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Assembly and function of DNA double
Assembly and function of DNA double

... DNA damage arises continuously as the result of intracellular metabolism and upon the exposure of cells to a multitude of genotoxic agents [1,2]. If left unrepaired, such insults can be lifethreatening for cells and organisms as they alter the content and organization of the genetic material. To ove ...
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... certain degree contributes to aggregation of the isolated EVs (Figure 1C). As a biological readout, isolated EVs associated with extracellular DNA were taken up by human mesenchymal stem cell in a time-dependent manner (Figure 1D and E). DISCUSSION: EVs carry multiple bioactive molecules, including ...
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... a systematic phenotypic analysis of genes that had been annotated as putative DNA replication/DNA repair regulators. One T-DNA insertion line (Salk_113285, hereafter referred to as rtel1-1) exhibited significant growth retardation of the young leaves and displayed a smaller final mature leaf size in c ...
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... The exact reaction conditions were important when carrying out UV endonuclease analysis. Since crude extracts were being used as a source of endonuclease, it was possible that these extracts could contain photoreactivating enzyme which would also have activity towards the dimer substrate. Photoreact ...
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DNA repair



DNA repair is a collection of processes by which a cell identifies and corrects damage to the DNA molecules that encode its genome. In human cells, both normal metabolic activities and environmental factors such as UV light and radiation can cause DNA damage, resulting in as many as 1 million individual molecular lesions per cell per day. Many of these lesions cause structural damage to the DNA molecule and can alter or eliminate the cell's ability to transcribe the gene that the affected DNA encodes. Other lesions induce potentially harmful mutations in the cell's genome, which affect the survival of its daughter cells after it undergoes mitosis. As a consequence, the DNA repair process is constantly active as it responds to damage in the DNA structure. When normal repair processes fail, and when cellular apoptosis does not occur, irreparable DNA damage may occur, including double-strand breaks and DNA crosslinkages (interstrand crosslinks or ICLs).The rate of DNA repair is dependent on many factors, including the cell type, the age of the cell, and the extracellular environment. A cell that has accumulated a large amount of DNA damage, or one that no longer effectively repairs damage incurred to its DNA, can enter one of three possible states: an irreversible state of dormancy, known as senescence cell suicide, also known as apoptosis or programmed cell death unregulated cell division, which can lead to the formation of a tumor that is cancerousThe DNA repair ability of a cell is vital to the integrity of its genome and thus to the normal functionality of that organism. Many genes that were initially shown to influence life span have turned out to be involved in DNA damage repair and protection.
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