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ECG Lecture Chapter 6
ECG Lecture Chapter 6

... • Complete dissociation between the atria and ventricles or complete heart block. • Atria are depolarizing but not conducting to ventricles so another pacemaker must take over in the ventricles. • True 3rd degree heart block will have the atria rate > the ventricular rate. ...
AA Inappropriate Sinus Tachycardia (IST) Info
AA Inappropriate Sinus Tachycardia (IST) Info

... must be excluded – these include anaemia, hyperthyroidism, phaeochromocytoma, diabetes induced autonomic dysfunction, fever, and dehydration ...
Cardioverting with Confidence
Cardioverting with Confidence

... Current Therapy ...
Atrial arrhythmias in the young: early onset atrial arrhythmias
Atrial arrhythmias in the young: early onset atrial arrhythmias

... Early onset atrial arrhythmias may be the first clinical manifestation of a muscular dystrophy syndrome. Appropriate clinical assessment and surveillance may uncover this primary cause and provide an opportunity for timely genetic counselling and family screening. ...
Sinus Nodal Dysfunction - Developing Anaesthesia
Sinus Nodal Dysfunction - Developing Anaesthesia

... inappropriate sinus bradycardia, sinus arrest, sinus node exit block, chronic atrial fibrillation with slow ventricular response and bradycardia-tachycardia syndrome. Although the term sick sinus syndrome remains popular, a more appropriate title is sinus nodal dysfunction (SND). The disease commonl ...
PPT
PPT

... By the time the slow antegrade conduction is complete, the fast pathway is no longer refractory, allowing retrograde conduction to occur. This “circus” mechanism is maintained as long as the relationship between fast and slow conduction, and fast/slow refractoriness persists. ...
EKG
EKG

... the heart rate. The release of adrenaline is controlled by the nervous system. The heart normally beats at around 72 times per minute and the sinus node speeds up during exertion, emotional stress, fever, etc., or whenever our body needs an extra boost of blood supply. In contrast, it and slows down ...
Part b
Part b

... (c) Second-degree heart block. (d) Ventricular fibrillation. These chaotic, grossly irregular ECG Some P waves are not conducted deflections are seen in acute through the AV node; hence more heart attack and electrical shock. P than QRS waves are seen. In this tracing, the ratio of P waves to QRS wa ...
A 93-Year-Old Woman with an Abnormal
A 93-Year-Old Woman with an Abnormal

... heart failure have electrolytes abnormalities and compromised renal function, which are predisposing factors for digoxin toxicity. Toxic levels of digoxin can induce cardiac arrhythmias; other manifestations of digoxin toxicity include nausea, vomiting, headache, fatigue, blurred vision, and altered ...
Malignant neurilemmoma of left atrium
Malignant neurilemmoma of left atrium

... slight back pain and numbness in the left leg. These echoes appearing behind the anterior mitral leaflet symptoms progressed rapidly and in April 1979, she during diastole, highly suggestive, but not absolutely was admitted with a suspected lumbar disc diagnostic, of a left atrial tumour. On cross-s ...
Atrial fibrillation and flutter in primary care
Atrial fibrillation and flutter in primary care

... Rate control is recommended for most people with AF Good rate control in AF can not only control symptoms but also ...
ALS course slide set - the Australian Resuscitation Council
ALS course slide set - the Australian Resuscitation Council

... 1. Is there any electrical activity? 2. What is the ventricular (QRS) rate? 3. Is the QRS rhythm regular or irregular? 4. Is the QRS width normal (narrow) or broad? 5. Is atrial activity present? (If so, what is it: P waves? Other atrial activity?) ...
3 Bipolar Limb Leads
3 Bipolar Limb Leads

... – Signals onset of atrial contraction QRS complex: ventricular depolarization – Signals onset of ventricular contraction.. T wave: repolarization of ventricles PR interval or PQ interval: 0.16 sec – Extends from start of atrial depolarization to start of ventricular depolarization (QRS complex) cont ...
Structural Heart Defects and Stroke Structural Heart Defects and
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... LV regional wall motion abnormality Aortic arch atheromatous plaques ...
ECG - WordPress.com
ECG - WordPress.com

... the rectangular looking wave somewhere near the left of the paper. It should be 10mm ...
Treatment of AF: Ablation or Pharmacotherapy: Which One and When?
Treatment of AF: Ablation or Pharmacotherapy: Which One and When?

... • Combination or ‘hybrid’ rhythm control therapy seems reasonable, although there is little evidence from controlled trials supporting its use • Antiarrhythmic drug therapy is commonly given for 8–12 weeks after ablation to reduce early recurrences of AF after catheter ablation, supported by a recen ...
File
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... the rectangular looking wave somewhere near the left of the paper. It should be 10mm ...
Diagnosis of Supraventricular Tachycardia
Diagnosis of Supraventricular Tachycardia

... irregular QRS complexes with no defined P wave. Atrial flutter (AFl): AFl may be a stable rhythm or a bridge arrhythmia between sinus rhythm and atrial fibrillation. It is an arrhythmia most commonly seen in men, the elderly and individuals with preexisting structural heart disease or chronic obstru ...
Post TIA, Post Stroke Prognosis
Post TIA, Post Stroke Prognosis

... • persons < 60 years with no other cardiac disorder (lone AF): relatively low risk for stroke • AF: abetting factor leading to formation of intra-atrial thrombi in patient with another heart disease • at risk: chronic sustained & intermittent ...
pocket guide to neonatal ecg interpretation, 3rd edition
pocket guide to neonatal ecg interpretation, 3rd edition

... b.  b.  b.  b.  b.  b.  b.  b.  b.  b.  b.  b.  b.  b.  c.  c.  c.  c.  c.  c.  c.  c.  c.  c.  c.  c.  c.  c.    2. a.    8. a.  14. a.  20. a.  26. a.  32. a.  38. a.  44. a.  50. a.  56. a.  62. a.  68. a.  74. a.  80 ...
Anemia LECTURE IN INTERNAL MEDICINE FOR IV COURSE
Anemia LECTURE IN INTERNAL MEDICINE FOR IV COURSE

... (Assessment of Structural Heart Disease) • Interviewing (complains, history) and physical examination with attention to coronary artery disease or myocardial infarctions, risk factors for coronary artery disease (CAD), and family history of sudden cardiac death are extremely important. • Auscultatio ...
Gender differences in cardiac electrophysiology and arrhythmias
Gender differences in cardiac electrophysiology and arrhythmias

... disappears, and women tend to develop elevated risk of stroke and poorer survival than their male counterparts, especially in those over 75 years of age. Accessory pathways causing re-entry tachycardia are more prevalent in men, while in women atrioventricular nodal re-entry tachycardia predominates ...
Left Atrial Volume
Left Atrial Volume

... of the burden of increased ventricular filling pressure. Typically ischemic heart disease (IHD) has a long period of subclinical dysfunction. Increased filling pressure, reflected by enlarged LAVI, is hypothesized to mirror the burden of subclinical and overt IHD. We hypothesized that a normal LAVI ...
Evaluation of Myocardial Blood Flow Reserve in Patients
Evaluation of Myocardial Blood Flow Reserve in Patients

... This study demonstrates a significant impairment in coronary blood flow reserve in most patients with idiopathic dilated cardiomyopathy despite normal epicardial coronary arteries. This change may prevent appropriate increases in coronary blood flow and thus lead to myocardial ischemia and progressi ...
Document
Document

... AF is the most common cardiac arrhythmia. 2.2 Million in USA & 5.0 Million worldwide. 1% of general population & 6% of those over 65 years. In the USA accounts for 875,000 hospitalisations and costs $6.6 Billion. Risk of stroke associated with AF is 5-12% per year. Risk of stroke may be reduced by w ...
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Atrial fibrillation



Atrial fibrillation (AF or A-fib) is an abnormal heart rhythm characterized by rapid and irregular beating. Often it starts as brief periods of abnormal beating which become longer and possibly constant over time. Most episodes have no symptoms. Occasionally there may be heart palpitations, fainting, shortness of breath, or chest pain. The disease increases the risk of heart failure, dementia, and stroke.Hypertension and valvular heart disease are the most common alterable risk factors for AF. Other heart-related risk factors include heart failure, coronary artery disease, cardiomyopathy, and congenital heart disease. In the developing world valvular heart disease often occurs as a result of rheumatic fever. Lung-related risk factors include COPD, obesity, and sleep apnea. Other factors include excess alcohol intake, diabetes mellitus, and thyrotoxicosis. However, half of cases are not associated with one of these risks. A diagnosis is made by feeling the pulse and may be confirmed using an electrocardiogram (ECG). The typical ECG shows no P waves and an irregular ventricular rate.AF is often treated with medications to slow the heart rate to a near normal range (known as rate control) or to convert the rhythm to normal sinus rhythm (known as rhythm control). Electrical cardioversion can also be used to convert AF to a normal sinus rhythm and is often used emergently if the person is unstable. Ablation may prevent recurrence in some people. Depending on the risk of stroke either aspirin or anti-clotting medications such as warfarin or a novel oral anticoagulant may be recommended. While these medications reduce this risk, they increase rates of major bleeding.Atrial fibrillation is the most common serious abnormal heart rhythm. In Europe and North America, as of 2014, it affects about 2% to 3% of the population. This is an increase from 0.4 to 1% of the population around 2005. In the developing world about 0.6% of males and 0.4% of females are affected. The percentage of people with AF increases with age with 0.14% under 50 years old, 4% between 60 and 70 years old, and 14% over 80 years old being affected. A-fib and atrial flutter resulted in 112,000 deaths in 2013, up from 29,000 in 1990. The first known report of an irregular pulse was by John Baptist Senac in 1749. This was first documented by ECG in 1909 by Thomas Lewis.
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