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CHEST INJURIES Philippe SCHERPEREEL Professor Emeritus TBILISI 2014 Centre Hospitalier Universitaire 59037 LILLE France E-mail : [email protected] CHEST INJURIES • MAIN CAUSE OF 25% OF DEATHS FROM TRAUMATIC ORIGIN: SECOND CAUSE OF MORTALITY • IMPORTANT PROGRESSES OF IMAGING FOR THE DIAGNOSIS: ECHOGRAPHY, SPIRALED CT SCAN, NMR • CONSERVATIVE TREATMENT: – CLOSED INJURIES: < 10% NEED SURGERY – PENETRATING WOUNDS: 15-30% REQUEST SURGERY BUT NON SURGICAL TECHNIQUES ARE MORE AND MORE USED – DAMAGE CONTROL SURGERY FOR THORACIC INJURIES: PACKING, OPEN CHEST, PULMONARY HILUM TWIST… • DEATHS – INMEDIATE: HEART AND MAJOR VESSELS RUPTURES – DELAYED: AIRWAY OBSTRUCTION, CARDIAC TAMPONADE, ASPIRATION BRONCHOPNEUMONITIS… TRAUMA LESIONS • THORACIC WALL:RIBS, STERNAL FRACTURES, FLAIL CHEST • PNEUMOTHORAX, HEMOTHORAX • DIAPHRAGM RUPTURE • PULMONARY CONTUSION • TRACHEO BRONCHIAL LESIONS • CARDIAC CONTUSION • CARDIAC TAMPONADE • AORTIC RUPTURE • MEDIASTINAL LESIONS FOUR STEPS 1. EVALUATION AND MANAGEMENT ON SITE (15 min ) 2. INITIAL EVALUATION AND MANAGEMENT AT THE EMERGENCY DEPARTMENT (30 min) 3. COMPLETE EVALUATION (1 – 3 HOURS) 4. FREQUENT RE-EVALUATIONS (24 HOURS) 1. EVALUATION AND IMMEDIATE MANAGEMENT • CLINICAL EXAMINATION: LIFE-THREATENING LESIONS – NEUROLOGICAL: CONSCIOUSNESS (Glasgow score) – CARDIO VASCULAR: • HEMORRHAGE • ARTERIAL PRESSURE • PERIPHERICAL PULSES – RESPIRATORY : VENTILATION • FIRST ACTIONS: – – – – AIRWAY VENOUS ACCESS ARTERIAL BLOOD PRESSURE CATHETER BIOLOGY: HEMOGLOBIN BLOOD GROUP CRITERIA OF INTUBATION IN CASE OF CHEST INJURY • VENTILATORY RATE > 25/min • HEART RATE > 100/min • SYSTOLIC ARTERIAL PRESSURE < 100 mmHg • RESPIRATORY FAILURE: PaO2 < 60 mmHg • and/or PaCO2 > 45 mmHg and/or pH < 7,20 ASSOCIATED LESIONS: – ABDOMINAL – NEUROLOGICAL (Glasgow Score ≤ 7) INTUBATION IF THE NUMBER OF CRITERIA ≥ 3 2. INITIAL EVALUATION AND MANAGEMENT • THORAX X RAY • ECHOGRAPHY – THORAX – HEART TOMODENSITOMETRY IS NOT PART OF THE INITIAL EVALUATION RIB, STERNUM AND SCAPULA FRACTURES • RIBS 1 - 3: – VIOLENT TRAUMA – ASSOCIATED LESIONS: HIGH MORTALITY Major vessels ruptures Medullary wounds, spinal lesions • RIBS 4 - 9 : – PULMONARY CONTUSION – HEMO, PNEUMOTHORAX • RIBS 10 – 12 : – ABOMINAL LESIONS? TENSION PNEUMOTHORAX SYMPTOMS: • SLIPPED MEDIASTIN • RESPIRATORY FAILURE • HYPOTENSION, TACHYCARDIA • HYPER SONORITY • JUGULAR DISTENSION • DELAYED CYANOSIS IMMEDIATE DECOMPRESSION MASSIVE HEMOTHORAX SYMPTOMS – Shock – Dull noise without ventilation – Cervical veins – Empty or distended MANAGEMENT – Fast fluid challenge – Drainage and X Ray ± auto transfusion – Surgery if the initial drainage is over 1 500 ml DIAPHRAGM RUPTURE CLINICAL SYMPTOMS • 5 TIMES MORE FREQUENT IN LEFT vs RIGHT SIDE • ASSOCIATION - 2/3 RIB FRACTURES ± FLAIL CHEST - ½ ABDOMINAL LESIONS LIVER, SPLEEN - < 5% SEVERE INTRA THORACIC WOUNDS • IMAGING - EMPTY VISCERA: BRIGHTNESS - SOLID VISCERA: OPAQUENESS RIGHT DIAPHRAGMATIC RUPTURE SURGERY RIGHT INTRA THORACIC OPAQUENESS LEFT DIAPHRAGMATIC RUPTURE STANDARD X - RAY WITH PRODUCT OF CONTRAST ASSOCIATION WITH AN HEMOTORAX DIAPHRAGMATIC RUPTURE TOMODENSITOMETRY FLAIL CHEST • DYSFUNCTION OF VENTILATORY MECHANISM PARADOXICAL MOVEMENTS OF THORAX : CHEST WALL DEPRESSION AT INSPIRATION • MANAGEMENT ANALGESIA : THORACIC EPIDURAL ANALGESIA INTERNAL PNEUMATIC STABILIZATION CONTINUOUS POSITIVE PRESSURE VENTILATION SURGICAL FIXATION FRACTURE OF THE STERNUM: HOOPING JUDET, SANCHEZ CLIPS, PINS PARADOXICAL VENTILATION FRACTURE OF THE STERNUM • SEAT BELT • STRONG IMPACT TRACHEO BRONCHIAL LESIONS PARTIAL OR COMPLETE LESIONS • FISSURE • FRACTURE • RUPTURE RARE OCCURENCE (1%), DIFFICULT DIAGNOSIS, MORE DELAYED, MORE FREQUENT LOCALIZATION: CARENA TRACHEO BRONCHIAL LESIONS CLINICAL SIGNS • TENSION PNEUMOTHORAX • CERVICO FACIAL SUBCUTANEOUS EMPHYSEMA THORACIC OR EXTENSIVE: SNOWY CREPITATION • MEDIASTINAL EMPHYSEMA • HEMOPTYSIA WHATEVER THE QUANTITY TRACHEO BRONCHIAL LESIONS TRACHEO BRONCHIAL LESIONS FIBEROPTIC ENDO SCOPY • INDICATIONS – Persistant air leakage – Isolated pneumothorax – Hemoptysis – Early ventilation failure • MANAGED IN A PATIENT – Hemodinamically stable – After pleural drainage – In the immediate vicinity of an operative theater thoracotomy in emergency DECISIONAL ALGORITHM SIGNS OF TRACHEAL BRONCHIAL LESIONS PNEUMOTHORAX DRAINAGE CONTROL OBSERVATION FIBEROPTIC ENDOSCOPY • FISSURE WITHOUT AIR LEAKAGE • DISTAL OR PARTIAL RUPTURE CONTROL • RUPTURE OR • COMPLETE FRACTURE • PROXIMAL THORACOTOMY LUNG CONTUSION CINICAL SIGNS • HEMOPTYSIS, HEMOPTOIC EDEMA IMAGING – Inhomogenous, glurred opacities, aeric bronchogrammes • GAZOMETRY: Hypoxemia • HEMOSTASIS: Frequent biological disseminated intra vascular coagulations (DIVC) • EVOLUTION: – SPONTANEOUS: Disappearance within 24-72 HOURS – COMPLICATIONS: • Broncho pulmonary infections • Air leakage • Pulmonary fibrosis LUNG CONTUSION INTEREST OF THE THORACIC TOMODENSITOMETRY Front pneumothorax Pulmonary contusion and hemotHorax EVOLUTION OF A LUNG CONTUSION PERICARDIAC LESIONS • PERICARDIAC RUPTURES RISK OF HEART LUXATION, IMMEDIATE OR DELAYED • CARDIAC TAMPONADE RISK OF SUDDEN DEATH - HYPOTENSION - DILATED CERVICAL VEINS - CARDIAC PULSELESS ACTIVITY MANAGEMENT - PERICARDIOCENTESIS - PERICARDIOTOMY PERICARDIAC EFFUSION CARDIAC CONTUSION FOLLOWING BLUNT CHEST TRAUMA DIAGNOSIS BASED UPON: 1. 2. 3. 4. CLINICAL SIGNS: PAIN, ARRYTHMIAS… E.C.G. BIOCHEMISTRY: CPK MB, TROPONIN E ECHOCARDIOGRAPHY+++ • SIZE AND KINETICS OF THE VENTRICLE, AKINESIA • SEARCH OF ASSOCIATED LESIONS 5. CORONARY ANGIOGRAPHY RADIONUCLIDE IMAGING ISOTOPIC ANGIOGRAPHY WITH TECHNECIUM CARDIAC CONTUSION EVOLUTION OF CARDIAC CONTUSIONS • USUALY FAVOURABLE WITHOUT CLINICAL OR • ELECTRICAL SEQUELAE COMPLICATIONS – EARLY: CARDIOGENIC SHOCK ARRITHMIAS AND ALTERATIONS OF CONDUCTION THROMBO EMBOLIES HEMOPERICARDIC TAMPONADE CARDIAC RUPTURES: SEPTUM, VALVULES – DELAYED: CARDIAC FAILURE VENTRICULAR ANEURYSM HEMOMEDIASTINUM • CLINICAL SIGNS – NON SPECIFIC: Retrosternal pain Interscapular pain Dyspnoea Collapse – EVOCATING: • DISPARITY IN PERIPHERAL PULSES ( MS, MI) • SYSTOLIC HEART MURMUR • NEUROLOGICAL DEFECT IN THE LOWER LEGS • ANURIA – LATELY: SIGNS OF COMPRESSION • OESOPHAGUS: DYSPHAGIA • TRACHEO BRONCHIAL: DYSPNOEA EVALUATION OF THE HEMOMEDIASTINUM ENLARGEMENT OF THE MEDIASTINUM TRAUMATIC LESIONS OF THE AORTA • FREQUENCE: 0,2 -2% OF THE THORACIC BLUNT • • TRAUMA SEVERITY: 12-20% OF THE DEATHS EVOLUTION BEFORE AORTIC RUPTURE AORTOGRAPHY SURGERY IN EMERGENCY SURGICAL TREATMENT TO-DAY AORTIC TRAUMA TOE – Trans Oesophageal Echography or Multislice computed tomography DELAYED SURGERY MEDICO SURGICAL TREATMENT MECHANISMS OF TRAUMATIC AORTIC LESIONS POLYTRAUMATISM AND DECELERATION ORGAN SPEED (km/h) REAL WEIGHT (kg) PARR Y GRANDE 1993 MASS IN DECELERATION (kg) 0 36 72 108 SPLEEN 0,25 2,5 10 22,5 HEART 0,35 2,5 14 31,5 BRAIN 1,5 15 60 135 LIVER 1,8 18 72 162 TOTAL BODY 70 700 2 800 6 300 RATIO 1 X10 X40 X90 STANDARD X - RAY • ENLARGEMENT OF THE UPPER MEDIASTINUM (67-80%) • DISAPPEARANCE OF THE AORTIC BUTTON (21-24%) • TRACHEAL DISPLACEMENT (3-12%) • HEMATOMA OF THE APEX OF THE LUNG (4-19%) • LEFT HEMOTHORAX (7-19%) TRAUMATIC RUPTURE OF THE AORTIC ISTHMUS AORTOGRAPHY ANGIO SCANNER RUPTURE OF THE AORTIC ISTHMUS NUCLEAR MAGNETIC RESONNANCE AORTOGRAPHY ENDOLEAK HEMATOMA TRANS OESOPHAGEAL IRM ECHOCARDIOGRAPHY MANAGEMENT BY ENDOSVASCULAR STENT • SUBACUTE OR CHRONIC AORTIC TRAUMA • WITHOUT SECONDARY RUPTURE • POSSIBLE COMPLICATIONS • OCCLUSION SUBCLAVIAN • ATELECTASIS • RISK ON THE LONG TERM (RUPTURE, PARAPLEGIA…) RUPTURE OF THE VESSELS OF THE BASIS OF THE NECK ANGIOGRAPHY SUMMARY • VERY FREQUENT, MOST OFTEN NON-LIFE- • • • • • THREATENING: THE RISK OF THE THORACIC TRAUMA IS THE DIFFICULTY TO RECOGNISE LESIONS: RARE (ex: Oesophagus) LIFE THREATENING (ex: rupture of vessels) IMPORTANCE OF THE INITIAL EVALUATION IMAGING ULTRASONOGRAPHY COMPUTED TOMOGRAPHY, N M R ANGIOGRAPHY NEED FOR ANALGESIA MANAGEMENT USUALY MORE CONSERVATIVE THAN SURGICAL, ENDOVASCULAR STENT NECESSITY OF PERMANENT RE ASSESSMENT OF THE PATIENT