Chronic stable angina
... • “Strangling of the chest” • Temporary imbalance between the coronary artery’s ability to supply oxygen and the cardiac muscle’s demand for oxygen • Ischemia limited in duration and does not cause permanent damage to myocardial tissue • Chronic stable angina: Pain usually relieved with Sublingual N ...
... • “Strangling of the chest” • Temporary imbalance between the coronary artery’s ability to supply oxygen and the cardiac muscle’s demand for oxygen • Ischemia limited in duration and does not cause permanent damage to myocardial tissue • Chronic stable angina: Pain usually relieved with Sublingual N ...
Metabolic syndrome and prognosis of ischemic mitral insufficiency
... Coronary artery disease (CAD) is graded in relation to the number of vessels wich were significant as stenostic (one, two, three, four and multi-vessel CAD). Patients underwent surgical (coronary artery baypass grafting (CABG)) and percutaneous revascularization (percutaneus coronary intervention (P ...
... Coronary artery disease (CAD) is graded in relation to the number of vessels wich were significant as stenostic (one, two, three, four and multi-vessel CAD). Patients underwent surgical (coronary artery baypass grafting (CABG)) and percutaneous revascularization (percutaneus coronary intervention (P ...
ACS - bjclearn
... ischemia due to a perfusion-dependent imbalance between supply and demand. – Myocardial ischemia does not cause immediate cell death but rather it occurs over a finite period of time. It can take at least 4 to 6 hours for complete necrosis of myocardial cells – This is dependent upon the presence of ...
... ischemia due to a perfusion-dependent imbalance between supply and demand. – Myocardial ischemia does not cause immediate cell death but rather it occurs over a finite period of time. It can take at least 4 to 6 hours for complete necrosis of myocardial cells – This is dependent upon the presence of ...
Supplemental Table 1
... indicated by biochemical markers of myocardial necrosis (e.g., a typical rise and gradual fall of cTnI concentrations), along with at least 1 of the following: ischemic symptoms, development of pathologic Q waves on the electrocardiogram or changes indicative of ischemia, coronary artery interventio ...
... indicated by biochemical markers of myocardial necrosis (e.g., a typical rise and gradual fall of cTnI concentrations), along with at least 1 of the following: ischemic symptoms, development of pathologic Q waves on the electrocardiogram or changes indicative of ischemia, coronary artery interventio ...
Coronary Heart Disease
... Coronary arteries and veins are found within heart muscle Blood Clots Coronary thrombosis Coronary embolism ...
... Coronary arteries and veins are found within heart muscle Blood Clots Coronary thrombosis Coronary embolism ...
Angina pectoris
... rest In some patients pain occurs predictably at a certain level of exertion ...
... rest In some patients pain occurs predictably at a certain level of exertion ...
Dr. Deng Xi Wei , MACC Chief Consultant in Cardiology Kiang Wu
... cause of death in industrialized countries and are expected to become so in emerging countries by 2020. ...
... cause of death in industrialized countries and are expected to become so in emerging countries by 2020. ...
half a decade of primary percutaneous
... less frequent (40.3% vs. 64.4%, p<0.005). All cause in-hospital death and composite of death, emergency Coronary Artery Bypass Grafts (CABG), Myocardial Infarction (MI) and stroke in group 2 was significantly higher than group 1 (12.3% versus 4.8%,p=0.02 and 15.7% versus 7.4%, p=0.04). Cardiogenic s ...
... less frequent (40.3% vs. 64.4%, p<0.005). All cause in-hospital death and composite of death, emergency Coronary Artery Bypass Grafts (CABG), Myocardial Infarction (MI) and stroke in group 2 was significantly higher than group 1 (12.3% versus 4.8%,p=0.02 and 15.7% versus 7.4%, p=0.04). Cardiogenic s ...
Acute Coronary Syndromes Algorithm
... Goals of Therapy in ACS Reduce the amount of myocardial necrosis that occurs in patients with MI, preserving left ventricular function and preventing heart failure Prevent major adverse cardiac events: death, nonfatal MI, and need for urgent revascularization Treat acute, life threatening compl ...
... Goals of Therapy in ACS Reduce the amount of myocardial necrosis that occurs in patients with MI, preserving left ventricular function and preventing heart failure Prevent major adverse cardiac events: death, nonfatal MI, and need for urgent revascularization Treat acute, life threatening compl ...
Unstable Angina and Non–ST Elevation Myocardial Infarction
... daily Ticagrelor a loading dose of 180 mg and 90 mg twice daily ...
... daily Ticagrelor a loading dose of 180 mg and 90 mg twice daily ...
For ranolazine study
... Ranolazine is provided efficiency of anti-ischemic/antianginal properties in patients with chronic angina without clinically significant changes in heart rate or blood pressure. Now we want to see the efficiency of ranolazine in obstructive coronary artery disease with left ventricular dysfunction. ...
... Ranolazine is provided efficiency of anti-ischemic/antianginal properties in patients with chronic angina without clinically significant changes in heart rate or blood pressure. Now we want to see the efficiency of ranolazine in obstructive coronary artery disease with left ventricular dysfunction. ...
Pre-Lecture Quiz
... 2. Cardiomyopathies involve mechanical rather than electrical etiologies of heart dysfunction. ...
... 2. Cardiomyopathies involve mechanical rather than electrical etiologies of heart dysfunction. ...
Levosimendan - Wellington ICU
... - also produces vasodilation by opening ATP-sensitive K+ channels in vascular smooth muscle -> reduces preload, afterload, improves O2 supply to myocardium, increases coronary blood flow and renal blood flow. - increases: Q, SV, HR and increases coronary blood flow. - decreases: SVR, SBP, PCWP, PAP, ...
... - also produces vasodilation by opening ATP-sensitive K+ channels in vascular smooth muscle -> reduces preload, afterload, improves O2 supply to myocardium, increases coronary blood flow and renal blood flow. - increases: Q, SV, HR and increases coronary blood flow. - decreases: SVR, SBP, PCWP, PAP, ...
Management of acute coronary syndrome
Management of acute coronary syndrome is targeted against the effects of reduced blood flow to the afflicted area of the heart muscle, usually because of a blood clot in one of the coronary arteries, the vessels that supply oxygenated blood to the myocardium. This is achieved with urgent hospitalization and medical therapy, including drugs that relieve chest pain and reduce the size of the infarct, and drugs that inhibit clot formation; for a subset of patients invasive measures are also employed (coronary angiography and percutaneous coronary intervention). Basic principles of management are the same for all types of acute coronary syndrome. However, some important aspects of treatment depend on the presence or absence of elevation of the ST segment on the electrocardiogram, which classifies cases upon presentation to either ST segment elevation myocardial infarction (STEMI) or non-ST elevation acute coronary syndrome (NST-ACS); the latter includes unstable angina and non-ST elevation myocardial infarction (NSTEMI). Treatment is generally more aggressive for STEMI patients, and reperfusion therapy is more often reserved for them. Long term therapy is necessary for prevention of recurrent events and complications.