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Fontan Procedure Part 2 By Dr. Madhusudan Raikar
Fontan Procedure Part 2 By Dr. Madhusudan Raikar

... ◦ Surgical ligation of the thoracic duct. ...
Modeling Cardiac Ventricular Activation
Modeling Cardiac Ventricular Activation

... While there is hardly any lack of electrocardiographic or magnetocardiographic data, detailed data on the anatomy and physiology of the conduction system or cellular interactions in the myocardium are scarce [Durrer et al., 1979]. We may even have access to high-quality recorded data on the endocard ...
Contemporary management of acute right ventricular failure: a
Contemporary management of acute right ventricular failure: a

... Because RV afterload is very low under normal conditions, blood flows from the RV into the pulmonary circulation both during systole and during the early part of diastole, leading to the absence of isovolumetric relaxation.15 ...
Cardiology - Stiftung KinderHerz
Cardiology - Stiftung KinderHerz

... and RVEF were found in an adult population that included patients with congenital heart disease [10]. Eyskens et al. [11] showed elevated RV systolic velocities in patients with ASDs and dilated RVs before percutaneous closure of the defect, which normalized within 24 hours after closure. Quantitati ...
Bicuspid Aortic Valve Disease Beyond the Aortic Root
Bicuspid Aortic Valve Disease Beyond the Aortic Root

... endothelium-derived nitric oxide synthase (eNOS) knockout mice had a significantly higher likelihood of developing BAV than control mice. This is corroborated by human studies documenting lower eNOS gene expression in the ascending aortas of BAV patients than in ascending aortas of control subjects ...
Nitroglycerin increases venous return but reduces CVP: a preload
Nitroglycerin increases venous return but reduces CVP: a preload

... extent from the arterioles and small arteries (one third). A common cause of increase in venous resistance is the venoconstriction in hypovolemia. Venous return can only occur when there is a pressure gradient. The CVP produces a backward force on the veins that impedes blood flow into the right atr ...
APPROACH TO AN INFANT WITH CYANOSIS –
APPROACH TO AN INFANT WITH CYANOSIS –

... Cyanotic heart disease with increased pulmonary flow will have a characteristic combination of mild cyanosis and severe CHF. This group is again divided into 2 sub groups. Group IV: CCHD. Increased PBF: Parallel circulation Transposition of great arteries (TGA) is the unique CHD belonging to this g ...
What does an indeterminate troponin really mean?
What does an indeterminate troponin really mean?

... “positive troponin” • There are clear standards for the interpretation of positive (>0.5) in setting of high suspicion of coronary artery disease. This leads to a protocalized approach with the common goal being early revascularization On the other hand there’s the gray area… • Troponin leak-a commo ...
Recent Advances in Neonatal Cardiac Surgery
Recent Advances in Neonatal Cardiac Surgery

... [45]. European studies suggest that screening is cost-effective in identifying newborns with CCHD [46,47]. Over half of babies discharged with CHD detected in the first year of life were discharged from the hospital with a normal routine neonatal examination and the 6 week exam missed one-third of t ...
Full version (PDF file)
Full version (PDF file)

... captopril was shown to exert additive effects in SHR, increasing NOS activity, eNOS protein expression in the aorta and decreasing conjugated dienes concentration in the kidneys, in contrast with monotherapeutic application (Vranková et al. 2009). As for the body weight loss after the combination of ...
Uncommon Conal Pathology in Complete Dextrotransposition of the
Uncommon Conal Pathology in Complete Dextrotransposition of the

... ventricular aspect of the ventricular septum characterized by: (1) smooth appearance; (2) a superior concave border, the posterior end of which receives the attachment of the papillary muscle of the conus; and (3) an inferior extremity receiving the medial end of the moderator band. In most cases of ...
Ruptured Aneurysm of the Right Sinus of Valsalva Into the Right
Ruptured Aneurysm of the Right Sinus of Valsalva Into the Right

... The most common coexisting congenital heart diseases are ventricular septal defect (VSD), usually subaortic (25-55%) and regurgitation of the aortic valve and rarely pulmonary stenosis, patent ductus arteriosus, atrial septal defect, subaortic stenosis and tetralogy of Fallot7. ASVs can remain silen ...
Survival after balloon atrial septostomy for complete transposition of
Survival after balloon atrial septostomy for complete transposition of

... analyses by Leanage et all( and Powell et al.17 Our findings are in agreement with these studies and others in that coarctation and persistent arterial duct were found to be significant risk factors.' 3 9 18 19 Analyses by Leanage and Powell also showed that age of septostomy, relative anaemia, the ...
A new concept of the contraction–extension
A new concept of the contraction–extension

... the left ventricular (LV) contraction–extension (C–E) property, which has an important relationship to LV function. Methods: Strain rate (SR) distribution within the posterior wall and interventricular septum was microscopically measured with a high accuracy of 821 ␮m in spatial resolution by using ...
Anomalous origin of the left coronary artery from the pulmonary
Anomalous origin of the left coronary artery from the pulmonary

... reimplantation of the left main coronary artery, and a CT-angiography one week after surgery confirmed the patency of the artery, Figure 3. During surgery the presence of an old infarction in the anterior myocardial wall was noted. Based on the clinical history, with a documented episode of ventricu ...
THE EFFECT OF EXERCISE ON MEAN LEFT VENTRICULAR
THE EFFECT OF EXERCISE ON MEAN LEFT VENTRICULAR

... mild mitral stenosis (valve area greater than 2.5 cm2), 1 with patent ductus arteriosus (pulmonary-systemic flow ratio, 1.2/1), 1 with hyperkinetic heart syndrome (9), and 1 with mild pulmonary hypertension of unknown etiology (pulmonary arterial pressure, 34/18 mm Hg). The failure group comprised 1 ...
full text pdf
full text pdf

... (12, 13). Studies have shown that abuse of AAS can induce hypertrophy of the left ventricle with disproportionate accumulation of extracellular collagen and interstitial fibrosis (14-17). Previous reports have further shown that chronic administration of supraphysiological doses of AAS can lead to i ...
Cardiac Iodine-123 Metaiodobenzylguanidine Imaging
Cardiac Iodine-123 Metaiodobenzylguanidine Imaging

... characteristics with survival was assessed: age; sex; underlying causes (ischemic or nonischemic); New York Heart Association (NYHA) functional class; heart rate; systolic and diastolic blood pressure; LVEF; presence of nonsustained ventricular tachycardia on Holter monitoring; echocardiography data ...
Predictors of right ventricular failure after left ventricular assist device
Predictors of right ventricular failure after left ventricular assist device

... setting of LVAD implantation has been recognized early, as evidenced by abundant number of attempts to identify independent risk factors and develop RVF predictor scores with a common purpose to improve patient selection and outcomes by recognizing potential need for biventricular assist device (BiV ...
Advanced EKG Interpretation PRESENTERS:
Advanced EKG Interpretation PRESENTERS:

... ♥Bradycardia is a slower than normal heartbeat. ♥Those with Heart blocks ♥Is a problem with the heart's electrical system and occurs when an electrical signal is slowed or disrupted as it moves through the heart. ♥Can happen as a result of aging, damage to the heart from a heart attack, or other con ...
Advanced EKG Interpretation - UT Health : The University of Toledo
Advanced EKG Interpretation - UT Health : The University of Toledo

... ♥Bradycardia is a slower than normal heartbeat. ♥Those with Heart blocks ♥Is a problem with the heart's electrical system and occurs when an electrical signal is slowed or disrupted as it moves through the heart. ♥Can happen as a result of aging, damage to the heart from a heart attack, or other con ...
How the ECG works
How the ECG works

... People tend to faff quite a lot about QRS axis deviations, but they are a fairly blunt-edged tool. Marked right axis deviation (e.g. +150o) may signify significant `right-sided' heart disease. Left axis deviation is not uncommon in inferior myocardial infarction, and if this is absent, the most like ...
Approach to infants and children with Cyanotic congenital heart
Approach to infants and children with Cyanotic congenital heart

... Cyanotic children may be classified into those who have decreased pulmonary blood flow (↓ PBF) or those with increased pulmonary blood flow (PBF.) This is primarily answered by history, physical examination and ultimately by chest x-ray. ...
Cardiac Anatomy and Physiologic Correlates
Cardiac Anatomy and Physiologic Correlates

... The aortic root and left ventricular outflow tract are include together because it is important to understand their close anatomical relations and coordinated physiologic function. The interleaflet triangles although considered part of the aortic root are beneath the leaflet annulus and are in fact ...
The Right Ventricular Function After Left Ventricular Assist Device
The Right Ventricular Function After Left Ventricular Assist Device

... the algorithm and divided into six segments (Figure 2). The speckletracking algorithm detects the QRS onset from the electrocardiographic signal to define the point of zero strain, amenable to correction, and calculates segmental and global strain. By temporal derivation of strain, the corresponding ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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