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Infectious bursal disease virus monoclonal antibody, clone
Infectious bursal disease virus monoclonal antibody, clone

... strain) derived from infected chicken bursas. Host: Mouse Reactivity: Chicken Applications: ELISA, IHC, S-ELISA, WB (See our web site product page for detailed applications information) Protocols: See our web site at http://www.abnova.com/support/protocols.asp or product page for detailed protocols ...
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... be started until this application has been reviewed and approved. Additional information may be requested before approval can be considered. Please allow at least one week for the review and approval process to be completed. Date: Project Title: ...
The first recorded case of HIV/AIDS in Australia was in Sydney in
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... In conjunction with a positive DNA PCR or a reactive EIA, the RNA quantitation may be diagnostic.  High levels of RNA are found during acute infection and in patients who are more likely to have disease progression.  Inhibition of cell-free HIV, as reflected by RNA copy number, is associated with ...
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The Natural History of Untreated HIV
The Natural History of Untreated HIV

... Adapted from Wada N et al. Am J Epidemiol 2013. ...
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HIV



The human immunodeficiency virus (HIV) is a lentivirus (a subgroup of retrovirus) that causes HIV infection and acquired immunodeficiency syndrome (AIDS). AIDS is a condition in humans in which progressive failure of the immune system allows life-threatening opportunistic infections and cancers to thrive. Without treatment, average survival time after infection with HIV is estimated to be 9 to 11 years, depending on the HIV subtype. Infection with HIV occurs by the transfer of blood, semen, vaginal fluid, pre-ejaculate, or breast milk. Within these bodily fluids, HIV is present as both free virus particles and virus within infected immune cells.HIV infects vital cells in the human immune system such as helper T cells (specifically CD4+ T cells), macrophages, and dendritic cells. HIV infection leads to low levels of CD4+ T cells through a number of mechanisms, including apoptosis of uninfected bystander cells, direct viral killing of infected cells, and killing of infected CD4+ T cells by CD8 cytotoxic lymphocytes that recognize infected cells. When CD4+ T cell numbers decline below a critical level, cell-mediated immunity is lost, and the body becomes progressively more susceptible to opportunistic infections.
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