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CT IMAGING OF A CASE WITH BIVENTRICULAR HYPERTROPHIC
CT IMAGING OF A CASE WITH BIVENTRICULAR HYPERTROPHIC

... CT IMAGING OF A CASE WITH BIVENTRICULAR HYPERTROPHY ...
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... embedded in a dense connective tissue matrix (Figures 3 a and b). With age, the amount of connective tissue increases with respect to the area occupied by the nodal cells.21 On the periphery of the node, specialized cells are mixed with those of the working myocardium (Figures 3a and b). In addition ...
pathology of inflammatory native valvular heart disease
pathology of inflammatory native valvular heart disease

... molecular mimicry to human tissue and are recognized by the immune system thus initiating an autoimmune response. Cross-reactivity between the M-protein and cardiac proteins is important. 9 The acute involvement of the heart in rheumatic fever is pancarditis with inflammation of the myocardium, peri ...
Reoperation for Tricuspid Regurgitation after Total Correction of
Reoperation for Tricuspid Regurgitation after Total Correction of

... However, the use of prosthetic rings poses unique problems in children. Growing children require a longitudinal management strategy to account for growth that is not a consideration in adult cardiac surgery. Therefore, every effort should be made to repair the native valve in children, even when it ...
PDF
PDF

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Biventricular and atrial diastolic function assessment using
Biventricular and atrial diastolic function assessment using

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Echocardiographic Investigation of Canine Myxomatous Mitral

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... provide information on cardiac abnormalities. However, in clinical routine, hemodynamics is mostly observed indirectly through global variables as the cardiac output in order to assess the cardiac performance. Indeed, a synthetic description of the available information and its relation with the hea ...
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... kinase (CK) levels in the past (as high as 1000 IU/L) attributed to endurance running. Echocardiography now revealed mild left and right ventricular enlargement with LV ejection fraction of 50%, moderate tricuspid regurgitation, and marked bi-atrial enlargement. Invasive EP testing was repeated in v ...
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Frog Heart Physiology
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... electrocardiographic abnormality, together with a short PR interval. Wolff-Parkinson-White syndrome(WPW) was initially reported in healthy young people with both preexcitation in the surface ECG and symptoms caused by tachyarrhythmias.1 The approximate incidence of WPW is 14/1000. Longitudinal studi ...
Coronary Sinus Catheter Placement
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BUKOVINІАN STATE MEDICAL UNIVERSITY
BUKOVINІАN STATE MEDICAL UNIVERSITY

... the kidney’s excretion of excess salt and fluid into the urine. While these drugs, which reduce congestion and swelling, are an essential part of heart failure therapy, patients can help the physician decrease the dosages of diuretics by limiting the amount of salt in their diet. Function of the imp ...
Evaluation of left ventricular systolic and diastolic functions in bipolar
Evaluation of left ventricular systolic and diastolic functions in bipolar

... disorder had died by the end of the study period. The mortality rate for cardiovascular disease was 1.59 for men and 1.47 for women [21]. A similar study found a twofold increased cardiovascular mortality rate for those with bipolar disorder compared with a cardiovascular mortality rate for individu ...
Echocardiographic Assessment of Aortic Stenosis
Echocardiographic Assessment of Aortic Stenosis

... mean gradient 40 mmHg provided that valve area does not exceed 1.0 cm2 at any flow rate.  Absence of contractile reserve (failure to increase SV or EF by 20%) is a predictor of a high surgical mortality and poor long-term outcome although valve replacement may improve LV function and outcome even i ...
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Lutembacher's syndrome



Lutembacher's syndrome is a form of congenital heart disease. Lutembacher's syndrome was first described by a French cardiologist by the name of Rene' Lutembacher (1884–1968) of Paris, France in 1916. Lutembacher syndrome is a rare disease that affects one of the chambers of the heart as well as a valve of the heart. Lutembacher's syndrome is known to affect females more often than males. Lutembacher is an extremely rare disease. Lutembacher's can affect children or adults; the person can either be born with the disorder or develop it later in life.Lutembacher affects more specifically the atria of the heart and the mitral or biscupid valve. The disorder itself is known more specifically as both congenital atrial septal defect (ASD) and acquired mitral stenosis (MS). Congenital (at birth) atrial septal defect refers to a hole being in the septum or wall that separates the two atria; this condition is usually seen in fetuses and infants. Mitral stenosis refers to mitral valve leaflets (or valve flaps) sticking to each other making the opening for blood to pass from the atrium to the ventricles very small. With the valve being so small, blood has difficulty passing through the left atrium into the left ventricle. There are several types of septal defects that may occur with Lutembacher's syndrome: ASD Ostium Secundum or ASD (Primium); Ostium Secundum is the most prevalent.Lutembacher is caused indirectly as the result of heart damage or disorders and not something that is necessarily infectious. Lutembacher's syndrome is caused by either birth defects where the heart fails to close all holes in the walls between the atria or from an episode of rheumatic fever where damage is done to the heart valves such as the mitral valve and resultant in an opening of heart wall between atria. With Lutembacher's syndrome, a fetus or infant is usually seen to have a hole in their heart wall (interatrial) separating their right and left atria. Normally during fetal development, blood bypasses the lungs and is oxygenated from the placenta. Blood passes from the umbilical cord and flows into the left atrium through an opening called the foramen ovale; the formaen ovale is a hole between the two atria. Once a baby is born and the lungs begin to fill with air and the blood flow of the heart changes, a tissue flap (somewhat like a trap door) called the septum primium closes the foramen ovale or hole between the two atria and becomes part of the atrial wall. The failure of the hole between the two atria to close after birth leads to a disorder called ASD primium. The most common problems with an opening found in the heart with Lutembacher's syndrome is Ostium Secundum. Ostium Secundum is a hole that is found within the flap of tissue (septum primium) that will eventually close the hole between the two atria after birth. With either type of ASD, ASD will usually cause the blood flow from the right atrium to skip going to the right ventricle and instead flow to the left atrium. If mitral stenosis (the hardening of flap of tissue known as a valve which opens and closes between the left atrium and ventricle to control blood flow) is also present, blood will flow into the right atrium through the hole between the atria wall instead of flowing into the left ventricle and systemic circulation. Eventually this leads to other problems such as the right ventricle failing and a reduced blood flow to the left ventricle.In addition to the ASD, acquired MS can be present either from an episode of rheumatic fever (the mother has or had rheumatic fever during the pregnancy) or the child being born with the disorder (congenital MS). With the combination of both ASD and MS, the heart can be under severe strain as it tries to move blood throughout the heart and lungs. To correct Lutembacher's syndrome, surgery is often done. There are several types of surgeries depending on the cause of Lutembacher's syndrome(ASD Primium or ASD Ostium Secundum with Mitral Stenosis): Suturing (stitching) or placing a patch of tissue (similar to skin grafting) over the hole to completely close the opening Reconstructing of the mitral and tricuspid valve while patching any holes in the heart Device closure of ASD (e.g. Amplatzer umbrella or CardioSEAL to seal the hole Percutaneous transcatheter therapy Transcatheter therapy of balloon valvuloplasty to correct MS↑ ↑ 2.0 2.1 2.2 2.3 2.4 ↑ 3.0 3.1 3.2 3.3 3.4 ↑ ↑ ↑ 6.0 6.1 6.2 6.3 ↑
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