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Cardiovascular 3 – Mechanical Properties of the Heart I
Cardiovascular 3 – Mechanical Properties of the Heart I

...  Larger forces are needed to put pressure on the ventricles as their volume increases.  This means that the L.V. is able to generate higher pressures with similar wall stress on the R.V.  In dilated cardiomyopathy, ventricles dilate and so the pressure generated with each pulse is low. 2. List th ...
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... Contraction spreads through the atrial wall until it reaches the Atrioventricular (AV) Node in the right atrial side of the interatrial septum just above the opening of the Coronary sinus. After a brief delay contraction passes to the ventricles. The AV Node is usually supplied by the distal right c ...
Sudden Cardiac Death
Sudden Cardiac Death

... Sudden cardiac arrest occurs when the electrical system to the heart malfunctions and suddenly becomes very irregular. The heart beats dangerously fast. The ventricles may flutter or quiver (ventricular fibrillation), and blood is not delivered to the body. In the first few minutes, the greatest con ...
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... Case 1 Management • A 63-year-old male presents with breathlessness x 3 days. ...
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... shifted upwards or deformed and excessive dilatation of the valve annulus. The functional disorder is caused by the regurgitation being clinically relevant. As soon as ventricular filling is impaired, left atrial pressure increases and pulmonary oedema can occur. This leads to clinical symptoms incl ...
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Supraventricular Tachycardia vs. Marfan`s Syndrome

... these tracts are usually only capable of retro- In summary, one can only speculate on the grade impulse transmission, in contrast with relationship between the father’s cardiac conthe pre-excitation syndromes (example: WPW dition and the proposed insured. Mortality syndrome) in which the accessory b ...
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... known as ventricular tachycardia (VT) or ventricular fibrillation (VF). These are diseases of the heart’s electrical conduction system that should not be confused with a heart attack (myocardial infarction), which is caused by a blocked blood vessel leading to loss of blood supply to a portion of th ...
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Ch 20 – The Heart

... a. controlled by papillary muscles on ventricular walls b. Attach to cusps of valves via chordae tendineae (heart strings) c. make “lub” sound when closing ...
A new and innovative surgical technique for the treatment of
A new and innovative surgical technique for the treatment of

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...  is an uncommon congenital heart defect with a prevelence of 0.3-0.5%, characterized primarily by abnormalities of the tricuspid valve and right ventricle.  EA does not affect fertility even in cyanotic women. ...
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Pseudoaneurysm of the Left Ventricle following Mitral Valve

... the ventricular vent suggests inherent weakness in the patient's myocardium and indeed postmortem examination revealed patchy myocardial fibrosis with coronary vessel disease. In the majority of previously reported cases of false aneurysms of the left ventricle following cardiotomy incision, the dia ...
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ITE Review: Cardiovascular

... -nonspecific ST/T wave and sinus tach are most common finding -look for right heart strain -peaked P in lead II -new RBBB, S1Q3T3 D-dimer -remember Well’s, negative test can not exclude moderate or high pretest! V/Q scan -need interpretation in the context of clinical suspicion -low-moderate pretest ...
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Double Inlet Left Ventricle
Double Inlet Left Ventricle

... In some patients, there is mild obstruction to either systemic or pulmonary blood flow. Blood flow through the aorta to the body may become restricted if the size of the ventricular septal defect is too small, resulting in serious illness. (The only route of blood flow to the aorta is across the ven ...
VALVULAR HEART DISEASE Case Goals and Objectives
VALVULAR HEART DISEASE Case Goals and Objectives

...  Partner trial: 3.8% vs 2.1%; at 2 yrs: 7.7% vs 4.9% – More frequent adverse events in surgical group-major bleeding, new onset afib – Increased risk of heart block (2 fold) – Paravalvular arotic regurgitation ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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