Download QUESTIONS IN PANCREAS AND DIABETES I

Survey
yes no Was this document useful for you?
   Thank you for your participation!

* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project

Document related concepts
no text concepts found
Transcript
QUESTIONS IN PANCREAS AND DIABETES
I-DEFINE
Hormone
Mixed gland
II-MENTION
-Exocrine Functions of the Pancreas
-Endocrine Functions of the Pancreas
- Hormones of the pancreas and function of each
III-FILL
-Hormones are each produced by separate, specialized cells of the pancreas, called the -----------------------------.
-Insulin is released at a basal rate by the ---------------------- of the islets of Langerhans. A rise in blood ----------------- is
the primary stimulus to increase insulin release. When blood glucose increases to more than --------------------------- of
blood, insulin secretion from the pancreas increases rapidly and then returns to baseline in ------------------.
- Glucagon is a protein hormone released from the------------------ of the islets of ------------------ in response to low
blood glucose levels . In most respects, glucagon works the opposite of insulin. Glucagon stimulates the used of fatty
acids as an energy source instead of glucose.
-Fasting blood glucose of 110 mg/100 mL on more than one occasion is diagnostic of ------------------.
- Hyperglycemia caused by an absolute lack of insulin is known as type --------------------------------.It results from ------------------- destruction of the beta cells of the islets of Langerhans.
-Hyperglycemia caused by cellular insensitivity to insulin is called type 2 diabetes .
-The number one risk factor for type 2 diabetes mellitus is-----------------------.
-------------------------- is an inflammation of the pancreas characterized by autodigestion of the pancreas by pancreatic
enzymes leading to areas of cell necrosis and hemorrhage.
IV-TRUE OR FALSE
-Non-fasting plasma glucose of greater than 200 mg/100 mL with symptoms of polyurea, polydipsia, and polyphagia
is diagnostic of diabetes.
-The genetic tendency to develop diabetes is strong.
-Glucose in the urine may or may not be indicative of diabetes.
- Under most conditions, glucose is not present in the urine of healthy, non-pregnant individuals.
-Diabetic Ketoacidosis is almost always restricted to type 1 diabetics
-Diabetic ketoacidosis may occur after physical stress such as pregnancy or an acute illness or trauma. But
sometimes it is the presenting symptom.
-Pancreatitis may occur as a result of blockage of the pancreatic duct, usually caused by a gallstone in the common
bile duct.
V-MCQ
Of Clinical Manifestations of diabetes
a- Increased rate of infections because of increased glucose concentration in mucus secretions, poor immune
function, and reduced blood flow.
b- Visual changes .
c- Paresthesias, or abnormalities in sensation.
d- Vaginal candidiasis (yeast infection),
e-Fatigue and muscle weakness
Long-Term Complications of diabetes
a- Microvascular damage occurs to the small arterioles, the capillaries, and the venules.
b- Vision Loss
c- Renal Damage
d- diabetic neuropathy.
e- all
*Clinical Manifestations of Pancreatic Cancer
a-It may be asymptomatic (until advanced). b- Pain may be an early complaint or may occur only with advanced
disease.
c-Advanced disease is associated with jaundice, d- Metastases to the brain and lung are common. e- Mortality is
nearly 100% within 5 years.