birth of child by the HIV infected | mother Violence Blood transfusion All the above| Choose the criteria for post contact prophylaxis of HIV infection. A medical failure during working| with the HIV |patient Birth of child by the HIV infected | mother Violence Blood transfusion All the above| Combinations of loss of body mass, sweating, recurrent аpthus stomatitis and girdle herpes and lymphadenopathy, allowed a physician to assume HIV-infection. It can be discovered at an additional inspection: Increased correlation of CD4/CD8 lymphocyte Diminished correlation of CD4/CD8 lymphocyte Change of neutrophil formula to the left Increased correlation of T8/T4 lymphocyte Diminished correlation of T8/T4 of lymphocyte Curent treatment for HIV infection consists of highly active antiretroviral therapy. Choose the correct combination of preparations: 1 NRTIs + 2 IP 1 NRTIs + 2 NNRTI 3 NRTIs 1 NRTIs +1 IP+ 2 NNRTI 2 NRTIs +2 IP Curent treatment for HIV infection consists of highly active artiretroviral therapy. Choose the correct combination of preparations: 1 NRTIs + 2 IP 2 NRTIs + 1 NNRTI 3 NRTIs 1 NRTIs + 1 IP+ 2 NNRTI 2 NRTIs + 2 IP Curent treatment for HIV infection consists of highly active antiretroviral therapy. Choose the correct combination of preparations: 1 NRTIs + 2 IP 1 NRTIs + 2 NNRTI 3 NRTIs 1 NRTIs +1 IP+ 2 NNRTI 2 NRTIs +2 IP Curent treatment for HIV infection consists of highly active artiretroviral therapy. Choose the correct combination of preparations: 1 NRTIs + 2 IP B. * C. D. E. 135. A. B. C. D. * E. 136. A. B. C. D. * E. 137. A. B. C. D. * E. 138. A. B. C. D. * E. 139. A. B. * C. D. E. 140. A. B. * C. D. E. 141. A. * B. C. 2 NRTIs + 1 NNRTI 3 NRTIs 1 NRTIs + 1 IP+ 2 NNRTI 2 NRTIs + 2 IP For today the effective methods of protection from HIV are: Vaccination and immunoprotein Chemoprophylactic Isolation of patients Safe sex and prevention of drug addiction Disinfection For today the effective methods of protection from HIV are: Vaccination and immunoprotein Chemoprophylactic Isolation of patients Safe sex and prevention of drug addiction Disinfection Genetic errors at replication of HIV is: Negative Rare Frequently enough Extraordinarily frequent Virions type one genetically changeable and second – no Genetic errors at replication of HIV is: Negative Rare Frequently enough Extraordinarily frequent Virions type one genetically changeable and second – no How many types of HIV are known? One Two Three Four Five How many types of HIV are known? One Two Three Four Five Immediately after a contact with blood and other biological liquids it is necessary to wash the muddy areas of skin with water and soap and to begin a postcontact prophylaxis, antiretroviral preparations not later than 24-36 hrs 36-48 hrs 48-60 hrs D. E. 142. A. * B. C. D. E. 143. A. B. C. D. * E. 144. A. B. C. D. * E. 145. A. B. C. * D. E. 146. A. B. C. * D. E. 147. A. * 60-72 hrs 72-86 hrs Immediately after a contact with blood and other biological liquids it is necessary to wash the muddy areas of skin with water and soap and to begin a postcontact prophylaxis, antiretroviral preparations not later than 24-36 hrs 36-48 hrs 48-60 hrs 60-72 hrs 72-86 hrs In the order of diminishing of probability of infection of HIV the transferred ways take place in such sequence: Sexual contact, contaminated blood transfusion, operation of tattoo, from breast milk, transplacental transmission Contaminated blood transfusion, transplacental transmission, sexual contact, operation of tattoo, postnatal from mother milk Transplatsental transmission, contaminated blood transfusion, postnatal from breast milk, sexual contact, operation of tattoo Contaminated blood transfusion, transplacental transmission, sexual contact, postnatal from breast milk, operation of tattoo Contaminated blood transfusion, sexual contact, transplacent transmission, operation of tattoo, postnatal from breast milk In the order of diminishing of probability of infection of HIV the transferred ways take place in such sequence: Sexual contact, contaminated blood transfusion, operation of tattoo, from breast milk, transplacental transmission Contaminated blood transfusion, transplacental transmission, sexual contact, operation of tattoo, postnatal from mother milk Transplatsental transmission, contaminated blood transfusion, postnatal from breast milk, sexual contact, operation of tattoo Contaminated blood transfusion, transplacental transmission, sexual contact, postnatal from breast milk, operation of tattoo Contaminated blood transfusion, sexual contact, transplacent transmission, operation of tattoo, postnatal from breast milk Name the main specific methods of diagnosis of HIV infection which is used in Ukraine? RPGA PLR IFA and ELISA Bioassey RIA Name the main specific methods of diagnosis of HIV infection which is used in Ukraine? RPGA PLR IFA and ELISA Bioassey RIA Name the most dangerous parenteral way of infection of HIV/AIDS? Infusion of donor blood and its preparations B. C. D. E. 148. A. * B. C. D. E. 149. A. * B. C. D. E. 150. A. * B. C. D. E. 151. A. B. C. D. * E. 152. A. B. C. D. * E. 153. A. B. C. D. E. * Transplantation of organs Injections of medications Diagnostic manipulations Intravenous introduction of drugs Name the most dangerous parenteral way of infection of HIV/AIDS? Infusion of donor blood and its preparations Transplantation of organs Injections of medications Diagnostic manipulations Intravenous introduction of drugs Name the source of HIV infection/AIDS? Human Warm-blooded animal Poultries Amphibious Fishes Name the source of HIV infection/AIDS? Human Warm-blooded animal Poultries Amphibious Fishes On a background of prolonged treatment for HIV patient, appeared ulcer on the mucous cell of mouth. At an objective examination doctor have found out erosions on the mucous, hyperemic and filling out mucus cell of oral cavity, tongue without raid, with a smooth surface. About what complication is possible to think? Leptospirosis Acute herpes Stevens-Johnson syndrome Candidos stomatitis Layel syndrome On a background of prolonged treatment for HIV patient, appeared ulcer on the mucous cell of mouth. At an objective examination doctor have found out erosions on the mucous, hyperemic and filling out mucus cell of oral cavity, tongue without raid, with a smooth surface. About what complication is possible to think? Leptospirosis Acute herpes Stevens-Johnson syndrome Candidos stomatitis Layel syndrome That is not characterised for I clinical stage of AIDS, in obedience to clinical classification of stages of HIV-infection for adults and teenagers, which is developed by the experts of WHO (in 2002)? Loss of mass of body 5 % from initial Purulent defect of skin and mucus (seborrhic dermatitis, mycotic defect of nails) An episode of herpes zoster during the last five years Risiding of infection of upper respiratory tracts (for example, bacterial sinuitis) Minimum defeats of mucus (recurrent ulcers of mucus shell of oral cavity) 154. A. B. C. D. E. * 155. A. B. C. * D. E. 156. A. B. C. * D. E. 157. A. B. C. * D. E. 158. A. B. C. D. E. * 159. A. * B. C. D. E. 160. A. * B. C. D. That is not characterised for I clinical stage of AIDS, in obedience to clinical classification of stages of HIV-infection for adults and teenagers, which is developed by the experts of WHO (in 2002)? Loss of mass of body 5 % from initial Purulent defect of skin and mucus (seborrhic dermatitis, mycotic defect of nails) An episode of herpes zoster during the last five years Risiding of infection of upper respiratory tracts (for example, bacterial sinuitis) Minimum defeats of mucus (recurrent ulcers of mucus shell of oral cavity) The basic way of transmission of exciter HIV infections/AIDS are such: Aerogene Alimentary Parententeral Through a kiss Bite of mosquito| The basic way of transmission of exciter HIV infections/AIDS are such: Aerogene Alimentary Parententeral Through a kiss Bite of mosquito The experts of WHO consider suspicious in relation to AIDS: Increase 3 and rmore lymph nodes in two anatomical topographic groups (except for inguinal) by sizes more than 2 cm in diameter, which lasts more than 3 months Increase 3 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Increase 2 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than 2 cm in diameter, which lasts more than 2 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 2 months The experts of WHO consider suspicious in relation to AIDS: Loss of weight (3 % and more) Loss of weight (5 % and more) Loss of weight (6 % and more) Loss of weight (9 % and more) Loss of weight (10 % and more) The experts of WHO consider suspicious in relation to AIDS: Diarrhea, which lasts more than 1 months Diarrhea, which lasts more than 2 months Diarrhea, which lasts more than 3 months Diarrhea, which lasts more than 4 months Diarrhea, which lasts more than 5 months The experts of WHO consider suspicious in relation to AIDS: Hypertermia, which lasts more than 1 months Hypertermia, which lasts more than 2 months Hypertermia, which lasts more than 3 months Hypertermia, which lasts more than 4 months E. 161. A. B. C. D. E. * 162. A. B. C. * D. E. 163. A. B. C. * D. E. 164. A. B. C. D. E. * 165. A. * B. C. D. E. 166. A. * B. C. D. E. 167. A. B. Hypertermia, which lasts more than 5 months The experts of WHO consider suspicious in relation to AIDS: Hypertermia, which lasts more than 1 months Diarrhea, which lasts more than 1 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Loss of weight (10 % and more) All the above The experts of WHO consider suspicious in relation to AIDS: Hypertermia, which lasts more than 21 months Diarrhea, which lasts more than 3 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Loss of weight (6 % and more) All the above The experts of WHO consider suspicious in relation to AIDS: Increase 3 and rmore lymph nodes in two anatomical topographic groups (except for inguinal) by sizes more than 2 cm in diameter, which lasts more than 3 months Increase 3 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Increase 2 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than 2 cm in diameter, which lasts more than 2 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 2 months The experts of WHO consider suspicious in relation to AIDS: Loss of weight (3 % and more) Loss of weight (5 % and more) Loss of weight (6 % and more) Loss of weight (9 % and more) Loss of weight (10 % and more) The experts of WHO consider suspicious in relation to AIDS: Diarrhea, which lasts more than 1 months Diarrhea, which lasts more than 2 months Diarrhea, which lasts more than 3 months Diarrhea, which lasts more than 4 months Diarrhea, which lasts more than 5 months The experts of WHO consider suspicious in relation to AIDS: Hypertermia, which lasts more than 1 months Hypertermia, which lasts more than 2 months Hypertermia, which lasts more than 3 months Hypertermia, which lasts more than 4 months Hypertermia, which lasts more than 5 months The experts of WHO consider suspicious in relation to AIDS: Hypertermia, which lasts more than 1 months Diarrhea, which lasts more than 1 months C. D. E. * 168. A. B. C. * D. E. 169. A. B. C. * D. E. 170. A. B. C. * D. E. 171. A. B. C. * D. E. 172. A. B. C. * D. E. 173. A. B. C. D. E. * Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Loss of weight (10 % and more) All the above The experts of WHO consider suspicious in relation to AIDS: Hypertermia, which lasts more than 21 months Diarrhea, which lasts more than 3 months Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than 1 cm in diameter, which lasts more than 3 months Loss of weight (6 % and more) All the above The planned inoculations for the children conduct in accordance with operating Calendar are all, except To tuberculosis To poliomyelitis To tuberculosis and salmoneliosis To tuberculosis and diphtheria To poliomyelitis and diphtheria The planned inoculations for the children conduct in accordance with operating Calendar are all, except To tuberculosis To poliomyelitis To tuberculosis and poliomyelitis To tuberculosis and diphtheria To poliomyelitis and diphtheria The planned inoculations of HIV infected| children| conduct in accordance with operating calendar are all, except To tuberculosis To poliomyelitis To tuberculosis and poliomyelitis To tuberculosis and diphtheria To poliomyelitis and diphtheria The planned inoculations of HIV infected| children| conduct in accordance with operating calendar are all, except To tuberculosis To poliomyelitis To tuberculosis and poliomyelitis To tuberculosis and diphtheria To poliomyelitis and diphtheria The sick grumbles about the prolonged cough, more than half-year, rising temperature of body to 38 °C, enlargement of peripheral lymphnode, frequent herpetic wide-spread pouring out with considerable lowering of body mass. In іmmunogram correlation of T-helper to T-suppression is 0,3. It takes place because infestant: Infects cells with the receptors of CD22 Induces proliferation of Т-helpers Induces proliferation of T-suppressors Infects cells from receptor CD8 Infects cells from receptor CD4 174. A. B. C. D. E. * 175. A. B. C. D. * E. 176. A. B. C. D. * E. 177. A. B. * C. D. E. 178. A. B. * C. D. E. 179. A. B. * C. D. E. 180. A. B. * C. D. E. 181. The sick grumbles about the prolonged cough, more than half-year, rising temperature of body to 38 °C, enlargement of peripheral lymphnode, frequent herpetic wide-spread pouring out with considerable lowering of body mass. In іmmunogram correlation of T-helper to T-suppression is 0,3. It takes place because infestant: Infects cells with the receptors of CD22 Induces proliferation of Т-helpers Induces proliferation of T-suppressors Infects cells from receptor CD8 Infects cells from receptor CD4 To what cells of blood the human immunodeficiency virus has most affected: Thrombocytes T-suppressor-cell Т-killer T-helper Leucocytes To what cells of blood the human immunodeficiency virus has most affected: Thrombocytes T-suppressor-cell Т-killer T-helper Leucocytes Violation of what stage of cell cycle does HIV predetermine lamivudin? Penetration of HIV into cells Reverse transcription Integration Transcription Translation Violation of what stage of cell cycle does HIV predetermine stavudin? Penetration of HIV in cells Reverse transcription Integration Transcription Translation Violation of what stage of cell cycle does HIV predetermine lamivudin? Penetration of HIV into cells Reverse transcription Integration Transcription Translation Violation of what stage of cell cycle does HIV predetermine stavudin? Penetration of HIV in cells Reverse transcription Integration Transcription Translation Violation of what stage of life cycle does HIV predetermine by Zidovudin? A. B. * C. D. E. 182. A. B. * C. D. E. 183. A. B. C. D. * E. 184. A. B. C. D. E. * 185. A. B. C. D. * E. 186. A. B. C. D. * E. 187. A. B. C. D. E. * 188. A. B. C. D. * Penetration of HIV in cells Reverse transcription Integration Transcription Translation Violation of what stage of life cycle does HIV predetermine by Zidovudin? Penetration of HIV in cells Reverse transcription Integration Transcription Translation What additional inspections must be conducted on patient with infectious mononucleosis? Reaction of Byurne and Rayta Khaddl'sona. IFA on HIV and test on rabbit-fever. Bacterioscopy on diphtheria and typhoid. IFA on HIV and bacterioscopy on diphtheria. Reaction of Paulya-Bunnelya and puncture of lymphatic node. What additional inspections must be conducted on patient with infectious mononucleosis? Reaction of Byurne and Rayta Khaddl'sona IFA on HIV and test on rabbit-fever Bacterioscopy on diphtheria and typhoid Reaction of Paulya-Bunnelya and puncture of lymphatic node Ig M and IgG to infectious mononucleosis What additional inspections must be conducted on patient with infectious mononucleosis? Reaction of Paulya-Bunnelya IFA on HIV and test on rabbit-fever Bacterioscopy on diphtheria and typhoid IFA on HCV Reaction of Byurne and puncture of lymphatic node What additional inspections must be conducted on patient with infectious mononucleosis? Reaction of Byurne and Rayta Khaddl'sona. IFA on HIV and test on rabbit-fever. Bacterioscopy on diphtheria and typhoid. IFA on HIV and bacterioscopy on diphtheria. Reaction of Paulya-Bunnelya and puncture of lymphatic node. What additional inspections must be conducted on patient with infectious mononucleosis? Reaction of Byurne and Rayta Khaddl'sona IFA on HIV and test on rabbit-fever Bacterioscopy on diphtheria and typhoid Reaction of Paulya-Bunnelya and puncture of lymphatic node Ig M and IgG to infectious mononucleosis What additional inspections must be conducted on patient with infectious mononucleosis? Reaction of Paulya-Bunnelya IFA on HIV and test on rabbit-fever Bacterioscopy on diphtheria and typhoid IFA on HCV E. 189. A. * B. C. D. E. 190. A. * B. C. D. E. 191. A. B. C. D. E. * 192. A. B. C. D. E. * 193. A. B. C. * D. E. 194. A. B. C. * D. E. 195. A. * B. C. D. E. 196. Reaction of Byurne and puncture of lymphatic node What antiretroviral preparation is taken as post contact prophylaxis after a contact with blood and other biological liquids? Azidotimidin Nevirapin Indinavir Saqvinavir Ifavirent What antiretroviral preparation is taken as post contact prophylaxis after a contact with blood and other biological liquids? Azidotimidin Nevirapin Indinavir Saqvinavir Ifavirent What cell of human body can HIV get into? Red corpuscles Neutrophilic leucocytes Monocyte T-lymphocte-killer T-cell helper What cell of human body can HIV get into? Red corpuscles Neutrophilic leucocytes Monocyte T-lymphocte-killer T-cell helper What cells are main target for HIV? T-suppressor-cell Т-kіller Т-helper D-cells 0-cells What cells are main target for HIV? T-suppressor-cell Т-kіller Т-helper D-cells 0-cells What cellular receptors of man can HIV stick to? CD4 CD8 CD95 CD40 CD3 What cellular receptors of man can HIV stick to? A. * B. C. D. E. 197. A. * B. C. D. E. 198. A. * B. C. D. E. 199. A. * B. C. D. E. 200. A. * B. C. D. E. 201. A. * B. C. D. E. 202. A. B. C. * D. E. 203. A. B. CD4 CD8 CD95 CD40 CD3 What clinical features of Kaposhi sarcoma in patients with AIDS? Will strike the persons of young and middle age Primary elements appear on a head and trunk Pouring out with necrosis and ulceration A sarcoma metastasis | in internal and marked high lethality All adopted features What clinical features of Kaposhi sarcoma in patients with AIDS? Will strike the persons of young and middle age Primary elements appear on a head and trunk Pouring out with necrosis and ulceration A sarcoma metastasis | in internal and marked high lethality All adopted features What disease is occupied by the second place after frequency of the first defects at AIDS? Sarcoma Kaposhi's Pneumocystis pneumonia Meningitis Encephalitis Lymphadenomas of cerebrum What disease is occupied by the second place after frequency of the first defects at AIDS? Sarcoma Kaposhi's Pneumocystis pneumonia Meningitis Encephalitis Lymphadenomas of cerebrum What disease is occupied by the second place after frequency of the first defects at AIDS? Sarcoma Kaposhi's Pneumocystis pneumonia Meningitis Encephalitis Lymphadenomas of cerebrum What dose of prophylaxis conducted by antiretroviral preparation after a contact with blood and other biological liquids? 600-800 mg/day 700-800 mg/day 800-1000 mg/day 1000-1100 mg/day 1100-1200 mg/day What dose of prophylaxis conducted by antiretroviral preparation after a contact with blood and other biological liquids? 600-800 mg/day 700-800 mg/day C. * D. E. 204. A. B. C. D. E. * 205. A. B. C. D. E. * 206. A. B. C. * D. E. 207. A. B. C. * D. E. 208. A. B. C. D. * E. 209. A. B. C. D. * E. 210. A. * B. C. D. 800-1000 mg/day 1000-1100 mg/day 1100-1200 mg/day What factors does influence on the level of CD4? Analytical variant Seasonal diseases pathema Some accidental diseases Treatment with corticosteroid All the above What factors does influence on the level of CD4? Analytical variant Seasonal diseases pathema Some accidental diseases Treatment with corticosteroid All the above ?what family of viruses does an exciter of HIV/AIDS belong to? Orto- and paramyxovirus Rabdovirus Retrovirus Herpesvirus Reovirus what family of viruses does an exciter of HIV/AIDS belong to? Orto- and paramyxovirus Rabdovirus Retrovirus Herpesvirus Reovirus What group of infectious diseases an exciter of HIV infection/AIDS belong to by L. Gromashevskij classification? Intestinal infection Infections of respiratory tract Blood infection Infection of external covers Behave to all indicated groups What group of infectious diseases an exciter of HIV infection/AIDS belong to by L. Gromashevskij classification? Intestinal infection Infections of respiratory tract Blood infection Infection of external covers Behave to all indicated groups What group of infectious diseases, does an exciter of HIV/AIDS belong to? Antroponozis Zoonosis Sapronosis Saprozoonozis E. 211. A. * B. C. D. E. 212. A. * B. C. D. E. 213. A. * B. C. D. E. 214. A. B. C. * D. E. 215. A. B. C. * D. E. 216. A. B. C. D. * E. Zooantroponozis What group of infectious diseases, does an exciter of HIV/AIDS belong to? Antroponozis Zoonosis Sapronosis Saprozoonozis Zooantroponozis What immunological changes will be found out for a patient with HIV infection? There is polyclonality B-сells activating, the amount of autoantibodies and immune complexes is increased There is polyclonality B-сells activating, the amount of autoantibodies and immune complexes diminishes There is polyclonality B-сells of oppression, the amount of autoantibodies and immune complexes diminishes There is polyclonality B-сells of oppression, the amount of autoantibodies and immune complexes is increased There is polyclonality B-сells activating, the amount| of autoantibodies and immune complexes is increased What immunological changes will be found out for a patient with HIV infection? There is polyclonality V-klitinna activating, the amount of autoantibodies and immune complexes is increased There is polyclonality V-klitinna activating, the amount of autoantibodies and immune complexes diminishes There is polyclonality V-klitinne of oppression, the amount of autoantibodies and immune complexes diminishes There is polyclonality V-klitinne of oppression, the amount of autoantibodies and immune complexes is increased There is polyclonality V-klitinna activating, the amount| of autoantibodies and immune complexes is increased What is the genome named that HIV plugged in the genome of cell of owner? Supervirus Provirus Particle of DNA Retrovirus Coxavirus What is the genome named that HIV plugged in the genome of cell of owner? Supervirus Provirus Particle of DNA Retrovirus Coxavirus What is the level of functional ability of patient at Ist clinical stage of AIDS? Decreased level of everyday activity Enhanceable level of everyday activity Variable level of everyday activity Normal level of everyday activity All the above| 217. A. B. C. D. * E. 218. A. B. C. D. * E. 219. A. B. C. D. * E. 220. A. B. C. D. * E. 221. A. B. * C. D. E. 222. A. B. * C. D. E. 223. A. B. * C. D. E. 224. A. * What is the level of functional ability of patient at Ist clinical stage of AIDS? Decreased level of everyday activity Enhanceable level of everyday activity Variable level of everyday activity Normal level of everyday activity All the above| What is the level of functional ability of patient at Ist clinical stage of AIDS? Decreased level of everyday activity Enhanceable level of everyday activity Variable level of everyday activity Normal level of everyday activity All the above| What is the time of prophylaxis conducted for antiretroviral preparation after a contact with blood and other biological liquids? During 1 wk During 2 wk During 3 wk During 4 wk During 5 wk What is the time of prophylaxis conducted for antiretroviral preparation after a contact with blood and other biological liquids? During 1 wk During 2 wk During 3 wk During 4 wk During 5 wk What is the underlaid immunodeficiency at HIV infection? Making progress diminishing amount of CD8 cell Making progress diminishing amount of CD4 cell Scaling down the amount of CD4 cell Scaling down the amount of CD8 cell Diminishing amount of CD4 to CD8 cell What is the underlaid immunodeficiency at HIV infection? Making progress diminishing amount of CD8 cell Making progress diminishing amount of CD4 cell Scaling down the amount of CD4 cell Scaling down the amount of CD8 cell Diminishing amount of CD4 to CD8 cell What is the underlaid immunodeficiency at HIV infection? Making progress diminishing amount of CD8 cell Making progress diminishing amount of CD4 cell Scaling down the amount of CD4 cell Scaling down the amount of CD8 cell Diminishing amount of CD4 to CD8 cell What laboratory signs are characteristic of AIDS-associative complex? Lowering of content of immunoproteins A and G B. C. D. E. 225. A. * B. C. D. E. 226. A. * B. C. D. E. 227. A. * B. C. D. E. 228. A. B. * C. D. E. 229. A. B. * C. D. E. 230. A. * B. C. D. E. 231. A. * B. C. Diminishing of correlation of CD4/CD8 below 1.0 Anaemia Leycopenia Increase level of circulatory immune complexes What laboratory signs are characteristic of AIDS-associative complex? Lowering of content of immunoproteins A and G Diminishing of correlation of CD4/CD8 below 1.0 Anaemia Leycopenia Increase level of circulatory immune complexes What medical professions carry the most potential threat of infection? Surgical and laboratory specialities, who contact with blood Therapeutic specialities Epidemiologists Sociologist Teachers of medical establishments What medical professions carry the most potential threat of infection? Surgical and laboratory specialities, who contact with blood Therapeutic specialities Epidemiologists Sociologist Teachers of medical establishments What sexual contact are the most dangerous for infection with HIV? Vaginal Anal Oral Lesbian Artificial impregnation What sexual contact are the most dangerous for infection with HIV? Vaginal Anal Oral Lesbian Artificial impregnation What аntiretroviral preparation is conduct as prophylaxis after contact with blood and other biological liquids? Zidovudin Viramin Fortovaze Saqvinavir Lopinavir What аntiretroviral preparation is conduct as prophylaxis after contact with blood and other biological liquids? Zidovudin Viramin Fortovaze D. E. 232. A. B. C. * D. E. 233. A. B. C. * D. E. 234. A. * B. C. D. E. 235. A. * B. C. D. E. 236. A. B. C. D. * E. 237. A. B. C. D. * E. 238. A. B. C. D. * E. Saqvinavir Lopinavir When do the plan caesarian section of HIV infected pregnant conducted with the purpose for decreasing of the risk of infecting the fetus? In 36 weeks In 37 weeks In 38 weeks In 39 weeks In 40 weeks When do the plan caesarian section of HIV infected pregnant conducted with the purpose for decreasing of the risk of infecting the fetus? In 36 weeks In 37 weeks In 38 weeks In 39 weeks In 40 weeks When does begin antiviral therapy for infant of HIV-infected women? In the first 8-12 hr after birth From 24-36 hr after birth Does not conduct From a month After diagnosis of AIDS When does begin antiviral therapy for infant of HIV-infected women? In the first 8-12 hr after birth From 24-36 hr after birth Does not conduct From a month After diagnosis of AIDS When is possible to diagnose AIDS? Only the content of CD4 less than 500 in 1 microlitre of blood Only the conten of CD4 less than 400 in 1 microlitre of blood Only the content of CD4 less than 300 in 1 microlitre of blood Only the content of CD4 less than 200 in 1 microolitre of blood Only the content of CD4 less than 100 in 1 microolitre of blood When is possible to diagnose AIDS? Only the content of CD4 less than 500 in 1 microlitre of blood Only the conten of CD4 less than 400 in 1 microlitre of blood Only the content of CD4 less than 300 in 1 microlitre of blood Only the content of CD4 less than 200 in 1 microolitre of blood Only the content of CD4 less than 100 in 1 microolitre of blood When is possible to diagnose AIDS? Only the content of CD4 less than 500 in 1 microlitre of blood Only the conten of CD4 less than 400 in 1 microlitre of blood Only the content of CD4 less than 300 in 1 microlitre of blood Only the content of CD4 less than 200 in 1 microolitre of blood Only the content of CD4 less than 100 in 1 microolitre of blood 239. A. B. C. * D. E. 240. A. B. C. * D. E. 241. A. B. * C. D. E. 242. A. B. * C. D. E. 243. A. B. C. D. E. * 244. A. B. C. D. E. * 245. A. B. C. D. E. * With the help of what molecules which is included in composition protien, a virus firmly contacts with the molecule of CD4: gp 100 gp 110 gp 120 gp 130 gp 140 With the help of what molecules which is included in composition protien, a virus firmly contacts with the molecule of CD4: gp 100 gp 110 gp 120 gp 130 gp 140 A patient concerned about attacks of fever, which are repeated every third day. There are jaundice of sclera and skin, hepatosplenomegaly. Which of the following diagnosis is most likely? Sepsis Malaria viral hepatitis Hemolytic anemia Leptospirosis a patient concerned about attacks of fever, which are repeated every third day. There are jaundice of sclera and skin, hepatosplenomegaly. Which of the following diagnosis is most likely? Sepsis Malaria viral hepatitis Hemolytic anemia Leptospirosis Activities for contacts with import case of malaria: Parasitoscopy of blood The direction of the contact in the detention facility for 5 days Chemoprophylaxis Vaccination Do not hold Activities for contacts with import case of malaria: Parasitoscopy of blood The direction of the contact in the detention facility for 5 days Chemoprophylaxis Vaccination Do not hold Contra-indication for the application of antibiotics are all, except. An increased sensitiveness to preparation Severy disorders of liver Severy disorders of kidneys Period of pregnancy and lactation Prolonged fever 246. A. B. C. D. E. * 247. A. B. C. D. E. * 248. A. B. C. * D. E. 249. A. B. C. * D. E. 250. A. B. C. D. E. * 251. A. B. C. D. E. * 252. A. B. C. D. * E. 253. A. Contra-indication for the application of antibiotics are all, except. An increased sensitiveness to preparation Severy disorders of liver Severy disorders of kidneys Period of pregnancy and lactation Prolonged fever Contra-indication for the application of antibiotics are all, except. An increased sensitiveness to preparation Severy disorders of liver Severy disorders of kidneys Period of pregnancy and lactation Prolonged fever Delagil appoint at the malaria in such doses: 0,5 g 3 per a day 3 days 0,5 g per a week In the first day 1 g, through 6 h 0,5 g 0,5 g per a day during a month 0,5 g 2 per a day 3 days Delagil appoint at the malaria in such doses: 0,5 g 3 per a day 3 days 0,5 g per a week In the first day 1 g, through 6 h 0,5 g 0,5 g per a day during a month 0,5 g 2 per a day 3 days During the treatment in a hospital of malaria the latest attack was happened. What is etiotropic treatment. Antibiotics Serum Delagil Primachin Delagil + Primachin During the treatment in a hospital of malaria the latest attack was happened. What is etiotropic treatment. Antibiotics Serum Delagil Primachin Delagil + Primachin During what time the lice can transfer the epidemic typhus? Up to 10 days Up to 15 days Up to 20 days Up to 30 days Up to 40 days During what time the lice can transfer the epidemic typhus? Up to 10 days B. C. D. * E. 254. A. B. C. D. * E. 255. A. B. C. D. * E. 256. A. B. C. D. * E. 257. A. B. C. D. * E. 258. A. B. C. D. * E. 259. A. B. * C. D. E. 260. A. B. * C. D. E. Up to 15 days Up to 20 days Up to 30 days Up to 40 days During what time the lice can transfer the epidemic typhus? Up to 10 days Up to 15 days Up to 20 days Up to 30 days Up to 40 days How prevent malaria infection? Follow the rules of personal hygiene Boiling of a water Vaccination Chemoprophylaxis Heating of a food How prevent malaria infection? Follow the rules of personal hygiene Boiling of a water Vaccination Chemoprophylaxis Heating of a food Immune modulator therapy of sepsis are all, except. Ronkoleykin Interferon Inductors of interferon Vaccine Normal human immune globuline Immune modulator therapy of sepsis are all, except. Ronkoleykin Interferon Inductors of interferon Vaccine Normal human immune globuline In a survey of the donor blood microhametosis was found. Assign treatment. Delagil Primachin Antibiotics Fluorochinolones Sulfanilamides In a survey of the donor blood microhametosis was found. Assign treatment. Delagil Primachin Antibiotics Fluorochinolones Sulfanilamides 261. A. B. C. D. E. * 262. A. B. C. D. E. * 263. A. * B. C. D. E. 264. A. * B. C. D. E. 265. A. * B. C. D. E. 266. A. * B. C. D. E. 267. A. B. * C. D. E. In the case of head pediculosis carry out sanitation: hair cut, followed by incineration, processing hair. What kind of preparation is used? 0,5 % solution of water emulsions karbofosa 0,5 % metilatsetofos 10 % solution of liquid neutral metilatsetofosa soap 0,5 % water emulsion dikrezilu 3 % soap RHTSG In the case of head pediculosis carry out sanitation: hair cut, followed by incineration, processing hair. What kind of preparation is used? 0,5 % solution of water emulsions karbofosa 0,5 % metilatsetofos 10 % solution of liquid neutral metilatsetofosa soap 0,5 % water emulsion dikrezilu 3 % soap RHTSG In what titre will be positive reaction of agglutination in patient with epidemic typhus in droplets Mosing test? 1:40 and above 1:80 and above 1:160 or higher 1:320 or higher 1:640 or higher In what titre will be positive reaction of agglutination in patient with epidemic typhus in droplets Mosing test? 1:40 and above 1:80 and above 1:160 or higher 1:320 or higher 1:640 or higher Indications for the appointment of hematoshizotropic antimalarial drugs: Attack of malaria Preventing of late relapse Prophylactic course after returning from malaria areas Ant recidive course for the reconvalescents During a check-up Indications for the appointment of hematoshizotropic antimalarial drugs: Attack of malaria Preventing of late relapse Prophylactic course after returning from malaria areas Ant recidive course for the reconvalescents During a check-up Indications for the appointment of histoshizotropic antimalarial drugs: Attack of malaria Prevention of late relapse Complications of malaria Chemoprophylaxis during staying in endemic areas During a check-up 268. A. B. * C. D. E. 269. A. B. C. D. E. * 270. A. B. C. D. E. * 271. A. B. * C. D. E. 272. A. B. * C. D. E. 273. A. * B. C. D. E. 274. A. * B. C. D. E. 275. A. B. C. Indications for the appointment of histoshizotropic antimalarial drugs: Attack of malaria Prevention of late relapse Complications of malaria Chemoprophylaxis during staying in endemic areas During a check-up Malaria must be differentiated primarily with such diseases: Cholangitis Pyelonephritis Sepsis viral hepatitis All of the above Malaria must be differentiated primarily with such diseases: Cholangitis Pyelonephritis Sepsis viral hepatitis All of the above On which period the maximal symptoms of epidemic typhus disease appear? At the incubation period At the 1th week of illness At the 2nd week of illness At the 3rd week of illness At the time of recovery On which period the maximal symptoms of epidemic typhus disease appear? At the incubation period At the 1th week of illness At the 2nd week of illness At the 3rd week of illness At the time of recovery Prevention of early relapses of malaria by: Delagil Immunoglobulin Antibiotics Primachin Glucocorticoids Prevention of early relapses of malaria by: Delagil Immunoglobulin Antibiotics Primachin Glucocorticoids Prevention of late relapse of malaria by: Delagil Immunoglobulin Antibiotics D. * E. 276. A. B. C. D. * E. 277. A. B. C. D. E. * 278. A. B. C. D. E. * 279. A. B. C. D. * E. 280. A. B. C. D. * E. 281. A. B. C. * D. E. 282. A. B. C. * D. E. 283. A. Primachin Glucocorticoids Prevention of late relapse of malaria by: Delagil Immunoglobulin Antibiotics Primachin Glucocorticoids Principles of etiotropic therapy of sepsis. Administration of antibiotics quick as possible Administration of antibiotics in maximal therapeutic doses In accordance to credible microbiological diagnosis An account of possible of therapeutic concentration of antibiotic in field of infection All the above Principles of etiotropic therapy of sepsis. Administration of antibiotics quick as possible Administration of antibiotics in maximal therapeutic doses In accordance to credible microbiological diagnosis An account of possible of therapeutic concentration of antibiotic in field of infection All the above Radical treatment of malaria include: 5 days therapy with delagil Primachin within 2 weeks Glucocorticoids Delagil + primachin Serum therapy Radical treatment of malaria include: 5 days therapy with delagil Primachin within 2 weeks Glucocorticoids Delagil + primachin Serum therapy Reconvalenscents after malaria may discharged from a clinic no earlier than: After 2 weeks to complete clinical recovery After 2 weeks of the conclusion of a radical course of therapy At the conclusion of a radical course of therapy with a negative result of parazitoscopy After 3 weeks with a negative blood culture results After 3 weeks, if the negative results of planting feces Reconvalenscents after malaria may discharged from a clinic no earlier than: After 2 weeks to complete clinical recovery After 2 weeks of the conclusion of a radical course of therapy At the conclusion of a radical course of therapy with a negative result of parazitoscopy After 3 weeks with a negative blood culture results After 3 weeks, if the negative results of planting feces Remedie for malaria prevention for those who have returned from disadvantaged areas: Human immunoglobulin B. C. D. * E. 284. A. B. C. D. * E. 285. A. * B. C. D. E. 286. A. * B. C. D. E. 287. A. B. C. D. * E. 288. A. B. C. D. * E. 289. A. B. C. D. E. * 290. A. B. C. D. E. * Interferon 6-day-prevention streptomyosin or tetracycline Primaquine 0,027 g 14 days All the above. Remedie for malaria prevention for those who have returned from disadvantaged areas: Human immunoglobulin Interferon 6-day-prevention streptomyosin or tetracycline Primaquine 0,027 g 14 days All the above. Rules of hospitalization of patients with malaria: In separate room In the respiratory infections department In the Meltserovsky‘s box Patients are not hospitalized In the intestinal infections department Rules of hospitalization of patients with malaria: In separate room In the respiratory infections department In the Meltserovsky‘s box Patients are not hospitalized In the intestinal infections department Specific complications of malaria, except: Hemoglobinuria fever The gap of the spleen Malaria‘s comma Perforation bowel Hemolytic anaemia Specific complications of malaria, except: Hemoglobinuria fever The gap of the spleen Malaria‘s comma Perforation bowel Hemolytic anaemia The diagnosis of malaria can be confirmed by : Microscopy of urine Hemoculture Bacteriology of stool Common blood analysis Parazitoscopy of blood The diagnosis of malaria can be confirmed by : Microscopy of urine Hemoculture Bacteriology of stool Common blood analysis Parazitoscopy of blood 291. A. B. C. D. * E. 292. A. B. C. D. * E. 293. A. B. C. * D. E. 294. A. B. C. * D. E. 295. A. B. C. * D. E. 296. A. B. C. * D. E. 297. A. B. C. D. E. * 298. A. B. C. The radical course of treatment of malaria includes: Five-day therapy of delagilum Prymahin during 2 weeks Delagilum + prymahin + fansydar Delagilum + prymahin Delagilum + fansydar The radical course of treatment of malaria includes: Five-day therapy of delagilum Prymahin during 2 weeks Delagilum + prymahin + fansydar Delagilum + prymahin Delagilum + fansydar There are etiotropic drags of malaria, except: Delagilum Prymahin Cerasyn Quinine Fansydar There are etiotropic drags of malaria, except: Delagilum Prymahin Cerasyn Quinine Fansydar What antibiotic is less effective in epidemic typhus? Tetracyclin Metacyclin Levomycetin Vibramycin Doxycyclin What antibiotic is less effective in epidemic typhus? Tetracyclin Metacyclin Levomycetin Vibramycin Doxycyclin What do you need for reatment of chlorochyn resistent forms of malaria? Bactrimum Dapson Fansydar Meflohin It is all above enumerated What do you need for reatment of chlorochyn resistent forms of malaria? Bactrimum Dapson Fansydar D. E. * 299. A. B. C. * D. E. 300. A. B. C. * D. E. 301. A. B. C. D. E. 302. A. * B. C. D. E. 303. A. B. C. D. E. 304. A. B. C. D. E. 305. A. B. * C. D. E. 306. A. Meflohin It is all above enumerated What do you need to give to the patients with malaria‘s coma? Glucocorticoids Antishok drags Intravenously delagil Blood transfusion Oxygen What do you need to give to the patients with malaria‘s coma? Glucocorticoids Antishok drags Intravenously delagil Blood transfusion Oxygen What group of infectious diseases epidemic typhus belong to? Intestinal Respiratory External covers Transmissive Blood What group of infectious diseases balantidosis belong to? Intestinal Respiratory External covers Transmissive Blood What group of infectious diseases malaria belong to? Intestinal Respiratory External covers Transmissive Blood What group of infectious diseases malaria belong to? Intestinal Respiratory External covers Transmissive Blood What is Brill's disease? Vertiacal borne Remote relapse typhus Early relapse typhus Re-infection rickettsia Self nozological unit What is Brill's disease? Vertiacal borne B. * C. D. E. 307. A. B. * C. D. E. 308. A. B. C. * D. E. 309. A. B. C. * D. E. 310. A. B. C. D. E. * 311. A. * B. C. D. E. 312. A. * B. C. D. E. 313. A. B. C. D. E. * Remote relapse typhus Early relapse typhus Re-infection rickettsia Self nozological unit What is Brill's disease? Vertiacal borne Remote relapse typhus Early relapse typhus Re-infection rickettsia Self nozological unit What is diagnostic titer response agglutination test with typhus rickettsia? 1:40 and above 1:80 and above 1:160 or higher 1:320 or higher 1:640 or higher What is diagnostic titer response agglutination test with typhus rickettsia? 1:40 and above 1:80 and above 1:160 or higher 1:320 or higher 1:640 or higher What is not typical for epidemic typhus exanthema? Disappearing with decreasing of temperature Never appear a new elements Roseola saved up to 6 days, petehii – 12 Leave a little pigmentation and poor peeling Few elements What is not typical for epidemic typhus exanthema? Arise on 7-10 days of illness Roseola-petehial kind Localized mainly on the lateral surface of the torso and limbs flexion surfaces It can grab hands and feet, but never on the face Abundant What is not typical for epidemic typhus exanthema? Arise on 7-10 days of illness Roseola-petehial kind Localized mainly on the lateral surface of the torso and limbs flexion surfaces It can grab hands and feet, but never on the face Abundant What is not typical for epidemic typhus exanthema? Disappearing with decreasing of temperature Never appear a new elements Roseola saved up to 6 days, petehii – 12 Leave a little pigmentation and poor peeling Few elements 314. A. B. C. D. * E. 315. A. B. C. D. * E. 316. A. B. C. * D. E. 317. A. B. C. * D. E. 318. A. B. C. * D. E. 319. A. B. C. D. * E. 320. A. B. C. D. * E. 321. A. B. C. What is not typical for severe epidemic typhus in the common blood analysis blood ? Neutrophyl leukocytosis Hypoeozinophylia Lymphopenia Anemia Increasing of ESR What is not typical for severe epidemic typhus in the common blood analysis blood ? Neutrophyl leukocytosis Hypoeozinophylia Lymphopenia Anemia Increasing of ESR What is not typical for the analysis of urine in the severe epidemic typhus? Proteinuria Single-cylinder hyaline Multiple granular cylinders A small number of erythrocytes A small number of leukocytes What is not typical for the analysis of urine in the severe epidemic typhus? Proteinuria Single hyaline cylinder Multiple granular cylinders A small number of erythrocytes A small number of leukocytes What is not typical for the analysis of urine in the severe epidemic typhus? Proteinuria Single hyaline cylinder Multiple granular cylinders A small number of erythrocytes A small number of leukocytes ?What is possible side effects at application of antibiotics are all, except. Stomach-ache Nausea, vomit Diarrhea Fever Skin rash What is possible side effects at application of antibiotics are all, except. Stomach-ache Nausea, vomit Diarrhea Fever Skin rash What is possible side effects at application of antibiotics are all, except. Stomach-ache Nausea, vomit Diarrhea D. * E. 322. A. * B. C. D. E. 323. A. * B. C. D. E. 324. A. B. C. D. E. * 325. A. B. C. D. E. * 326. A. B. C. D. E. * 327. A. B. C. D. * E. 328. A. B. C. D. * E. Fever Skin rash What is prophylaxis of epidemic typhus among the members of family with pediculosis in the focus of the disease? Monitoring and complete sanitation of the contact persons Chemoprophylaxis Antibiotic therapy Isolation of contact Check-up What is prophylaxis of epidemic typhus among the members of family with pediculosis in the focus of the disease? Monitoring and complete sanitation of the contact persons Chemoprophylaxis Antibiotic therapy Isolation of contact Check-up what is the Basic principles of antibiotics therapy. A selection of preparation from data of bacteriostatic Determination of dose, method and multiple of introduction the preparation Timeliness and definite duration of introduction input of antibiotic Combining antibiotics between itself for enhancement of antibacterial effect All the above what is the basic principles of antibiotics therapy? Selection of antibiotics after the studing of sensitiveness Selection of antibiotic according to the clinical diagnosis Choose the most active drug Choose the less toxic drug All the above what is the Basic principles of antibiotics therapy. A selection of preparation from data of bacteriostatic Determination of dose, method and multiple of introduction the preparation Timeliness and definite duration of introduction input of antibiotic Combining antibiotics between itself for enhancement of antibacterial effect All the above What is the diagnosis in reaction of agglutination with rickettsiae Prowazekii 1:640. Flu Typhoid fever Meningococcal infection Epidemic typhus Leptospirosis What is the diagnosis in reaction of agglutination with rickettsiae Prowazekii 1:640. Flu Typhoid fever Meningococcal infection Epidemic typhus Leptospirosis 329. A. B. C. * D. E. 330. A. B. * C. D. E. 331. A. B. C. * D. E. 332. A. B. * C. D. E. 333. A. B. C. * D. E. 334. A. B. C. * D. E. 335. A. B. C. D. E. * 336. A. B. C. What is the duration of observation of the contact persons in the focus of epidemic typhus: 21 days 25 days 51 days 72 days 3 months What is the duration of observation of the contact persons with the Brill‘s patient: 21 days 25 days 51 days 72 days 3 months What is the duration of observation of the contact persons in the focus of epidemic typhus: 21 days 25 days 51 days 72 days 3 months What is the duration of observation of the contact persons with the Brill‘s patient: 21 days 25 days 51 days 72 days 3 months What is the source of epidemic typhus? Patients with epidemic typhus Patients with disease Brill-Zinsser Patients with epidemic typhus and disease Brill-Zinsser Patients with Brill-Zinsser disease and Sachs disease Patients with epidemic typhus and abdominal typhoid What is the source of epidemic typhus? Patients with epidemic typhus Patients with disease Brill-Zinsser Patients with epidemic typhus and disease Brill-Zinsser Patients with Brill-Zinsser disease and Sachs disease Patients with epidemic typhus and abdominal typhoid What measures must be taken for people who stood in contact with malaria case: Microscopy of peripheral blood Direction of all patients for in an isolation for 5 days Chemical prophylaxis Street rounds Does not conduct What measures must be taken for people who stood in contact with malaria case: Microscopy of peripheral blood Direction of all patients for in an isolation for 5 days Chemical prophylaxis D. E. * 337. A. B. C. * D. E. 338. A. B. C. * D. E. 339. A. * B. C. D. E. 340. A. * B. C. D. E. 341. A. B. C. * D. E. 342. A. * B. C. D. E. 343. A. B. C. D. E. * 344. A. Street rounds Does not conduct What method used for identification of malaria: Stool culture test Hemoculture Microscopic assessment of blood Byurne test All above enumerated What method used for identification of malaria: Stool culture test Hemoculture Microscopic assessment of blood Byurne test All above enumerated When does begin treatment of patients with a malaria? Immediately after hospitalization After taking of material for research After raising of final diagnosis After the laboratory and instrumental result All answers are faithful When does begin treatment of patients with a malaria? Immediately after hospitalization After taking of material for research After raising of final diagnosis After the laboratory and instrumental result All answers are faithful When patient after epidemic typhus may been discharged from a clinic? After clinical recovering, but not earlier 12-days normal temperature After a full clinical recovering After clinical recovering, but not earlier 12-days after antibiotics therapy After 12-days normal body temperature After clinical recovering, but not earlier than the 9-day normal body temperature When patient after epidemic typhus may been discharged from a clinic? After clinical recovering, but not earlier 12-days normal temperature After a full clinical recovering After clinical recovering, but not earlier 12-days after antibiotics therapy After 12-days normal body temperature After clinical recovering, but not earlier than the 9-day normal body temperature When serological diagnosis is possible in patients with epidemic typhus? From the 1st day of illness Fromn the 2nd day illness From the 3-4th day illness From 4-5th day of illness From the 5 to 7th day of illness When serological diagnosis is possible in patients with epidemic typhus? From the 1st day of illness B. C. D. E. * 345. A. B. C. D. * E. F. G. H. I. J. 346. A. B. C. D. * E. F. G. H. I. J. 347. A. B. C. * D. E. 348. A. B. C. * D. E. 349. A. B. C. * D. E. Fromn the 2nd day illness From the 3-4th day illness From 4-5th day of illness From the 5 to 7th day of illness When the sick people gets epidemic typhus infection, which period is most dangerous epidemically? Fecal-oral Air-drop Contact Transmissive Blood Over the past 2 days, the incubation period and 2-3 days after lowering temperature All hectic period and 2-3 days after lowering temperature 2-3 days after lowering temperature Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature Over the past 2 days, the incubation period and the hectic period When the sick people gets epidemic typhus infection, which period is most dangerous epidemically? Fecal-oral Air-drop Contact Transmissive Blood Over the past 2 days, the incubation period and 2-3 days after lowering temperature All hectic period and 2-3 days after lowering temperature 2-3 days after lowering temperature Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature Over the past 2 days, the incubation period and the hectic period When we can stop the etiotropic treatment of the patient with epidemic typhus? Immediately after the normalization of body temperature After the normalization of the liver and spleen sizes After 2-days normal body temperature After the disappearance of roseola After 10 days disappearing of roseola When we can stop the etiotropic treatment of the patient with epidemic typhus? Immediately after the normalization of body temperature After the normalization of the liver and spleen sizes After 2-days normal body temperature After the disappearance of roseola After 10 days disappearing of roseola which of the following often occur in patient with epidemic typhus (tongue‘s tremor when protrusion that sticked on the lower teeth)? Heller's symptom Conjunctivitis Govorov-Godelje symptom Zorohovich-Chiari-Avtsyn symptom Rosenberg‘s enantema 350. A. B. C. * D. E. 351. A. B. C. D. E. * 352. A. B. C. D. * E. 353. A. B. C. D. * E. 354. A. B. C. D. E. * 355. A. B. C. D. E. * 356. A. B. C. D. E. * which of the following often occur in patient with epidemic typhus (tongue‘s tremor when protrusion that sticked on the lower teeth)? Heller's symptom Conjunctivitis Govorov-Godelje symptom Zorohovich-Chiari-Avtsyn symptom Rosenberg‘s enantema which of the following symptoms may occur in patient with epidemic typhus (petechiaes in the mucosal soft palate)? Heller's symptom Conjunctivitis Govorov-Godelje symptom Zorohovich-Chiari-Avtsyn symptom Rosenberg‘s enantema which of the following symptoms may occur in patient with epidemic typhus (petechiaes with the conjunctiva)? Heller's symptom Conjunctivitis Govorov-Godelje symptom Zorohovich-Ciari-Avtsyn symptom Rosenberg‘s enantema which of the following symptoms may occur in patient with epidemic typhus (petechiaes in the conjunctiva)? Heller's symptom Conjunctivitis Govorov-Godelje symptom Zorohovich-Ciari-Avtsyn symptom Rosenberg‘s enantema which of the following symptoms may occur in patient with epidemic typhus (petechiaes in the mucosal soft palate)? Heller's symptom Conjunctivitis Govorov-Godelje symptom Zorohovich-Chiari-Avtsyn symptom Rosenberg‘s enantema Who would be a subject for inspection on malaria? People who recovered of malaria Persons, returning from endemic regions of malaria Patients with fever more than 5 days Patients with spleenomegaly All above enumerated Who would be a subject for inspection on malaria? People who recovered of malaria Persons, returning from endemic regions of malaria Patients with fever more than 5 days Patients with spleenomegaly All above enumerated 357. A. B. * C. D. E. 358. A. B. * C. D. E. 359. A. B. C. * D. E. 360. A. B. C. * D. E. 361. A. B. C. * D. E. 362. A. B. C. * D. E. 363. A. B. C. * D. E. Why early relapse in malaria may develops ? Immune deficiency Presence of erythrocytic shizonts Presence of tissue shizonts Fresh contamination Presence of blood gamonts Why early relapse in malaria may develops ? Immune deficiency Presence of erythrocytic shizonts Presence of tissue shizonts Fresh contamination Presence of blood gamonts Why late relapses in malaria may develop ? Immune deficiency Presence of erythrocytic shizonts Presence of tissue shizonts Fresh contamination Presence of blood gamonts Why late relapses in malaria may develop ? Immune deficiency Presence of erythrocytic shizonts Presence of tissue shizonts Fresh contamination Presence of blood gamonts A patient is disturbed by attacks of fever. The icterus of sclera and skins is observed, hepatosplenomegaly is found on palpation. What is possible diagnosis accept? Leishmaniosis Leptospirosis Sepsis Malaria Viral hepatitis A patient is disturbed by attacks of fever. The icterus of sclera and skins is observed, hepatosplenomegaly is found on palpation. What is possible diagnosis accept? Leishmaniosis Leptospirosis Sepsis Malaria Viral hepatitis A patient sharply had a chill, head pain, vomits, temperature of body rose up to 38,5 °C. Till evening rigidity of muscles, Kernig’s symptom appeareD. Herpetic blisters are marked on mucous of lips and nosE. Neurological symptoms is not found out expresseD. What disease will you suspect? Herpetic encephalitis Abscess of brain Meningoencephalitis Hemorrhage in a brain Subarachnoid hemorrhage 364. A. B. C. * D. E. 365. A. B. C. * D. E. 366. A. B. C. * D. E. 367. A. * B. C. D. E. 368. A. B. C. D. E. * 369. A. B. C. D. E. * 370. A. B. C. D. E. * 371. A. A patient sharply had a chill, head pain, vomits, temperature of body rose up to 38,5 °C. Till evening rigidity of muscles, Kernig’s symptom appeared. Herpetic blisters are marked on mucous of lips and nose. Neurological symptoms is not found out expressed. What disease will you suspect? Herpetic encephalitis Abscess of brain Meningoencephalitis Hemorrhage in a brain Subarachnoid hemorrhage Basic mechanism of transmission of Lime disease: Air-drop Contact Transmissive Fecal-oral Vertical Basic mechanism of transmission of Lyme disease: Air-drop Contact Transmissive Fecal-oral Vertical Basic vector of Lime disease: Tick Mosquito Flea Mosquito All of bloodsucking insects Clinical stages of Lime-borreliosis: Common toxic Cardiological complications Neurological complications Artritic All above enumerated Clinical stages of Lime-borreliosis: Common toxic Cardiological complications Neurological complications Artritic All above enumerated Duration of activity of pliers depends on: Weather Regional naturally-geographical terms Kind of vector Activity of animals in different periods of year From all of transferred Duration of activity of pliers depends on: Weather B. C. D. E. * 372. A. B. C. D. E. * 373. A. B. C. D. E. * 374. A. B. * C. D. E. 375. A. B. C. * D. E. 376. A. B. C. * D. E. 377. A. B. C. * D. E. 378. A. B. C. * Regional naturally-geographical terms Kind of vector Activity of animals in different periods of year From all of transferred Etiological agent of meningitis are accept: Staphylococci Neisseria meningitides Mycobacterium tuberculosis Viruses Entamoeba histolytica ?Etiological agent of meningitis are accept: Staphylococci Neisseria meningitides Mycobacterium tuberculosis Viruses Entamoeba histolytica Examination is delivered in a cliniC. Objectively: temperature 40,2 °C. Expressed meningeal symptoms. Light, haptic, pain hypesthesiA. Blood: leucocytes – 5 thousand/l, ERS-19 mm/hr. A neurolymph under high pressure, turbid, lymphocytosis, some increasing of protein, chlorides. What Meningococcal meningitis is your diagnosis? Viral meningitis Tubercular meningitis Staphylococcus meningitis Pneumoccocal meningitis For endermic reaction of leishmaniosis diagnosis is used: Dyzenterin Antraksin Leishmanin Ornitin Brucellin For endermic reaction of leishmaniosis diagnosis is used: Dysenterin Antraxin Leishmanin Ornitin Brucellin For the treatment of acidosis in viral meningoencephalitis is better to usE. 10 % chloride solution 10-20 % glucose solution 4 % sodium bicarbonate solution Concentrated dry plasma Albumen For the treatment of acidosis in viral meningoencephalitis is better to usE. 10 % chloride solution 10-20 % glucose solution 4 % sodium bicarbonate solution D. E. 379. A. B. C. * D. E. 380. A. B. C. * D. E. 381. A. B. C. * D. E. 382. A. B. C. * D. E. 383. A. B. C. D. E. * 384. A. B. C. D. E. * 385. A. B. C. * D. E. 386. A. Concentrated dry plasma Albumen For treatment of leishmaniosis is used: Sulfanilamids Interferons Antibiotics Nitrofurans Hormones For treatment of leishmaniosis is used: Sulfanilamids Interferons Antibiotics Nitrofurans Hormones How is it possible to specify the diagnosis of viral meningoencephalitis after cerebrospinal puncturE. Some increasing of chlorides in CSF Presence of a lot of lymphocytes in CSF All the above Some increasing of protein in CSF Neurolymph under high pressure How is it possible to specify the diagnosis of viral meningoencephalitis after cerebrospinal puncturE. Some increasing of chlorides in CSF Presence of a lot of lymphocytes in CSF All the above Some increasing of protein in CSF Neurolymph under high pressure How many stages of the leishmania development do you now: 3 1 5 4 2 How many stages of the leishmania development do you now: 3 1 5 4 2 How to prevent of leishmaniosis? Follow the rules of personal hygiene Conduct heat preparing of food Vaccination Do not drink raw water Chemoprophylaxis How to prevent development of leishmaniosis? Follow the rules of personal hygiene B. C. * D. E. 387. A. * B. C. D. E. 388. A. * B. C. D. E. 389. A. * B. C. D. E. 390. A. * B. C. D. E. 391. A. B. C. D. E. * 392. A. B. C. D. E. * 393. A. * B. C. D. E. Conduct heat preparing of food Vaccination Do not drink raw water Chemoprophylaxis How to prevent Laim-borreliosis? Individual protecting from ticks Urgent antibiotic prophylaxis Vaccination Using of specific immunoglobuline All above enumerated How to prevent Laim-borreliosis? Individual protecting from ticks Urgent antibiotic prophylaxis Vaccination Using of specific immunoglobuline All above enumerated It is necessary to differentiate migrant erythema with: Erysipelas Anthrax Chicken-pox Herpetic infection Phlegmon It is necessary to differentiate migrant erythema with: Erysipelas Anthrax Chicken-pox Herpetic infection Phlegmon Leishmaniosis must be differentiated primarily with such disease: Sepsis Malaria Influenza Typhoid fever All of the above Leishmaniosis must be differentiated primarily with such disease: Sepsis Malaria Influenza Typhoid fever All of the above Lime disease can transmitted by ways, except for: Air-drop Contact Transmissive Fecal-oral Alimentary 394. A. * B. C. D. E. 395. A. * B. C. D. E. 396. A. B. C. D. * E. 397. A. B. C. D. E. * 398. A. B. * C. D. E. 399. A. * B. C. D. E. 400. A. * B. C. D. E. 401. A. B. * C. Lime disease can transmitted by ways, except for: Air-drop Contact Transmissive Fecal-oral Alimentary Mechanism of leishmaniosis transmission is: Transmissive Contact Alimentary All Air-droplets Mechanism of leishmaniosis transmission is: Transmissive Contact Alimentary All above it Air-droplets Methods of laboratory diagnostics of Laim-borreliosis: Selection of exciter (from blood, neurolymph, synovia, skin bioptat) Finding of borrelia antibodies (in blood, neurolymph) (IFA) Exposure of DNA of exciter (PCR) Darkfield microscopy All above enumerated Methods used for identification of sources of leishmaniosis: Stool culture Microscopy of tissue slades Urine culture Hemoculture All above enumerated Passing to exciters of Lime disease among pliers is carried out ways: Transovarian Sexual Air-drop Fecal-oral Vertical Passing to exciters of Lime disease among pliers is carried out ways: Transovarian Sexual Air-drop Fecal-oral Vertical Radical treatment of skin leishmaniosis include: Interferon Monomycin Glucantim D. E. 402. A. B. * C. D. E. 403. A. B. C. D. E. * 404. A. B. C. D. E. * 405. A. B. * C. D. E. 406. A. B. * C. D. E. 407. A. B. C. * D. E. 408. A. B. C. * D. E. 409. A. Penicillin Tetracyclin Radical treatment of skin leishmaniosis include: Interferon Monomycin Glucantim Penicillin Tetracyclin Radical treatment of visceral leishmaniosis include: Interferon Penicillin Tetracyclin Glucantim Monomycin Radical treatment of visceral leishmaniosis include: Interferon Penicillin Tetracyclin Glucantim Monomycin Rules of hospitalization of patients with leishmaniosis: Patients were not hospitalized In separate room In intestinal infections department In respiratory infections department In carantine box Rules of hospitalization of patients with leishmaniosis: Patients were not hospitalized In separate room In intestinal infections department In respiratory infections department In carantine box Source of meningoencephalitis are accept: People Ticks Fish Fleas Mosquitoes Source of meningoencephalitis are accept: People Ticks Fish Fleas Mosquitoes The basic clinical display of a skin leishmaniosis is: Abscess B. * C. D. E. 410. A. B. * C. D. E. 411. A. B. C. D. * E. 412. A. B. C. D. * E. 413. A. B. C. D. * E. 414. A. B. C. D. * E. 415. A. B. * C. D. E. 416. A. * B. Ulcer Phlegmon Vesiculs Hyperemia The basic clinical display of a skin leishmaniosis is: Abscess Ulcer Phlegmon Vesiculs Hyperemia The diagnosis of leishmaniosis can confirm after: Microscopy of blood Bacteriology of stool Hemoculture Microscopy of tissue punctates Microscopy of urine The diagnosis of leishmaniosis can confirm after: Microscopy of blood Bacteriology of stool Hemoculture Microscopy of tissue punctates Microscopy of urine The diagnostic reaction for leishmaniosis: Paul-Bunnel‘s reaction Rayt‘s reaction Vidal‘s reaction Reaction of latex agglutination Askoli reaction of precipitation The diagnostic reaction for leishmaniosis: Paul-Bunnel‘s reaction Rayt‘s reaction Vidal‘s reaction Reaction of latex agglutination Reaction of term precipitation of Askol The examination is delivered in a cliniC. Objectively: temperature 40,1 °C. Expressed meningeal symptoms. Light, haptic, pain hypesthesiA. Blood: leucocytes – 5 thousand/l, ERS-19 mm/hr. A neurolymph under high pressure, turbid, lymphocytosis, some increasing of protein, chlorides. What is your diagnosis? Meningococcal meningitis Viral meningitis Tubercular meningitis Staphylococcus meningitis Pneumoccocal meningitis The natural reservoir of exciter in case of Lime is, except for: Sick person Rodents C. D. E. 417. A. * B. C. D. E. 418. A. * B. C. D. E. 419. A. * B. C. D. E. 420. A. B. C. * D. E. 421. A. B. C. * D. E. 422. A. B. C. * D. E. 423. A. B. C. * D. Birds Pliers Cattle The natural reservoir of exciter in case of Lime is, except for: Sick person Rodents Birds Pliers Cattle The natural reservoir of exciter in case of Lime is, except for: Sick person Rodents Birds Pliers Cattle The natural reservoir of exciter in case of Lime is, except: Sick person Rodents Birds Pliers Cattle The patient concerned about fever, which is not periodical. Skin is pale, hepatosplenomegaly. What diagnose is most likely accept? Leishmaniosis Sepsis Malaria Leptospirosis Viral hepatitis The patient concerned about fever, which is not periodical. Skin is pale, hepatosplenomegaly. What diagnose is most likely accept? Leishmaniosis Sepsis Malaria Leptospirosis Viral hepatitis There are etiotropic drags for leishmaniosis treatment, except: Monomycin Glucantim Quinine Solustibozan Solusurmin There are etiotropic drags for leishmaniosis treatment, except: Monomycin Glucantim Quinine Solustibozan E. 424. A. B. C. D. * E. 425. A. B. C. D. * E. 426. A. B. C. * D. E. 427. A. B. C. * D. E. 428. A. B. C. * D. E. 429. A. B. C. * D. E. 430. A. B. C. * D. E. 431. A. B. Solusurmin Viral meningoencephalitis and DIC-syndrome require above all things. Administration of diuretics Administration of antihistamin Administration of vitamins Administration of analgetics Administration of heparin Viral meningoencephalitis and DIC-syndrome require above all things. Administration of diuretics Administration of antihistamin Administration of vitamins Administration of analgetics Administration of heparin What clinical forms of leishmaniosis do you know? Ulceral and erhythematous Papular and visceral Visceral and skin Ulceral and necrotic Bubous and skin What clinical forms of leishmaniosis do you know? Ulceral and erhythematous Papular and visceral Visceral and skin Ulceral and necrotic Bubous and skin What clinical forms of tick encephalitis present accept: Meningeal Meningoencephalitic General Feverish Meningoencephalopoliemielitic What clinical forms of tick encephalitis present accept: Meningeal Meningoencephalitic General Feverish Meningoencephalopoliemielitic What drugs of choice at etiotropic therapy of viral meningoencephalitis. Penicillins Amynoglicosides Interferons Phtorhinilons Nitrofuranes What drugs of choice at etiotropic therapy of viral meningoencephalitis. Penicillins Amynoglicosides C. * D. E. 432. A. B. * C. D. E. 433. A. B. * C. D. E. 434. A. B. C. D. E. * 435. A. B. C. D. E. * 436. A. B. C. D. * E. 437. A. B. C. D. * E. 438. A. * B. C. D. E. 439. Interferons Phtorhinilons Nitrofuranes What group of infectious diseases meningococcal infection belong to? Intestinal Respiratory External covers Blood Transmissive What group of infectious diseases meningococcal infection belong to? Intestinal Respiratory External covers Blood Transmissive What infectious diseases it is necessary differentiate visceral leishmaniosis with: Malaria Sepsis Influenza Leptospirozis All above enumerated What infectious diseases it is necessary differentiate visceral leishmaniosis with: Malaria Sepsis Influenza Leptospirozis All above enumerated What is etiological factor of antroponosis skin leishmaniosis: L. classica L. major L. brasiliensis L. tropica L. donovani What is etiological factor of antroponosis skin leishmaniosis: L. classica L. major L. brasiliensis L. tropica L. donovani What is etiological factor of visceral leishmaniosis: L. donovani L. major L. brasiliensis L. classica L. tropica What is etiological factor of visceral leishmaniosis: A. * B. C. D. E. 440. A. B. C. D. E. * 441. A. B. C. D. E. * 442. A. B. C. * D. E. 443. A. B. C. * D. E. 444. A. B. C. * D. E. 445. A. B. C. * D. E. 446. A. B. C. D. * L. donovani L. major L. brasiliensis L. classica L. tropica What is etiological factor of zoonosis skin leishmaniosis: L. donovani L. tropica L. classica L. brasiliensis L. major What is etiological factor of zoonosis skin leishmaniosis: L. donovani L. tropica L. classica L. brasiliensis L. major What is etiotropic treatment of leishmaniosis. Delagil Antibiotics Solusurmin Serum Primaquine What is etiotropic treatment of leishmaniosis. Delagil Antibiotics Solusurmin Serum Primaquine What is most characteristic symptom of the initial stage of Lime-borreliosis: Fever Catarrhal phenomena Migrant erythematic Dyspepsia phenomena Icterus What is most characteristic symptom of the initial stage of Lime-borreliosis: Fever Catarrhal phenomena Migrant erythematic Dyspepsia phenomena Icterus What is most effective antibiotics in meningococcal disease: Cephalosporines Phtorhinolons Aminoglicozides Penicillines E. 447. A. B. C. D. * E. 448. A. B. C. D. E. * 449. A. B. C. D. E. * 450. A. B. C. D. E. * 451. A. B. C. * D. E. 452. A. B. C. * D. E. 453. A. B. C. * D. E. 454. A. B. Macrolids What is most effective antibiotics in meningococcal disease: Cephalosporines Phtorhinolons Aminoglicozides Penicillines Macrolids What is prophylactic measures for high risk group? Immunization by inactivated vaccine Formulated vaccine Vitamin therapy Antibiotic therapy Immunization by live vaccine What is prophylaxy of meningococcal disease for high risk group? Immunization by inactivated vaccine Formulated vaccine Vitamin therapy Antibiotic therapy Immunization by live vaccine What is the method of laboratory diagnostics of Laym-borreliosis: Selection of exciter (from blood, neurolymph, synovia, skin bioptat) Finding of borrelia antibodies (in blood, neurolymph) (IFA) Exposure of DNA of exciter (PCR) Darkfield microscopy All above enumerated What is the origin of Lime disease: Simplest Viruses Borrelium Micoplasma Ricketsia What is the origin of Lime disease: Simplest Viruses Borrelia Micoplasma Rickettsia What is the origin of Lime disease: Simplest Viruses Borrelium Micoplasma Ricketsia What is the origin of Lime disease: Simplest Viruses C. * D. E. 455. A. B. C. D. E. * 456. A. B. C. D. E. * 457. A. B. * C. D. E. 458. A. B. * C. D. E. 459. A. B. * C. D. E. 460. A. B. * C. D. E. 461. A. B. C. D. E. * 462. Borrelium Micoplasma Ricketsia What is the origin of visceral leishmaniosis (kala-azar): L. classica L. tropica L. major L. brasiliensis L. donovani What is the origin of visceral leishmaniosis (kala-azar): L. classica L. tropica L. major L. brasiliensis L. donovani What is used as specific prophylaxis of viral meningoencephalitis. Immune globulin Vaccine Antibioticsnatoxin Serum Nothing What is used as specific prophylaxis of viral meningoencephalitis. Immune globulin Vaccine Antibioticsnatoxin Serum Nothing What laboratory methods should be taken for diagnosis of meningoencephalitis? Lumbar puncture Biopsy of tissues Urine examination Coprogram Serologic detection What laboratory methods should be taken for diagnosis of meningoencephalitis? Lumbar puncture Biopsy of tissues Urine examination Coprogram Serologic detection What material is necessary take for leishmaniosis diagnosis: Punctates from skin defect Punctates from spleen Punctates from lymph nodes Punctates from bone brain All above enumerated What material is necessary take for leishmaniosis diagnosis: A. B. C. D. E. * 463. A. B. C. D. E. * 464. A. B. C. D. E. * 465. A. B. C. * D. E. 466. A. B. C. * D. E. 467. A. B. * C. D. E. 468. A. B. C. D. * E. 469. A. B. C. D. * Punctates from skin defect Punctates from spleen Punctates from lymph nodes Punctates from bone brain All above enumerated What measures it’s necessary to perform in the focus of viral meningoencephalitis? Bacteriological inspection of contact Phagoprophylaxis Supervision during 2 weeks Chemoprophylaxis Immunization What measures it’s necessary to perform in the focus of viral meningoencephalitis? Bacteriological inspection of contact Phagoprophylaxis Supervision during 2 weeks Chemoprophylaxis Immunization What measures must be done in case of contact with leishmaniosis contact: Prophylaxis with antibiotics Chimioprophylaxis Do nothing Microscopy of tissue slades Vaccination What measures must be done in case of contact with leishmaniosis contact: Prophylaxis with antibiotics Chimioprophylaxis Do nothing Microscopy of tissue slades Vaccination What methods is used for identification of sources of leishmaniosis: Stool culture Microscopy of tissue slades Urine culture Hemoculture All above enumerated What organ demerged more frequent than all in patients with visceral leishmaniosis? Skin Lymphatic system Nervous system Gastrointestinal tract Lungs What organ demerged more frequent than all in patients with visceral leishmaniosis? Skin Lymphatic system Nervous system Gastrointestinal tract E. 470. A. B. * C. D. E. 471. A. B. * C. D. E. 472. A. B. * C. D. E. 473. A. B. * C. D. E. 474. A. B. C. D. * E. 475. A. B. C. D. * E. 476. A. B. C. D. E. * 477. A. B. Lungs What specific test is used for leishmaniosis diagnostic? Compliment fixation test Coetaneous allergic test Indirect hemaglutination test RIFA with anthrax antigen Hemaglutination test What specific test is used for leishmaniosis diagnostic? Compliment fixation test Coetaneous allergic test Indirect hemaglutination test RIFA with anthrax antigen Hemaglutination test When should start treatment of patient with leishmaniosis? After taking of material for research Immediately after hospitalization All answers are correct After clinical diagnosis After the laboratory and instrumental investig When should start treatment of patient with leishmaniosis? After taking of material for research Immediately after hospitalization All answers are correct After clinical diagnosis After the laboratory and instrumental investig Who are the source of leishmaniosis accept: Rodents Foxes Human Flies Wolves Who are the source of leishmaniosis accept: Rodents Foxes Human Flies Wolves Who would be a subject for inspection on leishmaniosis? Convalescents after leishmaniosis Persons, returning from endemic regions Patients with spleenomegaly Patients with fever more than 5 days All above enumerated Who would be a subject for inspection on leishmaniosis? Convalescents after leishmaniosis Persons, returning from endemic regions C. D. E. * 478. A. B. * C. D. E. 479. A. B. * C. D. E. 480. A. B. C. * D. E. 481. A. B. C. D. E. * 482. A. B. * C. D. E. 483. A. B. C. D. E. * 484. A. B. C. * D. E. Patients with spleenomegaly Patients with fever more than 5 days All above enumerated Wich of these symptoms are often present in patients with meningitis? Profuse watery diarrhea, vomiting, dehydratation, muscular cramps Fever, headache, stiff neck, vomiting, confusion, irritability Headache, dry cough, algor Abdominal pain, diarrhea, constipation, flatulence Algor, high temperature, headache Wich of these symptoms are often present in patients with meningitis? Profuse watery diarrhea, vomiting, dehydratation, muscular cramps Fever, headache, stiff neck, vomiting, confusion, irritability Headache, dry cough, algor Abdominal pain, diarrhea, constipation, flatulence Algor, high temperature, headache A kidney syndrome at haemorrhagic fever with kidneys syndrome shows up usually: Only laboratory changes Only on BRIDLES By pain in lumbar area, positive Pasternatsky symptom, development of oliguria By fever, polyuria, dyspepsia By paradoxical ischuria A patient has a heavy icteric form of leptospirosis. What from antibiotics is better to appoint as etiotropic therapy? Yunidoks Tetracyclinum Azitromicin Rovamicin Penicillin Agglutinines at a leptospirosis arrive at a maximal titre: On the third day of illness On the third week of illness and later On the fourth week of illness On the second month of illness To the second week of illness All of these have an epidemic dangerous at Rabies exept: Farm animals Wild rodents Domestic animals Foxes Reptiles Among the listed below choose the complication of the tetanus, which is not late: Contracture of muscles and joints Tetanus-kifozis Asphyxia Asthenic syndrome Chronic heterospecific diseases of lungs 485. A. B. * C. D. E. 486. A. B. C. D. * E. 487. A. B. C. D. * E. 488. A. B. C. * D. E. 489. A. B. C. D. * E. 490. A. * B. C. D. E. 491. A. * B. C. D. E. 492. Among the listed below choose the complication of the tetanus, which is not early: Tracheobronchitis Contracture of muscles and joints Asphyxia Myocarditis Pneumonia Among the listed below people who should receive an immediate prophylactic of the tetanus in form of AC-Antitoxin and AC IP injections after trauma? Man of 40 years, in anamnesis with 1 inoculation one year ago Pregnant woman of 30 years, in the second half of pregnancy Child, 7 months, instilled according to a calendar Retire man of 57 years, who is not instilled Child of 6 years, instilled according to a calendar Among the listed below what preparations are not etiological for tetanus? AC-anatoxin Medical horse serum Human immunoprotein Anticonvulsant preparations Penicillin An initial period at the hemorrhagic fever with a kidneys syndrome lasts: Few hours Day To three days Week Two weeks At a leptospirosis the exposure of antibodies is considered reliable in a titre: 1:70 and anymore 1:80 and anymore 1:60 and anymore 1:100 and anymore 1:40 and anymore At an objective review for the Congo hemorrhagic fever characteristically: Mucosal hyperemia of person Pallor of person Puffiness of person Ochrodermia of person Exanthema on face At patient, who acted to permanent establishment, on clinical epidemiological indexes of Lassa fever is suspected. What from the resulted clinical indexes are not characteristic for this disease? Hemolytic icterus Generalized lymphadenopathy Conjunctivitis Ulcerous pharyngitis Encephalopathy At patient, who acted to permanent establishment, the especially dangerous hemorrhagic fever is suspected. For the selection of exciter can be used all materials, except for? A. B. * C. D. E. 493. A. * B. C. D. E. 494. A. B. C. * D. E. 495. A. * B. C. D. E. 496. A. * B. C. D. E. 497. A. B. C. D. E. * 498. A. B. C. D. E. * 499. A. B. C. Blood Puncture from lymph nodes Urines Pleura liquid Swab from nasopharyng At treatment of patients with leptospirosis antibiotics consider most effective: Penicillin Macrolids Cefalosporins Ftorhinolons Sul'fanilamids Basic mechanism of transmission of Lime disease: Air-drop Contact Transmissive Fecal-oral Vertical Basic vector of Lyme disease: Tick Mosquito Flea Mosquito All of bloodsucking insects Citizen A., take participence in a business geologic expedition. Area is endemic according to Laim-borreliosis. How to prevent possible infection? Individual protecting from ticks Urgent antibiotic prophylaxis Vaccination Using of specific immunoglobuline All above enumerated Clinical stages of Lime-borreliosis: Common toxic Cardiological complications Neurological complications Artritic All above enumerated Confirm diagnosis of haemorrhagic fever with kidneys syndrome by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Virologic and serum methods Confirm the diagnosis of Congo hemorrhagic fever by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods D. E. * 500. A. B. C. D. E. * 501. A. B. C. D. E. * 502. A. B. C. D. E. * 503. A. B. C. D. E. * 504. A. B. * C. D. E. 505. A. B. C. D. E. * 506. A. B. C. * D. E. 507. Proper epidemiological information Virologic and serum methods Confirm the diagnosis of Ebola fever by a way of: Growth of viruses on chicken embryons Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Selection of virus on the Vero culture Confirm the diagnosis of Lassa hemorrhagic fever by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Virologic and serum methods Confirm the diagnosis of Marburg fever by a way of: Growth on chicken embryos Only bacteriological methods Bacteriological and serum methods Proper epidemiologys information Selection of virus on the Vero culture Confirm the diagnosis of Omsk fever by a way of: Growth of virus on chicken embryons Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Selection of virus on the Vero culture Daily allowance doses of penicillin at treatment of leptospirosis: 2-3 million units 3-12 million units 20 million units 40 million units Over 40 million units Duration of activity of pliers depends on: Weather Regional naturally-geographical terms Kind of vector Activity of animals in different periods of year From all of transferred Duration of the latent period in case of tetanus: 1-6 hours 1-4 days 5-14 days 1-6 weeks. 1-6 months During a walk in-field a fox attacked on a child, bit and disappear him feet and racemes of hands. An animal disappeared. What is medical tactic. A. B. * C. D. E. 508. A. B. C. D. E. * 509. A. B. C. * D. E. 510. A. B. C. * D. E. 511. A. B. C. D. * E. 512. A. B. C. D. * E. 513. A. B. C. D. * E. 514. A. * B. C. D. To process a wound surgically To process a wound soapy solution, to enter an antirabies immunoprotein and vaccine Preventive antirabies vaccination To process wounds surgically, to enter an antirabies immunoprotein and vaccine Preventive antirabies immunoprotein Etiotropic therapy of leptospirosis includes: Oxyhinolons Sulfanilamids Nitrofurans Antitocidns Antibiotics For confirmation of diagnosis of hemorragic fever with a kidney syndrome use: Bacteriological method Virological method Reaction of immunofluorescence Reaction of braking of hemagglutination Research of blood drop under a microscope For confirmation of yellow fever diagnosis use: Bacteriological analysis of blood Bacteriological examination of urine Virological hemanalysis Biochemical blood test Global analysis of blood For hydrophobia the source of infection can be a dog bite in all the cases, except for: Patient with hydrophobia Suspicion on hydrophobia Vagrant Month prior to the disease Last 10 days before the disease For the exciter of tetanus characteristic such properties, except: Formation of exokinesis Ability to propagate in anaerobic conditions Formation of spores Formation of gametes Gram positive For the initial period of the Congo hemorrhagic fever not characteristically: Fever Pains in joints and muscles Severe pain of head Oliguria Dizziness For the prophylaxis of leptospirosis use: Active vaccine Anavaccine Toxoid All E. 515. A. B. C. D. * E. 516. A. B. * C. D. E. 517. A. B. C. * D. E. 518. A. B. C. * D. E. 519. A. * B. C. D. E. 520. A. B. * C. D. E. 521. A. B. C. * D. E. 522. A. B. Antibiotics For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use: Glucocorticoids Anabolic steroid Disintoxication facilities Dihydration facilities Antihistaminics General view of patient with the hemorrhagic fever with a kidneys syndrome: Skinning covers Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk Hyperemia of person, scleritis, conjunctivitis Grayish color of person Icteric color of skin Hemograme in the second period of yellow fever: Leukocytosis Normal global analysis of blood Leukopenia, neutropenia Leukopenia, neutrophilosis Leukocytosis, lymphomonocytosis How long lasts the leptospirosis incubation period: 2 month 1-7 days 7-14 days 14-21 days 2-3 days How long the rash is present in case of haemorrhagic fever with kidneys syndrome? During all feverish period Before the reconvalescense Before development of clinical features of kidneys insufficiency During whole disease Appears yet in a latent period and disappears in the period of early reconvalescense illness? Paroxysm of fever Paroxysm of hydrophobia Hepatomegaly Spleenomegaly Presence of rash In case of tetanus the epidemiological measures are directed on: Elimination of the source of tetanus Treatment of the source of tetanus Specific prophylaxis Medicines prophylactics Nothing should be performed In hydrophobia the source of the virus can be: Wild animals Home animals C. D. E. * 523. A. B. C. D. E. * 524. A. B. C. * D. E. 525. A. B. * C. D. E. 526. A. B. C. * D. E. 527. A. B. C. * D. E. 528. A. * B. C. D. E. 529. A. * B. C. D. Bats Rodents All the answers are correct In most patients with Congo hemorrhagic fever temperature curve is: Wunderlich type Botkin type Undulating Intermittent Two-humped In the biochemical blood test at patients with the hemorrhagic fever with a kidneys syndrome not characteristically: High level of urea Decline of potassium level Bilirubinemia Increasing of kreatinine Increasing of nitrogen In the global analysis of blood in case of Congo hemorrhagic fever not characteristically: Leukocytosis Leukopenia Neutropenia Thrombocytopenia Increasing of ESR In the global analysis of blood: leucopenia, neutropenia, increasing of ESR. Intensive therapy without any effect and a patient died. Credible diagnosis? Viral hepatitis Leptospirosis Yellow fever Crimean fever Malaria In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is: High temperatures Pains in gastrocnemius muscles and positive Pasternatsky symptom Pains in joints and positive Pasternatsky symptom Hemorragic syndrome Dyspepsia phenomena It is necessary to differentiate migrant erythema with: Erysipelas Anthrax Chicken-pox Herpetic infection Phlegmon Lime disease can transmitted by ways, except for: Air-drop Contact Transmissive Fecal-oral E. 530. A. B. * C. D. E. 531. A. B. C. D. E. * 532. A. B. C. * D. E. 533. A. * B. C. D. E. 534. A. * B. C. D. E. 535. A. B. * C. D. E. 536. A. B. C. D. E. * 537. A. Alimentary Mechanism of transmission in case of tetanus: Intra muscular conduction Pin Insect conduction Faecally-oral Vertical conduction Methods of laboratory diagnostics of Lyme-borreliosis: Selection of exciter (from blood, neurolymph, synovia, skin bioptat) Finding of borrelia antibodies (in blood, neurolymph) (IFA) Exposure of DNA of exciter (PCR) Darkfield microscopy All enumerated Most characteristic symptom of the initial stage of Lime-borreliosis: Fever Catarrhal phenomena Migrant erythematic Dyspepsia phenomena Icterus Most characteristic symptoms of the hydrophobia are: Paroxysm of hydrophobia Apathy and depression Neuralgic pains on motion nervous barrels, the nearest to the place of bite Dyspepsia disorders Catarrhal phenomena Passing to exciters of Lime disease among pliers is carried out ways: Transovarian Sexual Air-drop Fecal-oral Vertical Patient A., 25 years old, is being treated because of tetanus concerning. Choose the specific treatment. Antibiotics Immune globulini Anticonvulsant medicine Cardiac preparations Respiratory analeptics Patient B., 37 years, had the rapid development of the severe tetanus after criminal abortion. What is the medical tactic? Anticonvulsant preparations Revision of the uterus cavity Analgesic therapy Antibiotics All answers are correct Phage symptom in case of yellow fever is: Pain in right iliac area B. C. * D. E. 538. A. B. * C. D. E. 539. A. B. * C. D. E. 540. A. * B. C. D. E. 541. A. B. C. D. * E. 542. A. * B. C. D. E. 543. A. B. C. D. E. * 544. A. B. C. D. Enanthema on a soft palate Replacement of tachicardia on expressed bradicardia Hemorrhages in a conjunctiva Yellow hands Pregnant woman of 22.years old delivered in the ambulance. How should be treated the child from the point of view of tetanus prophylactic? AC-antitoxin Anti tetanus serum Tetanus toxoid + anti tetanus serum Tetanus toxoid + anti tetanus serum + immunoprotein Anti tetanus serum + immunoprotein Rhabdovirus from an organism of the patient or animal is revealed to the flow: Last 20 days of latent period and during all the illness Last 7-10 days of latent period and during all the illness Last 7-10 days of latent period Last 7-10 days of latent period and at the beginning of illness During all the illness Specific prevention of Crimean-Congo haemorrhagic fever are: Vaccine and human immunoglobulin Serum Serum and human immunoglobulin Do not developed Antibacterial drugs Specific prevention of hemorrhagic fevers: The live vaccine Killed vaccine The specific immunoglobulin Do not developed Polivalent vaccine Tetanus might appear in case of: Trauma Mosquito bite Usage of stranger clothes Socialization with the sick people Usage of the water with poor quality Tetanus toxin consists of all units among the listed below, except: Tetanospasmin Tetanolysin Exotoxin Low-molecular fraction Enterotoxin The basic reservoir of rhabdovirus is: Pisces Reptiles Birds Weed-eaters E. * 545. A. * B. C. D. E. 546. A. B. C. D. * E. 547. A. * B. C. D. E. 548. A. B. C. D. E. * 549. A. B. C. D. * E. 550. A. B. C. D. * E. 551. A. B. C. D. E. * 552. A. * B. Carnivores The best terms of tetanus exciter cultivation: Anaerobic conditions Oxygen supply Presence of animal albumen in nutritive medium Low temperature 1 % peptone water The decline of arteriotony at a leptospirosis is not caused: By expansion of vessels under the action of toxin Hypovolemia By myocarditis Hypercalcgesty By adrenal insufficiency ?The exciter of tetanus is: Clostridia Escherichia Candida Virus Simplest The first symptom of prodromal period of hydrophobia is: Cough Nausea Vomiting Diarrhea Slight swelling and erethema of the scar The first symptom of prodromal period of hydrophobia is: Cough Nausea Vomiting Neurological pains in motion nervous barrels, the nearest to the place of bite Diarrhea The first symptom of prodromal period of hydrophobia is: Cough Nausea Vomiting Apathy and depression Diarrhea The main of antibiotics which is used in treatment of leptospirosis: Tetracyclin Tetraolean Erythromycin Streptomycin Penicillin The most characteristic symptom in the climax period of the Congo hemorrhagic fever is: Hemorrhagic syndrome Hepatic insufficiency C. D. E. 553. A. * B. C. D. E. 554. A. * B. C. D. E. 555. A. B. * C. D. E. 556. A. B. C. * D. E. 557. A. B. C. D. * E. 558. A. * B. C. D. E. 559. A. * B. C. Dyspepsia phenomena Sharp kidney insufficiency Мeningeal syndrome The natural reservoir of exciter in case of Lime is, except for: Sick person Rodents Birds Pliers Cattle The period of polyuria at haemorrhagic fever with kidneys syndrome is a sign of: Recovering Chronic process Unfavorable flow of illness Development of complications Complete convalescence The source of infection of Omsk‘s hemorrhagic fever are muskrat, water rats and other rodents. Who are the carriers? Bee and flea Pliers and flea Mosquitoes Fly Pliers and mosquitoes The spores of tetanus are saved: After boiling during 1 hour Under act of dry air at the temperature of 115 degrees C In soil during many years In 1 % solution of formalin during 6 hours All answers are correct Those who have been in contact with sick haemorrhagic fevers, as well as those who had bite by the ticks in endemic areas are introducing: Specific vaccine The specific immunoglobulin in doses of 10-15 ml vaccine The specific immunoglobulin in doses of 10-15 ml The specific immunoglobulin in doses 5-7,5 ml Nothing To what group of infections does the hydrophobia belong? Zoonosis Anthroponosis Capronosis Anthropozoonosis Caprozoonosis To you, as to the graduating student of medical university, is possibly to work upon termination of studies in Crimea. What endemic ticks infections is in this territory? Crimean hemorrhagic fever, tick encephalitis, Q-fever Crimean hemorrhagic fever, malaria, epidemic typhus Tick encephalitis, Ebola fever, Lassa fever, Crimean hemorrhagic fever D. E. 560. A. B. * C. D. E. 561. A. B. C. D. E. * 562. A. B. C. D. * E. 563. A. B. C. D. * E. 564. A. B. * C. D. E. 565. A. * B. C. D. E. 566. A. B. C. D. E. * 567. A. Q-fever, spotted fever, leptospirosis Q-fever, Crimean hemorrhagic fever, psittacosis, tick encephalitis Treatment of leptospirosis: Desintoxication, dehydratation, antibiotics, glukokorticosteroids Antileptospirosis immunoprotein, antibiotics, nosotropic facilities Antibiotics, rehadratation, sorbtion preparation, vitamins Diet № 7, antibiotics, desintoxication facilities Antibiotics, diuretic, interferons Unlike leptospirosis in case of yellow fever is absent: Hemorrhagic syndrome Kidney insufficiency Іntoxication syndrome Міalglic syndrome Hepatic insufficiency Urgent immunoprofilactic of tetanus in the case of trauma should be conducted in such period: 25 days from the moment of trauma 30 days from the moment of trauma In the first 10 days from the moment of trauma After the trauma Not mentioned Vaccinations against leptosprosis perform for: All Only villagers Only to the habitants of endemic districts Only to the persons busy in the stock-raising It is not conducted Vegetative form of exciter of tetanus is destroyed in such terms, except for: At a temperature of 100 °C At room temperature Under act of carbolic acid Under the action of oxygen Under act of antibiotics Violations of electrolyte balance show up at a leptospirosis: Metabolic acidosis By a metabolic alkalosis Respirator acidosis By a respiratory alkalosis All above enumerated What changes in biochemical blood test inherent for haemorrhagic fever with kidneys syndrome? Increase level of urea and bilirubin The level of urea and kreatinine falls The level of kreatinine grows and urea falls The level of urea grows and kreatinine falls The level of urea and kreatinine grows What changes in haemogram inherent Congo hemorrhagic fever? Normochomic anaemia, leucocytosis mononuclear B. C. * D. E. 568. A. B. C. D. * E. 569. A. B. C. * D. E. 570. A. B. C. * D. E. 571. A. B. C. * D. E. 572. A. B. C. * D. E. 573. A. B. C. * D. E. 574. A. B. C. * D. E. Erythrocytosis, lymphocytosis Hypochromic anemia, erythrofilosis Hypochromic anemia, neutrofilosis Hyperchromic anemia, neutrofilosis What changes in haemogram inherent for haemorrhagic fever with kidneys syndrome? Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia enhanceable ESR erythrocytosis, lymphocytosis,ESR is enhanceable Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia enhanceable ESR Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia enhanceable ESR Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR What etiothropic means use at treatment of haemorrhagic fever with kidneys syndrome: Benzylpenicillin Dopamine Virolex Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Ebola fever: Benzylpenicillin Dopamine Virolex Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Marburg fever: Benzypenicillin Dopamine Ribavirin Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Congo fever: Benzylpenicillin Dopamine Ribavirin Dexamethazone Etamsylatum What etiothropic means use at treatment of patients with Lassa fever: Benzylpenicillin Dopamine Ribavirin Dexamethazole Etamsylatum What etiothropic means use at treatment of patients with Crimea fever: Benzylpenicillin Dopamine Ribavirin Dexamethazone Etamsylatum 575. A. B. C. * D. E. 576. A. * B. C. D. E. 577. A. B. C. D. * E. 578. A. B. C. * D. E. 579. A. * B. C. D. E. 580. A. * B. C. D. E. 581. A. * B. C. D. E. 582. A. B. C. * What etiothropic means use at treatment of patients with Omsk fever: Benzylpenicillin Dopamine Ribavirin Dexamethazone Etamsylatum What from antibiotics are more effective at treatment of icteric form of leptospirosis: Penicillins Aminoglicozids Tetracyclins Macrolids Metrogil What from antibiotics more expedient to use for treatment of leptospirosis? Macrolids Tetracyclins Aminoglicosids Penicillins Metrogil What is the duration of excitation period of hydrophobia? 7-10 days 24 hours 2-3 days, sometimes to 6 days Not more than 2 days Up to 6 hours What is the duration of outpatient supervision after patients, recovered of tetanus? 2 years 3 months 1 month For the decreed groups of population for life time There is no such supervision at all What is the duration of the prodromal period for the hydrophobia? 1-3 days Up to 1 day 4-7 days 3-4 days 3-5 days What is the entrance for the hydrophobia? Damaged skin and mucous tissues Respiratory tracts Family ways Gastrointestinal tract Blood What is the first aid preparation for the patient with tetanus? Glucocorticoids Analgetics Anticonvulsant medicine D. E. 583. A. B. C. D. * E. 584. A. B. C. * D. E. 585. A. B. C. * D. E. 586. A. B. C. D. * E. 587. A. B. C. D. * E. 588. A. B. C. D. E. * 589. A. B. * C. D. E. 590. A. Surgical treatment of the wound Oxygen therapy What is the mechanism of transmission of hydrophobia? Transmissive Fecally-oral Air drop Wound Domestic contact What is the origin of Lime disease: Simplest Viruses Borrelium Micoplasma Ricketsia What is the receptivity of population to the tetanus? 0% 50 % Almost 100 % 10 % 70 % What is the sensitivity to the hydrophobia? 45 % 25 % 85 % 100 % 10 % What is typical for the Lassa hemorrhagic fever: Effect of cardiovascular system Development of acute hepatic insufficiency Hundred-per-cent lethality Defeat of breathing organs Development of paresis and paralysis What measures should be taken in relation to contact persons in case of tetanus? Vaccination Isolation of contacts Chemoprophylaxis Laboratory inspection They need no measures What periods of hydrophobia do you know? Incubation, depressions, excitation Incubation, depressions, excitation, paralytic Depression, excitation, paralytic Incubation, excitation, paralytic Incubation, depressions, paralytic What rashes in case of haemorrhagic fevers with kidneys syndrome? Roseola B. C. D. * E. 591. A. B. C. D. * E. 592. A. B. C. D. * E. 593. A. * B. C. D. E. 594. A. * B. C. D. E. 595. A. B. * C. D. E. 596. A. B. C. D. * E. 597. A. B. C. D. * E. Maculo-papular Punctuate Petechial Rashes is not characteristic What rashes present in case of Congo hemorrhagic fever? Roseola Maculo-papular Punctulate Petechial Rashes not is characteristic What rashes present in case of Crimea hemorrhagic fever? Roseola Maculo-papular Punctulate Petechial Rashes not is characteristic What temperature of the body is typical for the paralytic period? Hyperpyrexia Hypothermia High Normal Subfebril Whatever complication meets at the yellow fever: Liver insufficiency Kidney insufficiency Infectious-toxic shock Myocarditis Edema of lungs When from the beginning of vaccination an antibodies to the rhabdovirus appear? In a week In 2 weeks In a month After half of year Don’t produced Whether there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome: In an initial period It is not It is in all periods of disease It is in climax period It is in the period of recovering Which serotypes of leptospirosis caused the disease more frequent: L. interogans L. grippotyphosa L. canicola L. icterohaemorrhagia L. Pomona 598. A. B. C. * D. E. 599. A. B. C. * D. E. 600. A. * B. C. D. E. 601. A. B. C. * D. E. 602. A. B. C. D. * E. 603. A. * B. C. D. E. 604. A. * B. C. D. E. Who is the source of tetanus? Sick person Rodents Soil Insects Cattle Who is the source of the causal agent in the Crimean-Congo haemorrhagic fever? Rodents, cattle, birds Iksod and gamazov mites Rodents, cattle, birds, sick people The sick man, reconvalenc, bacteriocarries Rodents, cattle, birds, sick people, bacteriocarries Who of the listed below persons must take the conditional course of inoculations against hydrophobia? A teenager bitten by a dog which is on a leash, not instilled Man bitten by a fox which perished A child, scratched by a squirrel which disappeared in-field A woman, bitten by a cat ill with hydrophobia Man, who had a meal of undercooked of animal with hydrophobia With appearance of hemorrhagic syndrome at Congo fever temperature of body always: Normalize Grows critically Goes down Does not change Grows gradually With the purpose of immunotherapy it is better to apply at treatment of leptospirosis: Immunodepressants Antihistaminics Horse whey Antileptospirosis human immunoprotein Antileptospirosis neat immunoprotein You might be infected with a rhabdovirus in case of: Bite +salivation to the skin by an animal Infected meal Infected water Contact with the infected air Bite with the infected insect A painful lesion with a bright red, edematous, indurate appearance and an advancing, raised border that is sharply demarcated from the adjacent normal skin. Fever is a feature. What is the main mechanism of transmission of this disease? Airborne Alimentary Contact Transmisiv Vertical 605. A. B. * C. D. E. 606. A. * B. C. D. E. 607. A. B. C. * D. E. 608. A. B. C. * D. E. 609. A. B. * C. D. E. 610. A. B. C. D. E. * 611. A. A patient 60 years old for 2 days has disturbed severe pain in a right arm. On 3rd day appeared blisters, pouring out as a chain on the skin of shoulder, forearm and brush. Sensitiveness in the area of pouring out is mionectic. What disease can be diagnosed? Dermatitis Herpetic ganglionitis Neck-pectoral redicals Psoriasis Allergy A patient came with complaints of sickly erosions on his penis. From anamnesis frequent appearance of similar rashes is found out during a year. Objectively: on a balanus are the grouped blisters and erosions, soft on palpation. What is your diagnose? Recurrent herpes of ІІ type Vulvar pemphigus Primary syphyllis Shankoform pyoderma Recurrent herpes of ІІІ type A patient with temperature of body 40.0 °C, nonproductive cough, photophobia, puffiness of face, dots on gums, blushes on the mucus of cheeks your diagnosis? Tuberculosis Меningococcemia Measles Enteroviral infection Staphylococcal sepsis A patient’s temperature is 40 °C. There are deep and unproductive cough, photophobia, face puffiness whitish points on the mucous membrane of cheeks opposite molar teeth. What is the most possible diagnosis? Tuberculosis Meningococcemia Measles Enteroviral infection Staphylococcus sepsis All are the clinical signs of measles EXEPT: Acute beginning of high fever Icterus Maculo-papular rash Sequential appearance of rash Scaling And. 5 days after the isolation of the last patient 11 days 21 day 10 days No need for quarantine 5 days after isolation of the last child At a patient, 17 years: quinsy, temperature 38,2 °C, generilized lymphadenopathy (the first multiplied neck lymphatic knots which are located along m. sternocleidomastoideus), small icterus hepatospleenomegaly. What exciter causes this disease? Herpesvirus I type B. C. D. * E. 612. A. B. C. D. * E. 613. A. * B. C. D. E. 614. A. B. * C. D. E. 615. A. B. C. * D. E. 616. A. * B. C. D. E. 617. A. B. * C. D. E. 618. A. Herpesvirus ІІ type Herpesvirus ІІІ type Herpesvirus ІV type Herpesvirus V type At a patient, 17 years: quinsy. Temperature 38,2 °C, generilised lymphadenopathy (the first multiplied neck lymphatic knots which are located along m. sternocleidomastoideus), small icterus, hepatospleenomegaly. Previous diagnosis? Tuberculosis of lymphatic knots Bacterial quinsy Diphtheria Infectious mononucleosis Megacaryoblastoma At junior nurse, who works in child’s infectious department, herpes simplex was found. What should manager of department must do? Create a quarantine in the department To appoint an immunoprotein to the children Discharge all children from the department To appoint immunomodulators with a prophylactic purpose To inspect a junior nurse on a staphylococcus At the end of treatment of patients with erysipelas it is recommended to enter: 500 000 of Bicyllin-3 intramuscular 1 500 000 of Bicyllin-5 intramuscular 1 000 000 of Bicyllin-5 intramuscular 1 500 000 of Bicyllin-3 intramuscular 500 000 of Bicyllin-3 intramuscular Because of the risk of reinfection of erysipelas, prophylactic antibiotics are sometimes used after resolution of the initial condition. Which antibiotics did we use? Erythromycin Penicillin III Penicillin V Cephazolin Laevomicetyn Call the exciter of erysipelas. Streptococcus of group A Streptococcus of group B Streptococcus of group C Streptococcus of group D Streptococcus of group E Causing of erysipelas are: C. tetani E. coli Candida Epstein-Barr virus Hemolytic streptococcus group A Diagnosis of repeated erysipelas may be set if clinical signs appeared: In 2 years after erysipelas the last relapse of erysipelas B. C. D. * E. 619. A. B. * C. D. E. 620. A. B. * C. D. E. 621. A. B. * C. D. E. 622. A. B. * C. D. E. 623. A. B. * C. D. E. 624. A. B. * C. D. E. 625. A. B. * C. D. E. In 1 years after erysipelas the last relapse of erysipelas In 9 months after an initial appeal of erysipelas In 2 years after the initial disease In 1 years after the initial disease Did you need bacteriological examination of erysipelas? Yes No Only for the decreed persons Only for children Only at suspicion on diphtheria Did you need biological examination of erysipelas? Yes No Only for the decreed persons Only for children Only at suspicion on diphtheria Did you need serum examination of erysipelas? Yes No Only for the decreed persons Only for children Only at suspicion on diphtheria Ethyology of toxoplasmosis is: Sh. Flexneri Toxoplasma gondii Neisseria meningitides Corinebacterium diphtheria ?-hemolytic streptococcus ?Ethyology of toxoplasmosis is: Sh. Flexneri Toxoplasma gondii Neisseria meningitides Corinebacterium diphtheria Hemolytic streptococcus Ethyology of toxoplasmosis is: Sh. Flexneri T. gondii N. meningitides C. diphtheria S. aureus Ethyology of toxoplasmosis is: Virus Simplest Bacteria Helminthes Animals 626. A. B. * C. D. E. 627. A. B. * C. D. E. 628. A. B. C. D. E. * 629. A. B. C. * D. E. 630. A. B. C. D. E. * 631. A. B. * C. D. E. 632. A. B. C. * D. E. 633. A. B. Etiology of erysipelas is. Erysipelothrix rhusiopathiac beta-hemolytic streptococcus Bacillus anthracis Herpes zoster Herpes simplex ?Etiology of erysipelas is. Erysipelothrix rhusiopathiac B-hemolytic streptococcus Bacillus anthracis Herpes zoster Herpes simplex Factors of transmition of toxoplasmosis is: Vertical By ingestion of raw meat By ingestion of undercooked meat By cats All above it For how long a patient with complicated form of measles should be isolated: For 4 days from the beginning of rash For 7 days from the beginning of rash For 10 days from the beginning of rash For 17 days from the beginning For 20 days from the beginning of illness For specific pathogen of erysipelas following such properties: Formation of exotoxins Ability to multiply in anaerobiosis Formation of spores Formation of gametes Gram-positive For what disease characterize changes in a blood (presence of lymphomonocytes and a typical mononuclears)? Flu Infectious mononucleosis Measels AIDS Diphtheria From etiotropic (antistreptococcus) facilities the most effective are: Furazolidonum Gentamicin Benzilpenicilin and Oxacillinum Benzilpenicilin and Furazolidonum Doksiciklin and Gentamicin How long does the incubation period last in erysipelas? 1-5 days 5-10 days C. * D. E. 634. A. B. C. * D. E. 635. A. B. C. D. * E. 636. A. B. C. D. E. * F. G. H. I. J. 637. A. B. C. * D. E. 638. A. B. * C. D. E. 639. A. B. C. D. E. * 3-5 days 10-15 days 15-20 days How long does the incubation period last? 1-5 days 5-10 days 3-5 days 10-15 days 15-20 days How long is the incubation period of erysipelas? From a few hours to 5-7 days From a few hours to 7-10 days From a few hours to 2 days From a few hours to 3-5 days From a few hours to 14 days In childbirth is erysipelas of left shin. Anti-epidemic measures in the maternity ward are: No earlier than 2 years after disease No earlier than 3 years after disease In 9 months after the initial disease In 2 weeks after a tonsillitis (angina) In 3 weeks after a tonsillitis (angina) Not needed Quarantine in the department Chatting Quartz of air Isolation of sick, disinfection Indications for combined antibiotic therapy of erysipelas are: Initial disease Repeated wildfire Recurrent erysipelas Hard course To prevent relapse Measures of urgent prophylaxis for unvaccinated children who have never been ill with measles in case of exposure to an ill with measles Separation from the source Vaccination Administration of antibiotics Disinfection Does not exist Measures of urgent prophylaxis of measles for contacts which have never been ill, but were vaccinated against measles Separation from the source Vaccination Use of antibiotics Use of immunoglobulin No need to conduct 640. A. B. C. D. E. 641. A. * B. C. D. E. 642. A. B. C. D. E. * 643. A. B. C. D. E. 644. A. B. * C. D. E. 645. A. * B. C. D. E. 646. A. B. C. * D. Measures of urgent prophylaxis of measles for people who had been ill with measles, but never have been vaccinated Separation from the ill Vaccination Use of immunoglobulin Use of antibiotics No need to conduct Mechanism of transmission of erysipelas are often: Air Contact Transmissiv Fecal-oral Transplacental Mechanism of transmission of toxoplasmosis are often: Air Contact Transmissiv All above it Transplacental Methods of specific prophylaxis of scarlet fever: Isolation of ill Vaccination Use of antibiotics Disinfection Does not exist Morphological characteristics of the causative agent of erysipelas: Stik Cocc Ring Larva In a spiral Patient A., 40 years, complains of hyperemia and edema on a right cheek. At examination: temperature of body 38,7 °C, enlarged and painful lymphatic nodules on the right part of neck. A border between hyperemia and healthy skin is clear, there are bubbles with hemorrhagic content, and palpation is painful. What is clinical diagnosis? Erysipelas Anthrax Herpetic infection Chicken pox Phlegmon of cheek ?Patient A., complains of redness of skin and edema on the right cheek. During a review: Т 38,7 C, enlarged and painful lymphatic submandibular nodes on right side, border between red and normal skin is sharp, present of swallowing with dark content, palpation is painful. What is your previous Herpetic infection diagnosis? Anthrax, skin form Erysipelas, hemorrhagic form Varicella (chickenpox) E. 647. A. B. C. * D. E. 648. A. B. C. * D. E. 649. A. * B. C. D. E. 650. A. B. C. D. E. * 651. A. B. C. D. * E. 652. A. B. C. * D. E. 653. A. B. C. D. * Phlegmone of cheek Patient M., 30 years old, is treating ambulatory with uveitis. He complains of periodical subfebrill temperature, headaches, weakness; he is ill during 3 years. Clinical previous diagnose. Malaria Brucellosis Toxoplasmosis Amoebiasis Helmints Patient M., 30 years old, is treating ambulatory with uveitis. He complains of periodical subfebrill temperature, headaches, weakness; he is ill during 3 years. Clinical previous diagnose. Malaria Brucellosis Toxoplasmosis Amoebiasis Helmints Patient R., 31 years old, complains of rigor, fever (temperature 39 ?C), weakness, bright hyperemia with elevated border and edema of subcutaneous fat, painfull in periphery, appears on the left leg last day. What is clinical diagnosis? Erysipelas Anthrax Herpetic infection Chicken pox Phlegmon Patients with erysipelas working teacher in kindergarten. Anti-epidemic measures in the place of work are: Not needed Quarantine of the group The patient may be treated and continues to work Compulsory hospitalization of the patient Isolation of the patient to recovery Primary and secondary prevention of recurrences of erysipelas disease are: Avoid hypothermia Of antibiotics in sufficient Treatment that mycosis, thrombophlebitis-determine predisposition to erysipelas stop Bicylin-profilactyc after antibiotic therapy Avoid skin mikrotraum Rules of hospitalization of patients with infectious mononuclesis: Patients are not hospitalized In a chamber for the infections of respiratory tracts In a separate chamber In a chamber for the infections of external covers In a chamber for intestinal infections Term of contagious period of patient diagnosed with uncomplicated form of measles Until clinical recovery After rash starts disappearing Before appearance of rash 4 days from the beginning of rash E. 654. A. * B. C. D. E. 655. A. * B. C. D. E. 656. A. B. C. D. * E. 657. A. B. C. D. * E. 658. A. * B. C. D. E. 659. A. B. C. D. E. * 660. A. B. C. D. E. 661. 10 days from the beginning of illness The diagnosis of recurrent erysipelas can be set, if clinical displays appeared: In 2 years after a primary disease At the first In 6 months after a primary disease On other place In 2 weeks after the tonsillitis The diagnosis of recurrent erysipelas can be set, if clinical displays appeared: In 2 years after a primary disease At the first In 6 months after a primary disease On other place In 2 weeks after the tonsillitis The diagnosis of the repeated erysipelas can be set, if clinical displays appeared: In 2 years after the last recurrent erysipelas At the first In 6 months after a primary appeal In 2 years after a primary disease In a year after a primary disease The diagnosis of the repeated erysipelas can be set, if clinical displays appeared: In 2 years after the last recurrent erysipelas At the first In 6 months after a primary appeal In 2 years after a primary disease In a year after a primary disease The main complication of erysipelas: All above listed Glomerulonephritis Sepsis Elephantiasis Rheumatism The patient asked the doctor about the recurrence of erysipelas. He is a manager at the company. Terms treatment and access to work: Necessarily isolation Necessarily hospitalization Treatment in home, without access to work Can not works on this job No restrictions The source of exciter of cholera is: Man, patient with a tonsillitis (angina) Man, patient with moderate form cholera Man, patient with a scarlet fever Healthy carriers of streptococcus All is listed above There is the risk of reinfection of erysipelas, prophylactic antibiotics are sometimes used after resolution of the initial condition. Which antibiotics did we use? A. B. C. * D. E. 662. A. B. C. D. * E. 663. A. B. C. D. * E. 664. A. B. C. D. E. * 665. A. B. C. D. E. * 666. A. * B. C. D. E. 667. A. B. C. D. * E. 668. A. * B. C. D. Erythromycin Penicillin III Penicillin V Cephazolin Laevomicetyn To what kind of patients with erysipelas prednisolon should be prescribed? Patients with hemorrhagic-bulbous form of erysipelas Patients with accompanied thrombophlebitis Patients with severe intoxication Patients with often relapses of erysipelas and signs of lymphostasis For all patients To what kind of patients with erysipelas prednisolon should be prescribed? Patients with hemorrhagic-bulbous form of erysipelas Patients with accompanied thrombophlebitis Patients with severe intoxication Patients with often relapses of erysipelas and signs of lymphostasis For all patients Toxoplasmosis is transmitted by: Transplacentally By ingestion of raw By ingestion of undercooked meat By cats All above it Toxoplasmosis is transmitted by: Transplacentally By ingestion of raw By ingestion of undercooked meat By cats All above it What additional inspections must be conducted to the patient with infectious mononucleosis? IFA on HIV-infection, bacteriology inspection on diphtheria IFA on HIV-infection, bacteriology inspection on a rabbit-fever Bacteriology inspection on diphtheria and typhoid Reaction of Burne and Rihth-Heddlson Reaction of Paul-Bunnel and punction of lymphatic knot What additional test should hold for the patient with infectious mononucleosis? Burne and Wright-Hadlson‘s reactions ELISA-test, bacteriological test for tularemia Bacteriological test for diphtheria and typhoid fever ELISA-test, bacteriological test for diphtheria Paul-Burne reaction and lymph node puncture What are the anti epidemic measures in regards to people who were in contact with chicken-pox patient: Separation and limit of contacts with others Vaccination Use of antibiotics Disinfection E. 669. A. B. C. * D. E. 670. A. * B. C. D. E. 671. A. * B. C. D. E. 672. A. B. * C. D. E. 673. A. B. C. D. * E. 674. A. B. C. D. E. * 675. A. B. C. * D. E. 676. A. Does not exist What are the changes in the blood in patients with erysipelas? Changes are absent Limfomonocitosis, increase of ESR Neutrophilic leycositosis, increase of ESR Leycopeniya, increase of ESR Increase of ESR What are the main exciters of erysipelas. Streptococcus Staphylococcus Spirochetes Gonococcus Stick of Lefler What clinical form of erysipelas is in this patient? Eritematozis Eritematozo-bullezis Hemorrhagic Eritematozo-hemorrhagic Necrosis What complications can be after erysipelas? Myocarditis Endocarditis Inflammation of additional bosoms of nose Otitis Festering inflammation of neck lymphonoduss What complications can be after erysipelas? Myocarditis Otitis Parafaringeal abscesses Glomerulonephritis Encephalitis What complications can be after erysipelas? Pneumonia Pseudorheumatism Illness of Reyno Rheumatoceils Sepsis What disease is this? A painful lesion with a bright red, edematous, indurate appearance and an advancing, raised border that is sharply demarcated from the adjacent normal skin. Fever is a feature. Phlegmona Anthrax Erysipelas Thrombophlebitis Eczema What does the most characteristic syndrome appear in patients with in 1 days of diseases? Edema B. C. * D. E. 677. A. B. C. * D. E. 678. A. B. C. * D. E. 679. A. B. C. D. E. * 680. A. B. C. D. E. * 681. A. B. C. D. E. * 682. A. B. C. D. E. * 683. A. B. C. D. E. * Hyperemia Pain Stomach-ache Takhikardiya What does we use for the prophylaxis of reinfection of erysipelas? Incoming immunoproteins Incoming of small doses of hormones Incoming bicillin 1 per a month Incoming a vaccine Warning of skin lesion What does we use for the prophylaxis of reinfection of erysipelas? Incoming immunoproteins Incoming of small doses of hormones Incoming bicillin 1 per a month Incoming a vaccine Warning of skin lesion What examination should be performed to confirm the diagnosis of toxoplasmosis in pregnant women? Bacterioscopy of blood Biopsy materrial from lymph nodes, muscle or tissues Immunofluorescence test Burne test ELISA-test What examination should be performed to confirm the diagnosis of toxoplasmosis? Bacterioscopy of blood Biopsy materrial from lymph nodes, muscle or tissues Immunofluorescence test Burne test ELISA-test What examination should be performed to confirm the diagnosis of toxoplasmosis in pregnant women? Bacterioscopy of blood Biopsy materrial from lymph nodes, muscle or tissues Immunofluorescence test Burne test ELISA-test What examination should be performed to confirm the diagnosis of toxoplasmosis? Biological test Biopsy of lymph nodes Bacteriological test Burne‘s test ELISA-test What examination should be performed to confirm the diagnosis of toxoplasmosis in pregnant women? Biological test Biopsy of lymph nodes Bacteriological test Bacterioscopy of blood ELISA-test 684. A. B. * C. D. E. 685. A. B. C. D. E. * 686. A. * B. C. D. E. 687. A. B. C. * D. E. 688. A. B. C. * D. E. 689. A. B. C. D. * E. 690. A. * B. C. D. E. 691. A. B. * C. What from the following symptoms are not characteristic of infectious mononucleosis? Fever Defeat of kidneys Lymphadenopathy Tonsillitis Increasing of liver and spleen What is duration of contagious period for a patient with epidemic parotitis? 21 days First week of illness First 10 days from the beginning of disease Whole period of clinical symptoms First 9 days of disease. What is duration period of supervision after ill with scarlet fever? 7 days from time of contact 21 day Till patient’s rash is present Till patient is discharged from permanent establishment Not conducted What is the base prophylactic measures of erysipelas relapse? Administration of antistreptococcus immunoglobulin Usage of small dosage of hormones Bicillin prophylaxis once in a month Usage of vaccine Prevention of skin trauma and angina development What is the base prophylactic measures of erysipelas relapse? Administration of antistreptococcus immunoglobulin Usage of small dosage of hormones Bicillin prophylaxis once in a month Usage of vaccine Prevention of skin trauma and angina development What is the duration of contagious period for a patient diagnosed with scarlet fever? 10 days from the beginning of illness Until patient is discharged from the hospital Until rash is present Till the 22d day from the beginning of illness Not contagious What is the main mechanism of transmission of a erysipelas? Airborne Alimentary Contact Transmisiv Vertical What is the main mechanism of transmission of a toxoplasmosis? Airborne Fecal-oral Contact D. E. 692. A. B. C. * D. E. 693. A. B. C. * D. E. 694. A. B. * C. D. E. 695. A. B. * C. D. E. 696. A. B. C. * D. E. 697. A. B. C. D. E. * 698. A. B. C. D. * E. Transmisiv All above it What is the prevention of erysipelas recurrence? Introduction of antystreptococcus immunoglobulin The use of small doses of prednizolon Bicillin prophylaxis Serum Prevention of traumatization of skin and angina What is the prevention of recurrence of erysipelas? Introduction Ig The use hormones Bicylin-5 in a month Vaccine Nothing What kind of erysipelas do your now: Catarrhal, follicle and lacunars Catarrhal, erythemat-bulos, erythemat-hemoragic and bulos-hemoragic Catarrhal, erythemat-bulos, erythemat-hemoragic and necrotizing-ulcerous Catarrhal, erythemat-bulos, erythemat-hemoragic, bulos-hemoragic and necrotizing-ulcerous Catarrhal, erythemat-hemoragic and bulos-hemoragic What kind of erysipelas do your now: Catarrhal, erythemat-bulos, and bulos-hemoragic Catarrhal, erythemat-bulos, erythemat-hemoragic and bulos-hemoragic Catarrhal, erythemat-hemoragic and necrotizing-ulcerous Erythemat-bulos, erythemat-hemoragic, bulos-hemoragic and necrotizing-ulcerous Catarrhal, erythemat-hemoragic and bulos-hemoragic What kind of lymphonoduss are increased in patients with a erysipelas? All lymphonoduss Nothing Regionall Submandibular Anterolateral neck What measures should be taken in regards to persons, who were in contact with a patient diagnosed with epidemic parotitis? Observation after contact people during a maximal length of incubation period Quorantin in child's establishment Isolation of people who were in contact with ill from 11th to the 21t day of illness Isolation of children up to 10 years old, who were not ill with epidemic parotitis, for 21 day from a moment of contact All enumerated What the most possible complication occurs during infectious mononucleosis? Meningitis autoimmune alopecia encephalitis Splenic rupture Obstruction of respiratory tract 699. A. B. C. D. E. * 700. A. B. * C. D. E. 701. A. B. * C. D. E. 702. A. B. * C. D. E. 703. A. B. C. D. * E. 704. A. B. * C. D. E. 705. A. B. * C. D. E. 706. A. B. * C. Which antibiotic is more expedient in patient with erysipelas with benzylpenitsylin allergy? Ampicillin Amoxicillin Cefazolin Amokciklav Ciprofloxacin Which antibiotics are use as etiological treatment of toxoplasmsis: Penicillin Antyprotozoyni Cephalosporins Aminoglycosides Sulfonamides Which antibiotics are use as etiological treatment of pregnant women with toxoplasmosis: Penicillin Metronidasol Cephtriacson Amikacin Sulfanilamid Which antibiotics are use as etiological treatment of pregnant women toxoplasmsis: Aminohinol Spiramicin Fansydar Cephtriacson Sulfonamides Which antibiotics are use as etiological treatment of toxoplasmosis, except: Aminohinol Spiramicin Fansydar Ceftriaxon Sulfonamides Which antibiotics are use as etiological treatment of toxoplasmosis: Augmentin Tinidasol Cephtriaxon Avelox Nifuroxazid Which antibiotics are use as etiological treatment of pregnant women with toxoplasmosis: Penicillin Metronidasol Cephazolin Cyprofloxacin Biseptol Which antibiotics are use as etiological treatment of pregnant women toxoplasmosis: Avelox Spiramicin Fansydar D. E. 707. A. B. C. D. E. * 708. A. B. C. D. E. * 709. A. * B. C. D. E. 710. A. * B. C. D. E. 711. A. B. * C. D. E. 712. A. B. * C. D. E. 713. A. B. C. D. * E. 714. A. Cephtriacson Biseptol Which complications occurs in erysipelas? Sepsis Septical endomiocarditis Nephritis Trombophlebitis All the above Which complications occurs in erysipelas? Sepsis Septical endomiocarditis Nephritis Trombophlebitis All the above Which drags are use as etiological treatment of pregnant women toxoplasmosis: Immunoglobulin specific Immunoglobulin normal Interferon Imunophan Sulfonamides Which drags are use as etiological treatment of pregnant women toxoplasmosis: Immunoglobulin specific Serum Intron Imunophan Vaccine Which drags are use as etiological treatment of pregnant women toxoplasmosis, except: Immunoglobulin specific human Intron A Protephlazid Metronidasol Rovacid Which of antibiotics are use as etiological treatment of toxoplasmosis: Penicillin Metronidasol Cephazolin Cyprofloxacin Nifuroxazid Which of antibiotics are use as etiological treatment of toxoplasmosis, except: Metronidasol Spiramicin Fansydar Avelox Tinidasol Which of antibiotics are used as etiological treatment of toxoplasmsis: Penicillin B. * C. D. E. 715. A. B. * C. D. E. 716. A. B. * C. D. E. 717. A. B. C. D. * E. 718. A. B. * C. D. E. 719. A. * B. C. D. E. 720. A. B. * C. D. E. 721. A. B. C. * D. E. Antyprotozoyni Cephalosporins Aminoglycosides Sulfonamides Which of antibiotics are used as etiological treatment of pregnant women with toxoplasmosis: Penicillin Metronidasol Cephtriacson Amikacin Sulfanilamid Which of antibiotics are used as etiological treatment of pregnant women toxoplasmsis: Aminohinol Spiramicin Fansydar Cephtriacson Sulfonamides Which of antibiotics are used as etiological treatment of toxoplasmosis, except: Aminohinol Spiramicin Fansydar Cephtriacson Sulfonamides Which of drags are use as etiological treatment of pregnant women toxoplasmosis, except: Immunoglobulin specific Imunophan Protephlazid Metronidasol Rovamicini Which of drags are used as etiological treatment of pregnant women toxoplasmosis: Immunoglobulin specific Immunoglobulin normal Interferon Imunophan Sulfonamides Which of drags are used as etiological treatment of pregnant women toxoplasmosis, except: Immunoglobulin specific Imunophan Protephlazid Metronidasol Rovamicini Which patients with appropriate wildfire appoint prednisolone? In the primary form Repeated wildfire Often recurrent erysipelas If there are complications All patients 722. A. B. C. * D. E. 723. A. B. C. D. E. * 724. A. * B. C. D. E. 725. A. B. C. D. E. * 726. A. B. C. D. E. * 727. A. B. C. D. E. * 728. A. B. C. D. E. * 729. A. B. C. * Which patients with erysipelas assign appropriate prednisolone? Buloz-haemorrhagic form With concomitant tromboflebyt Often recurrent erysipelas Since phenomena limfostasis All patients Who is the source of erysipelas? Sick man with erysipelas Sick man with angina Sick man with scarlet fever Sick man with pneumonia All the above Who is the source of erysipelas? Sick man and carriers Rodents Soil Insects Cattle. Who is the source of erysipelas? Sick man with erysipelas Sick man with angina Sick man with scarlet fever Sick man with pneumonia All the above Who is the source of toxoplasmosis? Sick man and carriers Rodents Soil Insects Cattle Who is the source of toxoplasmosis? Sick man and carriers Rodents Soil Insects Cat Who is the source of toxoplasmosis? Sick man and carriers Rodents Soil Insects Pig An initial period at the hemorrhagic fever with a kidneys syndrome lasts: Few hours Day To three days D. E. 730. A. * B. C. D. E. 731. A. B. C. D. E. * 732. A. B. C. * D. E. 733. A. B. C. * D. E. 734. A. B. C. D. * E. 735. A. B. C. D. E. * 736. A. B. C. D. E. * 737. A. Week Two weeks At an objective examination for the Congo hemorrhagic fever character: Mucosal hyperemia of person Pallor of person Puffiness of person Ochrodermia of person Exanthema on face At I degree of dehydration the loss of liquid is: 0,5-1,5 % of body weight 6-9 % of body weight 3-6 % of body weight 5-8 % of body weight 1-3 % of body weight At what percent of fluid loss will be I degree of dehydration? -6 % of body weight 6-9 % of body weight 1-3 % of body weight 0,5-2 % of body weight 2-7 % of body weight At what percent of fluid loss will be II degree of dehydration? 3-6 % of body weight Over 10 % of body weight 6-9 % of body weight 4-8 % of body weight 10-15 % of body weight At what percentage of fluid loss will be IV degree of dehydration? 4-8 % of body weight 6-9 % of body weight 3-6 % of body weight Over 10 % of body weight Over 15 % of body weight Confirm the diagnosis of Congo hemorrhagic fever by a way of: virological methods bacteriological methods Bacteriological and serum methods epidemiological information Virologic and serum methods Confirm the diagnosis of Congo hemorrhagic fever by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Virologic and serum methods Confirm the diagnosis of Ebola fever by a way of: Growth of viruses on chicken embryos B. C. D. E. * 738. A. B. C. D. E. * 739. A. B. C. D. E. * 740. A. B. C. D. E. * 741. A. B. C. * D. E. 742. A. B. * C. D. E. 743. A. B. C. * D. E. 744. A. B. C. * D. E. Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Selection of virus on the Vero culture Confirm the diagnosis of hemorrhagic fever with kidneys syndrome by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Virologic and serum methods Confirm the diagnosis of Lassa hemorrhagic fever by a way of: Only virological methods Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Virologic and serum methods Confirm the diagnosis of Omsk fever by a way of: Growth of virus on chicken embryons Only bacteriological methods Bacteriological and serum methods Proper epidemiological information Selection of virus on the Vero culture Delagilum appoint at the malaria in such doses: 0,5 g 3 per a day 3 days 0,5 g per a week In the first day 1 g, through 6 h 0,5 g 0,5 g per a day during a month 0,5 g 2 per a day 3 days Especially dangerous for surroundings are patients with: Skin form of plague Pulmonary form of plague Skin-bubonic form of plague Bubonic form of plague Septic form of plague For a skin form of anthrax the most characteristically: Hyperemia Painful carbuncle Not painful carbuncle Painful noodles Vesicles and bulls For anthrax carbuncle the most characteristically: Ulcer with a festering bottom, roller on periphery and insignificant area of edema Ulcer with hyperemia on periphery without an edema Ulcer with a black scab, black color, second vesicles and area of edema around of ulcer Ulcer with a festering bottom, roller on periphery, second vesicles and area of edema Ulcer with serosis-hemorrhagic exudates, painful, with the area of edema around of ulcer 745. A. B. C. D. E. * 746. A. B. C. * D. E. 747. A. B. C. * D. E. 748. A. B. C. D. * E. 749. A. * B. C. D. E. 750. A. B. C. D. E. * 751. A. B. C. D. * E. 752. A. B. * C. For anthrax most characteristically: Change of stool Icterus of skin Catarrhal phenomena Meningeal phenomena Change of skin For confirmation of diagnosis of hemorrhagic fever with a kidney syndrome use: Bacteriological method Virological method Reaction of immunofluorescence Reaction of braking of hemagglutination Research of blood drop under a microscope For confirmation of yellow fever diagnosis use: Bacteriological analysis of blood Bacteriological examination of urine Virological hemanalysis Biochemical blood test Global analysis of blood For the initial period of the Congo hemorrhagic fever not characteristic: Fever Pains in joints and muscles Severe pain of head Oliguria Dizziness For the pulmonary form of anthrax characteristically: Foamy sputum with blood Glassy sputum with blood Foamy sputum without blood Foamy green sputum Like to «ferruginous» sputum For treatment of anthrax us: Sulfanilamids Nitrofurans Hormones Antiviral facilities Antibiotics For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use: Corticosteroids Anabolic steroids Disintoxication facilities Dehydration facilities Antihistaminics General view of patient with the hemorrhagic fever with a kidneys syndrome: Skinning covers Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk Hyperemia of person, scleritis, conjunctivitis D. E. 753. A. B. C. * D. E. 754. A. * B. C. D. E. 755. A. * B. C. D. E. 756. A. B. * C. D. E. 757. A. B. C. * D. E. 758. A. B. * C. D. E. 759. A. B. C. * D. E. 760. Grayish color of person Icteric color of skin Hemograme in the second period of yellow fever present: Leukocytosis Normal global analysis of blood Leukopenia, neutropenia Leukopenia, neutrophilosis Leukocytosis, lymphomonocytosis How long the rash is present in case of hemorrhagic fever with kidneys syndrome? During all feverish period Before the convalescence Before development of clinical features of kidneys insufficiency During whole disease Appears yet in a latent period and disappears in the period of early reconvalescense How many pandemics of plague were in history of mankind? Three Four One Two Five In a survey of donor blood found microhametosis. Assign treatment. Delagil Primaquine Antibiotics Fluoroquinolone sulfanilamide In the biochemical blood test at patients with the hemorrhagic fever with a kidneys syndrome not characteristic: High level of urea Decline of potassium level Bilirubinemia Increasing of kreatinine Increasing of nitrogen In the global analysis of blood in case of Congo hemorrhagic fever not characteristic: Leukocytosis Leukopenia Neutropenia Thrombocytopenia Increasing of ESR In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is: High temperatures Positive Pasternatsky’ symptom Pains in joints and positive Pasternatsky’ symptom Hemorragic syndrome Dyspepsia phenomena In which clinical forms of tularemia is valid the intracutaneous test? A. B. C. D. E. * 761. A. * B. C. D. E. 762. A. B. * C. D. E. 763. A. * B. C. D. E. 764. A. B. C. D. E. * 765. A. B. C. D. E. * 766. A. B. * C. D. E. 767. A. B. * C. D. Pulmonic Bubonic Eye-bubonic Ulcer-bubonic In all the clinical forms Indications for the appointment of hematoshizotrop antimalarial drugs: Attack of malaria Preventing late relapse Prophylactic course after returning from areas difficult to malaria Antyretsidiv course for the rekonvalescents During a check-up Indications for the appointment of histoshizotrop antimalarial drugs: Attack of malaria Preventing late relapse Complications of malaria Malaria chemoprophylaxis for a period of stay in endemic areas During a check-up It is necessary to appoint for successful treatment of anthrax: Antyanthrax immunoglobulin and penicillin Antyanthrax immunoglobulin and prednizolon Antyanthrax immunoglobulin and vyrolex Antyanthrax immunoglobulin and vermox Antyanthrax immunoglobulin and delagil Mechanism of transmission of anthrax are: Contact Alimentary Air-droplets’ Transmissiv All above it Patient V., 36 yrs. old has bubonic form of plague. What treatment do you suggest? Local antibiotic therapy Intravenous antibiotic therapy Desintoxication therapy Vitamins All the above Preparations for urgent prophylaxis of plague: Injection of human immunoglobulin Streptomycin or tetracycline Human immunoglobulin Dry living vaccine or tetracycline generations. Interferon Preparations for urgent prophylaxis of plague: Injection of human immunoglobulin Streptomycin or tetracycline Human immunoglobulin Dry living vaccine or tetracycline generations. E. 768. A. B. * C. D. E. 769. A. B. * C. D. E. 770. A. B. * C. D. E. 771. A. B. C. D. * E. 772. A. * B. C. D. E. 773. A. B. C. * D. E. 774. A. * B. C. D. E. 775. A. * Interferon Preparations for urgent prophylaxis of plague: Injection of human immunoglobulin Streptomycin or tetracycline Human immunoglobulin Dry living vaccine or tetracycline generations. Interferon Risk group of plague infection the most frequent is: Doctors Hunters Alcoholic Drug users Prostitutes Specify the measures of urgent prophylaxis of anthrax. Anti-anthrax immunoglobulin Penicillinum or tetracyclinum during 5 days Vaccination Medical supervision Biseptolum 5 days Symptom of Stefansky – it is: Enantema on a soft palate Enantema on a conjunctiva Shaking of tongue at an attempt to put out a tongue Shaking of edema like to jelly at pattering a hammer in the area of edema Painful of stomach in a right iliac area The alimentary mechanisms of the tularemia infection of the humans are the following: Due to water, food, straw and other substrata contaminated by the discharge of the animals sick with tularemia Contact with sick or dead rodents and hares Bites of the infected blood-sucking arthropods By means of a contact with sick animals During the belated agricultural work The basic clinical display of a skin form of anthrax is: Hyperemic of skins Vesiculs Ulcer Phlegmon Abscess The causative agent of plague is: Yersinia pestis Yersinia enterocolitica Yersinia pseudotuberculosis Bac. anthracis Pseudomonas mallei The causative agent of plague is: Yersinia pestis B. C. D. E. 776. A. B. * C. D. E. 777. A. B. C. D. E. * 778. A. * B. C. D. E. 779. A. B. C. * D. E. 780. A. B. * C. D. E. 781. A. B. C. D. E. * 782. A. B. C. D. Yersinia enterocolitica Yersinia pseudotuberculosis Bac. anthracis Pseudomonas mallei The contact mechanisms of the tularemia infection of the humans are the following: Due to water, food, straw and other substrata contaminated by the discharge of the animals sick with tularemia Contact with sick or dead rodents and hares Bites of the infected blood-sucking arthropods By means of a contact with sick man During the belated agricultural work The diagnosis of malaria can confirm: Microscopy of urine Hemoculture Bacteriology of stool The response action Parazitoscopy of blood The diagnostic endermic reaction of anthrax take: Antraksin Dizenterin Ornitin Malein Brucellin The diagnostic reaction of anthrax is: Rayt‘s reaction Vidal‘s reaction Reaction of term precipitation of Askoly Paul-Bunnel‘s reaction Reaction of agglutination-lysis The duration of incubation period at plague is: 2-6 h 2-6 d 10-15 d 17-21 d 1-6 w The duration of incubation period of plague is: 3 to 8 days; 2 to 12 days; 2 to 10 days; 1 to 8 days. 2 to 6 days; The duration of incubation period of plague is: 3 to 8 days; 2 to 12 days; 2 to 10 days; 1 to 8 days. E. * 783. A. B. C. * D. E. 784. A. B. C. * D. E. 785. A. B. C. * D. E. 786. A. B. C. * D. E. 787. A. * B. C. D. E. 788. A. * B. C. D. E. 789. A. B. C. D. * E. 790. A. B. 2 to 6 days; The etiological factor of anthrax is: Salmonella thyphi Erysipelothrix rhysiopothiac Bacillus anthracis Rickettsiosis sibirica Toxocara canis The main antibiotics, which used for tularemia treatment. Penicillines Vaccinotherapy Aminoglycosides Cephalosporines Methronidazol The measures of urgent prophylaxis of plague. Administration of human immunoglobulin Chlorochin (delagil) 0,25 g 2 times in week 6-day’s prophylaxis with streptomycin or tetracycline In first 5 days intake antibiotics of penicillin or tetracycline origin Іnterferon The measures of urgent prophylaxis of plague. Administration of human immunoglobulin Chlorochin (delagil) 0,25 g 2 times in week 6-day’s prophylaxis with streptomycin or tetracycline In first 5 days intake antibiotics of penicillin or tetracycline origin Іnterferon The most characteristic symptom in the climax period of the Congo hemorrhagic fever is: Hemorrhagic syndrome Hepatic insufficiency Dyspepsia phenomena Sharp kidney insufficiency Мeningeal syndrome The period of polyuria at haemorrhagic fever with kidneys syndrome is a sign of: Recovering Chronic process Unfavorable flow of illness Development of complications Complete convalescence The radical course of treatment of malaria includes: Five-day therapy of delagilum Prymahin during 2 weeks Delagilum + prymahin + fansydar Delagilum + prymahin Delagilum + fansydar The rules of hospitalization of patients with plague: To separate ward To ward for respiratory infections C. * D. E. 791. A. B. C. * D. E. 792. A. B. C. * D. E. 793. A. B. * C. D. E. 794. A. B. C. * D. E. 795. A. B. C. D. E. * 796. A. * B. C. D. E. 797. A. * B. To ward cubicle Patient’s are not hospitalized To ward for intestinal infections The source of infection of anthrax is more frequent than all: People Birds Home animals Rodents Fly The transmissional mechanisms of the tularemia infection of the humans are the following: Due to water, food, straw and other substrata contaminated by the discharge of the animals sick with tularemia Contact with sick or dead rodents and hares Bites of the infected blood-sucking arthropods By means of a contact with sick animals During the belated agricultural work The usual treatment for tularemia of streptomycin sulfate is 0,5 to 1 g injected once per day for 7 to 14 days or until the patient has been without fever for five to seven days. 1 to 2 g injected once per day for 7 to 14 days or until the patient has been without fever for five to seven days. 2 to 3 g injected once per day for 7 to 10 days or until the patient has been without fever for five to seven days. 2 to 3 g injected once per day for 5 to 7 days or until the patient has been without fever for five to seven days. 1 to 3 g injected once per day for 7 to 14. There are etiotropic drags of malaria, except: Delagilum Prymahin Cerasyn Quinine Fansidar There can be several main variants of the tularemia bubo outcome: Complete dissolving Suppuration Ulceration with the following scarring Sclerotization All the above To the internal-disseminated forms of plague belong: Primary-septic Bubonic Secondary-pulmonary Primary-pulmonary Intestinal To the internal-disseminated forms of plague belong: Primary-septic Bubonic C. D. E. 798. A. B. C. D. E. * 799. A. B. C. D. E. * 800. A. B. C. * D. E. 801. A. B. C. * D. E. 802. A. B. C. D. E. * 803. A. B. C. D. E. * 804. A. B. C. D. E. * 805. Secondary-pulmonary Primary-pulmonary Intestinal To the localized forms of plague belong: Intestinal Primary-septic Secondary-septic Primary-pulmonary Skin-bubonic To the localized forms of plague belong: Intestinal Primary-septic Secondary-septic Primary-pulmonary Bubonic To the localized forms of plague belong: Secondary-septic Primary-septic Skin Primary-pulmonary Intestinal To the localized forms of plague belong: Secondary-septic Primary-septic Skin Primary-pulmonary Intestinal To the localized forms of plague belong: Intestinal Primary-septic Secondary-septic Primary-pulmonary Skin-bubonic To the localized forms of plague belong: Intestinal Primary-septic Secondary-septic Primary-pulmonary Bubonic Tularemia is transmitted by: Air Food Contact Transmission All the above Tularemia may also be transmitted by such mechanisms except A. B. C. * D. E. 806. A. B. C. D. * E. 807. A. B. C. D. E. * 808. A. B. C. * D. E. 809. A. B. C. D. * E. 810. A. * B. C. D. E. 811. A. * B. C. D. E. 812. A. B. C. * D. Alimentary Transmissional Vertical Respirational Contct What anthrax prophylactic measures are entertained by farm workers? Vitamin therapy Immunization by inactivated vaccine Formulated vaccine Immunization by live vaccine Antibiotic therapy What changes in biochemical blood test inherent for hemorrhagic fever with kidneys syndrome? Increase level of urea and bilirubin The level of urea and kreatinine falls The level of kreatinine grows and urea falls The level of urea grows and kreatinine falls The level of urea and kreatinine increase What changes in blood analysis inherent at Congo hemorrhagic fever? Normochromic anaemia, leucocytosis mononuclear Erythrocytosis, lymphocytosis Hypochromic anemia, erythrophilosis Hypochromic anemia, neutrophilosis Hyperchromic anemia, neutrophilosis What changes in blood analysis inherent for hemorrhagic fever with kidneys syndrome? Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia increased ESR erythrocytosis, lymphocytosis,ESR is increased Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia increased ESR Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia increased ESR Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR What clinical form are not characterized for a rabbit-fever? Erytem Bubonic Eye-bubonic Ulcer-bubonic Pulmonic What complication is meet at the yellow fever: Liver insufficiency Kidney insufficiency Infectious-toxic shock Myocarditis Edema of lungs What does include a specific prophylaxis at a rabbit-fever? Tetracyclin Doxycyclin Vaccination by a living attenuated vaccine Bakteriofag E. 813. A. B. C. * D. E. 814. A. B. * C. D. E. 815. A. B. * C. D. E. 816. A. B. C. D. * E. 817. A. B. C. D. * E. 818. A. * B. C. D. E. 819. A. B. C. * D. E. 820. A. B. Vaccination by a chemical attenuated vaccine What drug did use for the treatment of small pox? Merapenem Flukonazol Specifically immunoglobulin Vitamin K Levomicetyn What drug did use for the treatment of plague? Amoxicillin Streptomycin Penicillin Biseptol 5-NOK What drug is first step of choice for the treatment of plague? Amoxicillin Streptomycin Penicillin Biseptol 5-NOK What drug use as the drug of choice to treat tularemia? Use kanamycin as the drug of choice. Use cyproay as the drug of choice. Use cephazolyn as the drug of choice. Use streptomycin as the drug of choice. Use levomicetyn as the drug of choice. What form of plague is highly fatal? Sylvatic; Bubonic; Septicemic; Pneumonic. Bubonic and septicemic What is the main feature of septicemic salmonelosis? Massive bacteriemia Headache Pain in the abdominal Throatache Bleeding What is the susceptibility of human to plague? Non susceptible 50 % Almost 100 % 10 % 70 % What is the susceptibility of human to plague? Non susceptible 50 % C. * D. E. 821. A. B. C. D. * E. 822. A. B. C. D. * E. 823. A. B. C. D. * E. 824. A. * B. C. D. E. 825. A. B. C. D. * E. 826. A. B. C. D. * E. 827. A. B. C. D. * E. 828. Almost 100 % 10 % 70 % What is typical for the Lassa hemorrhagic fever: Effect of cardiovascular system Development of acute hepatic insufficiency Hundred-per-cent lethality Defeat of breathing organs Development of paresis and paralysis What is typical for the Lassa hemorrhagic fever: Effect of cardiovascular system Development of acute hepatic insufficiency Hundred-per-cent lethality Defeat of breathing organs Development of paresis and paralysis What material is necessary take for diagnosis of anthrax: Spinal liquid Urine Saliva Content of carbuncle Nose swab What organ demerged more frequent than all in patients with anthrax? Skin Lights Gastrointestinal tract Lymphatic system Nervous system What rashes in case of haemorrhagic fevers with kidneys syndrome? Roseola Maculo-papular Punctuate Petechial Rashes is not characteristic What rashes present in case of Congo hemorrhagic fever? Roseola Maculo-papular Punctulate Petechial Rashes not is characteristic What rashes present in case of Crimea hemorrhagic fever? Roseola Maculo-papular Punctulate Petechial Rashes not is characteristic What specific test is used for anthrax diagnostic? A. B. C. * D. E. 829. A. B. C. * D. E. 830. A. B. C. D. E. * 831. A. B. C. * D. E. 832. A. * B. C. D. E. 833. A. B. C. D. * E. 834. A. B. C. D. * E. 835. A. * B. C. D. Compliment fixation test Indirect hemaglutination test Coetaneous test with antraxin Hemaglutination test RIFA with anthrax antigen What used for prophylaxis of small pox. Immunoglobulin Penicillinum or tetracyclinum during 5 days Vaccination Medical supervision Serum What your need for reatment of thlorohynresistent forms of malaria? Bactrimum Dapson Fansydar Meflohin It is all above enumerated What your need to give to the patients with malaria‘s comma? Glucocorticoids Antishok drags Intravenous delagilum Substitute blood transfusion Oxygen When does begin treatments of patients with a malaria? Immediately after hospitalization After taking of material for research After raising of final diagnosis After the laboratory and instrumental result All answers are faithful When there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome: In an initial period It is not It is in all periods of disease It is in climax period It is in the period of recovering Where may be find the tularemic granulloms: In liver In spleen In brain In lymphatic nodes In skin Which is preliminary diagnosis? Anginous-bubonic form of tularemia Oropharyngeal form of Diphtheria The Vensan-Simanovsky’s tonsilitis The Dyuge-Shtryumpelya’s tonsilitis E. 836. A. * B. C. D. E. 837. A. B. C. * D. E. 838. A. B. C. D. * E. 839. A. B. * C. D. E. 840. A. B. * C. D. E. 841. A. B. C. D. * E. 842. A. * B. C. D. E. 843. A. B. Cataral tonsilitis Which method are the most often used to diagnose the rabbit-fever in the people? An intracutaneous allergic test with tularin An intracutaneous allergic test with antraxin ELISA test with specifical antibody Allergic test of Byurne Test of Cuverkalov Which of microorganismes causes the tularemia: Shigella tularensis Riketsua tularensis Francisella tularensis Salmonella tularensis Clostridia tularensis Which of microorganismes causes the tularemia: Legionella Corinebacterium diphtheriae Orthomyxoviridae Francisella tularensis Leptospira Which specifical serological methods is use for tularemia diagnostics Indirect hemagglutination test Infradermic test with tularin Compliment fiction rest Agglutination reaction Hemagglutination reaction Who are the infection source of anthrax. Birds Wild animals Fly Human Rodents Who is the reservoir of causative agent of plague in nature? Birds Insects Fresh-water fish Rodents Cattle Who is the source of infection at a rabbit-fever? Cattle, rodents, hare, water-rats, rabbits Guinea-pigs Man Leeches and shellfishes Cats With appearance of hemorrhagic syndrome at Congo fever temperature of body always: Normal Grows critically C. * D. E. 844. A. B. C. D. * E. 845. A. * B. C. D. E. 846. A. B. * C. D. E. 847. A. B. C. D. * E. 848. A. B. C. * D. E. 849. A. * B. C. D. E. 850. A. B. * C. D. E. 851. Goes down Does not change Grows gradually With what diseases it is necessary to differentiate anthrax: Leptospirozis Typhoid fever Dermatitis Carbuncle Meningococcal infection Y. pestis is transmitted more frequently by: Flea Water Air Food storage Tick Your mast begins to treat patients with a plague: Immediately after hospitalization Immediately after hospitalization, carrying out only material for research After raising of final diagnosis After laboratory and instrumental diagnostics All answers are faithful When there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome: In an initial period It is not It is in all periods of disease It is in climax period It is in the period of recovering When you can stopped etiotropic medications treatment of the patient with epidemic typhus? Immediately after the normalization of body temperature After the normalization of the liver and spleen After a 2-day normal body temperature After the disappearance of roseola Within 10 days after the disappearance of roseola Y. pestis is transmitted more frequently by: Flea Water Air Food storage Tick Your mast begins to treat patients with a plague: Immediately after hospitalization Immediately after hospitalization, carrying out only material for research After raising of final diagnosis After laboratory and instrumental diagnostics All answers are faithful You must begin to treat patients with a plague: A. B. * C. D. E. Immediately after hospitalization Immediately after hospitalization, carrying out only material for research After raising of final diagnosis After laboratory and instrumental diagnostics All answers are faithful