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An Update on Brain Disease - Vet Education Webinar Membership
An Update on Brain Disease - Vet Education Webinar Membership

... Cerebellar abiotrophies differ from cerebellar hypoplasia (the main differential) in that they are conditions in which the cerebellum develops normally but then begins to degenerate. Classically these conditions are breed related, autosomal recessive in their mode of inheritance (where known), progr ...
Understanding Genetics and Alzheimer`s Disease
Understanding Genetics and Alzheimer`s Disease

... • Research has not confirmed whether specific vitamins, substances or supplements can prevent or slow down Alzheimer’s disease. Before trying any of these, be sure to discuss the possible risks and benefits with your doctor. • Sporadic Alzheimer’s disease usually does not run in families. However, ...
Digestive Ch23-part 2
Digestive Ch23-part 2

... Copyright © 2010 Pearson Education, Inc. ...
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Protective Effect of Honey on Acetaminophen Induced Liver Toxicity

... previous study in which honey was observed to have a potent hepatoprotective action upon APAP-induced oxidative stress and liver toxicity in rat as demonstrated by a significant decrease in serum ALP and AST and total bilirubin level in rat with APAP hepatotoxicity (Ayyavu et al., 2009). It seems th ...
Neurotropic B Vitamins and Their Role in Nerve Care
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... vegetarians, increased demand (e.g. pregnancy), or increased loss or malfunction associated with certain disease states (e.g. renal disease). Many conditions that cause B vitamin deficiency are chronic and/or irreversible, such as diabetes, renal impairment, gastrointestinal diseases, and advanced a ...
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View the complete presentation (12.3 MB PDF)

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... Reactions are divided in two phases: Phase I: oxidation and reduction reactions mediated via cytochrome P450 oxidases Phase II: conjugation of phase I metabolites with glucuronate, sulphate or glutathione ...
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AP151--DIGESTIVE SYSTEM Part 2--f13
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... alcohol) or impairment of its function may lead to many complications in one’s health. There is a no rational therapy available for liver disorders and it is a still challenge to modern medicine1. Hepatic injury can be life threatening when the entirely or most of the liver is exposed to any hepatot ...
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... dominant 1:10,000-50,000 but 1:1000 in N. Sweden (Lapland) b. Precipitated by phenytoin, phenobarbital drugs, alcohol, fasting, hormones estrogen, stress, infection or lead poisoning 3. Biochemistry of neurotoxicity a. heme deficiency   P450 drug metabolism   tryptophan dioxygenase brain tryp ...
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Information on Celiac Disease/Gluten Free Diet

... • Lactose Intolerance due to inflammation and damage to intestinal mucosa • Skin Rash or itchy skin (dermatitis herpetiformis) Methods of Diagnosis: • Small bowel biopsy – check villi for damage. • Physicians can test the blood (Celiac Blood Panel) to measure levels of antibodies to endomysium and t ...
Gluten Free Diets
Gluten Free Diets

THE SCARBROUGH CLINIC
THE SCARBROUGH CLINIC

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Water Soluble Vitamins

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Introduction to Google Adwords
Introduction to Google Adwords

... Low urinary sulfate is an indication that total body glutathione is low due to chronic demand and sulfur-containing amino acids are needed. The amino acid N-acetylcysteine is one effective agent for raising your glutathione and sulfate levels. Another sulfur-containing nutrient, lipoic acid, should ...
High carbohydrate diets and Alzheimer`s disease
High carbohydrate diets and Alzheimer`s disease

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Wilson's disease



Wilson's disease or hepatolenticular degeneration is an autosomal recessive genetic disorder in which copper accumulates in tissues; this manifests as neurological or psychiatric symptoms and liver disease. It is treated with medication that reduces copper absorption or removes the excess copper from the body, but occasionally a liver transplant is required.The condition is due to mutations in the Wilson disease protein (ATP7B) gene. A single abnormal copy of the gene is present in 1 in 100 people, who do not develop any symptoms (they are carriers). If a child inherits the gene from both parents, the child may develop Wilson's disease. Symptoms usually appear between the ages of 6 and 20 years, but cases in much older people have been described. Wilson's disease occurs in 1 to 4 per 100,000 people. It is named after Samuel Alexander Kinnier Wilson (1878–1937), the British neurologist who first described the condition in 1912.
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