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Hairy cell leukemia is a chronic Lymphoprolifrative disorder. in 1952
Hairy cell leukemia is a chronic Lymphoprolifrative disorder. in 1952

Gene Therapy for Primary Immunodeficiency Diseases: Should we do it?
Gene Therapy for Primary Immunodeficiency Diseases: Should we do it?

... •Typical SCID, due to defects that include IL2RG (X-linked), ADA, IL7R, JAK3, RAG1, RAG2, DCLRE1C (Artemis), TCRD, TCRE, TCRZ, and CD45 • Leaky SCID or Omenn syndrome • Variant SCID, with low T-cells but no defect in a known SCID gene • Syndromes with variably affected cellular immunity that may be ...
hybridoma technology for production of monoclonal antibodies
hybridoma technology for production of monoclonal antibodies

... Hybridomas are cells that have been engineered to produce a desired antibody in large amounts. To produce monoclonal antibodies, Bcells are removed from the spleen of an animal that has been challenged with the relevant antigen. These B-cells are then fused with myeloma tumor cells that can grow ind ...
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... cells; identifies and neutralises pathogens Antigen:  substance that can  provoke an immune response Clone: group of identical decendents Epitope: the specific piece of the antigen to which an  antibody binds.  Humoral: relating to fluids Innate:  present from birth ...
2005 Scientific Summary - Myasthenia Gravis Foundation of America
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... Patients who have seronegative MG and who have antibodies against MuSK can be referred to as having MuSK-MG to distinguish them from patients who have anti-AChR antibodies (AChR-MG). MuSK-MG can be associated with severe bulbar and facial weakness and tongue atrophy. The authors studied 15 MuSK-MG p ...
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... our body cells. They are glycoproteins, synthesized at the rough endoplasmic reticulum and then sent to the Golgi apparatus for final processing and packaging in a vesicle. They are transported to the surface of the cell membrane in a vesicle where they are finally integrated into the cell membrane. ...
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... membrane, the immunological mechanisms so far described only have a very limited effect in eliminating them. However, the cell wall deficient bacterial forms are evidently rendered recognisable to the immune system by the specific SANUKEHL preparations. ...
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... produce IL-2 and interferon, and consequently fail to stimulate CD-8 killer cells. This immunosuppression can be overcome by viral load but does not prevent treatment with LTCI. thymic involution.6 Thus there appears to be a firm rationale for thymus-derived immunotherapeutic factors such as LTCI. I ...
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... SCID is actually a set of more than a dozen different disorders, all of which result in a failure of affected infants to develop T cells. It is a syndrome that results from a variety of genetic causes. Although most newborns with SCID appear healthy, the disorder is present at birth. Typically SCID in ...
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... This graph illustrates the speed and strength of the immune response (measured by concentration of antibodies in the blood) after an initial and secondary exposure to a pathogen. Can you explain what is shown by the graph? Imagine two scenarios: one where the first exposure is a real infection and o ...
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... went on to further therapy, 3 of whom received an allogeneic bone marrow transplant. The fourth had refractory Tcell ALL aberrantly expressing CD19, entered remission after CTL019, and subsequently underwent donor lymphocyte infusion. She remains in remission >1 year later. The fifth patient develop ...
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... APC Presentation, T cell response Deamidated gliadin interacts with HLA DQ2 or HLA DQ8 on antigen presenting cells (APCs). Deamidated gliadin is presented to CD4 T cells. CD4 T cells produce cytokines (such as IFNγ) which cause tissue damage. ...
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... Name the system that defends the body against pathogens? Explain the non-specific response and the specific response of the body. 4. COPY: Primary line of defense = skin mucous, membranes, tears, digestive enzymes non – specific response Secondary line of defense=phagocytic white blood cells (engulf ...
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... has fully re-grown from stem cells in the transplanted graft. In ATIR101™, T-cells that would cause GVHD are eliminated from the donor lymphocytes using Kiadis Pharma’s photodepletion technology, minimizing the risk of GVHD and eliminating the need for prophylactic immune-suppression. At the same ti ...
cancer immunology - Cell Signaling Technology
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Immunomodulatory Activity of Dalbergia Latifolia on Swis Albino Mice
Immunomodulatory Activity of Dalbergia Latifolia on Swis Albino Mice

... A number of different cells are considered phagocytes. The most common type is the neutrophil, which primarily fights bacteria. If doctors are worried about a bacterial infection, they might order a blood test to see if a patient has an increased number of neutrophils triggered by the infection. Oth ...
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Immunosuppressive drug

For a list of immunosuppressive drugs, see the transplant rejection page.Immunosuppressive drugs or immunosuppressive agents or antirejection medications are drugs that inhibit or prevent activity of the immune system. They are used in immunosuppressive therapy to: Prevent the rejection of transplanted organs and tissues (e.g., bone marrow, heart, kidney, liver) Treat autoimmune diseases or diseases that are most likely of autoimmune origin (e.g., rheumatoid arthritis, multiple sclerosis, myasthenia gravis, systemic lupus erythematosus, sarcoidosis, focal segmental glomerulosclerosis, Crohn's disease, Behcet's Disease, pemphigus, and ulcerative colitis). Treat some other non-autoimmune inflammatory diseases (e.g., long term allergic asthma control).A common side-effect of many immunosuppressive drugs is immunodeficiency, because the majority of them act non-selectively, resulting in increased susceptibility to infections and decreased cancer immunosurveillance. There are also other side-effects, such as hypertension, dyslipidemia, hyperglycemia, peptic ulcers, lipodystrophy, moon face, liver and kidney injury. The immunosuppressive drugs also interact with other medicines and affect their metabolism and action. Actual or suspected immunosuppressive agents can be evaluated in terms of their effects on lymphocyte subpopulations in tissues using immunohistochemistry.Immunosuppressive drugs can be classified into five groups: glucocorticoids cytostatics antibodies drugs acting on immunophilins other drugs.
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