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Transcript 
Myocardial Perfusion Imaging:
One day rest stress protocol.
After the IV administration of 10
mCi Tc 99m sestamibi /
tetrofosmin, SPECT images of
the heart are acquired. At peak
exercise or pharmacologic
(dipyridamole/ adenosine/
dobutamine) stress, 30-40 mCi
of the tracer is injected and
Gated SPECT images of the
heart are acquired. The images
are reconstructed using filtered
back projection and displayed
in the standard format.
Perfusion and function are
quantitated and EF calculated
using MPI software. Normal
study showing no definite
evidence of reversible or fixed
defects and normal left
ventricular function.
No reversible defects. Normal Function
No reversible defects. Normal Function. Inferior wall attenuation
No reversible defects. Normal Function. Inferior wall attenuation
A fixed anterior wall
defect which could be an
attenuation artifact
(breast attenuation in
females) or a scar from
prior MI. Correlation with
Gated images is helpful
in differentiation. Scar
shows hypokinesis and
an artifact shows normal
wall motion and
thickening
No reversible defects. Normal function. Artifactual extracardiac uptake in stomach
No reversible defects. Normal function. Artifactual extracardiac uptake in colon
No reversible defects. Normal Function. Patient motion and Arm attenuation
Circumflex and RCA Ischemia
RCA and Circumflex Ischemia
LAD and RCA Ischemia
Reversible defect in the inferior wall and apex. Normal function
Reversible defect in the lateral wall. Normal function
RCA Infarct. No Ischemia. Global Hypokinesis. Ischemic Cardiomyopathy
RCA infarct. No Ischemia.
Reversible lateral wall defect. Transient ischemic dilatation.
Septal Hypokinesis
Fixed defects in the inferior wall and apex. Reversible defect in anterior wall.
Dilated left ventricle. Global hypokinesis. Septal dyskinesis. Ischemic CMP
Partially reversible defect in the anterior wall. Reversible defect in lateral
wall. Dilated left ventricle. Anterior hypokinesis, Septal dyskinesis
Predominantly fixed defect in the lateral wall with partial
reversibility.Global hypokinesis. Dilated left ventricle
Fixed defect in the septum and apex. Normal function.
? hibernating myocardium in the LAD territory.
Fixed apical and septal defect. Global hypokinesis and apical dyskinesis/aneurysm
No reversible defects. Normal Function. LVH
Subtle anterior Ischemia. Dilated ventricle. Septal dyskinesis from prior CABG
No reversible defects. Non-Ischemic Cardiomyopathy
No Ischemia. Normal Function. Right Ventricular Hypertrophy
Partially reversible apical defect. Normal Function. Dextrocardia
Dextrocardia
No reversible defects. Normal function. Uptake in the right
axillary adenopathy from NHL.
Normal Planar Study
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