Download ID_3859_Viral hepatitis-AIDS-Infectiou_English_sem_10

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E.
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E.
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All the following medicines are hepatoprotective agents except:
Carsil
Silibor
Legalon
Amicacinum
Arginine
All the following medicines are hepatoprotective agents except:
Essenciale
Silibor
Legalon
Atenolol
Hepadif
All the following medicines are hepatoprotective agents except:
Carsil
Silibor
Legalon
Lomusol
Hepabene
All the following medicines are interferons except:
Intron
Pegasis
Reaferon
Leukinferon
Cycloferon
All the following medicines are interferons except:
Intron
Roferon
Reaferon
Leukinferon
Ceftriaxon
All the following medicines are interferons except:
Intron
Roferon
Reaferon
Laferobion
Legalon
All the hepatitis have parenteral route of transmission except:
A
B
C
D
TTV
All the hepatitis have parenteral route of transmission except:
E
B
C
D.
E.
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D
TTV
All the hepatitis have parenteral route of transmission except:
A and E
B and D
C and B
D and C
TTV
All the hepatitis have parenteral route of transmission except:
A
B
C
D
TTV
As etiotropic therapy of acute and chronic viral hepatitis B utillize:
Corticosteroid
Immunomodulate preparations
Cytostatics
Antibiotics
Antiviral preparations
As etiotropic therapy of sharp and chronic viral hepatitis B utillize:
Corticosteroid
Immunomodulate preparations
Cytostatics
Antibiotics
Antiviral preparations
Basic principles of antiviral therapy for viral hepatitis.
Individual selection of dose and rhythm of application of preparations
Duration of introduction of preparations
Control of amount of erytrocytes, leucocytes and thrombocytes, in blood
Control of iron level in blood
All the above
Basic principles of antiviral therapy for viral hepatitis.
Individual selection of dose and rhythm of application of preparations
Duration of introduction of preparations
Control of amount of erytrocytes, leucocytes and thrombocytes, in blood
Control of iron level in blood
All the above
Before the appearance of jaundice in a patient 16 years old, during 2 days there were an increasing of
the body temperature (38,5 °C), headache, dull ache disturbed whole body. Name the variant of
pre-icteric period of viral hepatitis for this patient?
Influenza-like
Astenovegetative
Artralgic
Dyspeptic
Allergic
16.
A. *
B.
C.
D.
E.
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C.
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C.
Choose the indexes of efficiency of interferon therapy.
Disappearance| of markers of viral replication
Improvement of the general state
Normalization of the liver size
Disappearance of icterus
All the above
Choose the indexes of efficiency of interferon therapy.
Improvement of the general state
Normalization of activity of ALaT
Normalization of the liver size
Disappearance of icterus
All the above
Choose the indexes of efficiency of interferon therapy.
Disappearance| of markers of viral replication
Improvement of the general state
Normalization of the liver size
Disappearance of icterus
All the above
Choose the indexes of efficiency of interferon therapy.
Improvement of the general state
Normalization of activity of ALaT
Normalization of the liver size
Disappearance of icterus
All the above
Choose the indexes of efficiency of interferon therapy.
Improvement of the general state
Normalization of activity of ALaT
Normalization of the liver size
Disappearance of icterus
All the above
Choose the remedies for etiotropic therapy for viral hepatitis.
Antibiotics
Interferon
Probiotics
Vaccine
Normal human immunoprotein
Choose the remedies for etiotropic therapy for viral hepatitis.
Antibiotics
Interferon
Probiotics
Vaccine
Normal human immunoprotein
Choose the remedies for etiotropic therapy for viral hepatitis.
Antibiotics
Interferon
Probiotics
D.
E.
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A. *
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B.
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Vaccine
Normal human immunoprotein
Choose the remedies for etiotropic therapy of viral hepatitis.
Ribavirin
Vaccine
Normal human immunoprotein
Hepatoprotector
Glucocorticoid
Choose the remedies for etiotropic therapy of viral hepatitis.
Ribavirin
Vaccine
Normal human immunoprotein
Hepatoprotector
Glucocorticoid
Chronic course is common for viral hepatitis except:
A
B
C
D
B+C
Chronic course is common for viral hepatitis except:
A
B
C
D
B+C
Chronic course is common for viral hepatitis except:
A
B
C
D
B+C
Chronic course is common for viral hepatitis except:
A
B
C
D
B+C
Contra-indications for antiviral therapy of viral hepatitis.
Contra-indications for antiviral therapy of viral hepatitis.
ecompensatory cirrhosis of liver
Autoimmune disease
Alcoholism and other drug addictions
Coinfection by HIV
All the above
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Contra-indications for antiviral therapy of viral hepatitis.
Contra-indications for antiviral therapy of viral hepatitis.
ecompensatory cirrhosis of liver
Autoimmune disease
Alcoholism and other drug addictions
Coinfection by HIV
All the above
Diagnosed a patient: chronic hepatitis in the stage of integration. What markers will be in patient in
this stage disease?
Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing
of stercobilin of excrements. What is the type of icterus?
Haemolitic
Parenchymatous
Transport
Extraliver
Mechanical
Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing
of stercobilin of excrements. What is the type of icterus?
Haemolitic
Parenchymatous
Transport
Extraliver
Mechanical
Direct bilirubin is increased , in urine there is significant increase of bilirubin and urobilin, increasing
of stercobilin of excrements. What is the type of icterus?
Haemolitic
Parenchymatous
Transport
Extraliver
Mechanical
Etiotropic therapy of viral hepatitis is.
Ribavirin
Interferon
Inductors of interferon
Zefix
All the above.
Etiotropic therapy of viral hepatitis is.
Ribavirin
Interferon
Inductors of interferon
Zefix
All enumerated.
Factors which are indications of successful interferon therapy in HV infections are all, except.
Level of ALaT not more than 2-3 norm
Low titre of HCV after the treatment
C.
D.
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Absence of cholestasis
2th and 4th genotypes of HCV
Expressed fibrosis
Factors which are indications of successful interferon therapy in HV infections are all, except.
Level of ALaT not more than 2-3 norm
Low titre of HCV after the treatment
Absence of cholestasis
2th and 4th genotypes of HCV
Expressed fibrosis
Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical
supervision after isolation.
A medical supervision during 6 month
Biochemical inspection
Abstain from hard physical load
Temporal contra-indications for prophylactic inoculations
All the above
Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical
supervision after isolation.
Medical supervision during 6 months, periodic biochemical inspections.
Control bacteriological examinations
Full labor investigation
To continue prophylactic inoculations
Supervision is not needed
Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical
supervision after isolation.
A medical supervision during 6 month
Biochemical inspection
Abstain from hard physical load
Temporal contra-indications for prophylactic inoculations
All the above
Give recommendation for a patient in reconvalensent period of viral hepatitis during a clinical
supervision after isolation.
Medical supervision during 6 months, periodic biochemical inspections.
Control bacteriological examinations
Full labor investigation
To continue prophylactic inoculations
Supervision is not needed
Indirect action of interferon therapy are all except.
Influenza-like syndrome
Nausea
Depression
Intensification of autoimmune diseases
Progress of fibrosis
Indirect action of interferon therapy are all except.
Influenza-like syndrome
Nausea
C.
D.
E. *
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A.
B.
C.
D.
E. *
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B.
C.
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E. *
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B.
C.
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B.
C.
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B.
C.
Depression
Intensification of autoimmune diseases
Progress of fibrosis
Indirect action of interferon therapy are all except.
Influenza-like syndrome
Nausea
Depression
Intensification of autoimmune diseases
Progress of fibrosis
Indirect action of interferon therapy are all except.
Influenza-like syndrome
Nausea
Depression
Intensification of autoimmune diseases
Progress of fibrosis
Indirect action of interferon therapy.
Influenza-like syndrome
Nausea
Itching
Para-hypnosis
All the above
Indirect action of interferon therapy.
Influenza-like syndrome
Nausea
Itching
Para-hypnosis
All the above
Indirect action of interferons.
Flatulence
Diarrhea
Nausea
Depression
All the above
Indirect action of interferons.
Flatulence
Diarrhea
Nausea
Depression
All the above
On the average 15 to 30 % of all population of the planet suffer from some pathology of liver.
Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the
world there are about 2 million people with acute viral hepatitis. What % of all cases will develop
chronic form.
100 %
50 %
25 %
D. *
E.
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B.
C.
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B.
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A.
B.
10 %
1%
On the average 15 to 30 % of all population of the planet suffer from some pathology of liver.
Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the
world there are about 2 million people with acute viral hepatitis. What % of all cases will develop
chronic form.
100 %
50 %
25 %
10 %
1%
On the average 15 to 30 % of all population of the planet suffer from some pathology of liver.
Prevalence of hepatitis and cirrhosis in the European countries is about 1 % of adults. Annually in the
world there are about 2 million people with acute viral hepatitis. What % of all cases will develop
chronic form.
100 %
50 %
25 %
10 %
1%
That characteristic of a partial answer of interferon therapy are all, except.
Disappearance of markers of viral replication
Normalization of activity of ALaT is upon completion of course of therapy
Disappearance of icterus
Normalization the state of patient
Normalization of the size of liver
That characteristic of a partial answer of interferon therapy are all, except.
Disappearance of markers of viral replication
Normalization of activity of ALaT is upon completion of course of therapy
Disappearance of icterus
Normalization the state of patient
Normalization of the size of liver
That characteristic of a partial answer of interferon therapy are all, except.
Disappearance of markers of viral replication
Normalization of activity of ALaT is upon completion of course of therapy
Disappearance of icterus
Normalization the state of patient
Normalization of the size of liver
That characteristic of a partial answer of interferon therapy are all, except.
Disappearance of markers of viral replication
Normalization of activity of ALaT is upon completion of course of therapy
Disappearance of icterus
Normalization the state of patient
Normalization of the size of liver
The characteristic of an unsteady answer of interferon therapy are.
Disappearance of markers of viral replication upon completion of course of therapy
Normalization of activity of ALaT during the course of therapy
C.
D.
E. *
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A.
B.
C.
D.
E. *
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B.
C.
D.
E. *
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B.
C.
D.
E. *
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B.
C.
D.
E. *
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B.
C.
D.
E. *
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B.
C.
D.
E. *
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B.
C.
D.
E. *
69.
An origin of relapse in next 6 months
Disappearance of icterus
All the above
The characteristic of an unsteady answer of interferon therapy are.
Disappearance of markers of viral replication upon completion of course of therapy
Normalization of activity of ALaT during the course of therapy
An origin of relapse in next 6 months
Disappearance of icterus
All enumerated
The characteristic of an unsteady answer of interferon therapy are.
Disappearance of markers of viral replication upon completion of course of therapy
Normalization of activity of ALaT during the course of therapy
An origin of relapse in next 6 months
Disappearance of icterus
All the above
The criteria for application of etiotropic therapy in viral hepatitis is.
Protracted motion of HBV, HVD
Any form of HV
Biochemical activity
Presence of virus replication
All the above.
The criteria for application of etiotropic therapy for the patient with HCV.
Clinical displays are insignificant
Icterus is absent
Moderate biochemical activity
There is anti-HCV in blood
RNA of HCV +
The criteria for application of etiotropic therapy in viral hepatitis is.
Protracted motion of HBV, HVD
Any form of HV
Biochemical activity
Presence of virus replication
All the above.
The criteria for application of etiotropic therapy for the patient with HCV.
Clinical displays are insignificant
Icterus is absent
Moderate biochemical activity
There is anti-HCV in blood
RNA of HCV +
Types of answer for interferon therapy are.
Stable remission
Unsteady
Partial answer
Absence of answer
All the above
Types of answer for interferon therapy are.
A.
B.
C.
D.
E. *
70.
A.
B.
C.
D.
E. *
71.
A.
B. *
C.
D.
E.
72.
A.
B. *
C.
D.
E.
73.
A.
B. *
C.
D.
E.
74.
A. *
B.
C.
D.
E.
75.
A. *
B.
C.
D.
E.
76.
A. *
B.
C.
D.
Stable remission
Unsteady
Partial answer
Absence of answer
All the above
Types of answer for interferon therapy are.
Stable remission
Unsteady
Partial answer
Absence of answer
All the above
What group of infectious diseases hepatitis A belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis A belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis A belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis B belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis B belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis B belong to:
External covers
Intestinal
Blood
Wound
E.
77.
A. *
B.
C.
D.
E.
78.
A. *
B.
C.
D.
E.
79.
A. *
B.
C.
D.
E.
80.
A. *
B.
C.
D.
E.
81.
A. *
B.
C.
D.
E.
82.
A. *
B.
C.
D.
E.
83.
A.
B. *
C.
D.
E.
84.
A.
B. *
Transmissive
What group of infectious diseases hepatitis C belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis C belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis C belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis D belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis D belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis D belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis E belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What group of infectious diseases hepatitis E belong to:
External covers
Intestinal
C.
D.
E.
85.
A.
B. *
C.
D.
E.
86.
A. *
B.
C.
D.
E.
87.
A. *
B.
C.
D.
E.
88.
A. *
B.
C.
D.
E.
89.
A.
B. *
C.
D.
E.
90.
A.
B. *
C.
D.
E.
91.
A.
B. *
C.
D.
E.
92.
Blood
Wound
Transmissive
What group of infectious diseases hepatitis E belong to:
External covers
Intestinal
Blood
Wound
Transmissive
What is incubation period for hepatitis A:
45 days
180 days
360 days
90 days
25 days
What is incubation period for hepatitis A:
45 days
180 days
360 days
90 days
25 days
What is incubation period for hepatitis A:
45 days
180 days
360 days
90 days
25 days
What is incubation period for hepatitis B:
45 days
180 days
360 days
90 days
25 days
What is incubation period for hepatitis B:
45 days
180 days
360 days
90 days
25 days
What is incubation period for hepatitis B:
45 days
180 days
360 days
90 days
25 days
What is incubation period for hepatitis B:
A.
B. *
C.
D.
E.
93.
A. *
B.
C.
D.
E.
94.
A. *
B.
C.
D.
E.
95.
A.
B.
C.
D. *
E.
96.
A.
B.
C.
D. *
E.
97.
A.
B.
C.
D. *
E.
98.
A.
B.
C.
D. *
E.
99.
A. *
B.
C.
D.
45 days
180 days
360 days
90 days
25 days
What is mechanism of transmission in viral hepatitis C:
Contact
Transmissive
Alimentary
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis D:
Contact
Transmissive
Alimentary
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis E:
Contact
Sexual
Alimentary
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis A:
Contact
Transmissive
Vertical
Fecal-oral
Air-drop
What is mechanism of transmission in salmonelosis:
Contact
Transmissive
Alimentary
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis A:
Contact
Transmissive
Vertical
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis B:
Contact
Transmissive
Alimentary
Fecal-oral
E.
100.
A. *
B.
C.
D.
E.
101.
A. *
B.
C.
D.
E.
102.
A.
B.
C.
D. *
E.
103.
A.
B.
C. *
D.
E.
104.
A.
B.
C. *
D.
E.
105.
A.
B.
C. *
D.
E.
106.
A.
B.
C.
D.
E. *
107.
A.
B.
Air-drop
What is mechanism of transmission in viral hepatitis C:
Contact
Transmissive
Alimentary
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis D:
Contact
Transmissive
Alimentary
Fecal-oral
Air-drop
What is mechanism of transmission in viral hepatitis E:
Contact
Sexual
Alimentary
Fecal-oral
Air-drop
What is transmissive factor for hepatitis A:
Blood
Semen
Water
Air
Milk of mother
What is transmissive factor for hepatitis A:
Blood
Semen
Water
Air
Milk of mother
What is transmissive factor for hepatitis A:
Blood
Semen
Water
Air
Milk of mother
What is transmissive factor for hepatitis B:
Food
Milk
Water
Air
Milk of mother
What is transmissive factor for hepatitis B:
Food
Milk
C.
D.
E. *
108.
A.
B.
C.
D.
E. *
109.
A.
B.
C.
D. *
E.
110.
A.
B.
C.
D. *
E.
111.
A.
B.
C.
D. *
E.
112.
A.
B.
C.
D. *
E.
113.
A.
B.
C.
D. *
E.
114.
A.
B.
Water
Air
Milk of mother
What is transmissive factor for hepatitis B:
Food
Milk
Water
Air
Milk of mother
What laboratory and instrumental examinations are needed for confirming the diagnosis of viral
hepatitis.
Complete analysis of blood
Ultrasound of abdominal region
Determination of activity of aminotransferase
Determination of antigen of viruses
Duodenal probing
What laboratory and instrumental examinations are needed for confirming the diagnosis of viral
hepatitis.
Complete analysis of blood
Ultrasound of abdominal region
Determination of activity of aminotransferase
Determination of antigen of viruses
Duodenal probing
What laboratory and instrumental examinations are needed for confirming the diagnosis of viral
hepatitis.
Complete analysis of blood
Ultrasound of abdominal region
Determination of activity of aminotransferase
Determination of antigen of viruses
Duodenal probing
What laboratory and instrumental examinations are needed for confirming the diagnosis of viral
hepatitis.
Complete analysis of blood
Ultrasound of abdominal region
Determination of activity of aminotransferase
Determination of antigen of viruses
Duodenal probing
What laboratory work-up is needed for confirming the diagnosis of viral hepatitis.
Total analysis of blood
Determination of level of bilirubin
Determination of activity of aminotransferase
Determination of markers of HV in IFA
All the above
What laboratory work-up is needed for confirming the diagnosis of viral hepatitis.
Total analysis of blood
Determination of level of bilirubin
C.
D. *
E.
115.
A.
B.
C.
D. *
E.
116.
A.
B.
C.
D. *
E.
117.
A.
B.
C.
D.
E. *
118.
A.
B.
C.
D.
E. *
119.
A.
B.
C.
D.
E. *
120.
A.
B.
C.
D.
E. *
121.
A.
B. *
C.
D.
Determination of activity of aminotransferase
Determination of markers of HV in IFA
All the above
What laboratory work-up is needed for confirming the diagnosis of viral hepatitis.
Total analysis of blood
Determination of level of bilirubin
Determination of activity of aminotransferase
Determination of markers of HV in IFA
All enumerated
What laboratory work-up is needed for confirming the diagnosis of viral hepatitis.
Total analysis of blood
Determination of level of bilirubin
Determination of activity of aminotransferase
Determination of markers of HV in IFA
All the above
When is interferon therapy effective in the the patient.
Normalization of the state of patient
Normalization of activity of ALaT upon completion of course of therapy
Disappearance of icterus
Normalization of the size of liver
The markers of viral replication, are determined upon completion of course of therapy
When is interferon therapy effective in the the patient.
Normalization of the state of patient
Normalization of activity of ALaT upon completion of course of therapy
Disappearance of icterus
Normalization of the size of liver
The markers of viral replication, are determined upon completion of course of therapy
When is interferon therapy effective in the the patient.
Normalization of the state of patient
Normalization of activity of ALaT upon completion of course of therapy
Disappearance of icterus
Normalization of the size of liver
The markers of viral replication, are determined upon completion of course of therapy
When is interferon therapy effective in the the patient.
Normalization of the state of patient
Normalization of activity of ALaT upon completion of course of therapy
Disappearance of icterus
Normalization of the size of liver
The markers of viral replication, are determined upon completion of course of therapy
Combinations of loss of body mass, sweating, recurrent аpthus stomatitis and girdle herpes and
lymphadenopathy, allowed a physician to assume HIV-infection. It can be discovered at an additional
inspection:
Increased correlation of CD4/CD8 lymphocyte
Diminished correlation of CD4/CD8 lymphocyte
Change of neutrophil formula to the left
Increased correlation of T8/T4 lymphocyte
E.
122.
A. *
B.
C.
D.
E.
123.
A. *
B.
C.
D.
E.
124.
A.
B.
C.
D.
E. *
125.
A.
B.
C.
D.
E. *
126.
A.
B.
C.
D.
E.
127.
A.
B.
Diminished correlation of T8/T4 of lymphocyte
A sick entered permanent establishment with complaints about general weakness, increase of
temperature, pain in throat. Objectively: the mucus cell of retropharynx is bright red, on oral cavity
are raids of gum-blush, taken off easily, discovered enlargement of all groups of lymphnode, 2-3cm
in a diameter, dense, elastic little painful, not soldered between itself. Liver is enlarged on 3cm,
spleen – on 2cm. In blood present leucocytosis and lymphomonocytosis. What is probable diagnosis?
Infectious mononucleosis
Diphtheria
Acute leukosis
Quinsies
Adenovirus infection
A sick entered permanent establishment with complaints about general weakness, increase of
temperature, pain in throat. Objectively: the mucus cell of retropharynx is bright red, on oral cavity
are raids of gum-blush, taken off easily, discovered enlargement of all groups of lymphnode, 2-3cm
in a diameter, dense, elastic little painful, not soldered between itself. Liver is enlarged on 3cm,
spleen – on 2cm. In blood present leucocytosis and lymphomonocytosis. What is probable diagnosis?
Infectious mononucleosis
Diphtheria
Acute leukosis
Quinsies
Adenovirus infection
A youth with the catarrhal phenomena found out enlargement of axillary and submandibular
lymphatic node, hyperplasia of oral cavity with the magnificent raid of gum-blush on them as points
and spots, presence of mucus cell pouring out, increased liver and spleen size.What additional
researches must be appointed to do for diagnosis?
A test to HIV
IFA for the exposure of antibodies toHIV
IFA for the exposure of antibodies to the virus of Epstien-Bar
Analysis in the presence of mononuclear antibodies
All the above
A youth with the catarrhal phenomena found out enlargement of axillary and submandibular
lymphatic node, hyperplasia of oral cavity with the magnificent raid of gum-blush on them as points
and spots, presence of mucus cell pouring out, increased liver and spleen size.What additional
researches must be appointed to do for diagnosis?
A test to HIV
IFA for the exposure of antibodies toHIV
IFA for the exposure of antibodies to the virus of Epstien-Bar
Analysis in the presence of mononuclear antibodies
All the above
Call the groups of possible risk of HIV infection:
Only homo- and bisexual, prostitutes and other persons who conduct disorderly sexual life:
Only drug addicts who enter drugs parenterally
Only recipeint of blood, its preparations, sperm and organs
Only patients with venereal diseases and parenteral viral hepatitis and from the HIV infected mothers
All enumerated
Choose the criteria for post contact prophylaxis of HIV infection.
A medical failure during working| with the HIV |patient
Birth of child by the HIV infected | mother
C.
D.
E. *
128.
A.
B.
C.
D.
E. *
129.
A.
B.
C.
D.
E. *
130.
A.
B. *
C.
D.
E.
131.
A.
B.
C. *
D.
E.
132.
A.
B. *
C.
D.
E.
133.
A.
B.
C. *
D.
E.
134.
A.
Violence
Blood transfusion
All the above|
Choose the criteria for post contact prophylaxis of HIV infection.
A medical failure during working| with the HIV |patient
Birth of child by the HIV infected | mother
Violence
Blood transfusion
All the above|
Choose the criteria for post contact prophylaxis of HIV infection.
A medical failure during working| with the HIV |patient
Birth of child by the HIV infected | mother
Violence
Blood transfusion
All the above|
Combinations of loss of body mass, sweating, recurrent аpthus stomatitis and girdle herpes and
lymphadenopathy, allowed a physician to assume HIV-infection. It can be discovered at an additional
inspection:
Increased correlation of CD4/CD8 lymphocyte
Diminished correlation of CD4/CD8 lymphocyte
Change of neutrophil formula to the left
Increased correlation of T8/T4 lymphocyte
Diminished correlation of T8/T4 of lymphocyte
Curent treatment for HIV infection consists of highly active antiretroviral therapy. Choose the correct
combination of preparations:
1 NRTIs + 2 IP
1 NRTIs + 2 NNRTI
3 NRTIs
1 NRTIs +1 IP+ 2 NNRTI
2 NRTIs +2 IP
Curent treatment for HIV infection consists of highly active artiretroviral therapy. Choose the correct
combination of preparations:
1 NRTIs + 2 IP
2 NRTIs + 1 NNRTI
3 NRTIs
1 NRTIs + 1 IP+ 2 NNRTI
2 NRTIs + 2 IP
Curent treatment for HIV infection consists of highly active antiretroviral therapy. Choose the correct
combination of preparations:
1 NRTIs + 2 IP
1 NRTIs + 2 NNRTI
3 NRTIs
1 NRTIs +1 IP+ 2 NNRTI
2 NRTIs +2 IP
Curent treatment for HIV infection consists of highly active artiretroviral therapy. Choose the correct
combination of preparations:
1 NRTIs + 2 IP
B. *
C.
D.
E.
135.
A.
B.
C.
D. *
E.
136.
A.
B.
C.
D. *
E.
137.
A.
B.
C.
D. *
E.
138.
A.
B.
C.
D. *
E.
139.
A.
B. *
C.
D.
E.
140.
A.
B. *
C.
D.
E.
141.
A. *
B.
C.
2 NRTIs + 1 NNRTI
3 NRTIs
1 NRTIs + 1 IP+ 2 NNRTI
2 NRTIs + 2 IP
For today the effective methods of protection from HIV are:
Vaccination and immunoprotein
Chemoprophylactic
Isolation of patients
Safe sex and prevention of drug addiction
Disinfection
For today the effective methods of protection from HIV are:
Vaccination and immunoprotein
Chemoprophylactic
Isolation of patients
Safe sex and prevention of drug addiction
Disinfection
Genetic errors at replication of HIV is:
Negative
Rare
Frequently enough
Extraordinarily frequent
Virions type one genetically changeable and second – no
Genetic errors at replication of HIV is:
Negative
Rare
Frequently enough
Extraordinarily frequent
Virions type one genetically changeable and second – no
How many types of HIV are known?
One
Two
Three
Four
Five
How many types of HIV are known?
One
Two
Three
Four
Five
Immediately after a contact with blood and other biological liquids it is necessary to wash the muddy
areas of skin with water and soap and to begin a postcontact prophylaxis, antiretroviral preparations
not later than
24-36 hrs
36-48 hrs
48-60 hrs
D.
E.
142.
A. *
B.
C.
D.
E.
143.
A.
B.
C.
D. *
E.
144.
A.
B.
C.
D. *
E.
145.
A.
B.
C. *
D.
E.
146.
A.
B.
C. *
D.
E.
147.
A. *
60-72 hrs
72-86 hrs
Immediately after a contact with blood and other biological liquids it is necessary to wash the muddy
areas of skin with water and soap and to begin a postcontact prophylaxis, antiretroviral preparations
not later than
24-36 hrs
36-48 hrs
48-60 hrs
60-72 hrs
72-86 hrs
In the order of diminishing of probability of infection of HIV the transferred ways take place in such
sequence:
Sexual contact, contaminated blood transfusion, operation of tattoo, from breast milk, transplacental
transmission
Contaminated blood transfusion, transplacental transmission, sexual contact, operation of tattoo,
postnatal from mother milk
Transplatsental transmission, contaminated blood transfusion, postnatal from breast milk, sexual
contact, operation of tattoo
Contaminated blood transfusion, transplacental transmission, sexual contact, postnatal from breast
milk, operation of tattoo
Contaminated blood transfusion, sexual contact, transplacent transmission, operation of tattoo,
postnatal from breast milk
In the order of diminishing of probability of infection of HIV the transferred ways take place in such
sequence:
Sexual contact, contaminated blood transfusion, operation of tattoo, from breast milk, transplacental
transmission
Contaminated blood transfusion, transplacental transmission, sexual contact, operation of tattoo,
postnatal from mother milk
Transplatsental transmission, contaminated blood transfusion, postnatal from breast milk, sexual
contact, operation of tattoo
Contaminated blood transfusion, transplacental transmission, sexual contact, postnatal from breast
milk, operation of tattoo
Contaminated blood transfusion, sexual contact, transplacent transmission, operation of tattoo,
postnatal from breast milk
Name the main specific methods of diagnosis of HIV infection which is used in Ukraine?
RPGA
PLR
IFA and ELISA
Bioassey
RIA
Name the main specific methods of diagnosis of HIV infection which is used in Ukraine?
RPGA
PLR
IFA and ELISA
Bioassey
RIA
Name the most dangerous parenteral way of infection of HIV/AIDS?
Infusion of donor blood and its preparations
B.
C.
D.
E.
148.
A. *
B.
C.
D.
E.
149.
A. *
B.
C.
D.
E.
150.
A. *
B.
C.
D.
E.
151.
A.
B.
C.
D. *
E.
152.
A.
B.
C.
D. *
E.
153.
A.
B.
C.
D.
E. *
Transplantation of organs
Injections of medications
Diagnostic manipulations
Intravenous introduction of drugs
Name the most dangerous parenteral way of infection of HIV/AIDS?
Infusion of donor blood and its preparations
Transplantation of organs
Injections of medications
Diagnostic manipulations
Intravenous introduction of drugs
Name the source of HIV infection/AIDS?
Human
Warm-blooded animal
Poultries
Amphibious
Fishes
Name the source of HIV infection/AIDS?
Human
Warm-blooded animal
Poultries
Amphibious
Fishes
On a background of prolonged treatment for HIV patient, appeared ulcer on the mucous cell of
mouth. At an objective examination doctor have found out erosions on the mucous, hyperemic and
filling out mucus cell of oral cavity, tongue without raid, with a smooth surface. About what
complication is possible to think?
Leptospirosis
Acute herpes
Stevens-Johnson syndrome
Candidos stomatitis
Layel syndrome
On a background of prolonged treatment for HIV patient, appeared ulcer on the mucous cell of
mouth. At an objective examination doctor have found out erosions on the mucous, hyperemic and
filling out mucus cell of oral cavity, tongue without raid, with a smooth surface. About what
complication is possible to think?
Leptospirosis
Acute herpes
Stevens-Johnson syndrome
Candidos stomatitis
Layel syndrome
That is not characterised for I clinical stage of AIDS, in obedience to clinical classification of stages
of HIV-infection for adults and teenagers, which is developed by the experts of WHO (in 2002)?
Loss of mass of body 5 % from initial
Purulent defect of skin and mucus (seborrhic dermatitis, mycotic defect of nails)
An episode of herpes zoster during the last five years
Risiding of infection of upper respiratory tracts (for example, bacterial sinuitis)
Minimum defeats of mucus (recurrent ulcers of mucus shell of oral cavity)
154.
A.
B.
C.
D.
E. *
155.
A.
B.
C. *
D.
E.
156.
A.
B.
C. *
D.
E.
157.
A.
B.
C. *
D.
E.
158.
A.
B.
C.
D.
E. *
159.
A. *
B.
C.
D.
E.
160.
A. *
B.
C.
D.
That is not characterised for I clinical stage of AIDS, in obedience to clinical classification of stages
of HIV-infection for adults and teenagers, which is developed by the experts of WHO (in 2002)?
Loss of mass of body 5 % from initial
Purulent defect of skin and mucus (seborrhic dermatitis, mycotic defect of nails)
An episode of herpes zoster during the last five years
Risiding of infection of upper respiratory tracts (for example, bacterial sinuitis)
Minimum defeats of mucus (recurrent ulcers of mucus shell of oral cavity)
The basic way of transmission of exciter HIV infections/AIDS are such:
Aerogene
Alimentary
Parententeral
Through a kiss
Bite of mosquito|
The basic way of transmission of exciter HIV infections/AIDS are such:
Aerogene
Alimentary
Parententeral
Through a kiss
Bite of mosquito
The experts of WHO consider suspicious in relation to AIDS:
Increase 3 and rmore lymph nodes in two anatomical topographic groups (except for inguinal) by
sizes more than 2 cm in diameter, which lasts more than 3 months
Increase 3 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Increase 2 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than
2 cm in diameter, which lasts more than 2 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 2 months
The experts of WHO consider suspicious in relation to AIDS:
Loss of weight (3 % and more)
Loss of weight (5 % and more)
Loss of weight (6 % and more)
Loss of weight (9 % and more)
Loss of weight (10 % and more)
The experts of WHO consider suspicious in relation to AIDS:
Diarrhea, which lasts more than 1 months
Diarrhea, which lasts more than 2 months
Diarrhea, which lasts more than 3 months
Diarrhea, which lasts more than 4 months
Diarrhea, which lasts more than 5 months
The experts of WHO consider suspicious in relation to AIDS:
Hypertermia, which lasts more than 1 months
Hypertermia, which lasts more than 2 months
Hypertermia, which lasts more than 3 months
Hypertermia, which lasts more than 4 months
E.
161.
A.
B.
C.
D.
E. *
162.
A.
B.
C. *
D.
E.
163.
A.
B.
C. *
D.
E.
164.
A.
B.
C.
D.
E. *
165.
A. *
B.
C.
D.
E.
166.
A. *
B.
C.
D.
E.
167.
A.
B.
Hypertermia, which lasts more than 5 months
The experts of WHO consider suspicious in relation to AIDS:
Hypertermia, which lasts more than 1 months
Diarrhea, which lasts more than 1 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Loss of weight (10 % and more)
All the above
The experts of WHO consider suspicious in relation to AIDS:
Hypertermia, which lasts more than 21 months
Diarrhea, which lasts more than 3 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Loss of weight (6 % and more)
All the above
The experts of WHO consider suspicious in relation to AIDS:
Increase 3 and rmore lymph nodes in two anatomical topographic groups (except for inguinal) by
sizes more than 2 cm in diameter, which lasts more than 3 months
Increase 3 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Increase 2 and more lymph nodes in two anatomical groups (except for inguinal), by sizes more than
2 cm in diameter, which lasts more than 2 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 2 months
The experts of WHO consider suspicious in relation to AIDS:
Loss of weight (3 % and more)
Loss of weight (5 % and more)
Loss of weight (6 % and more)
Loss of weight (9 % and more)
Loss of weight (10 % and more)
The experts of WHO consider suspicious in relation to AIDS:
Diarrhea, which lasts more than 1 months
Diarrhea, which lasts more than 2 months
Diarrhea, which lasts more than 3 months
Diarrhea, which lasts more than 4 months
Diarrhea, which lasts more than 5 months
The experts of WHO consider suspicious in relation to AIDS:
Hypertermia, which lasts more than 1 months
Hypertermia, which lasts more than 2 months
Hypertermia, which lasts more than 3 months
Hypertermia, which lasts more than 4 months
Hypertermia, which lasts more than 5 months
The experts of WHO consider suspicious in relation to AIDS:
Hypertermia, which lasts more than 1 months
Diarrhea, which lasts more than 1 months
C.
D.
E. *
168.
A.
B.
C. *
D.
E.
169.
A.
B.
C. *
D.
E.
170.
A.
B.
C. *
D.
E.
171.
A.
B.
C. *
D.
E.
172.
A.
B.
C. *
D.
E.
173.
A.
B.
C.
D.
E. *
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Loss of weight (10 % and more)
All the above
The experts of WHO consider suspicious in relation to AIDS:
Hypertermia, which lasts more than 21 months
Diarrhea, which lasts more than 3 months
Increase 2 and more lymph nodes in two аnatomical groups (except for inguinal), by sizes more than
1 cm in diameter, which lasts more than 3 months
Loss of weight (6 % and more)
All the above
The planned inoculations for the children conduct in accordance with operating Calendar are all,
except
To tuberculosis
To poliomyelitis
To tuberculosis and salmoneliosis
To tuberculosis and diphtheria
To poliomyelitis and diphtheria
The planned inoculations for the children conduct in accordance with operating Calendar are all,
except
To tuberculosis
To poliomyelitis
To tuberculosis and poliomyelitis
To tuberculosis and diphtheria
To poliomyelitis and diphtheria
The planned inoculations of HIV infected| children| conduct in accordance with operating calendar
are all, except
To tuberculosis
To poliomyelitis
To tuberculosis and poliomyelitis
To tuberculosis and diphtheria
To poliomyelitis and diphtheria
The planned inoculations of HIV infected| children| conduct in accordance with operating calendar
are all, except
To tuberculosis
To poliomyelitis
To tuberculosis and poliomyelitis
To tuberculosis and diphtheria
To poliomyelitis and diphtheria
The sick grumbles about the prolonged cough, more than half-year, rising temperature of body to 38
°C, enlargement of peripheral lymphnode, frequent herpetic wide-spread pouring out with
considerable lowering of body mass. In іmmunogram correlation of T-helper to T-suppression is 0,3.
It takes place because infestant:
Infects cells with the receptors of CD22
Induces proliferation of Т-helpers
Induces proliferation of T-suppressors
Infects cells from receptor CD8
Infects cells from receptor CD4
174.
A.
B.
C.
D.
E. *
175.
A.
B.
C.
D. *
E.
176.
A.
B.
C.
D. *
E.
177.
A.
B. *
C.
D.
E.
178.
A.
B. *
C.
D.
E.
179.
A.
B. *
C.
D.
E.
180.
A.
B. *
C.
D.
E.
181.
The sick grumbles about the prolonged cough, more than half-year, rising temperature of body to 38
°C, enlargement of peripheral lymphnode, frequent herpetic wide-spread pouring out with
considerable lowering of body mass. In іmmunogram correlation of T-helper to T-suppression is 0,3.
It takes place because infestant:
Infects cells with the receptors of CD22
Induces proliferation of Т-helpers
Induces proliferation of T-suppressors
Infects cells from receptor CD8
Infects cells from receptor CD4
To what cells of blood the human immunodeficiency virus has most affected:
Thrombocytes
T-suppressor-cell
Т-killer
T-helper
Leucocytes
To what cells of blood the human immunodeficiency virus has most affected:
Thrombocytes
T-suppressor-cell
Т-killer
T-helper
Leucocytes
Violation of what stage of cell cycle does HIV predetermine lamivudin?
Penetration of HIV into cells
Reverse transcription
Integration
Transcription
Translation
Violation of what stage of cell cycle does HIV predetermine stavudin?
Penetration of HIV in cells
Reverse transcription
Integration
Transcription
Translation
Violation of what stage of cell cycle does HIV predetermine lamivudin?
Penetration of HIV into cells
Reverse transcription
Integration
Transcription
Translation
Violation of what stage of cell cycle does HIV predetermine stavudin?
Penetration of HIV in cells
Reverse transcription
Integration
Transcription
Translation
Violation of what stage of life cycle does HIV predetermine by Zidovudin?
A.
B. *
C.
D.
E.
182.
A.
B. *
C.
D.
E.
183.
A.
B.
C.
D. *
E.
184.
A.
B.
C.
D.
E. *
185.
A.
B.
C.
D. *
E.
186.
A.
B.
C.
D. *
E.
187.
A.
B.
C.
D.
E. *
188.
A.
B.
C.
D. *
Penetration of HIV in cells
Reverse transcription
Integration
Transcription
Translation
Violation of what stage of life cycle does HIV predetermine by Zidovudin?
Penetration of HIV in cells
Reverse transcription
Integration
Transcription
Translation
What additional inspections must be conducted on patient with infectious mononucleosis?
Reaction of Byurne and Rayta Khaddl'sona.
IFA on HIV and test on rabbit-fever.
Bacterioscopy on diphtheria and typhoid.
IFA on HIV and bacterioscopy on diphtheria.
Reaction of Paulya-Bunnelya and puncture of lymphatic node.
What additional inspections must be conducted on patient with infectious mononucleosis?
Reaction of Byurne and Rayta Khaddl'sona
IFA on HIV and test on rabbit-fever
Bacterioscopy on diphtheria and typhoid
Reaction of Paulya-Bunnelya and puncture of lymphatic node
Ig M and IgG to infectious mononucleosis
What additional inspections must be conducted on patient with infectious mononucleosis?
Reaction of Paulya-Bunnelya
IFA on HIV and test on rabbit-fever
Bacterioscopy on diphtheria and typhoid
IFA on HCV
Reaction of Byurne and puncture of lymphatic node
What additional inspections must be conducted on patient with infectious mononucleosis?
Reaction of Byurne and Rayta Khaddl'sona.
IFA on HIV and test on rabbit-fever.
Bacterioscopy on diphtheria and typhoid.
IFA on HIV and bacterioscopy on diphtheria.
Reaction of Paulya-Bunnelya and puncture of lymphatic node.
What additional inspections must be conducted on patient with infectious mononucleosis?
Reaction of Byurne and Rayta Khaddl'sona
IFA on HIV and test on rabbit-fever
Bacterioscopy on diphtheria and typhoid
Reaction of Paulya-Bunnelya and puncture of lymphatic node
Ig M and IgG to infectious mononucleosis
What additional inspections must be conducted on patient with infectious mononucleosis?
Reaction of Paulya-Bunnelya
IFA on HIV and test on rabbit-fever
Bacterioscopy on diphtheria and typhoid
IFA on HCV
E.
189.
A. *
B.
C.
D.
E.
190.
A. *
B.
C.
D.
E.
191.
A.
B.
C.
D.
E. *
192.
A.
B.
C.
D.
E. *
193.
A.
B.
C. *
D.
E.
194.
A.
B.
C. *
D.
E.
195.
A. *
B.
C.
D.
E.
196.
Reaction of Byurne and puncture of lymphatic node
What antiretroviral preparation is taken as post contact prophylaxis after a contact with blood and
other biological liquids?
Azidotimidin
Nevirapin
Indinavir
Saqvinavir
Ifavirent
What antiretroviral preparation is taken as post contact prophylaxis after a contact with blood and
other biological liquids?
Azidotimidin
Nevirapin
Indinavir
Saqvinavir
Ifavirent
What cell of human body can HIV get into?
Red corpuscles
Neutrophilic leucocytes
Monocyte
T-lymphocte-killer
T-cell helper
What cell of human body can HIV get into?
Red corpuscles
Neutrophilic leucocytes
Monocyte
T-lymphocte-killer
T-cell helper
What cells are main target for HIV?
T-suppressor-cell
Т-kіller
Т-helper
D-cells
0-cells
What cells are main target for HIV?
T-suppressor-cell
Т-kіller
Т-helper
D-cells
0-cells
What cellular receptors of man can HIV stick to?
CD4
CD8
CD95
CD40
CD3
What cellular receptors of man can HIV stick to?
A. *
B.
C.
D.
E.
197.
A. *
B.
C.
D.
E.
198.
A. *
B.
C.
D.
E.
199.
A. *
B.
C.
D.
E.
200.
A. *
B.
C.
D.
E.
201.
A. *
B.
C.
D.
E.
202.
A.
B.
C. *
D.
E.
203.
A.
B.
CD4
CD8
CD95
CD40
CD3
What clinical features of Kaposhi sarcoma in patients with AIDS?
Will strike the persons of young and middle age
Primary elements appear on a head and trunk
Pouring out with necrosis and ulceration
A sarcoma metastasis | in internal and marked high lethality
All adopted features
What clinical features of Kaposhi sarcoma in patients with AIDS?
Will strike the persons of young and middle age
Primary elements appear on a head and trunk
Pouring out with necrosis and ulceration
A sarcoma metastasis | in internal and marked high lethality
All adopted features
What disease is occupied by the second place after frequency of the first defects at AIDS?
Sarcoma Kaposhi's
Pneumocystis pneumonia
Meningitis
Encephalitis
Lymphadenomas of cerebrum
What disease is occupied by the second place after frequency of the first defects at AIDS?
Sarcoma Kaposhi's
Pneumocystis pneumonia
Meningitis
Encephalitis
Lymphadenomas of cerebrum
What disease is occupied by the second place after frequency of the first defects at AIDS?
Sarcoma Kaposhi's
Pneumocystis pneumonia
Meningitis
Encephalitis
Lymphadenomas of cerebrum
What dose of prophylaxis conducted by antiretroviral preparation after a contact with blood and other
biological liquids?
600-800 mg/day
700-800 mg/day
800-1000 mg/day
1000-1100 mg/day
1100-1200 mg/day
What dose of prophylaxis conducted by antiretroviral preparation after a contact with blood and other
biological liquids?
600-800 mg/day
700-800 mg/day
C. *
D.
E.
204.
A.
B.
C.
D.
E. *
205.
A.
B.
C.
D.
E. *
206.
A.
B.
C. *
D.
E.
207.
A.
B.
C. *
D.
E.
208.
A.
B.
C.
D. *
E.
209.
A.
B.
C.
D. *
E.
210.
A. *
B.
C.
D.
800-1000 mg/day
1000-1100 mg/day
1100-1200 mg/day
What factors does influence on the level of CD4?
Analytical variant
Seasonal diseases pathema
Some accidental diseases
Treatment with corticosteroid
All the above
What factors does influence on the level of CD4?
Analytical variant
Seasonal diseases pathema
Some accidental diseases
Treatment with corticosteroid
All the above
?what family of viruses does an exciter of HIV/AIDS belong to?
Orto- and paramyxovirus
Rabdovirus
Retrovirus
Herpesvirus
Reovirus
what family of viruses does an exciter of HIV/AIDS belong to?
Orto- and paramyxovirus
Rabdovirus
Retrovirus
Herpesvirus
Reovirus
What group of infectious diseases an exciter of HIV infection/AIDS belong to by L. Gromashevskij
classification?
Intestinal infection
Infections of respiratory tract
Blood infection
Infection of external covers
Behave to all indicated groups
What group of infectious diseases an exciter of HIV infection/AIDS belong to by L. Gromashevskij
classification?
Intestinal infection
Infections of respiratory tract
Blood infection
Infection of external covers
Behave to all indicated groups
What group of infectious diseases, does an exciter of HIV/AIDS belong to?
Antroponozis
Zoonosis
Sapronosis
Saprozoonozis
E.
211.
A. *
B.
C.
D.
E.
212.
A. *
B.
C.
D.
E.
213.
A. *
B.
C.
D.
E.
214.
A.
B.
C. *
D.
E.
215.
A.
B.
C. *
D.
E.
216.
A.
B.
C.
D. *
E.
Zooantroponozis
What group of infectious diseases, does an exciter of HIV/AIDS belong to?
Antroponozis
Zoonosis
Sapronosis
Saprozoonozis
Zooantroponozis
What immunological changes will be found out for a patient with HIV infection?
There is polyclonality B-сells activating, the amount of autoantibodies and immune complexes is
increased
There is polyclonality B-сells activating, the amount of autoantibodies and immune complexes
diminishes
There is polyclonality B-сells of oppression, the amount of autoantibodies and immune complexes
diminishes
There is polyclonality B-сells of oppression, the amount of autoantibodies and immune complexes is
increased
There is polyclonality B-сells activating, the amount| of autoantibodies and immune complexes is
increased
What immunological changes will be found out for a patient with HIV infection?
There is polyclonality V-klitinna activating, the amount of autoantibodies and immune complexes is
increased
There is polyclonality V-klitinna activating, the amount of autoantibodies and immune complexes
diminishes
There is polyclonality V-klitinne of oppression, the amount of autoantibodies and immune complexes
diminishes
There is polyclonality V-klitinne of oppression, the amount of autoantibodies and immune complexes
is increased
There is polyclonality V-klitinna activating, the amount| of autoantibodies and immune complexes is
increased
What is the genome named that HIV plugged in the genome of cell of owner?
Supervirus
Provirus
Particle of DNA
Retrovirus
Coxavirus
What is the genome named that HIV plugged in the genome of cell of owner?
Supervirus
Provirus
Particle of DNA
Retrovirus
Coxavirus
What is the level of functional ability of patient at Ist clinical stage of AIDS?
Decreased level of everyday activity
Enhanceable level of everyday activity
Variable level of everyday activity
Normal level of everyday activity
All the above|
217.
A.
B.
C.
D. *
E.
218.
A.
B.
C.
D. *
E.
219.
A.
B.
C.
D. *
E.
220.
A.
B.
C.
D. *
E.
221.
A.
B. *
C.
D.
E.
222.
A.
B. *
C.
D.
E.
223.
A.
B. *
C.
D.
E.
224.
A. *
What is the level of functional ability of patient at Ist clinical stage of AIDS?
Decreased level of everyday activity
Enhanceable level of everyday activity
Variable level of everyday activity
Normal level of everyday activity
All the above|
What is the level of functional ability of patient at Ist clinical stage of AIDS?
Decreased level of everyday activity
Enhanceable level of everyday activity
Variable level of everyday activity
Normal level of everyday activity
All the above|
What is the time of prophylaxis conducted for antiretroviral preparation after a contact with blood
and other biological liquids?
During 1 wk
During 2 wk
During 3 wk
During 4 wk
During 5 wk
What is the time of prophylaxis conducted for antiretroviral preparation after a contact with blood
and other biological liquids?
During 1 wk
During 2 wk
During 3 wk
During 4 wk
During 5 wk
What is the underlaid immunodeficiency at HIV infection?
Making progress diminishing amount of CD8 cell
Making progress diminishing amount of CD4 cell
Scaling down the amount of CD4 cell
Scaling down the amount of CD8 cell
Diminishing amount of CD4 to CD8 cell
What is the underlaid immunodeficiency at HIV infection?
Making progress diminishing amount of CD8 cell
Making progress diminishing amount of CD4 cell
Scaling down the amount of CD4 cell
Scaling down the amount of CD8 cell
Diminishing amount of CD4 to CD8 cell
What is the underlaid immunodeficiency at HIV infection?
Making progress diminishing amount of CD8 cell
Making progress diminishing amount of CD4 cell
Scaling down the amount of CD4 cell
Scaling down the amount of CD8 cell
Diminishing amount of CD4 to CD8 cell
What laboratory signs are characteristic of AIDS-associative complex?
Lowering of content of immunoproteins A and G
B.
C.
D.
E.
225.
A. *
B.
C.
D.
E.
226.
A. *
B.
C.
D.
E.
227.
A. *
B.
C.
D.
E.
228.
A.
B. *
C.
D.
E.
229.
A.
B. *
C.
D.
E.
230.
A. *
B.
C.
D.
E.
231.
A. *
B.
C.
Diminishing of correlation of CD4/CD8 below 1.0
Anaemia
Leycopenia
Increase level of circulatory immune complexes
What laboratory signs are characteristic of AIDS-associative complex?
Lowering of content of immunoproteins A and G
Diminishing of correlation of CD4/CD8 below 1.0
Anaemia
Leycopenia
Increase level of circulatory immune complexes
What medical professions carry the most potential threat of infection?
Surgical and laboratory specialities, who contact with blood
Therapeutic specialities
Epidemiologists
Sociologist
Teachers of medical establishments
What medical professions carry the most potential threat of infection?
Surgical and laboratory specialities, who contact with blood
Therapeutic specialities
Epidemiologists
Sociologist
Teachers of medical establishments
What sexual contact are the most dangerous for infection with HIV?
Vaginal
Anal
Oral
Lesbian
Artificial impregnation
What sexual contact are the most dangerous for infection with HIV?
Vaginal
Anal
Oral
Lesbian
Artificial impregnation
What аntiretroviral preparation is conduct as prophylaxis after contact with blood and other
biological liquids?
Zidovudin
Viramin
Fortovaze
Saqvinavir
Lopinavir
What аntiretroviral preparation is conduct as prophylaxis after contact with blood and other
biological liquids?
Zidovudin
Viramin
Fortovaze
D.
E.
232.
A.
B.
C. *
D.
E.
233.
A.
B.
C. *
D.
E.
234.
A. *
B.
C.
D.
E.
235.
A. *
B.
C.
D.
E.
236.
A.
B.
C.
D. *
E.
237.
A.
B.
C.
D. *
E.
238.
A.
B.
C.
D. *
E.
Saqvinavir
Lopinavir
When do the plan caesarian section of HIV infected pregnant conducted with the purpose for
decreasing of the risk of infecting the fetus?
In 36 weeks
In 37 weeks
In 38 weeks
In 39 weeks
In 40 weeks
When do the plan caesarian section of HIV infected pregnant conducted with the purpose for
decreasing of the risk of infecting the fetus?
In 36 weeks
In 37 weeks
In 38 weeks
In 39 weeks
In 40 weeks
When does begin antiviral therapy for infant of HIV-infected women?
In the first 8-12 hr after birth
From 24-36 hr after birth
Does not conduct
From a month
After diagnosis of AIDS
When does begin antiviral therapy for infant of HIV-infected women?
In the first 8-12 hr after birth
From 24-36 hr after birth
Does not conduct
From a month
After diagnosis of AIDS
When is possible to diagnose AIDS?
Only the content of CD4 less than 500 in 1 microlitre of blood
Only the conten of CD4 less than 400 in 1 microlitre of blood
Only the content of CD4 less than 300 in 1 microlitre of blood
Only the content of CD4 less than 200 in 1 microolitre of blood
Only the content of CD4 less than 100 in 1 microolitre of blood
When is possible to diagnose AIDS?
Only the content of CD4 less than 500 in 1 microlitre of blood
Only the conten of CD4 less than 400 in 1 microlitre of blood
Only the content of CD4 less than 300 in 1 microlitre of blood
Only the content of CD4 less than 200 in 1 microolitre of blood
Only the content of CD4 less than 100 in 1 microolitre of blood
When is possible to diagnose AIDS?
Only the content of CD4 less than 500 in 1 microlitre of blood
Only the conten of CD4 less than 400 in 1 microlitre of blood
Only the content of CD4 less than 300 in 1 microlitre of blood
Only the content of CD4 less than 200 in 1 microolitre of blood
Only the content of CD4 less than 100 in 1 microolitre of blood
239.
A.
B.
C. *
D.
E.
240.
A.
B.
C. *
D.
E.
241.
A.
B. *
C.
D.
E.
242.
A.
B. *
C.
D.
E.
243.
A.
B.
C.
D.
E. *
244.
A.
B.
C.
D.
E. *
245.
A.
B.
C.
D.
E. *
With the help of what molecules which is included in composition protien, a virus firmly contacts
with the molecule of CD4:
gp 100
gp 110
gp 120
gp 130
gp 140
With the help of what molecules which is included in composition protien, a virus firmly contacts
with the molecule of CD4:
gp 100
gp 110
gp 120
gp 130
gp 140
A patient concerned about attacks of fever, which are repeated every third day. There are jaundice of
sclera and skin, hepatosplenomegaly. Which of the following diagnosis is most likely?
Sepsis
Malaria
viral hepatitis
Hemolytic anemia
Leptospirosis
a patient concerned about attacks of fever, which are repeated every third day. There are jaundice of
sclera and skin, hepatosplenomegaly. Which of the following diagnosis is most likely?
Sepsis
Malaria
viral hepatitis
Hemolytic anemia
Leptospirosis
Activities for contacts with import case of malaria:
Parasitoscopy of blood
The direction of the contact in the detention facility for 5 days
Chemoprophylaxis
Vaccination
Do not hold
Activities for contacts with import case of malaria:
Parasitoscopy of blood
The direction of the contact in the detention facility for 5 days
Chemoprophylaxis
Vaccination
Do not hold
Contra-indication for the application of antibiotics are all, except.
An increased sensitiveness to preparation
Severy disorders of liver
Severy disorders of kidneys
Period of pregnancy and lactation
Prolonged fever
246.
A.
B.
C.
D.
E. *
247.
A.
B.
C.
D.
E. *
248.
A.
B.
C. *
D.
E.
249.
A.
B.
C. *
D.
E.
250.
A.
B.
C.
D.
E. *
251.
A.
B.
C.
D.
E. *
252.
A.
B.
C.
D. *
E.
253.
A.
Contra-indication for the application of antibiotics are all, except.
An increased sensitiveness to preparation
Severy disorders of liver
Severy disorders of kidneys
Period of pregnancy and lactation
Prolonged fever
Contra-indication for the application of antibiotics are all, except.
An increased sensitiveness to preparation
Severy disorders of liver
Severy disorders of kidneys
Period of pregnancy and lactation
Prolonged fever
Delagil appoint at the malaria in such doses:
0,5 g 3 per a day 3 days
0,5 g per a week
In the first day 1 g, through 6 h 0,5 g
0,5 g per a day during a month
0,5 g 2 per a day 3 days
Delagil appoint at the malaria in such doses:
0,5 g 3 per a day 3 days
0,5 g per a week
In the first day 1 g, through 6 h 0,5 g
0,5 g per a day during a month
0,5 g 2 per a day 3 days
During the treatment in a hospital of malaria the latest attack was happened. What is etiotropic
treatment.
Antibiotics
Serum
Delagil
Primachin
Delagil + Primachin
During the treatment in a hospital of malaria the latest attack was happened. What is etiotropic
treatment.
Antibiotics
Serum
Delagil
Primachin
Delagil + Primachin
During what time the lice can transfer the epidemic typhus?
Up to 10 days
Up to 15 days
Up to 20 days
Up to 30 days
Up to 40 days
During what time the lice can transfer the epidemic typhus?
Up to 10 days
B.
C.
D. *
E.
254.
A.
B.
C.
D. *
E.
255.
A.
B.
C.
D. *
E.
256.
A.
B.
C.
D. *
E.
257.
A.
B.
C.
D. *
E.
258.
A.
B.
C.
D. *
E.
259.
A.
B. *
C.
D.
E.
260.
A.
B. *
C.
D.
E.
Up to 15 days
Up to 20 days
Up to 30 days
Up to 40 days
During what time the lice can transfer the epidemic typhus?
Up to 10 days
Up to 15 days
Up to 20 days
Up to 30 days
Up to 40 days
How prevent malaria infection?
Follow the rules of personal hygiene
Boiling of a water
Vaccination
Chemoprophylaxis
Heating of a food
How prevent malaria infection?
Follow the rules of personal hygiene
Boiling of a water
Vaccination
Chemoprophylaxis
Heating of a food
Immune modulator therapy of sepsis are all, except.
Ronkoleykin
Interferon
Inductors of interferon
Vaccine
Normal human immune globuline
Immune modulator therapy of sepsis are all, except.
Ronkoleykin
Interferon
Inductors of interferon
Vaccine
Normal human immune globuline
In a survey of the donor blood microhametosis was found. Assign treatment.
Delagil
Primachin
Antibiotics
Fluorochinolones
Sulfanilamides
In a survey of the donor blood microhametosis was found. Assign treatment.
Delagil
Primachin
Antibiotics
Fluorochinolones
Sulfanilamides
261.
A.
B.
C.
D.
E. *
262.
A.
B.
C.
D.
E. *
263.
A. *
B.
C.
D.
E.
264.
A. *
B.
C.
D.
E.
265.
A. *
B.
C.
D.
E.
266.
A. *
B.
C.
D.
E.
267.
A.
B. *
C.
D.
E.
In the case of head pediculosis carry out sanitation: hair cut, followed by incineration, processing
hair. What kind of preparation is used?
0,5 % solution of water emulsions karbofosa
0,5 % metilatsetofos
10 % solution of liquid neutral metilatsetofosa soap
0,5 % water emulsion dikrezilu
3 % soap RHTSG
In the case of head pediculosis carry out sanitation: hair cut, followed by incineration, processing
hair. What kind of preparation is used?
0,5 % solution of water emulsions karbofosa
0,5 % metilatsetofos
10 % solution of liquid neutral metilatsetofosa soap
0,5 % water emulsion dikrezilu
3 % soap RHTSG
In what titre will be positive reaction of agglutination in patient with epidemic typhus in droplets
Mosing test?
1:40 and above
1:80 and above
1:160 or higher
1:320 or higher
1:640 or higher
In what titre will be positive reaction of agglutination in patient with epidemic typhus in droplets
Mosing test?
1:40 and above
1:80 and above
1:160 or higher
1:320 or higher
1:640 or higher
Indications for the appointment of hematoshizotropic antimalarial drugs:
Attack of malaria
Preventing of late relapse
Prophylactic course after returning from malaria areas
Ant recidive course for the reconvalescents
During a check-up
Indications for the appointment of hematoshizotropic antimalarial drugs:
Attack of malaria
Preventing of late relapse
Prophylactic course after returning from malaria areas
Ant recidive course for the reconvalescents
During a check-up
Indications for the appointment of histoshizotropic antimalarial drugs:
Attack of malaria
Prevention of late relapse
Complications of malaria
Chemoprophylaxis during staying in endemic areas
During a check-up
268.
A.
B. *
C.
D.
E.
269.
A.
B.
C.
D.
E. *
270.
A.
B.
C.
D.
E. *
271.
A.
B. *
C.
D.
E.
272.
A.
B. *
C.
D.
E.
273.
A. *
B.
C.
D.
E.
274.
A. *
B.
C.
D.
E.
275.
A.
B.
C.
Indications for the appointment of histoshizotropic antimalarial drugs:
Attack of malaria
Prevention of late relapse
Complications of malaria
Chemoprophylaxis during staying in endemic areas
During a check-up
Malaria must be differentiated primarily with such diseases:
Cholangitis
Pyelonephritis
Sepsis
viral hepatitis
All of the above
Malaria must be differentiated primarily with such diseases:
Cholangitis
Pyelonephritis
Sepsis
viral hepatitis
All of the above
On which period the maximal symptoms of epidemic typhus disease appear?
At the incubation period
At the 1th week of illness
At the 2nd week of illness
At the 3rd week of illness
At the time of recovery
On which period the maximal symptoms of epidemic typhus disease appear?
At the incubation period
At the 1th week of illness
At the 2nd week of illness
At the 3rd week of illness
At the time of recovery
Prevention of early relapses of malaria by:
Delagil
Immunoglobulin
Antibiotics
Primachin
Glucocorticoids
Prevention of early relapses of malaria by:
Delagil
Immunoglobulin
Antibiotics
Primachin
Glucocorticoids
Prevention of late relapse of malaria by:
Delagil
Immunoglobulin
Antibiotics
D. *
E.
276.
A.
B.
C.
D. *
E.
277.
A.
B.
C.
D.
E. *
278.
A.
B.
C.
D.
E. *
279.
A.
B.
C.
D. *
E.
280.
A.
B.
C.
D. *
E.
281.
A.
B.
C. *
D.
E.
282.
A.
B.
C. *
D.
E.
283.
A.
Primachin
Glucocorticoids
Prevention of late relapse of malaria by:
Delagil
Immunoglobulin
Antibiotics
Primachin
Glucocorticoids
Principles of etiotropic therapy of sepsis.
Administration of antibiotics quick as possible
Administration of antibiotics in maximal therapeutic doses
In accordance to credible microbiological diagnosis
An account of possible of therapeutic concentration of antibiotic in field of infection
All the above
Principles of etiotropic therapy of sepsis.
Administration of antibiotics quick as possible
Administration of antibiotics in maximal therapeutic doses
In accordance to credible microbiological diagnosis
An account of possible of therapeutic concentration of antibiotic in field of infection
All the above
Radical treatment of malaria include:
5 days therapy with delagil
Primachin within 2 weeks
Glucocorticoids
Delagil + primachin
Serum therapy
Radical treatment of malaria include:
5 days therapy with delagil
Primachin within 2 weeks
Glucocorticoids
Delagil + primachin
Serum therapy
Reconvalenscents after malaria may discharged from a clinic no earlier than:
After 2 weeks to complete clinical recovery
After 2 weeks of the conclusion of a radical course of therapy
At the conclusion of a radical course of therapy with a negative result of parazitoscopy
After 3 weeks with a negative blood culture results
After 3 weeks, if the negative results of planting feces
Reconvalenscents after malaria may discharged from a clinic no earlier than:
After 2 weeks to complete clinical recovery
After 2 weeks of the conclusion of a radical course of therapy
At the conclusion of a radical course of therapy with a negative result of parazitoscopy
After 3 weeks with a negative blood culture results
After 3 weeks, if the negative results of planting feces
Remedie for malaria prevention for those who have returned from disadvantaged areas:
Human immunoglobulin
B.
C.
D. *
E.
284.
A.
B.
C.
D. *
E.
285.
A. *
B.
C.
D.
E.
286.
A. *
B.
C.
D.
E.
287.
A.
B.
C.
D. *
E.
288.
A.
B.
C.
D. *
E.
289.
A.
B.
C.
D.
E. *
290.
A.
B.
C.
D.
E. *
Interferon
6-day-prevention streptomyosin or tetracycline
Primaquine 0,027 g 14 days
All the above.
Remedie for malaria prevention for those who have returned from disadvantaged areas:
Human immunoglobulin
Interferon
6-day-prevention streptomyosin or tetracycline
Primaquine 0,027 g 14 days
All the above.
Rules of hospitalization of patients with malaria:
In separate room
In the respiratory infections department
In the Meltserovsky‘s box
Patients are not hospitalized
In the intestinal infections department
Rules of hospitalization of patients with malaria:
In separate room
In the respiratory infections department
In the Meltserovsky‘s box
Patients are not hospitalized
In the intestinal infections department
Specific complications of malaria, except:
Hemoglobinuria fever
The gap of the spleen
Malaria‘s comma
Perforation bowel
Hemolytic anaemia
Specific complications of malaria, except:
Hemoglobinuria fever
The gap of the spleen
Malaria‘s comma
Perforation bowel
Hemolytic anaemia
The diagnosis of malaria can be confirmed by :
Microscopy of urine
Hemoculture
Bacteriology of stool
Common blood analysis
Parazitoscopy of blood
The diagnosis of malaria can be confirmed by :
Microscopy of urine
Hemoculture
Bacteriology of stool
Common blood analysis
Parazitoscopy of blood
291.
A.
B.
C.
D. *
E.
292.
A.
B.
C.
D. *
E.
293.
A.
B.
C. *
D.
E.
294.
A.
B.
C. *
D.
E.
295.
A.
B.
C. *
D.
E.
296.
A.
B.
C. *
D.
E.
297.
A.
B.
C.
D.
E. *
298.
A.
B.
C.
The radical course of treatment of malaria includes:
Five-day therapy of delagilum
Prymahin during 2 weeks
Delagilum + prymahin + fansydar
Delagilum + prymahin
Delagilum + fansydar
The radical course of treatment of malaria includes:
Five-day therapy of delagilum
Prymahin during 2 weeks
Delagilum + prymahin + fansydar
Delagilum + prymahin
Delagilum + fansydar
There are etiotropic drags of malaria, except:
Delagilum
Prymahin
Cerasyn
Quinine
Fansydar
There are etiotropic drags of malaria, except:
Delagilum
Prymahin
Cerasyn
Quinine
Fansydar
What antibiotic is less effective in epidemic typhus?
Tetracyclin
Metacyclin
Levomycetin
Vibramycin
Doxycyclin
What antibiotic is less effective in epidemic typhus?
Tetracyclin
Metacyclin
Levomycetin
Vibramycin
Doxycyclin
What do you need for reatment of chlorochyn resistent forms of malaria?
Bactrimum
Dapson
Fansydar
Meflohin
It is all above enumerated
What do you need for reatment of chlorochyn resistent forms of malaria?
Bactrimum
Dapson
Fansydar
D.
E. *
299.
A.
B.
C. *
D.
E.
300.
A.
B.
C. *
D.
E.
301.
A.
B.
C.
D.
E.
302.
A. *
B.
C.
D.
E.
303.
A.
B.
C.
D.
E.
304.
A.
B.
C.
D.
E.
305.
A.
B. *
C.
D.
E.
306.
A.
Meflohin
It is all above enumerated
What do you need to give to the patients with malaria‘s coma?
Glucocorticoids
Antishok drags
Intravenously delagil
Blood transfusion
Oxygen
What do you need to give to the patients with malaria‘s coma?
Glucocorticoids
Antishok drags
Intravenously delagil
Blood transfusion
Oxygen
What group of infectious diseases epidemic typhus belong to?
Intestinal
Respiratory
External covers
Transmissive
Blood
What group of infectious diseases balantidosis belong to?
Intestinal
Respiratory
External covers
Transmissive
Blood
What group of infectious diseases malaria belong to?
Intestinal
Respiratory
External covers
Transmissive
Blood
What group of infectious diseases malaria belong to?
Intestinal
Respiratory
External covers
Transmissive
Blood
What is Brill's disease?
Vertiacal borne
Remote relapse typhus
Early relapse typhus
Re-infection rickettsia
Self nozological unit
What is Brill's disease?
Vertiacal borne
B. *
C.
D.
E.
307.
A.
B. *
C.
D.
E.
308.
A.
B.
C. *
D.
E.
309.
A.
B.
C. *
D.
E.
310.
A.
B.
C.
D.
E. *
311.
A. *
B.
C.
D.
E.
312.
A. *
B.
C.
D.
E.
313.
A.
B.
C.
D.
E. *
Remote relapse typhus
Early relapse typhus
Re-infection rickettsia
Self nozological unit
What is Brill's disease?
Vertiacal borne
Remote relapse typhus
Early relapse typhus
Re-infection rickettsia
Self nozological unit
What is diagnostic titer response agglutination test with typhus rickettsia?
1:40 and above
1:80 and above
1:160 or higher
1:320 or higher
1:640 or higher
What is diagnostic titer response agglutination test with typhus rickettsia?
1:40 and above
1:80 and above
1:160 or higher
1:320 or higher
1:640 or higher
What is not typical for epidemic typhus exanthema?
Disappearing with decreasing of temperature
Never appear a new elements
Roseola saved up to 6 days, petehii – 12
Leave a little pigmentation and poor peeling
Few elements
What is not typical for epidemic typhus exanthema?
Arise on 7-10 days of illness
Roseola-petehial kind
Localized mainly on the lateral surface of the torso and limbs flexion surfaces
It can grab hands and feet, but never on the face
Abundant
What is not typical for epidemic typhus exanthema?
Arise on 7-10 days of illness
Roseola-petehial kind
Localized mainly on the lateral surface of the torso and limbs flexion surfaces
It can grab hands and feet, but never on the face
Abundant
What is not typical for epidemic typhus exanthema?
Disappearing with decreasing of temperature
Never appear a new elements
Roseola saved up to 6 days, petehii – 12
Leave a little pigmentation and poor peeling
Few elements
314.
A.
B.
C.
D. *
E.
315.
A.
B.
C.
D. *
E.
316.
A.
B.
C. *
D.
E.
317.
A.
B.
C. *
D.
E.
318.
A.
B.
C. *
D.
E.
319.
A.
B.
C.
D. *
E.
320.
A.
B.
C.
D. *
E.
321.
A.
B.
C.
What is not typical for severe epidemic typhus in the common blood analysis blood ?
Neutrophyl leukocytosis
Hypoeozinophylia
Lymphopenia
Anemia
Increasing of ESR
What is not typical for severe epidemic typhus in the common blood analysis blood ?
Neutrophyl leukocytosis
Hypoeozinophylia
Lymphopenia
Anemia
Increasing of ESR
What is not typical for the analysis of urine in the severe epidemic typhus?
Proteinuria
Single-cylinder hyaline
Multiple granular cylinders
A small number of erythrocytes
A small number of leukocytes
What is not typical for the analysis of urine in the severe epidemic typhus?
Proteinuria
Single hyaline cylinder
Multiple granular cylinders
A small number of erythrocytes
A small number of leukocytes
What is not typical for the analysis of urine in the severe epidemic typhus?
Proteinuria
Single hyaline cylinder
Multiple granular cylinders
A small number of erythrocytes
A small number of leukocytes
?What is possible side effects at application of antibiotics are all, except.
Stomach-ache
Nausea, vomit
Diarrhea
Fever
Skin rash
What is possible side effects at application of antibiotics are all, except.
Stomach-ache
Nausea, vomit
Diarrhea
Fever
Skin rash
What is possible side effects at application of antibiotics are all, except.
Stomach-ache
Nausea, vomit
Diarrhea
D. *
E.
322.
A. *
B.
C.
D.
E.
323.
A. *
B.
C.
D.
E.
324.
A.
B.
C.
D.
E. *
325.
A.
B.
C.
D.
E. *
326.
A.
B.
C.
D.
E. *
327.
A.
B.
C.
D. *
E.
328.
A.
B.
C.
D. *
E.
Fever
Skin rash
What is prophylaxis of epidemic typhus among the members of family with pediculosis in the focus
of the disease?
Monitoring and complete sanitation of the contact persons
Chemoprophylaxis
Antibiotic therapy
Isolation of contact
Check-up
What is prophylaxis of epidemic typhus among the members of family with pediculosis in the focus
of the disease?
Monitoring and complete sanitation of the contact persons
Chemoprophylaxis
Antibiotic therapy
Isolation of contact
Check-up
what is the Basic principles of antibiotics therapy.
A selection of preparation from data of bacteriostatic
Determination of dose, method and multiple of introduction the preparation
Timeliness and definite duration of introduction input of antibiotic
Combining antibiotics between itself for enhancement of antibacterial effect
All the above
what is the basic principles of antibiotics therapy?
Selection of antibiotics after the studing of sensitiveness
Selection of antibiotic according to the clinical diagnosis
Choose the most active drug
Choose the less toxic drug
All the above
what is the Basic principles of antibiotics therapy.
A selection of preparation from data of bacteriostatic
Determination of dose, method and multiple of introduction the preparation
Timeliness and definite duration of introduction input of antibiotic
Combining antibiotics between itself for enhancement of antibacterial effect
All the above
What is the diagnosis in reaction of agglutination with rickettsiae Prowazekii 1:640.
Flu
Typhoid fever
Meningococcal infection
Epidemic typhus
Leptospirosis
What is the diagnosis in reaction of agglutination with rickettsiae Prowazekii 1:640.
Flu
Typhoid fever
Meningococcal infection
Epidemic typhus
Leptospirosis
329.
A.
B.
C. *
D.
E.
330.
A.
B. *
C.
D.
E.
331.
A.
B.
C. *
D.
E.
332.
A.
B. *
C.
D.
E.
333.
A.
B.
C. *
D.
E.
334.
A.
B.
C. *
D.
E.
335.
A.
B.
C.
D.
E. *
336.
A.
B.
C.
What is the duration of observation of the contact persons in the focus of epidemic typhus:
21 days
25 days
51 days
72 days
3 months
What is the duration of observation of the contact persons with the Brill‘s patient:
21 days
25 days
51 days
72 days
3 months
What is the duration of observation of the contact persons in the focus of epidemic typhus:
21 days
25 days
51 days
72 days
3 months
What is the duration of observation of the contact persons with the Brill‘s patient:
21 days
25 days
51 days
72 days
3 months
What is the source of epidemic typhus?
Patients with epidemic typhus
Patients with disease Brill-Zinsser
Patients with epidemic typhus and disease Brill-Zinsser
Patients with Brill-Zinsser disease and Sachs disease
Patients with epidemic typhus and abdominal typhoid
What is the source of epidemic typhus?
Patients with epidemic typhus
Patients with disease Brill-Zinsser
Patients with epidemic typhus and disease Brill-Zinsser
Patients with Brill-Zinsser disease and Sachs disease
Patients with epidemic typhus and abdominal typhoid
What measures must be taken for people who stood in contact with malaria case:
Microscopy of peripheral blood
Direction of all patients for in an isolation for 5 days
Chemical prophylaxis
Street rounds
Does not conduct
What measures must be taken for people who stood in contact with malaria case:
Microscopy of peripheral blood
Direction of all patients for in an isolation for 5 days
Chemical prophylaxis
D.
E. *
337.
A.
B.
C. *
D.
E.
338.
A.
B.
C. *
D.
E.
339.
A. *
B.
C.
D.
E.
340.
A. *
B.
C.
D.
E.
341.
A.
B.
C. *
D.
E.
342.
A. *
B.
C.
D.
E.
343.
A.
B.
C.
D.
E. *
344.
A.
Street rounds
Does not conduct
What method used for identification of malaria:
Stool culture test
Hemoculture
Microscopic assessment of blood
Byurne test
All above enumerated
What method used for identification of malaria:
Stool culture test
Hemoculture
Microscopic assessment of blood
Byurne test
All above enumerated
When does begin treatment of patients with a malaria?
Immediately after hospitalization
After taking of material for research
After raising of final diagnosis
After the laboratory and instrumental result
All answers are faithful
When does begin treatment of patients with a malaria?
Immediately after hospitalization
After taking of material for research
After raising of final diagnosis
After the laboratory and instrumental result
All answers are faithful
When patient after epidemic typhus may been discharged from a clinic?
After clinical recovering, but not earlier 12-days normal temperature
After a full clinical recovering
After clinical recovering, but not earlier 12-days after antibiotics therapy
After 12-days normal body temperature
After clinical recovering, but not earlier than the 9-day normal body temperature
When patient after epidemic typhus may been discharged from a clinic?
After clinical recovering, but not earlier 12-days normal temperature
After a full clinical recovering
After clinical recovering, but not earlier 12-days after antibiotics therapy
After 12-days normal body temperature
After clinical recovering, but not earlier than the 9-day normal body temperature
When serological diagnosis is possible in patients with epidemic typhus?
From the 1st day of illness
Fromn the 2nd day illness
From the 3-4th day illness
From 4-5th day of illness
From the 5 to 7th day of illness
When serological diagnosis is possible in patients with epidemic typhus?
From the 1st day of illness
B.
C.
D.
E. *
345.
A.
B.
C.
D. *
E.
F.
G.
H.
I.
J.
346.
A.
B.
C.
D. *
E.
F.
G.
H.
I.
J.
347.
A.
B.
C. *
D.
E.
348.
A.
B.
C. *
D.
E.
349.
A.
B.
C. *
D.
E.
Fromn the 2nd day illness
From the 3-4th day illness
From 4-5th day of illness
From the 5 to 7th day of illness
When the sick people gets epidemic typhus infection, which period is most dangerous epidemically?
Fecal-oral
Air-drop
Contact
Transmissive
Blood
Over the past 2 days, the incubation period and 2-3 days after lowering temperature
All hectic period and 2-3 days after lowering temperature
2-3 days after lowering temperature
Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature
Over the past 2 days, the incubation period and the hectic period
When the sick people gets epidemic typhus infection, which period is most dangerous epidemically?
Fecal-oral
Air-drop
Contact
Transmissive
Blood
Over the past 2 days, the incubation period and 2-3 days after lowering temperature
All hectic period and 2-3 days after lowering temperature
2-3 days after lowering temperature
Over the past 2 days, the incubation period, all febrile period and 2-3 days after lowering temperature
Over the past 2 days, the incubation period and the hectic period
When we can stop the etiotropic treatment of the patient with epidemic typhus?
Immediately after the normalization of body temperature
After the normalization of the liver and spleen sizes
After 2-days normal body temperature
After the disappearance of roseola
After 10 days disappearing of roseola
When we can stop the etiotropic treatment of the patient with epidemic typhus?
Immediately after the normalization of body temperature
After the normalization of the liver and spleen sizes
After 2-days normal body temperature
After the disappearance of roseola
After 10 days disappearing of roseola
which of the following often occur in patient with epidemic typhus (tongue‘s tremor when protrusion
that sticked on the lower teeth)?
Heller's symptom
Conjunctivitis
Govorov-Godelje symptom
Zorohovich-Chiari-Avtsyn symptom
Rosenberg‘s enantema
350.
A.
B.
C. *
D.
E.
351.
A.
B.
C.
D.
E. *
352.
A.
B.
C.
D. *
E.
353.
A.
B.
C.
D. *
E.
354.
A.
B.
C.
D.
E. *
355.
A.
B.
C.
D.
E. *
356.
A.
B.
C.
D.
E. *
which of the following often occur in patient with epidemic typhus (tongue‘s tremor when protrusion
that sticked on the lower teeth)?
Heller's symptom
Conjunctivitis
Govorov-Godelje symptom
Zorohovich-Chiari-Avtsyn symptom
Rosenberg‘s enantema
which of the following symptoms may occur in patient with epidemic typhus (petechiaes in the
mucosal soft palate)?
Heller's symptom
Conjunctivitis
Govorov-Godelje symptom
Zorohovich-Chiari-Avtsyn symptom
Rosenberg‘s enantema
which of the following symptoms may occur in patient with epidemic typhus (petechiaes with the
conjunctiva)?
Heller's symptom
Conjunctivitis
Govorov-Godelje symptom
Zorohovich-Ciari-Avtsyn symptom
Rosenberg‘s enantema
which of the following symptoms may occur in patient with epidemic typhus (petechiaes in the
conjunctiva)?
Heller's symptom
Conjunctivitis
Govorov-Godelje symptom
Zorohovich-Ciari-Avtsyn symptom
Rosenberg‘s enantema
which of the following symptoms may occur in patient with epidemic typhus (petechiaes in the
mucosal soft palate)?
Heller's symptom
Conjunctivitis
Govorov-Godelje symptom
Zorohovich-Chiari-Avtsyn symptom
Rosenberg‘s enantema
Who would be a subject for inspection on malaria?
People who recovered of malaria
Persons, returning from endemic regions of malaria
Patients with fever more than 5 days
Patients with spleenomegaly
All above enumerated
Who would be a subject for inspection on malaria?
People who recovered of malaria
Persons, returning from endemic regions of malaria
Patients with fever more than 5 days
Patients with spleenomegaly
All above enumerated
357.
A.
B. *
C.
D.
E.
358.
A.
B. *
C.
D.
E.
359.
A.
B.
C. *
D.
E.
360.
A.
B.
C. *
D.
E.
361.
A.
B.
C. *
D.
E.
362.
A.
B.
C. *
D.
E.
363.
A.
B.
C. *
D.
E.
Why early relapse in malaria may develops ?
Immune deficiency
Presence of erythrocytic shizonts
Presence of tissue shizonts
Fresh contamination
Presence of blood gamonts
Why early relapse in malaria may develops ?
Immune deficiency
Presence of erythrocytic shizonts
Presence of tissue shizonts
Fresh contamination
Presence of blood gamonts
Why late relapses in malaria may develop ?
Immune deficiency
Presence of erythrocytic shizonts
Presence of tissue shizonts
Fresh contamination
Presence of blood gamonts
Why late relapses in malaria may develop ?
Immune deficiency
Presence of erythrocytic shizonts
Presence of tissue shizonts
Fresh contamination
Presence of blood gamonts
A patient is disturbed by attacks of fever. The icterus of sclera and skins is observed,
hepatosplenomegaly is found on palpation. What is possible diagnosis accept?
Leishmaniosis
Leptospirosis
Sepsis
Malaria
Viral hepatitis
A patient is disturbed by attacks of fever. The icterus of sclera and skins is observed,
hepatosplenomegaly is found on palpation. What is possible diagnosis accept?
Leishmaniosis
Leptospirosis
Sepsis
Malaria
Viral hepatitis
A patient sharply had a chill, head pain, vomits, temperature of body rose up to 38,5 °C. Till evening
rigidity of muscles, Kernig’s symptom appeareD. Herpetic blisters are marked on mucous of lips and
nosE. Neurological symptoms is not found out expresseD. What disease will you suspect?
Herpetic encephalitis
Abscess of brain
Meningoencephalitis
Hemorrhage in a brain
Subarachnoid hemorrhage
364.
A.
B.
C. *
D.
E.
365.
A.
B.
C. *
D.
E.
366.
A.
B.
C. *
D.
E.
367.
A. *
B.
C.
D.
E.
368.
A.
B.
C.
D.
E. *
369.
A.
B.
C.
D.
E. *
370.
A.
B.
C.
D.
E. *
371.
A.
A patient sharply had a chill, head pain, vomits, temperature of body rose up to 38,5 °C. Till evening
rigidity of muscles, Kernig’s symptom appeared. Herpetic blisters are marked on mucous of lips and
nose. Neurological symptoms is not found out expressed. What disease will you suspect?
Herpetic encephalitis
Abscess of brain
Meningoencephalitis
Hemorrhage in a brain
Subarachnoid hemorrhage
Basic mechanism of transmission of Lime disease:
Air-drop
Contact
Transmissive
Fecal-oral
Vertical
Basic mechanism of transmission of Lyme disease:
Air-drop
Contact
Transmissive
Fecal-oral
Vertical
Basic vector of Lime disease:
Tick
Mosquito
Flea
Mosquito
All of bloodsucking insects
Clinical stages of Lime-borreliosis:
Common toxic
Cardiological complications
Neurological complications
Artritic
All above enumerated
Clinical stages of Lime-borreliosis:
Common toxic
Cardiological complications
Neurological complications
Artritic
All above enumerated
Duration of activity of pliers depends on:
Weather
Regional naturally-geographical terms
Kind of vector
Activity of animals in different periods of year
From all of transferred
Duration of activity of pliers depends on:
Weather
B.
C.
D.
E. *
372.
A.
B.
C.
D.
E. *
373.
A.
B.
C.
D.
E. *
374.
A.
B. *
C.
D.
E.
375.
A.
B.
C. *
D.
E.
376.
A.
B.
C. *
D.
E.
377.
A.
B.
C. *
D.
E.
378.
A.
B.
C. *
Regional naturally-geographical terms
Kind of vector
Activity of animals in different periods of year
From all of transferred
Etiological agent of meningitis are accept:
Staphylococci
Neisseria meningitides
Mycobacterium tuberculosis
Viruses
Entamoeba histolytica
?Etiological agent of meningitis are accept:
Staphylococci
Neisseria meningitides
Mycobacterium tuberculosis
Viruses
Entamoeba histolytica
Examination is delivered in a cliniC. Objectively: temperature 40,2 °C. Expressed meningeal
symptoms. Light, haptic, pain hypesthesiA. Blood: leucocytes – 5 thousand/l, ERS-19 mm/hr. A
neurolymph under high pressure, turbid, lymphocytosis, some increasing of protein, chlorides. What
Meningococcal
meningitis
is your diagnosis?
Viral meningitis
Tubercular meningitis
Staphylococcus meningitis
Pneumoccocal meningitis
For endermic reaction of leishmaniosis diagnosis is used:
Dyzenterin
Antraksin
Leishmanin
Ornitin
Brucellin
For endermic reaction of leishmaniosis diagnosis is used:
Dysenterin
Antraxin
Leishmanin
Ornitin
Brucellin
For the treatment of acidosis in viral meningoencephalitis is better to usE.
10 % chloride solution
10-20 % glucose solution
4 % sodium bicarbonate solution
Concentrated dry plasma
Albumen
For the treatment of acidosis in viral meningoencephalitis is better to usE.
10 % chloride solution
10-20 % glucose solution
4 % sodium bicarbonate solution
D.
E.
379.
A.
B.
C. *
D.
E.
380.
A.
B.
C. *
D.
E.
381.
A.
B.
C. *
D.
E.
382.
A.
B.
C. *
D.
E.
383.
A.
B.
C.
D.
E. *
384.
A.
B.
C.
D.
E. *
385.
A.
B.
C. *
D.
E.
386.
A.
Concentrated dry plasma
Albumen
For treatment of leishmaniosis is used:
Sulfanilamids
Interferons
Antibiotics
Nitrofurans
Hormones
For treatment of leishmaniosis is used:
Sulfanilamids
Interferons
Antibiotics
Nitrofurans
Hormones
How is it possible to specify the diagnosis of viral meningoencephalitis after cerebrospinal puncturE.
Some increasing of chlorides in CSF
Presence of a lot of lymphocytes in CSF
All the above
Some increasing of protein in CSF
Neurolymph under high pressure
How is it possible to specify the diagnosis of viral meningoencephalitis after cerebrospinal puncturE.
Some increasing of chlorides in CSF
Presence of a lot of lymphocytes in CSF
All the above
Some increasing of protein in CSF
Neurolymph under high pressure
How many stages of the leishmania development do you now:
3
1
5
4
2
How many stages of the leishmania development do you now:
3
1
5
4
2
How to prevent of leishmaniosis?
Follow the rules of personal hygiene
Conduct heat preparing of food
Vaccination
Do not drink raw water
Chemoprophylaxis
How to prevent development of leishmaniosis?
Follow the rules of personal hygiene
B.
C. *
D.
E.
387.
A. *
B.
C.
D.
E.
388.
A. *
B.
C.
D.
E.
389.
A. *
B.
C.
D.
E.
390.
A. *
B.
C.
D.
E.
391.
A.
B.
C.
D.
E. *
392.
A.
B.
C.
D.
E. *
393.
A. *
B.
C.
D.
E.
Conduct heat preparing of food
Vaccination
Do not drink raw water
Chemoprophylaxis
How to prevent Laim-borreliosis?
Individual protecting from ticks
Urgent antibiotic prophylaxis
Vaccination
Using of specific immunoglobuline
All above enumerated
How to prevent Laim-borreliosis?
Individual protecting from ticks
Urgent antibiotic prophylaxis
Vaccination
Using of specific immunoglobuline
All above enumerated
It is necessary to differentiate migrant erythema with:
Erysipelas
Anthrax
Chicken-pox
Herpetic infection
Phlegmon
It is necessary to differentiate migrant erythema with:
Erysipelas
Anthrax
Chicken-pox
Herpetic infection
Phlegmon
Leishmaniosis must be differentiated primarily with such disease:
Sepsis
Malaria
Influenza
Typhoid fever
All of the above
Leishmaniosis must be differentiated primarily with such disease:
Sepsis
Malaria
Influenza
Typhoid fever
All of the above
Lime disease can transmitted by ways, except for:
Air-drop
Contact
Transmissive
Fecal-oral
Alimentary
394.
A. *
B.
C.
D.
E.
395.
A. *
B.
C.
D.
E.
396.
A.
B.
C.
D. *
E.
397.
A.
B.
C.
D.
E. *
398.
A.
B. *
C.
D.
E.
399.
A. *
B.
C.
D.
E.
400.
A. *
B.
C.
D.
E.
401.
A.
B. *
C.
Lime disease can transmitted by ways, except for:
Air-drop
Contact
Transmissive
Fecal-oral
Alimentary
Mechanism of leishmaniosis transmission is:
Transmissive
Contact
Alimentary
All
Air-droplets
Mechanism of leishmaniosis transmission is:
Transmissive
Contact
Alimentary
All above it
Air-droplets
Methods of laboratory diagnostics of Laim-borreliosis:
Selection of exciter (from blood, neurolymph, synovia, skin bioptat)
Finding of borrelia antibodies (in blood, neurolymph) (IFA)
Exposure of DNA of exciter (PCR)
Darkfield microscopy
All above enumerated
Methods used for identification of sources of leishmaniosis:
Stool culture
Microscopy of tissue slades
Urine culture
Hemoculture
All above enumerated
Passing to exciters of Lime disease among pliers is carried out ways:
Transovarian
Sexual
Air-drop
Fecal-oral
Vertical
Passing to exciters of Lime disease among pliers is carried out ways:
Transovarian
Sexual
Air-drop
Fecal-oral
Vertical
Radical treatment of skin leishmaniosis include:
Interferon
Monomycin
Glucantim
D.
E.
402.
A.
B. *
C.
D.
E.
403.
A.
B.
C.
D.
E. *
404.
A.
B.
C.
D.
E. *
405.
A.
B. *
C.
D.
E.
406.
A.
B. *
C.
D.
E.
407.
A.
B.
C. *
D.
E.
408.
A.
B.
C. *
D.
E.
409.
A.
Penicillin
Tetracyclin
Radical treatment of skin leishmaniosis include:
Interferon
Monomycin
Glucantim
Penicillin
Tetracyclin
Radical treatment of visceral leishmaniosis include:
Interferon
Penicillin
Tetracyclin
Glucantim
Monomycin
Radical treatment of visceral leishmaniosis include:
Interferon
Penicillin
Tetracyclin
Glucantim
Monomycin
Rules of hospitalization of patients with leishmaniosis:
Patients were not hospitalized
In separate room
In intestinal infections department
In respiratory infections department
In carantine box
Rules of hospitalization of patients with leishmaniosis:
Patients were not hospitalized
In separate room
In intestinal infections department
In respiratory infections department
In carantine box
Source of meningoencephalitis are accept:
People
Ticks
Fish
Fleas
Mosquitoes
Source of meningoencephalitis are accept:
People
Ticks
Fish
Fleas
Mosquitoes
The basic clinical display of a skin leishmaniosis is:
Abscess
B. *
C.
D.
E.
410.
A.
B. *
C.
D.
E.
411.
A.
B.
C.
D. *
E.
412.
A.
B.
C.
D. *
E.
413.
A.
B.
C.
D. *
E.
414.
A.
B.
C.
D. *
E.
415.
A.
B. *
C.
D.
E.
416.
A. *
B.
Ulcer
Phlegmon
Vesiculs
Hyperemia
The basic clinical display of a skin leishmaniosis is:
Abscess
Ulcer
Phlegmon
Vesiculs
Hyperemia
The diagnosis of leishmaniosis can confirm after:
Microscopy of blood
Bacteriology of stool
Hemoculture
Microscopy of tissue punctates
Microscopy of urine
The diagnosis of leishmaniosis can confirm after:
Microscopy of blood
Bacteriology of stool
Hemoculture
Microscopy of tissue punctates
Microscopy of urine
The diagnostic reaction for leishmaniosis:
Paul-Bunnel‘s reaction
Rayt‘s reaction
Vidal‘s reaction
Reaction of latex agglutination
Askoli reaction of precipitation
The diagnostic reaction for leishmaniosis:
Paul-Bunnel‘s reaction
Rayt‘s reaction
Vidal‘s reaction
Reaction of latex agglutination
Reaction of term precipitation of Askol
The examination is delivered in a cliniC. Objectively: temperature 40,1 °C. Expressed meningeal
symptoms. Light, haptic, pain hypesthesiA. Blood: leucocytes – 5 thousand/l, ERS-19 mm/hr. A
neurolymph under high pressure, turbid, lymphocytosis, some increasing of protein, chlorides. What
is your diagnosis?
Meningococcal meningitis
Viral meningitis
Tubercular meningitis
Staphylococcus meningitis
Pneumoccocal meningitis
The natural reservoir of exciter in case of Lime is, except for:
Sick person
Rodents
C.
D.
E.
417.
A. *
B.
C.
D.
E.
418.
A. *
B.
C.
D.
E.
419.
A. *
B.
C.
D.
E.
420.
A.
B.
C. *
D.
E.
421.
A.
B.
C. *
D.
E.
422.
A.
B.
C. *
D.
E.
423.
A.
B.
C. *
D.
Birds
Pliers
Cattle
The natural reservoir of exciter in case of Lime is, except for:
Sick person
Rodents
Birds
Pliers
Cattle
The natural reservoir of exciter in case of Lime is, except for:
Sick person
Rodents
Birds
Pliers
Cattle
The natural reservoir of exciter in case of Lime is, except:
Sick person
Rodents
Birds
Pliers
Cattle
The patient concerned about fever, which is not periodical. Skin is pale, hepatosplenomegaly. What
diagnose is most likely accept?
Leishmaniosis
Sepsis
Malaria
Leptospirosis
Viral hepatitis
The patient concerned about fever, which is not periodical. Skin is pale, hepatosplenomegaly. What
diagnose is most likely accept?
Leishmaniosis
Sepsis
Malaria
Leptospirosis
Viral hepatitis
There are etiotropic drags for leishmaniosis treatment, except:
Monomycin
Glucantim
Quinine
Solustibozan
Solusurmin
There are etiotropic drags for leishmaniosis treatment, except:
Monomycin
Glucantim
Quinine
Solustibozan
E.
424.
A.
B.
C.
D. *
E.
425.
A.
B.
C.
D. *
E.
426.
A.
B.
C. *
D.
E.
427.
A.
B.
C. *
D.
E.
428.
A.
B.
C. *
D.
E.
429.
A.
B.
C. *
D.
E.
430.
A.
B.
C. *
D.
E.
431.
A.
B.
Solusurmin
Viral meningoencephalitis and DIC-syndrome require above all things.
Administration of diuretics
Administration of antihistamin
Administration of vitamins
Administration of analgetics
Administration of heparin
Viral meningoencephalitis and DIC-syndrome require above all things.
Administration of diuretics
Administration of antihistamin
Administration of vitamins
Administration of analgetics
Administration of heparin
What clinical forms of leishmaniosis do you know?
Ulceral and erhythematous
Papular and visceral
Visceral and skin
Ulceral and necrotic
Bubous and skin
What clinical forms of leishmaniosis do you know?
Ulceral and erhythematous
Papular and visceral
Visceral and skin
Ulceral and necrotic
Bubous and skin
What clinical forms of tick encephalitis present accept:
Meningeal
Meningoencephalitic
General
Feverish
Meningoencephalopoliemielitic
What clinical forms of tick encephalitis present accept:
Meningeal
Meningoencephalitic
General
Feverish
Meningoencephalopoliemielitic
What drugs of choice at etiotropic therapy of viral meningoencephalitis.
Penicillins
Amynoglicosides
Interferons
Phtorhinilons
Nitrofuranes
What drugs of choice at etiotropic therapy of viral meningoencephalitis.
Penicillins
Amynoglicosides
C. *
D.
E.
432.
A.
B. *
C.
D.
E.
433.
A.
B. *
C.
D.
E.
434.
A.
B.
C.
D.
E. *
435.
A.
B.
C.
D.
E. *
436.
A.
B.
C.
D. *
E.
437.
A.
B.
C.
D. *
E.
438.
A. *
B.
C.
D.
E.
439.
Interferons
Phtorhinilons
Nitrofuranes
What group of infectious diseases meningococcal infection belong to?
Intestinal
Respiratory
External covers
Blood
Transmissive
What group of infectious diseases meningococcal infection belong to?
Intestinal
Respiratory
External covers
Blood
Transmissive
What infectious diseases it is necessary differentiate visceral leishmaniosis with:
Malaria
Sepsis
Influenza
Leptospirozis
All above enumerated
What infectious diseases it is necessary differentiate visceral leishmaniosis with:
Malaria
Sepsis
Influenza
Leptospirozis
All above enumerated
What is etiological factor of antroponosis skin leishmaniosis:
L. classica
L. major
L. brasiliensis
L. tropica
L. donovani
What is etiological factor of antroponosis skin leishmaniosis:
L. classica
L. major
L. brasiliensis
L. tropica
L. donovani
What is etiological factor of visceral leishmaniosis:
L. donovani
L. major
L. brasiliensis
L. classica
L. tropica
What is etiological factor of visceral leishmaniosis:
A. *
B.
C.
D.
E.
440.
A.
B.
C.
D.
E. *
441.
A.
B.
C.
D.
E. *
442.
A.
B.
C. *
D.
E.
443.
A.
B.
C. *
D.
E.
444.
A.
B.
C. *
D.
E.
445.
A.
B.
C. *
D.
E.
446.
A.
B.
C.
D. *
L. donovani
L. major
L. brasiliensis
L. classica
L. tropica
What is etiological factor of zoonosis skin leishmaniosis:
L. donovani
L. tropica
L. classica
L. brasiliensis
L. major
What is etiological factor of zoonosis skin leishmaniosis:
L. donovani
L. tropica
L. classica
L. brasiliensis
L. major
What is etiotropic treatment of leishmaniosis.
Delagil
Antibiotics
Solusurmin
Serum
Primaquine
What is etiotropic treatment of leishmaniosis.
Delagil
Antibiotics
Solusurmin
Serum
Primaquine
What is most characteristic symptom of the initial stage of Lime-borreliosis:
Fever
Catarrhal phenomena
Migrant erythematic
Dyspepsia phenomena
Icterus
What is most characteristic symptom of the initial stage of Lime-borreliosis:
Fever
Catarrhal phenomena
Migrant erythematic
Dyspepsia phenomena
Icterus
What is most effective antibiotics in meningococcal disease:
Cephalosporines
Phtorhinolons
Aminoglicozides
Penicillines
E.
447.
A.
B.
C.
D. *
E.
448.
A.
B.
C.
D.
E. *
449.
A.
B.
C.
D.
E. *
450.
A.
B.
C.
D.
E. *
451.
A.
B.
C. *
D.
E.
452.
A.
B.
C. *
D.
E.
453.
A.
B.
C. *
D.
E.
454.
A.
B.
Macrolids
What is most effective antibiotics in meningococcal disease:
Cephalosporines
Phtorhinolons
Aminoglicozides
Penicillines
Macrolids
What is prophylactic measures for high risk group?
Immunization by inactivated vaccine
Formulated vaccine
Vitamin therapy
Antibiotic therapy
Immunization by live vaccine
What is prophylaxy of meningococcal disease for high risk group?
Immunization by inactivated vaccine
Formulated vaccine
Vitamin therapy
Antibiotic therapy
Immunization by live vaccine
What is the method of laboratory diagnostics of Laym-borreliosis:
Selection of exciter (from blood, neurolymph, synovia, skin bioptat)
Finding of borrelia antibodies (in blood, neurolymph) (IFA)
Exposure of DNA of exciter (PCR)
Darkfield microscopy
All above enumerated
What is the origin of Lime disease:
Simplest
Viruses
Borrelium
Micoplasma
Ricketsia
What is the origin of Lime disease:
Simplest
Viruses
Borrelia
Micoplasma
Rickettsia
What is the origin of Lime disease:
Simplest
Viruses
Borrelium
Micoplasma
Ricketsia
What is the origin of Lime disease:
Simplest
Viruses
C. *
D.
E.
455.
A.
B.
C.
D.
E. *
456.
A.
B.
C.
D.
E. *
457.
A.
B. *
C.
D.
E.
458.
A.
B. *
C.
D.
E.
459.
A.
B. *
C.
D.
E.
460.
A.
B. *
C.
D.
E.
461.
A.
B.
C.
D.
E. *
462.
Borrelium
Micoplasma
Ricketsia
What is the origin of visceral leishmaniosis (kala-azar):
L. classica
L. tropica
L. major
L. brasiliensis
L. donovani
What is the origin of visceral leishmaniosis (kala-azar):
L. classica
L. tropica
L. major
L. brasiliensis
L. donovani
What is used as specific prophylaxis of viral meningoencephalitis.
Immune globulin
Vaccine
Antibioticsnatoxin
Serum
Nothing
What is used as specific prophylaxis of viral meningoencephalitis.
Immune globulin
Vaccine
Antibioticsnatoxin
Serum
Nothing
What laboratory methods should be taken for diagnosis of meningoencephalitis?
Lumbar puncture
Biopsy of tissues
Urine examination
Coprogram
Serologic detection
What laboratory methods should be taken for diagnosis of meningoencephalitis?
Lumbar puncture
Biopsy of tissues
Urine examination
Coprogram
Serologic detection
What material is necessary take for leishmaniosis diagnosis:
Punctates from skin defect
Punctates from spleen
Punctates from lymph nodes
Punctates from bone brain
All above enumerated
What material is necessary take for leishmaniosis diagnosis:
A.
B.
C.
D.
E. *
463.
A.
B.
C.
D.
E. *
464.
A.
B.
C.
D.
E. *
465.
A.
B.
C. *
D.
E.
466.
A.
B.
C. *
D.
E.
467.
A.
B. *
C.
D.
E.
468.
A.
B.
C.
D. *
E.
469.
A.
B.
C.
D. *
Punctates from skin defect
Punctates from spleen
Punctates from lymph nodes
Punctates from bone brain
All above enumerated
What measures it’s necessary to perform in the focus of viral meningoencephalitis?
Bacteriological inspection of contact
Phagoprophylaxis
Supervision during 2 weeks
Chemoprophylaxis
Immunization
What measures it’s necessary to perform in the focus of viral meningoencephalitis?
Bacteriological inspection of contact
Phagoprophylaxis
Supervision during 2 weeks
Chemoprophylaxis
Immunization
What measures must be done in case of contact with leishmaniosis contact:
Prophylaxis with antibiotics
Chimioprophylaxis
Do nothing
Microscopy of tissue slades
Vaccination
What measures must be done in case of contact with leishmaniosis contact:
Prophylaxis with antibiotics
Chimioprophylaxis
Do nothing
Microscopy of tissue slades
Vaccination
What methods is used for identification of sources of leishmaniosis:
Stool culture
Microscopy of tissue slades
Urine culture
Hemoculture
All above enumerated
What organ demerged more frequent than all in patients with visceral leishmaniosis?
Skin
Lymphatic system
Nervous system
Gastrointestinal tract
Lungs
What organ demerged more frequent than all in patients with visceral leishmaniosis?
Skin
Lymphatic system
Nervous system
Gastrointestinal tract
E.
470.
A.
B. *
C.
D.
E.
471.
A.
B. *
C.
D.
E.
472.
A.
B. *
C.
D.
E.
473.
A.
B. *
C.
D.
E.
474.
A.
B.
C.
D. *
E.
475.
A.
B.
C.
D. *
E.
476.
A.
B.
C.
D.
E. *
477.
A.
B.
Lungs
What specific test is used for leishmaniosis diagnostic?
Compliment fixation test
Coetaneous allergic test
Indirect hemaglutination test
RIFA with anthrax antigen
Hemaglutination test
What specific test is used for leishmaniosis diagnostic?
Compliment fixation test
Coetaneous allergic test
Indirect hemaglutination test
RIFA with anthrax antigen
Hemaglutination test
When should start treatment of patient with leishmaniosis?
After taking of material for research
Immediately after hospitalization
All answers are correct
After clinical diagnosis
After the laboratory and instrumental investig
When should start treatment of patient with leishmaniosis?
After taking of material for research
Immediately after hospitalization
All answers are correct
After clinical diagnosis
After the laboratory and instrumental investig
Who are the source of leishmaniosis accept:
Rodents
Foxes
Human
Flies
Wolves
Who are the source of leishmaniosis accept:
Rodents
Foxes
Human
Flies
Wolves
Who would be a subject for inspection on leishmaniosis?
Convalescents after leishmaniosis
Persons, returning from endemic regions
Patients with spleenomegaly
Patients with fever more than 5 days
All above enumerated
Who would be a subject for inspection on leishmaniosis?
Convalescents after leishmaniosis
Persons, returning from endemic regions
C.
D.
E. *
478.
A.
B. *
C.
D.
E.
479.
A.
B. *
C.
D.
E.
480.
A.
B.
C. *
D.
E.
481.
A.
B.
C.
D.
E. *
482.
A.
B. *
C.
D.
E.
483.
A.
B.
C.
D.
E. *
484.
A.
B.
C. *
D.
E.
Patients with spleenomegaly
Patients with fever more than 5 days
All above enumerated
Wich of these symptoms are often present in patients with meningitis?
Profuse watery diarrhea, vomiting, dehydratation, muscular cramps
Fever, headache, stiff neck, vomiting, confusion, irritability
Headache, dry cough, algor
Abdominal pain, diarrhea, constipation, flatulence
Algor, high temperature, headache
Wich of these symptoms are often present in patients with meningitis?
Profuse watery diarrhea, vomiting, dehydratation, muscular cramps
Fever, headache, stiff neck, vomiting, confusion, irritability
Headache, dry cough, algor
Abdominal pain, diarrhea, constipation, flatulence
Algor, high temperature, headache
A kidney syndrome at haemorrhagic fever with kidneys syndrome shows up usually:
Only laboratory changes
Only on BRIDLES
By pain in lumbar area, positive Pasternatsky symptom, development of oliguria
By fever, polyuria, dyspepsia
By paradoxical ischuria
A patient has a heavy icteric form of leptospirosis. What from antibiotics is better to appoint as
etiotropic therapy?
Yunidoks
Tetracyclinum
Azitromicin
Rovamicin
Penicillin
Agglutinines at a leptospirosis arrive at a maximal titre:
On the third day of illness
On the third week of illness and later
On the fourth week of illness
On the second month of illness
To the second week of illness
All of these have an epidemic dangerous at Rabies exept:
Farm animals
Wild rodents
Domestic animals
Foxes
Reptiles
Among the listed below choose the complication of the tetanus, which is not late:
Contracture of muscles and joints
Tetanus-kifozis
Asphyxia
Asthenic syndrome
Chronic heterospecific diseases of lungs
485.
A.
B. *
C.
D.
E.
486.
A.
B.
C.
D. *
E.
487.
A.
B.
C.
D. *
E.
488.
A.
B.
C. *
D.
E.
489.
A.
B.
C.
D. *
E.
490.
A. *
B.
C.
D.
E.
491.
A. *
B.
C.
D.
E.
492.
Among the listed below choose the complication of the tetanus, which is not early:
Tracheobronchitis
Contracture of muscles and joints
Asphyxia
Myocarditis
Pneumonia
Among the listed below people who should receive an immediate prophylactic of the tetanus in form
of AC-Antitoxin and AC IP injections after trauma?
Man of 40 years, in anamnesis with 1 inoculation one year ago
Pregnant woman of 30 years, in the second half of pregnancy
Child, 7 months, instilled according to a calendar
Retire man of 57 years, who is not instilled
Child of 6 years, instilled according to a calendar
Among the listed below what preparations are not etiological for tetanus?
AC-anatoxin
Medical horse serum
Human immunoprotein
Anticonvulsant preparations
Penicillin
An initial period at the hemorrhagic fever with a kidneys syndrome lasts:
Few hours
Day
To three days
Week
Two weeks
At a leptospirosis the exposure of antibodies is considered reliable in a titre:
1:70 and anymore
1:80 and anymore
1:60 and anymore
1:100 and anymore
1:40 and anymore
At an objective review for the Congo hemorrhagic fever characteristically:
Mucosal hyperemia of person
Pallor of person
Puffiness of person
Ochrodermia of person
Exanthema on face
At patient, who acted to permanent establishment, on clinical epidemiological indexes of Lassa fever
is suspected. What from the resulted clinical indexes are not characteristic for this disease?
Hemolytic icterus
Generalized lymphadenopathy
Conjunctivitis
Ulcerous pharyngitis
Encephalopathy
At patient, who acted to permanent establishment, the especially dangerous hemorrhagic fever is
suspected. For the selection of exciter can be used all materials, except for?
A.
B. *
C.
D.
E.
493.
A. *
B.
C.
D.
E.
494.
A.
B.
C. *
D.
E.
495.
A. *
B.
C.
D.
E.
496.
A. *
B.
C.
D.
E.
497.
A.
B.
C.
D.
E. *
498.
A.
B.
C.
D.
E. *
499.
A.
B.
C.
Blood
Puncture from lymph nodes
Urines
Pleura liquid
Swab from nasopharyng
At treatment of patients with leptospirosis antibiotics consider most effective:
Penicillin
Macrolids
Cefalosporins
Ftorhinolons
Sul'fanilamids
Basic mechanism of transmission of Lime disease:
Air-drop
Contact
Transmissive
Fecal-oral
Vertical
Basic vector of Lyme disease:
Tick
Mosquito
Flea
Mosquito
All of bloodsucking insects
Citizen A., take participence in a business geologic expedition. Area is endemic according to
Laim-borreliosis. How to prevent possible infection?
Individual protecting from ticks
Urgent antibiotic prophylaxis
Vaccination
Using of specific immunoglobuline
All above enumerated
Clinical stages of Lime-borreliosis:
Common toxic
Cardiological complications
Neurological complications
Artritic
All above enumerated
Confirm diagnosis of haemorrhagic fever with kidneys syndrome by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Virologic and serum methods
Confirm the diagnosis of Congo hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
D.
E. *
500.
A.
B.
C.
D.
E. *
501.
A.
B.
C.
D.
E. *
502.
A.
B.
C.
D.
E. *
503.
A.
B.
C.
D.
E. *
504.
A.
B. *
C.
D.
E.
505.
A.
B.
C.
D.
E. *
506.
A.
B.
C. *
D.
E.
507.
Proper epidemiological information
Virologic and serum methods
Confirm the diagnosis of Ebola fever by a way of:
Growth of viruses on chicken embryons
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Selection of virus on the Vero culture
Confirm the diagnosis of Lassa hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Virologic and serum methods
Confirm the diagnosis of Marburg fever by a way of:
Growth on chicken embryos
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiologys information
Selection of virus on the Vero culture
Confirm the diagnosis of Omsk fever by a way of:
Growth of virus on chicken embryons
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Selection of virus on the Vero culture
Daily allowance doses of penicillin at treatment of leptospirosis:
2-3 million units
3-12 million units
20 million units
40 million units
Over 40 million units
Duration of activity of pliers depends on:
Weather
Regional naturally-geographical terms
Kind of vector
Activity of animals in different periods of year
From all of transferred
Duration of the latent period in case of tetanus:
1-6 hours
1-4 days
5-14 days
1-6 weeks.
1-6 months
During a walk in-field a fox attacked on a child, bit and disappear him feet and racemes of hands. An
animal disappeared. What is medical tactic.
A.
B. *
C.
D.
E.
508.
A.
B.
C.
D.
E. *
509.
A.
B.
C. *
D.
E.
510.
A.
B.
C. *
D.
E.
511.
A.
B.
C.
D. *
E.
512.
A.
B.
C.
D. *
E.
513.
A.
B.
C.
D. *
E.
514.
A. *
B.
C.
D.
To process a wound surgically
To process a wound soapy solution, to enter an antirabies immunoprotein and vaccine
Preventive antirabies vaccination
To process wounds surgically, to enter an antirabies immunoprotein and vaccine
Preventive antirabies immunoprotein
Etiotropic therapy of leptospirosis includes:
Oxyhinolons
Sulfanilamids
Nitrofurans
Antitocidns
Antibiotics
For confirmation of diagnosis of hemorragic fever with a kidney syndrome use:
Bacteriological method
Virological method
Reaction of immunofluorescence
Reaction of braking of hemagglutination
Research of blood drop under a microscope
For confirmation of yellow fever diagnosis use:
Bacteriological analysis of blood
Bacteriological examination of urine
Virological hemanalysis
Biochemical blood test
Global analysis of blood
For hydrophobia the source of infection can be a dog bite in all the cases, except for:
Patient with hydrophobia
Suspicion on hydrophobia
Vagrant
Month prior to the disease
Last 10 days before the disease
For the exciter of tetanus characteristic such properties, except:
Formation of exokinesis
Ability to propagate in anaerobic conditions
Formation of spores
Formation of gametes
Gram positive
For the initial period of the Congo hemorrhagic fever not characteristically:
Fever
Pains in joints and muscles
Severe pain of head
Oliguria
Dizziness
For the prophylaxis of leptospirosis use:
Active vaccine
Anavaccine
Toxoid
All
E.
515.
A.
B.
C.
D. *
E.
516.
A.
B. *
C.
D.
E.
517.
A.
B.
C. *
D.
E.
518.
A.
B.
C. *
D.
E.
519.
A. *
B.
C.
D.
E.
520.
A.
B. *
C.
D.
E.
521.
A.
B.
C. *
D.
E.
522.
A.
B.
Antibiotics
For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use:
Glucocorticoids
Anabolic steroid
Disintoxication facilities
Dihydration facilities
Antihistaminics
General view of patient with the hemorrhagic fever with a kidneys syndrome:
Skinning covers
Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk
Hyperemia of person, scleritis, conjunctivitis
Grayish color of person
Icteric color of skin
Hemograme in the second period of yellow fever:
Leukocytosis
Normal global analysis of blood
Leukopenia, neutropenia
Leukopenia, neutrophilosis
Leukocytosis, lymphomonocytosis
How long lasts the leptospirosis incubation period:
2 month
1-7 days
7-14 days
14-21 days
2-3 days
How long the rash is present in case of haemorrhagic fever with kidneys syndrome?
During all feverish period
Before the reconvalescense
Before development of clinical features of kidneys insufficiency
During whole disease
Appears yet in a latent period and disappears in the period of early reconvalescense
illness?
Paroxysm of fever
Paroxysm of hydrophobia
Hepatomegaly
Spleenomegaly
Presence of rash
In case of tetanus the epidemiological measures are directed on:
Elimination of the source of tetanus
Treatment of the source of tetanus
Specific prophylaxis
Medicines prophylactics
Nothing should be performed
In hydrophobia the source of the virus can be:
Wild animals
Home animals
C.
D.
E. *
523.
A.
B.
C.
D.
E. *
524.
A.
B.
C. *
D.
E.
525.
A.
B. *
C.
D.
E.
526.
A.
B.
C. *
D.
E.
527.
A.
B.
C. *
D.
E.
528.
A. *
B.
C.
D.
E.
529.
A. *
B.
C.
D.
Bats
Rodents
All the answers are correct
In most patients with Congo hemorrhagic fever temperature curve is:
Wunderlich type
Botkin type
Undulating
Intermittent
Two-humped
In the biochemical blood test at patients with the hemorrhagic fever with a kidneys syndrome not
characteristically:
High level of urea
Decline of potassium level
Bilirubinemia
Increasing of kreatinine
Increasing of nitrogen
In the global analysis of blood in case of Congo hemorrhagic fever not characteristically:
Leukocytosis
Leukopenia
Neutropenia
Thrombocytopenia
Increasing of ESR
In the global analysis of blood: leucopenia, neutropenia, increasing of ESR. Intensive therapy without
any effect and a patient died. Credible diagnosis?
Viral hepatitis
Leptospirosis
Yellow fever
Crimean fever
Malaria
In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is:
High temperatures
Pains in gastrocnemius muscles and positive Pasternatsky symptom
Pains in joints and positive Pasternatsky symptom
Hemorragic syndrome
Dyspepsia phenomena
It is necessary to differentiate migrant erythema with:
Erysipelas
Anthrax
Chicken-pox
Herpetic infection
Phlegmon
Lime disease can transmitted by ways, except for:
Air-drop
Contact
Transmissive
Fecal-oral
E.
530.
A.
B. *
C.
D.
E.
531.
A.
B.
C.
D.
E. *
532.
A.
B.
C. *
D.
E.
533.
A. *
B.
C.
D.
E.
534.
A. *
B.
C.
D.
E.
535.
A.
B. *
C.
D.
E.
536.
A.
B.
C.
D.
E. *
537.
A.
Alimentary
Mechanism of transmission in case of tetanus:
Intra muscular conduction
Pin
Insect conduction
Faecally-oral
Vertical conduction
Methods of laboratory diagnostics of Lyme-borreliosis:
Selection of exciter (from blood, neurolymph, synovia, skin bioptat)
Finding of borrelia antibodies (in blood, neurolymph) (IFA)
Exposure of DNA of exciter (PCR)
Darkfield microscopy
All enumerated
Most characteristic symptom of the initial stage of Lime-borreliosis:
Fever
Catarrhal phenomena
Migrant erythematic
Dyspepsia phenomena
Icterus
Most characteristic symptoms of the hydrophobia are:
Paroxysm of hydrophobia
Apathy and depression
Neuralgic pains on motion nervous barrels, the nearest to the place of bite
Dyspepsia disorders
Catarrhal phenomena
Passing to exciters of Lime disease among pliers is carried out ways:
Transovarian
Sexual
Air-drop
Fecal-oral
Vertical
Patient A., 25 years old, is being treated because of tetanus concerning. Choose the specific
treatment.
Antibiotics
Immune globulini
Anticonvulsant medicine
Cardiac preparations
Respiratory analeptics
Patient B., 37 years, had the rapid development of the severe tetanus after criminal abortion. What is
the medical tactic?
Anticonvulsant preparations
Revision of the uterus cavity
Analgesic therapy
Antibiotics
All answers are correct
Phage symptom in case of yellow fever is:
Pain in right iliac area
B.
C. *
D.
E.
538.
A.
B. *
C.
D.
E.
539.
A.
B. *
C.
D.
E.
540.
A. *
B.
C.
D.
E.
541.
A.
B.
C.
D. *
E.
542.
A. *
B.
C.
D.
E.
543.
A.
B.
C.
D.
E. *
544.
A.
B.
C.
D.
Enanthema on a soft palate
Replacement of tachicardia on expressed bradicardia
Hemorrhages in a conjunctiva
Yellow hands
Pregnant woman of 22.years old delivered in the ambulance. How should be treated the child from
the point of view of tetanus prophylactic?
AC-antitoxin
Anti tetanus serum
Tetanus toxoid + anti tetanus serum
Tetanus toxoid + anti tetanus serum + immunoprotein
Anti tetanus serum + immunoprotein
Rhabdovirus from an organism of the patient or animal is revealed to the flow:
Last 20 days of latent period and during all the illness
Last 7-10 days of latent period and during all the illness
Last 7-10 days of latent period
Last 7-10 days of latent period and at the beginning of illness
During all the illness
Specific prevention of Crimean-Congo haemorrhagic fever are:
Vaccine and human immunoglobulin
Serum
Serum and human immunoglobulin
Do not developed
Antibacterial drugs
Specific prevention of hemorrhagic fevers:
The live vaccine
Killed vaccine
The specific immunoglobulin
Do not developed
Polivalent vaccine
Tetanus might appear in case of:
Trauma
Mosquito bite
Usage of stranger clothes
Socialization with the sick people
Usage of the water with poor quality
Tetanus toxin consists of all units among the listed below, except:
Tetanospasmin
Tetanolysin
Exotoxin
Low-molecular fraction
Enterotoxin
The basic reservoir of rhabdovirus is:
Pisces
Reptiles
Birds
Weed-eaters
E. *
545.
A. *
B.
C.
D.
E.
546.
A.
B.
C.
D. *
E.
547.
A. *
B.
C.
D.
E.
548.
A.
B.
C.
D.
E. *
549.
A.
B.
C.
D. *
E.
550.
A.
B.
C.
D. *
E.
551.
A.
B.
C.
D.
E. *
552.
A. *
B.
Carnivores
The best terms of tetanus exciter cultivation:
Anaerobic conditions
Oxygen supply
Presence of animal albumen in nutritive medium
Low temperature
1 % peptone water
The decline of arteriotony at a leptospirosis is not caused:
By expansion of vessels under the action of toxin
Hypovolemia
By myocarditis
Hypercalcgesty
By adrenal insufficiency
?The exciter of tetanus is:
Clostridia
Escherichia
Candida
Virus
Simplest
The first symptom of prodromal period of hydrophobia is:
Cough
Nausea
Vomiting
Diarrhea
Slight swelling and erethema of the scar
The first symptom of prodromal period of hydrophobia is:
Cough
Nausea
Vomiting
Neurological pains in motion nervous barrels, the nearest to the place of bite
Diarrhea
The first symptom of prodromal period of hydrophobia is:
Cough
Nausea
Vomiting
Apathy and depression
Diarrhea
The main of antibiotics which is used in treatment of leptospirosis:
Tetracyclin
Tetraolean
Erythromycin
Streptomycin
Penicillin
The most characteristic symptom in the climax period of the Congo hemorrhagic fever is:
Hemorrhagic syndrome
Hepatic insufficiency
C.
D.
E.
553.
A. *
B.
C.
D.
E.
554.
A. *
B.
C.
D.
E.
555.
A.
B. *
C.
D.
E.
556.
A.
B.
C. *
D.
E.
557.
A.
B.
C.
D. *
E.
558.
A. *
B.
C.
D.
E.
559.
A. *
B.
C.
Dyspepsia phenomena
Sharp kidney insufficiency
Мeningeal syndrome
The natural reservoir of exciter in case of Lime is, except for:
Sick person
Rodents
Birds
Pliers
Cattle
The period of polyuria at haemorrhagic fever with kidneys syndrome is a sign of:
Recovering
Chronic process
Unfavorable flow of illness
Development of complications
Complete convalescence
The source of infection of Omsk‘s hemorrhagic fever are muskrat, water rats and other rodents. Who
are the carriers?
Bee and flea
Pliers and flea
Mosquitoes
Fly
Pliers and mosquitoes
The spores of tetanus are saved:
After boiling during 1 hour
Under act of dry air at the temperature of 115 degrees C
In soil during many years
In 1 % solution of formalin during 6 hours
All answers are correct
Those who have been in contact with sick haemorrhagic fevers, as well as those who had bite by the
ticks in endemic areas are introducing:
Specific vaccine
The specific immunoglobulin in doses of 10-15 ml vaccine
The specific immunoglobulin in doses of 10-15 ml
The specific immunoglobulin in doses 5-7,5 ml
Nothing
To what group of infections does the hydrophobia belong?
Zoonosis
Anthroponosis
Capronosis
Anthropozoonosis
Caprozoonosis
To you, as to the graduating student of medical university, is possibly to work upon termination of
studies in Crimea. What endemic ticks infections is in this territory?
Crimean hemorrhagic fever, tick encephalitis, Q-fever
Crimean hemorrhagic fever, malaria, epidemic typhus
Tick encephalitis, Ebola fever, Lassa fever, Crimean hemorrhagic fever
D.
E.
560.
A.
B. *
C.
D.
E.
561.
A.
B.
C.
D.
E. *
562.
A.
B.
C.
D. *
E.
563.
A.
B.
C.
D. *
E.
564.
A.
B. *
C.
D.
E.
565.
A. *
B.
C.
D.
E.
566.
A.
B.
C.
D.
E. *
567.
A.
Q-fever, spotted fever, leptospirosis
Q-fever, Crimean hemorrhagic fever, psittacosis, tick encephalitis
Treatment of leptospirosis:
Desintoxication, dehydratation, antibiotics, glukokorticosteroids
Antileptospirosis immunoprotein, antibiotics, nosotropic facilities
Antibiotics, rehadratation, sorbtion preparation, vitamins
Diet № 7, antibiotics, desintoxication facilities
Antibiotics, diuretic, interferons
Unlike leptospirosis in case of yellow fever is absent:
Hemorrhagic syndrome
Kidney insufficiency
Іntoxication syndrome
Міalglic syndrome
Hepatic insufficiency
Urgent immunoprofilactic of tetanus in the case of trauma should be conducted in such period:
25 days from the moment of trauma
30 days from the moment of trauma
In the first 10 days from the moment of trauma
After the trauma
Not mentioned
Vaccinations against leptosprosis perform for:
All
Only villagers
Only to the habitants of endemic districts
Only to the persons busy in the stock-raising
It is not conducted
Vegetative form of exciter of tetanus is destroyed in such terms, except for:
At a temperature of 100 °C
At room temperature
Under act of carbolic acid
Under the action of oxygen
Under act of antibiotics
Violations of electrolyte balance show up at a leptospirosis:
Metabolic acidosis
By a metabolic alkalosis
Respirator acidosis
By a respiratory alkalosis
All above enumerated
What changes in biochemical blood test inherent for haemorrhagic fever with kidneys syndrome?
Increase level of urea and bilirubin
The level of urea and kreatinine falls
The level of kreatinine grows and urea falls
The level of urea grows and kreatinine falls
The level of urea and kreatinine grows
What changes in haemogram inherent Congo hemorrhagic fever?
Normochomic anaemia, leucocytosis mononuclear
B.
C. *
D.
E.
568.
A.
B.
C.
D. *
E.
569.
A.
B.
C. *
D.
E.
570.
A.
B.
C. *
D.
E.
571.
A.
B.
C. *
D.
E.
572.
A.
B.
C. *
D.
E.
573.
A.
B.
C. *
D.
E.
574.
A.
B.
C. *
D.
E.
Erythrocytosis, lymphocytosis
Hypochromic anemia, erythrofilosis
Hypochromic anemia, neutrofilosis
Hyperchromic anemia, neutrofilosis
What changes in haemogram inherent for haemorrhagic fever with kidneys syndrome?
Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia enhanceable
ESR
erythrocytosis,
lymphocytosis,ESR is enhanceable
Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia enhanceable ESR
Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia enhanceable ESR
Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR
What etiothropic means use at treatment of haemorrhagic fever with kidneys syndrome:
Benzylpenicillin
Dopamine
Virolex
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Ebola fever:
Benzylpenicillin
Dopamine
Virolex
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Marburg fever:
Benzypenicillin
Dopamine
Ribavirin
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Congo fever:
Benzylpenicillin
Dopamine
Ribavirin
Dexamethazone
Etamsylatum
What etiothropic means use at treatment of patients with Lassa fever:
Benzylpenicillin
Dopamine
Ribavirin
Dexamethazole
Etamsylatum
What etiothropic means use at treatment of patients with Crimea fever:
Benzylpenicillin
Dopamine
Ribavirin
Dexamethazone
Etamsylatum
575.
A.
B.
C. *
D.
E.
576.
A. *
B.
C.
D.
E.
577.
A.
B.
C.
D. *
E.
578.
A.
B.
C. *
D.
E.
579.
A. *
B.
C.
D.
E.
580.
A. *
B.
C.
D.
E.
581.
A. *
B.
C.
D.
E.
582.
A.
B.
C. *
What etiothropic means use at treatment of patients with Omsk fever:
Benzylpenicillin
Dopamine
Ribavirin
Dexamethazone
Etamsylatum
What from antibiotics are more effective at treatment of icteric form of leptospirosis:
Penicillins
Aminoglicozids
Tetracyclins
Macrolids
Metrogil
What from antibiotics more expedient to use for treatment of leptospirosis?
Macrolids
Tetracyclins
Aminoglicosids
Penicillins
Metrogil
What is the duration of excitation period of hydrophobia?
7-10 days
24 hours
2-3 days, sometimes to 6 days
Not more than 2 days
Up to 6 hours
What is the duration of outpatient supervision after patients, recovered of tetanus?
2 years
3 months
1 month
For the decreed groups of population for life time
There is no such supervision at all
What is the duration of the prodromal period for the hydrophobia?
1-3 days
Up to 1 day
4-7 days
3-4 days
3-5 days
What is the entrance for the hydrophobia?
Damaged skin and mucous tissues
Respiratory tracts
Family ways
Gastrointestinal tract
Blood
What is the first aid preparation for the patient with tetanus?
Glucocorticoids
Analgetics
Anticonvulsant medicine
D.
E.
583.
A.
B.
C.
D. *
E.
584.
A.
B.
C. *
D.
E.
585.
A.
B.
C. *
D.
E.
586.
A.
B.
C.
D. *
E.
587.
A.
B.
C.
D. *
E.
588.
A.
B.
C.
D.
E. *
589.
A.
B. *
C.
D.
E.
590.
A.
Surgical treatment of the wound
Oxygen therapy
What is the mechanism of transmission of hydrophobia?
Transmissive
Fecally-oral
Air drop
Wound
Domestic contact
What is the origin of Lime disease:
Simplest
Viruses
Borrelium
Micoplasma
Ricketsia
What is the receptivity of population to the tetanus?
0%
50 %
Almost 100 %
10 %
70 %
What is the sensitivity to the hydrophobia?
45 %
25 %
85 %
100 %
10 %
What is typical for the Lassa hemorrhagic fever:
Effect of cardiovascular system
Development of acute hepatic insufficiency
Hundred-per-cent lethality
Defeat of breathing organs
Development of paresis and paralysis
What measures should be taken in relation to contact persons in case of tetanus?
Vaccination
Isolation of contacts
Chemoprophylaxis
Laboratory inspection
They need no measures
What periods of hydrophobia do you know?
Incubation, depressions, excitation
Incubation, depressions, excitation, paralytic
Depression, excitation, paralytic
Incubation, excitation, paralytic
Incubation, depressions, paralytic
What rashes in case of haemorrhagic fevers with kidneys syndrome?
Roseola
B.
C.
D. *
E.
591.
A.
B.
C.
D. *
E.
592.
A.
B.
C.
D. *
E.
593.
A. *
B.
C.
D.
E.
594.
A. *
B.
C.
D.
E.
595.
A.
B. *
C.
D.
E.
596.
A.
B.
C.
D. *
E.
597.
A.
B.
C.
D. *
E.
Maculo-papular
Punctuate
Petechial
Rashes is not characteristic
What rashes present in case of Congo hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
Petechial
Rashes not is characteristic
What rashes present in case of Crimea hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
Petechial
Rashes not is characteristic
What temperature of the body is typical for the paralytic period?
Hyperpyrexia
Hypothermia
High
Normal
Subfebril
Whatever complication meets at the yellow fever:
Liver insufficiency
Kidney insufficiency
Infectious-toxic shock
Myocarditis
Edema of lungs
When from the beginning of vaccination an antibodies to the rhabdovirus appear?
In a week
In 2 weeks
In a month
After half of year
Don’t produced
Whether there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome:
In an initial period
It is not
It is in all periods of disease
It is in climax period
It is in the period of recovering
Which serotypes of leptospirosis caused the disease more frequent:
L. interogans
L. grippotyphosa
L. canicola
L. icterohaemorrhagia
L. Pomona
598.
A.
B.
C. *
D.
E.
599.
A.
B.
C. *
D.
E.
600.
A. *
B.
C.
D.
E.
601.
A.
B.
C. *
D.
E.
602.
A.
B.
C.
D. *
E.
603.
A. *
B.
C.
D.
E.
604.
A. *
B.
C.
D.
E.
Who is the source of tetanus?
Sick person
Rodents
Soil
Insects
Cattle
Who is the source of the causal agent in the Crimean-Congo haemorrhagic fever?
Rodents, cattle, birds
Iksod and gamazov mites
Rodents, cattle, birds, sick people
The sick man, reconvalenc, bacteriocarries
Rodents, cattle, birds, sick people, bacteriocarries
Who of the listed below persons must take the conditional course of inoculations against
hydrophobia?
A teenager bitten by a dog which is on a leash, not instilled
Man bitten by a fox which perished
A child, scratched by a squirrel which disappeared in-field
A woman, bitten by a cat ill with hydrophobia
Man, who had a meal of undercooked of animal with hydrophobia
With appearance of hemorrhagic syndrome at Congo fever temperature of body always:
Normalize
Grows critically
Goes down
Does not change
Grows gradually
With the purpose of immunotherapy it is better to apply at treatment of leptospirosis:
Immunodepressants
Antihistaminics
Horse whey
Antileptospirosis human immunoprotein
Antileptospirosis neat immunoprotein
You might be infected with a rhabdovirus in case of:
Bite +salivation to the skin by an animal
Infected meal
Infected water
Contact with the infected air
Bite with the infected insect
A painful lesion with a bright red, edematous, indurate appearance and an advancing, raised border
that is sharply demarcated from the adjacent normal skin. Fever is a feature. What is the main
mechanism of transmission of this disease?
Airborne
Alimentary
Contact
Transmisiv
Vertical
605.
A.
B. *
C.
D.
E.
606.
A. *
B.
C.
D.
E.
607.
A.
B.
C. *
D.
E.
608.
A.
B.
C. *
D.
E.
609.
A.
B. *
C.
D.
E.
610.
A.
B.
C.
D.
E. *
611.
A.
A patient 60 years old for 2 days has disturbed severe pain in a right arm. On 3rd day appeared
blisters, pouring out as a chain on the skin of shoulder, forearm and brush. Sensitiveness in the area
of pouring out is mionectic. What disease can be diagnosed?
Dermatitis
Herpetic ganglionitis
Neck-pectoral redicals
Psoriasis
Allergy
A patient came with complaints of sickly erosions on his penis. From anamnesis frequent appearance
of similar rashes is found out during a year. Objectively: on a balanus are the grouped blisters and
erosions, soft on palpation. What is your diagnose?
Recurrent herpes of ІІ type
Vulvar pemphigus
Primary syphyllis
Shankoform pyoderma
Recurrent herpes of ІІІ type
A patient with temperature of body 40.0 °C, nonproductive cough, photophobia, puffiness of face,
dots on gums, blushes on the mucus of cheeks your diagnosis?
Tuberculosis
Меningococcemia
Measles
Enteroviral infection
Staphylococcal sepsis
A patient’s temperature is 40 °C. There are deep and unproductive cough, photophobia, face
puffiness whitish points on the mucous membrane of cheeks opposite molar teeth. What is the most
possible diagnosis?
Tuberculosis
Meningococcemia
Measles
Enteroviral infection
Staphylococcus sepsis
All are the clinical signs of measles EXEPT:
Acute beginning of high fever
Icterus
Maculo-papular rash
Sequential appearance of rash
Scaling
And. 5 days after the isolation of the last patient
11 days
21 day
10 days
No need for quarantine
5 days after isolation of the last child
At a patient, 17 years: quinsy, temperature 38,2 °C, generilized lymphadenopathy (the first multiplied
neck lymphatic knots which are located along m. sternocleidomastoideus), small icterus
hepatospleenomegaly. What exciter causes this disease?
Herpesvirus I type
B.
C.
D. *
E.
612.
A.
B.
C.
D. *
E.
613.
A. *
B.
C.
D.
E.
614.
A.
B. *
C.
D.
E.
615.
A.
B.
C. *
D.
E.
616.
A. *
B.
C.
D.
E.
617.
A.
B. *
C.
D.
E.
618.
A.
Herpesvirus ІІ type
Herpesvirus ІІІ type
Herpesvirus ІV type
Herpesvirus V type
At a patient, 17 years: quinsy. Temperature 38,2 °C, generilised lymphadenopathy (the first
multiplied neck lymphatic knots which are located along m. sternocleidomastoideus), small icterus,
hepatospleenomegaly. Previous diagnosis?
Tuberculosis of lymphatic knots
Bacterial quinsy
Diphtheria
Infectious mononucleosis
Megacaryoblastoma
At junior nurse, who works in child’s infectious department, herpes simplex was found. What should
manager of department must do?
Create a quarantine in the department
To appoint an immunoprotein to the children
Discharge all children from the department
To appoint immunomodulators with a prophylactic purpose
To inspect a junior nurse on a staphylococcus
At the end of treatment of patients with erysipelas it is recommended to enter:
500 000 of Bicyllin-3 intramuscular
1 500 000 of Bicyllin-5 intramuscular
1 000 000 of Bicyllin-5 intramuscular
1 500 000 of Bicyllin-3 intramuscular
500 000 of Bicyllin-3 intramuscular
Because of the risk of reinfection of erysipelas, prophylactic antibiotics are sometimes used after
resolution of the initial condition. Which antibiotics did we use?
Erythromycin
Penicillin III
Penicillin V
Cephazolin
Laevomicetyn
Call the exciter of erysipelas.
Streptococcus of group A
Streptococcus of group B
Streptococcus of group C
Streptococcus of group D
Streptococcus of group E
Causing of erysipelas are:
C. tetani
E. coli
Candida
Epstein-Barr virus
Hemolytic streptococcus group A
Diagnosis of repeated erysipelas may be set if clinical signs appeared:
In 2 years after erysipelas the last relapse of erysipelas
B.
C.
D. *
E.
619.
A.
B. *
C.
D.
E.
620.
A.
B. *
C.
D.
E.
621.
A.
B. *
C.
D.
E.
622.
A.
B. *
C.
D.
E.
623.
A.
B. *
C.
D.
E.
624.
A.
B. *
C.
D.
E.
625.
A.
B. *
C.
D.
E.
In 1 years after erysipelas the last relapse of erysipelas
In 9 months after an initial appeal of erysipelas
In 2 years after the initial disease
In 1 years after the initial disease
Did you need bacteriological examination of erysipelas?
Yes
No
Only for the decreed persons
Only for children
Only at suspicion on diphtheria
Did you need biological examination of erysipelas?
Yes
No
Only for the decreed persons
Only for children
Only at suspicion on diphtheria
Did you need serum examination of erysipelas?
Yes
No
Only for the decreed persons
Only for children
Only at suspicion on diphtheria
Ethyology of toxoplasmosis is:
Sh. Flexneri
Toxoplasma gondii
Neisseria meningitides
Corinebacterium diphtheria
?-hemolytic streptococcus
?Ethyology of toxoplasmosis is:
Sh. Flexneri
Toxoplasma gondii
Neisseria meningitides
Corinebacterium diphtheria
Hemolytic streptococcus
Ethyology of toxoplasmosis is:
Sh. Flexneri
T. gondii
N. meningitides
C. diphtheria
S. aureus
Ethyology of toxoplasmosis is:
Virus
Simplest
Bacteria
Helminthes
Animals
626.
A.
B. *
C.
D.
E.
627.
A.
B. *
C.
D.
E.
628.
A.
B.
C.
D.
E. *
629.
A.
B.
C. *
D.
E.
630.
A.
B.
C.
D.
E. *
631.
A.
B. *
C.
D.
E.
632.
A.
B.
C. *
D.
E.
633.
A.
B.
Etiology of erysipelas is.
Erysipelothrix rhusiopathiac
beta-hemolytic streptococcus
Bacillus anthracis
Herpes zoster
Herpes simplex
?Etiology of erysipelas is.
Erysipelothrix rhusiopathiac
B-hemolytic streptococcus
Bacillus anthracis
Herpes zoster
Herpes simplex
Factors of transmition of toxoplasmosis is:
Vertical
By ingestion of raw meat
By ingestion of undercooked meat
By cats
All above it
For how long a patient with complicated form of measles should be isolated:
For 4 days from the beginning of rash
For 7 days from the beginning of rash
For 10 days from the beginning of rash
For 17 days from the beginning
For 20 days from the beginning of illness
For specific pathogen of erysipelas following such properties:
Formation of exotoxins
Ability to multiply in anaerobiosis
Formation of spores
Formation of gametes
Gram-positive
For what disease characterize changes in a blood (presence of lymphomonocytes and a typical
mononuclears)?
Flu
Infectious mononucleosis
Measels
AIDS
Diphtheria
From etiotropic (antistreptococcus) facilities the most effective are:
Furazolidonum
Gentamicin
Benzilpenicilin and Oxacillinum
Benzilpenicilin and Furazolidonum
Doksiciklin and Gentamicin
How long does the incubation period last in erysipelas?
1-5 days
5-10 days
C. *
D.
E.
634.
A.
B.
C. *
D.
E.
635.
A.
B.
C.
D. *
E.
636.
A.
B.
C.
D.
E. *
F.
G.
H.
I.
J.
637.
A.
B.
C. *
D.
E.
638.
A.
B. *
C.
D.
E.
639.
A.
B.
C.
D.
E. *
3-5 days
10-15 days
15-20 days
How long does the incubation period last?
1-5 days
5-10 days
3-5 days
10-15 days
15-20 days
How long is the incubation period of erysipelas?
From a few hours to 5-7 days
From a few hours to 7-10 days
From a few hours to 2 days
From a few hours to 3-5 days
From a few hours to 14 days
In childbirth is erysipelas of left shin. Anti-epidemic measures in the maternity ward are:
No earlier than 2 years after disease
No earlier than 3 years after disease
In 9 months after the initial disease
In 2 weeks after a tonsillitis (angina)
In 3 weeks after a tonsillitis (angina)
Not needed
Quarantine in the department
Chatting
Quartz of air
Isolation of sick, disinfection
Indications for combined antibiotic therapy of erysipelas are:
Initial disease
Repeated wildfire
Recurrent erysipelas
Hard course
To prevent relapse
Measures of urgent prophylaxis for unvaccinated children who have never been ill with measles in
case of exposure to an ill with measles
Separation from the source
Vaccination
Administration of antibiotics
Disinfection
Does not exist
Measures of urgent prophylaxis of measles for contacts which have never been ill, but were
vaccinated against measles
Separation from the source
Vaccination
Use of antibiotics
Use of immunoglobulin
No need to conduct
640.
A.
B.
C.
D.
E.
641.
A. *
B.
C.
D.
E.
642.
A.
B.
C.
D.
E. *
643.
A.
B.
C.
D.
E.
644.
A.
B. *
C.
D.
E.
645.
A. *
B.
C.
D.
E.
646.
A.
B.
C. *
D.
Measures of urgent prophylaxis of measles for people who had been ill with measles, but never have
been vaccinated
Separation from the ill
Vaccination
Use of immunoglobulin
Use of antibiotics
No need to conduct
Mechanism of transmission of erysipelas are often:
Air
Contact
Transmissiv
Fecal-oral
Transplacental
Mechanism of transmission of toxoplasmosis are often:
Air
Contact
Transmissiv
All above it
Transplacental
Methods of specific prophylaxis of scarlet fever:
Isolation of ill
Vaccination
Use of antibiotics
Disinfection
Does not exist
Morphological characteristics of the causative agent of erysipelas:
Stik
Cocc
Ring
Larva
In a spiral
Patient A., 40 years, complains of hyperemia and edema on a right cheek. At examination:
temperature of body 38,7 °C, enlarged and painful lymphatic nodules on the right part of neck. A
border between hyperemia and healthy skin is clear, there are bubbles with hemorrhagic content, and
palpation is painful. What is clinical diagnosis?
Erysipelas
Anthrax
Herpetic infection
Chicken pox
Phlegmon of cheek
?Patient A., complains of redness of skin and edema on the right cheek. During a review: Т 38,7 C,
enlarged and painful lymphatic submandibular nodes on right side, border between red and normal
skin is sharp, present of swallowing with dark content, palpation is painful. What is your previous
Herpetic
infection
diagnosis?
Anthrax, skin form
Erysipelas, hemorrhagic form
Varicella (chickenpox)
E.
647.
A.
B.
C. *
D.
E.
648.
A.
B.
C. *
D.
E.
649.
A. *
B.
C.
D.
E.
650.
A.
B.
C.
D.
E. *
651.
A.
B.
C.
D. *
E.
652.
A.
B.
C. *
D.
E.
653.
A.
B.
C.
D. *
Phlegmone of cheek
Patient M., 30 years old, is treating ambulatory with uveitis. He complains of periodical subfebrill
temperature, headaches, weakness; he is ill during 3 years. Clinical previous diagnose.
Malaria
Brucellosis
Toxoplasmosis
Amoebiasis
Helmints
Patient M., 30 years old, is treating ambulatory with uveitis. He complains of periodical subfebrill
temperature, headaches, weakness; he is ill during 3 years. Clinical previous diagnose.
Malaria
Brucellosis
Toxoplasmosis
Amoebiasis
Helmints
Patient R., 31 years old, complains of rigor, fever (temperature 39 ?C), weakness, bright hyperemia
with elevated border and edema of subcutaneous fat, painfull in periphery, appears on the left leg last
day. What is clinical diagnosis?
Erysipelas
Anthrax
Herpetic infection
Chicken pox
Phlegmon
Patients with erysipelas working teacher in kindergarten. Anti-epidemic measures in the place of
work are:
Not needed
Quarantine of the group
The patient may be treated and continues to work
Compulsory hospitalization of the patient
Isolation of the patient to recovery
Primary and secondary prevention of recurrences of erysipelas disease are:
Avoid hypothermia
Of antibiotics in sufficient
Treatment that mycosis, thrombophlebitis-determine predisposition to erysipelas stop
Bicylin-profilactyc after antibiotic therapy
Avoid skin mikrotraum
Rules of hospitalization of patients with infectious mononuclesis:
Patients are not hospitalized
In a chamber for the infections of respiratory tracts
In a separate chamber
In a chamber for the infections of external covers
In a chamber for intestinal infections
Term of contagious period of patient diagnosed with uncomplicated form of measles
Until clinical recovery
After rash starts disappearing
Before appearance of rash
4 days from the beginning of rash
E.
654.
A. *
B.
C.
D.
E.
655.
A. *
B.
C.
D.
E.
656.
A.
B.
C.
D. *
E.
657.
A.
B.
C.
D. *
E.
658.
A. *
B.
C.
D.
E.
659.
A.
B.
C.
D.
E. *
660.
A.
B.
C.
D.
E.
661.
10 days from the beginning of illness
The diagnosis of recurrent erysipelas can be set, if clinical displays appeared:
In 2 years after a primary disease
At the first
In 6 months after a primary disease
On other place
In 2 weeks after the tonsillitis
The diagnosis of recurrent erysipelas can be set, if clinical displays appeared:
In 2 years after a primary disease
At the first
In 6 months after a primary disease
On other place
In 2 weeks after the tonsillitis
The diagnosis of the repeated erysipelas can be set, if clinical displays appeared:
In 2 years after the last recurrent erysipelas
At the first
In 6 months after a primary appeal
In 2 years after a primary disease
In a year after a primary disease
The diagnosis of the repeated erysipelas can be set, if clinical displays appeared:
In 2 years after the last recurrent erysipelas
At the first
In 6 months after a primary appeal
In 2 years after a primary disease
In a year after a primary disease
The main complication of erysipelas:
All above listed
Glomerulonephritis
Sepsis
Elephantiasis
Rheumatism
The patient asked the doctor about the recurrence of erysipelas. He is a manager at the company.
Terms treatment and access to work:
Necessarily isolation
Necessarily hospitalization
Treatment in home, without access to work
Can not works on this job
No restrictions
The source of exciter of cholera is:
Man, patient with a tonsillitis (angina)
Man, patient with moderate form cholera
Man, patient with a scarlet fever
Healthy carriers of streptococcus
All is listed above
There is the risk of reinfection of erysipelas, prophylactic antibiotics are sometimes used after
resolution of the initial condition. Which antibiotics did we use?
A.
B.
C. *
D.
E.
662.
A.
B.
C.
D. *
E.
663.
A.
B.
C.
D. *
E.
664.
A.
B.
C.
D.
E. *
665.
A.
B.
C.
D.
E. *
666.
A. *
B.
C.
D.
E.
667.
A.
B.
C.
D. *
E.
668.
A. *
B.
C.
D.
Erythromycin
Penicillin III
Penicillin V
Cephazolin
Laevomicetyn
To what kind of patients with erysipelas prednisolon should be prescribed?
Patients with hemorrhagic-bulbous form of erysipelas
Patients with accompanied thrombophlebitis
Patients with severe intoxication
Patients with often relapses of erysipelas and signs of lymphostasis
For all patients
To what kind of patients with erysipelas prednisolon should be prescribed?
Patients with hemorrhagic-bulbous form of erysipelas
Patients with accompanied thrombophlebitis
Patients with severe intoxication
Patients with often relapses of erysipelas and signs of lymphostasis
For all patients
Toxoplasmosis is transmitted by:
Transplacentally
By ingestion of raw
By ingestion of undercooked meat
By cats
All above it
Toxoplasmosis is transmitted by:
Transplacentally
By ingestion of raw
By ingestion of undercooked meat
By cats
All above it
What additional inspections must be conducted to the patient with infectious mononucleosis?
IFA on HIV-infection, bacteriology inspection on diphtheria
IFA on HIV-infection, bacteriology inspection on a rabbit-fever
Bacteriology inspection on diphtheria and typhoid
Reaction of Burne and Rihth-Heddlson
Reaction of Paul-Bunnel and punction of lymphatic knot
What additional test should hold for the patient with infectious mononucleosis?
Burne and Wright-Hadlson‘s reactions
ELISA-test, bacteriological test for tularemia
Bacteriological test for diphtheria and typhoid fever
ELISA-test, bacteriological test for diphtheria
Paul-Burne reaction and lymph node puncture
What are the anti epidemic measures in regards to people who were in contact with chicken-pox
patient:
Separation and limit of contacts with others
Vaccination
Use of antibiotics
Disinfection
E.
669.
A.
B.
C. *
D.
E.
670.
A. *
B.
C.
D.
E.
671.
A. *
B.
C.
D.
E.
672.
A.
B. *
C.
D.
E.
673.
A.
B.
C.
D. *
E.
674.
A.
B.
C.
D.
E. *
675.
A.
B.
C. *
D.
E.
676.
A.
Does not exist
What are the changes in the blood in patients with erysipelas?
Changes are absent
Limfomonocitosis, increase of ESR
Neutrophilic leycositosis, increase of ESR
Leycopeniya, increase of ESR
Increase of ESR
What are the main exciters of erysipelas.
Streptococcus
Staphylococcus
Spirochetes
Gonococcus
Stick of Lefler
What clinical form of erysipelas is in this patient?
Eritematozis
Eritematozo-bullezis
Hemorrhagic
Eritematozo-hemorrhagic
Necrosis
What complications can be after erysipelas?
Myocarditis
Endocarditis
Inflammation of additional bosoms of nose
Otitis
Festering inflammation of neck lymphonoduss
What complications can be after erysipelas?
Myocarditis
Otitis
Parafaringeal abscesses
Glomerulonephritis
Encephalitis
What complications can be after erysipelas?
Pneumonia
Pseudorheumatism
Illness of Reyno
Rheumatoceils
Sepsis
What disease is this? A painful lesion with a bright red, edematous, indurate appearance and an
advancing, raised border that is sharply demarcated from the adjacent normal skin. Fever is a feature.
Phlegmona
Anthrax
Erysipelas
Thrombophlebitis
Eczema
What does the most characteristic syndrome appear in patients with in 1 days of diseases?
Edema
B.
C. *
D.
E.
677.
A.
B.
C. *
D.
E.
678.
A.
B.
C. *
D.
E.
679.
A.
B.
C.
D.
E. *
680.
A.
B.
C.
D.
E. *
681.
A.
B.
C.
D.
E. *
682.
A.
B.
C.
D.
E. *
683.
A.
B.
C.
D.
E. *
Hyperemia
Pain
Stomach-ache
Takhikardiya
What does we use for the prophylaxis of reinfection of erysipelas?
Incoming immunoproteins
Incoming of small doses of hormones
Incoming bicillin 1 per a month
Incoming a vaccine
Warning of skin lesion
What does we use for the prophylaxis of reinfection of erysipelas?
Incoming immunoproteins
Incoming of small doses of hormones
Incoming bicillin 1 per a month
Incoming a vaccine
Warning of skin lesion
What examination should be performed to confirm the diagnosis of toxoplasmosis in pregnant
women?
Bacterioscopy
of blood
Biopsy materrial from lymph nodes, muscle or tissues
Immunofluorescence test
Burne test
ELISA-test
What examination should be performed to confirm the diagnosis of toxoplasmosis?
Bacterioscopy of blood
Biopsy materrial from lymph nodes, muscle or tissues
Immunofluorescence test
Burne test
ELISA-test
What examination should be performed to confirm the diagnosis of toxoplasmosis in pregnant
women?
Bacterioscopy of blood
Biopsy materrial from lymph nodes, muscle or tissues
Immunofluorescence test
Burne test
ELISA-test
What examination should be performed to confirm the diagnosis of toxoplasmosis?
Biological test
Biopsy of lymph nodes
Bacteriological test
Burne‘s test
ELISA-test
What examination should be performed to confirm the diagnosis of toxoplasmosis in pregnant
women?
Biological test
Biopsy of lymph nodes
Bacteriological test
Bacterioscopy of blood
ELISA-test
684.
A.
B. *
C.
D.
E.
685.
A.
B.
C.
D.
E. *
686.
A. *
B.
C.
D.
E.
687.
A.
B.
C. *
D.
E.
688.
A.
B.
C. *
D.
E.
689.
A.
B.
C.
D. *
E.
690.
A. *
B.
C.
D.
E.
691.
A.
B. *
C.
What from the following symptoms are not characteristic of infectious mononucleosis?
Fever
Defeat of kidneys
Lymphadenopathy
Tonsillitis
Increasing of liver and spleen
What is duration of contagious period for a patient with epidemic parotitis?
21 days
First week of illness
First 10 days from the beginning of disease
Whole period of clinical symptoms
First 9 days of disease.
What is duration period of supervision after ill with scarlet fever?
7 days from time of contact
21 day
Till patient’s rash is present
Till patient is discharged from permanent establishment
Not conducted
What is the base prophylactic measures of erysipelas relapse?
Administration of antistreptococcus immunoglobulin
Usage of small dosage of hormones
Bicillin prophylaxis once in a month
Usage of vaccine
Prevention of skin trauma and angina development
What is the base prophylactic measures of erysipelas relapse?
Administration of antistreptococcus immunoglobulin
Usage of small dosage of hormones
Bicillin prophylaxis once in a month
Usage of vaccine
Prevention of skin trauma and angina development
What is the duration of contagious period for a patient diagnosed with scarlet fever?
10 days from the beginning of illness
Until patient is discharged from the hospital
Until rash is present
Till the 22d day from the beginning of illness
Not contagious
What is the main mechanism of transmission of a erysipelas?
Airborne
Alimentary
Contact
Transmisiv
Vertical
What is the main mechanism of transmission of a toxoplasmosis?
Airborne
Fecal-oral
Contact
D.
E.
692.
A.
B.
C. *
D.
E.
693.
A.
B.
C. *
D.
E.
694.
A.
B. *
C.
D.
E.
695.
A.
B. *
C.
D.
E.
696.
A.
B.
C. *
D.
E.
697.
A.
B.
C.
D.
E. *
698.
A.
B.
C.
D. *
E.
Transmisiv
All above it
What is the prevention of erysipelas recurrence?
Introduction of antystreptococcus immunoglobulin
The use of small doses of prednizolon
Bicillin prophylaxis
Serum
Prevention of traumatization of skin and angina
What is the prevention of recurrence of erysipelas?
Introduction Ig
The use hormones
Bicylin-5 in a month
Vaccine
Nothing
What kind of erysipelas do your now:
Catarrhal, follicle and lacunars
Catarrhal, erythemat-bulos, erythemat-hemoragic and bulos-hemoragic
Catarrhal, erythemat-bulos, erythemat-hemoragic and necrotizing-ulcerous
Catarrhal, erythemat-bulos, erythemat-hemoragic, bulos-hemoragic and necrotizing-ulcerous
Catarrhal, erythemat-hemoragic and bulos-hemoragic
What kind of erysipelas do your now:
Catarrhal, erythemat-bulos, and bulos-hemoragic
Catarrhal, erythemat-bulos, erythemat-hemoragic and bulos-hemoragic
Catarrhal, erythemat-hemoragic and necrotizing-ulcerous
Erythemat-bulos, erythemat-hemoragic, bulos-hemoragic and necrotizing-ulcerous
Catarrhal, erythemat-hemoragic and bulos-hemoragic
What kind of lymphonoduss are increased in patients with a erysipelas?
All lymphonoduss
Nothing
Regionall
Submandibular
Anterolateral neck
What measures should be taken in regards to persons, who were in contact with a patient diagnosed
with epidemic parotitis?
Observation after contact people during a maximal length of incubation period
Quorantin in child's establishment
Isolation of people who were in contact with ill from 11th to the 21t day of illness
Isolation of children up to 10 years old, who were not ill with epidemic parotitis, for 21 day from a
moment of contact
All enumerated
What the most possible complication occurs during infectious mononucleosis?
Meningitis
autoimmune alopecia
encephalitis
Splenic rupture
Obstruction of respiratory tract
699.
A.
B.
C.
D.
E. *
700.
A.
B. *
C.
D.
E.
701.
A.
B. *
C.
D.
E.
702.
A.
B. *
C.
D.
E.
703.
A.
B.
C.
D. *
E.
704.
A.
B. *
C.
D.
E.
705.
A.
B. *
C.
D.
E.
706.
A.
B. *
C.
Which antibiotic is more expedient in patient with erysipelas with benzylpenitsylin allergy?
Ampicillin
Amoxicillin
Cefazolin
Amokciklav
Ciprofloxacin
Which antibiotics are use as etiological treatment of toxoplasmsis:
Penicillin
Antyprotozoyni
Cephalosporins
Aminoglycosides
Sulfonamides
Which antibiotics are use as etiological treatment of pregnant women with toxoplasmosis:
Penicillin
Metronidasol
Cephtriacson
Amikacin
Sulfanilamid
Which antibiotics are use as etiological treatment of pregnant women toxoplasmsis:
Aminohinol
Spiramicin
Fansydar
Cephtriacson
Sulfonamides
Which antibiotics are use as etiological treatment of toxoplasmosis, except:
Aminohinol
Spiramicin
Fansydar
Ceftriaxon
Sulfonamides
Which antibiotics are use as etiological treatment of toxoplasmosis:
Augmentin
Tinidasol
Cephtriaxon
Avelox
Nifuroxazid
Which antibiotics are use as etiological treatment of pregnant women with toxoplasmosis:
Penicillin
Metronidasol
Cephazolin
Cyprofloxacin
Biseptol
Which antibiotics are use as etiological treatment of pregnant women toxoplasmosis:
Avelox
Spiramicin
Fansydar
D.
E.
707.
A.
B.
C.
D.
E. *
708.
A.
B.
C.
D.
E. *
709.
A. *
B.
C.
D.
E.
710.
A. *
B.
C.
D.
E.
711.
A.
B. *
C.
D.
E.
712.
A.
B. *
C.
D.
E.
713.
A.
B.
C.
D. *
E.
714.
A.
Cephtriacson
Biseptol
Which complications occurs in erysipelas?
Sepsis
Septical endomiocarditis
Nephritis
Trombophlebitis
All the above
Which complications occurs in erysipelas?
Sepsis
Septical endomiocarditis
Nephritis
Trombophlebitis
All the above
Which drags are use as etiological treatment of pregnant women toxoplasmosis:
Immunoglobulin specific
Immunoglobulin normal
Interferon
Imunophan
Sulfonamides
Which drags are use as etiological treatment of pregnant women toxoplasmosis:
Immunoglobulin specific
Serum
Intron
Imunophan
Vaccine
Which drags are use as etiological treatment of pregnant women toxoplasmosis, except:
Immunoglobulin specific human
Intron A
Protephlazid
Metronidasol
Rovacid
Which of antibiotics are use as etiological treatment of toxoplasmosis:
Penicillin
Metronidasol
Cephazolin
Cyprofloxacin
Nifuroxazid
Which of antibiotics are use as etiological treatment of toxoplasmosis, except:
Metronidasol
Spiramicin
Fansydar
Avelox
Tinidasol
Which of antibiotics are used as etiological treatment of toxoplasmsis:
Penicillin
B. *
C.
D.
E.
715.
A.
B. *
C.
D.
E.
716.
A.
B. *
C.
D.
E.
717.
A.
B.
C.
D. *
E.
718.
A.
B. *
C.
D.
E.
719.
A. *
B.
C.
D.
E.
720.
A.
B. *
C.
D.
E.
721.
A.
B.
C. *
D.
E.
Antyprotozoyni
Cephalosporins
Aminoglycosides
Sulfonamides
Which of antibiotics are used as etiological treatment of pregnant women with toxoplasmosis:
Penicillin
Metronidasol
Cephtriacson
Amikacin
Sulfanilamid
Which of antibiotics are used as etiological treatment of pregnant women toxoplasmsis:
Aminohinol
Spiramicin
Fansydar
Cephtriacson
Sulfonamides
Which of antibiotics are used as etiological treatment of toxoplasmosis, except:
Aminohinol
Spiramicin
Fansydar
Cephtriacson
Sulfonamides
Which of drags are use as etiological treatment of pregnant women toxoplasmosis, except:
Immunoglobulin specific
Imunophan
Protephlazid
Metronidasol
Rovamicini
Which of drags are used as etiological treatment of pregnant women toxoplasmosis:
Immunoglobulin specific
Immunoglobulin normal
Interferon
Imunophan
Sulfonamides
Which of drags are used as etiological treatment of pregnant women toxoplasmosis, except:
Immunoglobulin specific
Imunophan
Protephlazid
Metronidasol
Rovamicini
Which patients with appropriate wildfire appoint prednisolone?
In the primary form
Repeated wildfire
Often recurrent erysipelas
If there are complications
All patients
722.
A.
B.
C. *
D.
E.
723.
A.
B.
C.
D.
E. *
724.
A. *
B.
C.
D.
E.
725.
A.
B.
C.
D.
E. *
726.
A.
B.
C.
D.
E. *
727.
A.
B.
C.
D.
E. *
728.
A.
B.
C.
D.
E. *
729.
A.
B.
C. *
Which patients with erysipelas assign appropriate prednisolone?
Buloz-haemorrhagic form
With concomitant tromboflebyt
Often recurrent erysipelas
Since phenomena limfostasis
All patients
Who is the source of erysipelas?
Sick man with erysipelas
Sick man with angina
Sick man with scarlet fever
Sick man with pneumonia
All the above
Who is the source of erysipelas?
Sick man and carriers
Rodents
Soil
Insects
Cattle.
Who is the source of erysipelas?
Sick man with erysipelas
Sick man with angina
Sick man with scarlet fever
Sick man with pneumonia
All the above
Who is the source of toxoplasmosis?
Sick man and carriers
Rodents
Soil
Insects
Cattle
Who is the source of toxoplasmosis?
Sick man and carriers
Rodents
Soil
Insects
Cat
Who is the source of toxoplasmosis?
Sick man and carriers
Rodents
Soil
Insects
Pig
An initial period at the hemorrhagic fever with a kidneys syndrome lasts:
Few hours
Day
To three days
D.
E.
730.
A. *
B.
C.
D.
E.
731.
A.
B.
C.
D.
E. *
732.
A.
B.
C. *
D.
E.
733.
A.
B.
C. *
D.
E.
734.
A.
B.
C.
D. *
E.
735.
A.
B.
C.
D.
E. *
736.
A.
B.
C.
D.
E. *
737.
A.
Week
Two weeks
At an objective examination for the Congo hemorrhagic fever character:
Mucosal hyperemia of person
Pallor of person
Puffiness of person
Ochrodermia of person
Exanthema on face
At I degree of dehydration the loss of liquid is:
0,5-1,5 % of body weight
6-9 % of body weight
3-6 % of body weight
5-8 % of body weight
1-3 % of body weight
At what percent of fluid loss will be I degree of dehydration?
-6 % of body weight
6-9 % of body weight
1-3 % of body weight
0,5-2 % of body weight
2-7 % of body weight
At what percent of fluid loss will be II degree of dehydration?
3-6 % of body weight
Over 10 % of body weight
6-9 % of body weight
4-8 % of body weight
10-15 % of body weight
At what percentage of fluid loss will be IV degree of dehydration?
4-8 % of body weight
6-9 % of body weight
3-6 % of body weight
Over 10 % of body weight
Over 15 % of body weight
Confirm the diagnosis of Congo hemorrhagic fever by a way of:
virological methods
bacteriological methods
Bacteriological and serum methods
epidemiological information
Virologic and serum methods
Confirm the diagnosis of Congo hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Virologic and serum methods
Confirm the diagnosis of Ebola fever by a way of:
Growth of viruses on chicken embryos
B.
C.
D.
E. *
738.
A.
B.
C.
D.
E. *
739.
A.
B.
C.
D.
E. *
740.
A.
B.
C.
D.
E. *
741.
A.
B.
C. *
D.
E.
742.
A.
B. *
C.
D.
E.
743.
A.
B.
C. *
D.
E.
744.
A.
B.
C. *
D.
E.
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Selection of virus on the Vero culture
Confirm the diagnosis of hemorrhagic fever with kidneys syndrome by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Virologic and serum methods
Confirm the diagnosis of Lassa hemorrhagic fever by a way of:
Only virological methods
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Virologic and serum methods
Confirm the diagnosis of Omsk fever by a way of:
Growth of virus on chicken embryons
Only bacteriological methods
Bacteriological and serum methods
Proper epidemiological information
Selection of virus on the Vero culture
Delagilum appoint at the malaria in such doses:
0,5 g 3 per a day 3 days
0,5 g per a week
In the first day 1 g, through 6 h 0,5 g
0,5 g per a day during a month
0,5 g 2 per a day 3 days
Especially dangerous for surroundings are patients with:
Skin form of plague
Pulmonary form of plague
Skin-bubonic form of plague
Bubonic form of plague
Septic form of plague
For a skin form of anthrax the most characteristically:
Hyperemia
Painful carbuncle
Not painful carbuncle
Painful noodles
Vesicles and bulls
For anthrax carbuncle the most characteristically:
Ulcer with a festering bottom, roller on periphery and insignificant area of edema
Ulcer with hyperemia on periphery without an edema
Ulcer with a black scab, black color, second vesicles and area of edema around of ulcer
Ulcer with a festering bottom, roller on periphery, second vesicles and area of edema
Ulcer with serosis-hemorrhagic exudates, painful, with the area of edema around of ulcer
745.
A.
B.
C.
D.
E. *
746.
A.
B.
C. *
D.
E.
747.
A.
B.
C. *
D.
E.
748.
A.
B.
C.
D. *
E.
749.
A. *
B.
C.
D.
E.
750.
A.
B.
C.
D.
E. *
751.
A.
B.
C.
D. *
E.
752.
A.
B. *
C.
For anthrax most characteristically:
Change of stool
Icterus of skin
Catarrhal phenomena
Meningeal phenomena
Change of skin
For confirmation of diagnosis of hemorrhagic fever with a kidney syndrome use:
Bacteriological method
Virological method
Reaction of immunofluorescence
Reaction of braking of hemagglutination
Research of blood drop under a microscope
For confirmation of yellow fever diagnosis use:
Bacteriological analysis of blood
Bacteriological examination of urine
Virological hemanalysis
Biochemical blood test
Global analysis of blood
For the initial period of the Congo hemorrhagic fever not characteristic:
Fever
Pains in joints and muscles
Severe pain of head
Oliguria
Dizziness
For the pulmonary form of anthrax characteristically:
Foamy sputum with blood
Glassy sputum with blood
Foamy sputum without blood
Foamy green sputum
Like to «ferruginous» sputum
For treatment of anthrax us:
Sulfanilamids
Nitrofurans
Hormones
Antiviral facilities
Antibiotics
For treatment of patients with the hemorrhagic fever with a kidney syndrome does not use:
Corticosteroids
Anabolic steroids
Disintoxication facilities
Dehydration facilities
Antihistaminics
General view of patient with the hemorrhagic fever with a kidneys syndrome:
Skinning covers
Pallor of nasolabial triangle, hyperemia of neck and overhead half of trunk
Hyperemia of person, scleritis, conjunctivitis
D.
E.
753.
A.
B.
C. *
D.
E.
754.
A. *
B.
C.
D.
E.
755.
A. *
B.
C.
D.
E.
756.
A.
B. *
C.
D.
E.
757.
A.
B.
C. *
D.
E.
758.
A.
B. *
C.
D.
E.
759.
A.
B.
C. *
D.
E.
760.
Grayish color of person
Icteric color of skin
Hemograme in the second period of yellow fever present:
Leukocytosis
Normal global analysis of blood
Leukopenia, neutropenia
Leukopenia, neutrophilosis
Leukocytosis, lymphomonocytosis
How long the rash is present in case of hemorrhagic fever with kidneys syndrome?
During all feverish period
Before the convalescence
Before development of clinical features of kidneys insufficiency
During whole disease
Appears yet in a latent period and disappears in the period of early reconvalescense
How many pandemics of plague were in history of mankind?
Three
Four
One
Two
Five
In a survey of donor blood found microhametosis. Assign treatment.
Delagil
Primaquine
Antibiotics
Fluoroquinolone
sulfanilamide
In the biochemical blood test at patients with the hemorrhagic fever with a kidneys syndrome not
characteristic:
High level of urea
Decline of potassium level
Bilirubinemia
Increasing of kreatinine
Increasing of nitrogen
In the global analysis of blood in case of Congo hemorrhagic fever not characteristic:
Leukocytosis
Leukopenia
Neutropenia
Thrombocytopenia
Increasing of ESR
In the initial period of hemorrhagic fever with a kidney syndrome a characteristic sign is:
High temperatures
Positive Pasternatsky’ symptom
Pains in joints and positive Pasternatsky’ symptom
Hemorragic syndrome
Dyspepsia phenomena
In which clinical forms of tularemia is valid the intracutaneous test?
A.
B.
C.
D.
E. *
761.
A. *
B.
C.
D.
E.
762.
A.
B. *
C.
D.
E.
763.
A. *
B.
C.
D.
E.
764.
A.
B.
C.
D.
E. *
765.
A.
B.
C.
D.
E. *
766.
A.
B. *
C.
D.
E.
767.
A.
B. *
C.
D.
Pulmonic
Bubonic
Eye-bubonic
Ulcer-bubonic
In all the clinical forms
Indications for the appointment of hematoshizotrop antimalarial drugs:
Attack of malaria
Preventing late relapse
Prophylactic course after returning from areas difficult to malaria
Antyretsidiv course for the rekonvalescents
During a check-up
Indications for the appointment of histoshizotrop antimalarial drugs:
Attack of malaria
Preventing late relapse
Complications of malaria
Malaria chemoprophylaxis for a period of stay in endemic areas
During a check-up
It is necessary to appoint for successful treatment of anthrax:
Antyanthrax immunoglobulin and penicillin
Antyanthrax immunoglobulin and prednizolon
Antyanthrax immunoglobulin and vyrolex
Antyanthrax immunoglobulin and vermox
Antyanthrax immunoglobulin and delagil
Mechanism of transmission of anthrax are:
Contact
Alimentary
Air-droplets’
Transmissiv
All above it
Patient V., 36 yrs. old has bubonic form of plague. What treatment do you suggest?
Local antibiotic therapy
Intravenous antibiotic therapy
Desintoxication therapy
Vitamins
All the above
Preparations for urgent prophylaxis of plague:
Injection of human immunoglobulin
Streptomycin or tetracycline
Human immunoglobulin
Dry living vaccine or tetracycline generations.
Interferon
Preparations for urgent prophylaxis of plague:
Injection of human immunoglobulin
Streptomycin or tetracycline
Human immunoglobulin
Dry living vaccine or tetracycline generations.
E.
768.
A.
B. *
C.
D.
E.
769.
A.
B. *
C.
D.
E.
770.
A.
B. *
C.
D.
E.
771.
A.
B.
C.
D. *
E.
772.
A. *
B.
C.
D.
E.
773.
A.
B.
C. *
D.
E.
774.
A. *
B.
C.
D.
E.
775.
A. *
Interferon
Preparations for urgent prophylaxis of plague:
Injection of human immunoglobulin
Streptomycin or tetracycline
Human immunoglobulin
Dry living vaccine or tetracycline generations.
Interferon
Risk group of plague infection the most frequent is:
Doctors
Hunters
Alcoholic
Drug users
Prostitutes
Specify the measures of urgent prophylaxis of anthrax.
Anti-anthrax immunoglobulin
Penicillinum or tetracyclinum during 5 days
Vaccination
Medical supervision
Biseptolum 5 days
Symptom of Stefansky – it is:
Enantema on a soft palate
Enantema on a conjunctiva
Shaking of tongue at an attempt to put out a tongue
Shaking of edema like to jelly at pattering a hammer in the area of edema
Painful of stomach in a right iliac area
The alimentary mechanisms of the tularemia infection of the humans are the following:
Due to water, food, straw and other substrata contaminated by the discharge of the animals sick with
tularemia
Contact with sick or dead rodents and hares
Bites of the infected blood-sucking arthropods
By means of a contact with sick animals
During the belated agricultural work
The basic clinical display of a skin form of anthrax is:
Hyperemic of skins
Vesiculs
Ulcer
Phlegmon
Abscess
The causative agent of plague is:
Yersinia pestis
Yersinia enterocolitica
Yersinia pseudotuberculosis
Bac. anthracis
Pseudomonas mallei
The causative agent of plague is:
Yersinia pestis
B.
C.
D.
E.
776.
A.
B. *
C.
D.
E.
777.
A.
B.
C.
D.
E. *
778.
A. *
B.
C.
D.
E.
779.
A.
B.
C. *
D.
E.
780.
A.
B. *
C.
D.
E.
781.
A.
B.
C.
D.
E. *
782.
A.
B.
C.
D.
Yersinia enterocolitica
Yersinia pseudotuberculosis
Bac. anthracis
Pseudomonas mallei
The contact mechanisms of the tularemia infection of the humans are the following:
Due to water, food, straw and other substrata contaminated by the discharge of the animals sick with
tularemia
Contact with sick or dead rodents and hares
Bites of the infected blood-sucking arthropods
By means of a contact with sick man
During the belated agricultural work
The diagnosis of malaria can confirm:
Microscopy of urine
Hemoculture
Bacteriology of stool
The response action
Parazitoscopy of blood
The diagnostic endermic reaction of anthrax take:
Antraksin
Dizenterin
Ornitin
Malein
Brucellin
The diagnostic reaction of anthrax is:
Rayt‘s reaction
Vidal‘s reaction
Reaction of term precipitation of Askoly
Paul-Bunnel‘s reaction
Reaction of agglutination-lysis
The duration of incubation period at plague is:
2-6 h
2-6 d
10-15 d
17-21 d
1-6 w
The duration of incubation period of plague is:
3 to 8 days;
2 to 12 days;
2 to 10 days;
1 to 8 days.
2 to 6 days;
The duration of incubation period of plague is:
3 to 8 days;
2 to 12 days;
2 to 10 days;
1 to 8 days.
E. *
783.
A.
B.
C. *
D.
E.
784.
A.
B.
C. *
D.
E.
785.
A.
B.
C. *
D.
E.
786.
A.
B.
C. *
D.
E.
787.
A. *
B.
C.
D.
E.
788.
A. *
B.
C.
D.
E.
789.
A.
B.
C.
D. *
E.
790.
A.
B.
2 to 6 days;
The etiological factor of anthrax is:
Salmonella thyphi
Erysipelothrix rhysiopothiac
Bacillus anthracis
Rickettsiosis sibirica
Toxocara canis
The main antibiotics, which used for tularemia treatment.
Penicillines
Vaccinotherapy
Aminoglycosides
Cephalosporines
Methronidazol
The measures of urgent prophylaxis of plague.
Administration of human immunoglobulin
Chlorochin (delagil) 0,25 g 2 times in week
6-day’s prophylaxis with streptomycin or tetracycline
In first 5 days intake antibiotics of penicillin or tetracycline origin
Іnterferon
The measures of urgent prophylaxis of plague.
Administration of human immunoglobulin
Chlorochin (delagil) 0,25 g 2 times in week
6-day’s prophylaxis with streptomycin or tetracycline
In first 5 days intake antibiotics of penicillin or tetracycline origin
Іnterferon
The most characteristic symptom in the climax period of the Congo hemorrhagic fever is:
Hemorrhagic syndrome
Hepatic insufficiency
Dyspepsia phenomena
Sharp kidney insufficiency
Мeningeal syndrome
The period of polyuria at haemorrhagic fever with kidneys syndrome is a sign of:
Recovering
Chronic process
Unfavorable flow of illness
Development of complications
Complete convalescence
The radical course of treatment of malaria includes:
Five-day therapy of delagilum
Prymahin during 2 weeks
Delagilum + prymahin + fansydar
Delagilum + prymahin
Delagilum + fansydar
The rules of hospitalization of patients with plague:
To separate ward
To ward for respiratory infections
C. *
D.
E.
791.
A.
B.
C. *
D.
E.
792.
A.
B.
C. *
D.
E.
793.
A.
B. *
C.
D.
E.
794.
A.
B.
C. *
D.
E.
795.
A.
B.
C.
D.
E. *
796.
A. *
B.
C.
D.
E.
797.
A. *
B.
To ward cubicle
Patient’s are not hospitalized
To ward for intestinal infections
The source of infection of anthrax is more frequent than all:
People
Birds
Home animals
Rodents
Fly
The transmissional mechanisms of the tularemia infection of the humans are the following:
Due to water, food, straw and other substrata contaminated by the discharge of the animals sick with
tularemia
Contact with sick or dead rodents and hares
Bites of the infected blood-sucking arthropods
By means of a contact with sick animals
During the belated agricultural work
The usual treatment for tularemia of streptomycin sulfate is
0,5 to 1 g injected once per day for 7 to 14 days or until the patient has been without fever for five to
seven days.
1 to 2 g injected once per day for 7 to 14 days or until the patient has been without fever for five to
seven days.
2 to 3 g injected once per day for 7 to 10 days or until the patient has been without fever for five to
seven days.
2 to 3 g injected once per day for 5 to 7 days or until the patient has been without fever for five to
seven days.
1 to 3 g injected once per day for 7 to 14.
There are etiotropic drags of malaria, except:
Delagilum
Prymahin
Cerasyn
Quinine
Fansidar
There can be several main variants of the tularemia bubo outcome:
Complete dissolving
Suppuration
Ulceration with the following scarring
Sclerotization
All the above
To the internal-disseminated forms of plague belong:
Primary-septic
Bubonic
Secondary-pulmonary
Primary-pulmonary
Intestinal
To the internal-disseminated forms of plague belong:
Primary-septic
Bubonic
C.
D.
E.
798.
A.
B.
C.
D.
E. *
799.
A.
B.
C.
D.
E. *
800.
A.
B.
C. *
D.
E.
801.
A.
B.
C. *
D.
E.
802.
A.
B.
C.
D.
E. *
803.
A.
B.
C.
D.
E. *
804.
A.
B.
C.
D.
E. *
805.
Secondary-pulmonary
Primary-pulmonary
Intestinal
To the localized forms of plague belong:
Intestinal
Primary-septic
Secondary-septic
Primary-pulmonary
Skin-bubonic
To the localized forms of plague belong:
Intestinal
Primary-septic
Secondary-septic
Primary-pulmonary
Bubonic
To the localized forms of plague belong:
Secondary-septic
Primary-septic
Skin
Primary-pulmonary
Intestinal
To the localized forms of plague belong:
Secondary-septic
Primary-septic
Skin
Primary-pulmonary
Intestinal
To the localized forms of plague belong:
Intestinal
Primary-septic
Secondary-septic
Primary-pulmonary
Skin-bubonic
To the localized forms of plague belong:
Intestinal
Primary-septic
Secondary-septic
Primary-pulmonary
Bubonic
Tularemia is transmitted by:
Air
Food
Contact
Transmission
All the above
Tularemia may also be transmitted by such mechanisms except
A.
B.
C. *
D.
E.
806.
A.
B.
C.
D. *
E.
807.
A.
B.
C.
D.
E. *
808.
A.
B.
C. *
D.
E.
809.
A.
B.
C.
D. *
E.
810.
A. *
B.
C.
D.
E.
811.
A. *
B.
C.
D.
E.
812.
A.
B.
C. *
D.
Alimentary
Transmissional
Vertical
Respirational
Contct
What anthrax prophylactic measures are entertained by farm workers?
Vitamin therapy
Immunization by inactivated vaccine
Formulated vaccine
Immunization by live vaccine
Antibiotic therapy
What changes in biochemical blood test inherent for hemorrhagic fever with kidneys syndrome?
Increase level of urea and bilirubin
The level of urea and kreatinine falls
The level of kreatinine grows and urea falls
The level of urea grows and kreatinine falls
The level of urea and kreatinine increase
What changes in blood analysis inherent at Congo hemorrhagic fever?
Normochromic anaemia, leucocytosis mononuclear
Erythrocytosis, lymphocytosis
Hypochromic anemia, erythrophilosis
Hypochromic anemia, neutrophilosis
Hyperchromic anemia, neutrophilosis
What changes in blood analysis inherent for hemorrhagic fever with kidneys syndrome?
Normochromic anaemia, leucocytosis with atypical mononucleosis, thrombocytopenia increased ESR
erythrocytosis, lymphocytosis,ESR is increased
Normochromic anaemia, leucopenia with neutrophylosis, thrombocytopenia increased ESR
Hypochromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia increased ESR
Hyperchromic anaemia, leucocytosis with neutrophylosis, thrombocytopenia mionectic ESR
What clinical form are not characterized for a rabbit-fever?
Erytem
Bubonic
Eye-bubonic
Ulcer-bubonic
Pulmonic
What complication is meet at the yellow fever:
Liver insufficiency
Kidney insufficiency
Infectious-toxic shock
Myocarditis
Edema of lungs
What does include a specific prophylaxis at a rabbit-fever?
Tetracyclin
Doxycyclin
Vaccination by a living attenuated vaccine
Bakteriofag
E.
813.
A.
B.
C. *
D.
E.
814.
A.
B. *
C.
D.
E.
815.
A.
B. *
C.
D.
E.
816.
A.
B.
C.
D. *
E.
817.
A.
B.
C.
D. *
E.
818.
A. *
B.
C.
D.
E.
819.
A.
B.
C. *
D.
E.
820.
A.
B.
Vaccination by a chemical attenuated vaccine
What drug did use for the treatment of small pox?
Merapenem
Flukonazol
Specifically immunoglobulin
Vitamin K
Levomicetyn
What drug did use for the treatment of plague?
Amoxicillin
Streptomycin
Penicillin
Biseptol
5-NOK
What drug is first step of choice for the treatment of plague?
Amoxicillin
Streptomycin
Penicillin
Biseptol
5-NOK
What drug use as the drug of choice to treat tularemia?
Use kanamycin as the drug of choice.
Use cyproay as the drug of choice.
Use cephazolyn as the drug of choice.
Use streptomycin as the drug of choice.
Use levomicetyn as the drug of choice.
What form of plague is highly fatal?
Sylvatic;
Bubonic;
Septicemic;
Pneumonic.
Bubonic and septicemic
What is the main feature of septicemic salmonelosis?
Massive bacteriemia
Headache
Pain in the abdominal
Throatache
Bleeding
What is the susceptibility of human to plague?
Non susceptible
50 %
Almost 100 %
10 %
70 %
What is the susceptibility of human to plague?
Non susceptible
50 %
C. *
D.
E.
821.
A.
B.
C.
D. *
E.
822.
A.
B.
C.
D. *
E.
823.
A.
B.
C.
D. *
E.
824.
A. *
B.
C.
D.
E.
825.
A.
B.
C.
D. *
E.
826.
A.
B.
C.
D. *
E.
827.
A.
B.
C.
D. *
E.
828.
Almost 100 %
10 %
70 %
What is typical for the Lassa hemorrhagic fever:
Effect of cardiovascular system
Development of acute hepatic insufficiency
Hundred-per-cent lethality
Defeat of breathing organs
Development of paresis and paralysis
What is typical for the Lassa hemorrhagic fever:
Effect of cardiovascular system
Development of acute hepatic insufficiency
Hundred-per-cent lethality
Defeat of breathing organs
Development of paresis and paralysis
What material is necessary take for diagnosis of anthrax:
Spinal liquid
Urine
Saliva
Content of carbuncle
Nose swab
What organ demerged more frequent than all in patients with anthrax?
Skin
Lights
Gastrointestinal tract
Lymphatic system
Nervous system
What rashes in case of haemorrhagic fevers with kidneys syndrome?
Roseola
Maculo-papular
Punctuate
Petechial
Rashes is not characteristic
What rashes present in case of Congo hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
Petechial
Rashes not is characteristic
What rashes present in case of Crimea hemorrhagic fever?
Roseola
Maculo-papular
Punctulate
Petechial
Rashes not is characteristic
What specific test is used for anthrax diagnostic?
A.
B.
C. *
D.
E.
829.
A.
B.
C. *
D.
E.
830.
A.
B.
C.
D.
E. *
831.
A.
B.
C. *
D.
E.
832.
A. *
B.
C.
D.
E.
833.
A.
B.
C.
D. *
E.
834.
A.
B.
C.
D. *
E.
835.
A. *
B.
C.
D.
Compliment fixation test
Indirect hemaglutination test
Coetaneous test with antraxin
Hemaglutination test
RIFA with anthrax antigen
What used for prophylaxis of small pox.
Immunoglobulin
Penicillinum or tetracyclinum during 5 days
Vaccination
Medical supervision
Serum
What your need for reatment of thlorohynresistent forms of malaria?
Bactrimum
Dapson
Fansydar
Meflohin
It is all above enumerated
What your need to give to the patients with malaria‘s comma?
Glucocorticoids
Antishok drags
Intravenous delagilum
Substitute blood transfusion
Oxygen
When does begin treatments of patients with a malaria?
Immediately after hospitalization
After taking of material for research
After raising of final diagnosis
After the laboratory and instrumental result
All answers are faithful
When there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome:
In an initial period
It is not
It is in all periods of disease
It is in climax period
It is in the period of recovering
Where may be find the tularemic granulloms:
In liver
In spleen
In brain
In lymphatic nodes
In skin
Which is preliminary diagnosis?
Anginous-bubonic form of tularemia
Oropharyngeal form of Diphtheria
The Vensan-Simanovsky’s tonsilitis
The Dyuge-Shtryumpelya’s tonsilitis
E.
836.
A. *
B.
C.
D.
E.
837.
A.
B.
C. *
D.
E.
838.
A.
B.
C.
D. *
E.
839.
A.
B. *
C.
D.
E.
840.
A.
B. *
C.
D.
E.
841.
A.
B.
C.
D. *
E.
842.
A. *
B.
C.
D.
E.
843.
A.
B.
Cataral tonsilitis
Which method are the most often used to diagnose the rabbit-fever in the people?
An intracutaneous allergic test with tularin
An intracutaneous allergic test with antraxin
ELISA test with specifical antibody
Allergic test of Byurne
Test of Cuverkalov
Which of microorganismes causes the tularemia:
Shigella tularensis
Riketsua tularensis
Francisella tularensis
Salmonella tularensis
Clostridia tularensis
Which of microorganismes causes the tularemia:
Legionella
Corinebacterium diphtheriae
Orthomyxoviridae
Francisella tularensis
Leptospira
Which specifical serological methods is use for tularemia diagnostics
Indirect hemagglutination test
Infradermic test with tularin
Compliment fiction rest
Agglutination reaction
Hemagglutination reaction
Who are the infection source of anthrax.
Birds
Wild animals
Fly
Human
Rodents
Who is the reservoir of causative agent of plague in nature?
Birds
Insects
Fresh-water fish
Rodents
Cattle
Who is the source of infection at a rabbit-fever?
Cattle, rodents, hare, water-rats, rabbits
Guinea-pigs
Man
Leeches and shellfishes
Cats
With appearance of hemorrhagic syndrome at Congo fever temperature of body always:
Normal
Grows critically
C. *
D.
E.
844.
A.
B.
C.
D. *
E.
845.
A. *
B.
C.
D.
E.
846.
A.
B. *
C.
D.
E.
847.
A.
B.
C.
D. *
E.
848.
A.
B.
C. *
D.
E.
849.
A. *
B.
C.
D.
E.
850.
A.
B. *
C.
D.
E.
851.
Goes down
Does not change
Grows gradually
With what diseases it is necessary to differentiate anthrax:
Leptospirozis
Typhoid fever
Dermatitis
Carbuncle
Meningococcal infection
Y. pestis is transmitted more frequently by:
Flea
Water
Air
Food storage
Tick
Your mast begins to treat patients with a plague:
Immediately after hospitalization
Immediately after hospitalization, carrying out only material for research
After raising of final diagnosis
After laboratory and instrumental diagnostics
All answers are faithful
When there is violation of diuresis at patients with hemorrhagic fever with a kidneys syndrome:
In an initial period
It is not
It is in all periods of disease
It is in climax period
It is in the period of recovering
When you can stopped etiotropic medications treatment of the patient with epidemic typhus?
Immediately after the normalization of body temperature
After the normalization of the liver and spleen
After a 2-day normal body temperature
After the disappearance of roseola
Within 10 days after the disappearance of roseola
Y. pestis is transmitted more frequently by:
Flea
Water
Air
Food storage
Tick
Your mast begins to treat patients with a plague:
Immediately after hospitalization
Immediately after hospitalization, carrying out only material for research
After raising of final diagnosis
After laboratory and instrumental diagnostics
All answers are faithful
You must begin to treat patients with a plague:
A.
B. *
C.
D.
E.
Immediately after hospitalization
Immediately after hospitalization, carrying out only material for research
After raising of final diagnosis
After laboratory and instrumental diagnostics
All answers are faithful